Search results for "Review"

showing 10 items of 4492 documents

Changes in Serine Racemase-Dependent Modulation of NMDA Receptor: Impact on Physiological and Pathological Brain Aging

2018

International audience; The N-methyl-D-Aspartate glutamate receptors (NMDARs) are pivotal for the functional and morphological plasticity that are required in neuronal networks for efficient brain activities and notably for cognitive-related abilities. Because NMDARs are heterogeneous in subunit composition and associated with multiple functional regulatory sites, their efficacy is under the tonic influence of numerous allosteric modulations, whose dysfunction generally represents the first step generating pathological states. Among the enzymatic candidates, serine racemase (SR) has recently gathered an increasing interest considering that it tightly regulates the production of D-serine, an…

0301 basic medicine[SDV]Life Sciences [q-bio]Allosteric regulation[SHS.PSY]Humanities and Social Sciences/PsychologyglutamateDiseaseReviewBiologyBiochemistry Genetics and Molecular Biology (miscellaneous)BiochemistryNMDA receptors[SHS.PSY] Humanities and Social Sciences/Psychology03 medical and health sciences0302 clinical medicineserine racemasemedicineMolecular BiosciencesAmyotrophic lateral sclerosislong term potentiationMolecular BiologyPathologicallcsh:QH301-705.5ComputingMilieux_MISCELLANEOUS[SCCO.NEUR]Cognitive science/Neuroscience[SCCO.NEUR] Cognitive science/NeuroscienceagingGlutamate receptorLong-term potentiationAlzheimer's diseasemedicine.diseaseMESH: NMDA receptors serine racemase aging Alzheimer’s disease D-serine long term potentiation glutamate[SDV] Life Sciences [q-bio]030104 developmental biologylcsh:Biology (General)d-serineSerine racemaseNMDA receptor[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Neuroscience030217 neurology & neurosurgeryFrontiers in Molecular Biosciences
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Neuronal Cytoskeleton in Intellectual Disability: From Systems Biology and Modeling to Therapeutic Opportunities

2021

Intellectual disability (ID) is a pathological condition characterized by limited intellectual functioning and adaptive behaviors. It affects 1–3% of the worldwide population, and no pharmacological therapies are currently available. More than 1000 genes have been found mutated in ID patients pointing out that, despite the common phenotype, the genetic bases are highly heterogeneous and apparently unrelated. Bibliomic analysis reveals that ID genes converge onto a few biological modules, including cytoskeleton dynamics, whose regulation depends on Rho GTPases transduction. Genetic variants exert their effects at different levels in a hierarchical arrangement, starting from the molecular lev…

0301 basic medicineactin cytoskeletonReview0302 clinical medicineBorderline intellectual functioningIntellectual disabilityDisabilità Intellettiva GTPasi CitoscheletroBiology (General)CytoskeletonSpectroscopyNeuronseducation.field_of_studysystems biologyCognitionGeneral MedicinePhenotypeComputer Science ApplicationsChemistryPhenotypeintellectual disabilitySignal TransductionBoolean modelingQH301-705.5NeurogenesisIn silicoSystems biologyPopulationBiologyCatalysismicrotubulesInorganic Chemistry03 medical and health sciencesmedicineAnimalsHumansPhysical and Theoretical ChemistryeducationQD1-999Molecular BiologyGTPase signalingsmall Rho GTPasesOrganic Chemistrypharmacological modulationprotein:protein interaction networkActin cytoskeletonmedicine.disease030104 developmental biologySynapsesneuronal networksNeuroscience030217 neurology & neurosurgery
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Advance on Resveratrol Application in Bone Regeneration: Progress and Perspectives for Use in Oral and Maxillofacial Surgery

2019

The natural polyphenol Resveratrol (RSV) claims numerous positive effects on health due to the well documented biological effects demonstrating its potential as a disease-preventing agent and as adjuvant for treatment of a wide variety of chronic diseases. Since several studies, both in vitro and in vivo, have highlighted the protective bone aptitude of RSV both as promoter of osteoblasts’ proliferation and antagonist of osteoclasts’ differentiation, they could be interesting in view of applications in the field of dentistry and maxillofacial surgery. This review has brought together experimental findings on the use of RSV in the regeneration of bone tissue comprising also its application a…

0301 basic medicinealveolar bone lossBone Regenerationmedicine.medical_treatmentlcsh:QR1-502ReviewResveratrolBioinformaticsBone tissueBiochemistrylcsh:Microbiology03 medical and health scienceschemistry.chemical_compoundresveratrol scaffold0302 clinical medicineIn vivoSettore MED/28 - Malattie OdontostomatologicheAnimalsHumansMedicineBone regenerationMolecular Biologybone-regenerationbone defectbusiness.industryRegeneration (biology)alveolar bone locraniofacial tissue030206 dentistryBone defectSurgery Oral030104 developmental biologymedicine.anatomical_structurechemistryResveratrolSettore CHIM/09 - Farmaceutico Tecnologico ApplicativoChronic DiseaseOral and maxillofacial surgerybusinessAdjuvant
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Physiological Functions of the β-Site Amyloid Precursor Protein Cleaving Enzyme 1 and 2

2017

BACE1 was discovered as the β-secretase for initiating the cleavage of amyloid precursor protein (APP) at the β-secretase site, while its close homology BACE2 cleaves APP within the β-amyloid (Aβ) domain region and shows distinct cleavage preferences in vivo. Inhibition of BACE1 proteolytic activity has been confirmed to decrease Aβ generation and amyloid deposition, and thus specific inhibition of BACE1 by small molecules is a current focus for Alzheimer’s disease therapy. While BACE1 inhibitors are being tested in advanced clinical trials, knowledge regarding the properties and physiological functions of BACE is highly important and this review summarizes advancements in BACE1 research ov…

0301 basic medicineamyloid plaquessecretaseReviewamyloid precursor proteinBiology03 medical and health sciencesCellular and Molecular Neurosciencemental disordersAmyloid precursor proteinaspartic proteaseBACE substratesGlucose homeostasisMolecular Biologychemistry.chemical_classificationNeurogenesisBACE2P3 peptideBACE1Biochemistry of Alzheimer's disease030104 developmental biologyEnzymechemistryBiochemistrySynaptic plasticitybiology.proteinAmyloid precursor protein secretaseAlzheimer’s diseaseNeuroscienceFrontiers in Molecular Neuroscience
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The Impact of Microbiota on the Pathogenesis of Amyotrophic Lateral Sclerosis and the Possible Benefits of Polyphenols. An Overview

2021

The relationship between gut microbiota and neurodegenerative diseases is becoming clearer. Among said diseases amyotrophic lateral sclerosis (ALS) stands out due to its severity and, as with other chronic pathologies that cause neurodegeneration, gut microbiota could play a fundamental role in its pathogenesis. Therefore, polyphenols could be a therapeutic alternative due to their anti-inflammatory action and probiotic effect. Thus, the objective of our narrative review was to identify those bacteria that could have connection with the mentioned disease (ALS) and to analyze the benefits produced by administering polyphenols. Therefore, an extensive search was carried out selecting the most…

0301 basic medicineamyotrophic lateral sclerosisEndocrinology Diabetes and Metabolismlcsh:QR1-502ReviewDiseaseGut floraBioinformaticsBiochemistrylcsh:MicrobiologyPathogenesis03 medical and health sciences0302 clinical medicineLactobacillusmicrobiotaMedicineAmyotrophic lateral sclerosisMolecular BiologypolyphenolsBifidobacteriumbiologybusiness.industryNeurodegenerationfood and beveragesmedicine.diseasebiology.organism_classification030104 developmental biologybusiness030217 neurology & neurosurgeryAkkermansia muciniphilaMetabolites
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Oxidative Stress, Neuroinflammation and Mitochondria in the Pathophysiology of Amyotrophic Lateral Sclerosis

2020

Amyotrophic lateral sclerosis (ALS) is a progressive motor neuron (MN) disease. Its primary cause remains elusive, although a combination of different causal factors cannot be ruled out. There is no cure, and prognosis is poor. Most patients with ALS die due to disease-related complications, such as respiratory failure, within three years of diagnosis. While the underlying mechanisms are unclear, different cell types (microglia, astrocytes, macrophages and T cell subsets) appear to play key roles in the pathophysiology of the disease. Neuroinflammation and oxidative stress pave the way leading to neurodegeneration and MN death. ALS-associated mitochondrial dysfunction occurs at different le…

0301 basic medicineamyotrophic lateral sclerosisPhysiologyClinical BiochemistryReviewDiseaseMitochondrionmedicine.disease_causeBiochemistryneuroinflammationNeurologia03 medical and health sciences0302 clinical medicineoxidative stressMedicineAmyotrophic lateral sclerosisMolecular BiologyNeuroinflammationMicrogliabusiness.industrylcsh:RM1-950NeurodegenerationCell Biologymedicine.diseasePatologiaPathophysiologymitochondrialcsh:Therapeutics. Pharmacology030104 developmental biologymedicine.anatomical_structuremotor neuron diseasebusinessNeuroscience030217 neurology & neurosurgeryOxidative stressAntioxidants
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Therapeutic alternative of the ketogenic Mediterranean diet to improve mitochondrial activity in Amyotrophic Lateral Sclerosis (ALS): A Comprehensive…

2019

Abstract Amyotrophic lateral sclerosis (ALS) is an incurable neurodegenerative disease which is pathogenically based on the mitochondrial alteration of motor neurons, causing progressive neuron death. While ALS is characterized by enormous oxidative stress, the Mediterranean diet has been seen to have high antioxidant power. Therefore, the aim of this study is to determine how the Mediterranean diet can improve mitochondrial activity, establishing the specific nutrients and, in addition, observing the pathogenic mechanisms related to the disease that would achieve this improvement. To this end, a comprehensive review of the literature was performed using PubMed. KBs have been observed to ha…

0301 basic medicineamyotrophic lateral sclerosismitochondria ; mediterranean diet ; amyotrophic lateral sclerosis ; ketone bodiesMediterranean dietReviewslcsh:TX341-641ReviewDiseaseMitochondrionBioinformaticsmedicine.disease_causeNeuroprotection03 medical and health sciences0302 clinical medicineKetogenesisMedicineAmyotrophic lateral sclerosisbusiness.industryfood and beveragesmediterranean dietmedicine.diseasemitochondria030104 developmental biologyketone bodiesbusinessNeuron deathlcsh:Nutrition. Foods and food supply030217 neurology & neurosurgeryOxidative stressFood ScienceFood Science & Nutrition
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Antioxidant Alternatives in the Treatment of Amyotrophic Lateral Sclerosis: A Comprehensive Review

2020

Amyotrophic lateral sclerosis (ALS) is a neurodegenerative disease that produces a selective loss of the motor neurons of the spinal cord, brain stem and motor cortex. Oxidative stress (OS) associated with mitochondrial dysfunction and the deterioration of the electron transport chain has been shown to be a factor that contributes to neurodegeneration and plays a potential role in the pathogenesis of ALS. The regions of the central nervous system affected have high levels of reactive oxygen species (ROS) and reduced antioxidant defences. Scientific studies propose treatment with antioxidants to combat the characteristic OS and the regeneration of nicotinamide adenine dinucleotide (NAD+) lev…

0301 basic medicineamyotrophic lateral sclerosispterostilbenePterostilbenePhysiologyCentral nervous systemReviewPharmacologyNicotinamide adenine dinucleotidemedicine.disease_causelcsh:Physiology03 medical and health scienceschemistry.chemical_compound0302 clinical medicinePhysiology (medical)mitochondrial dysfunctionmedicineoxidative stressneurodegenerative diseasesAmyotrophic lateral sclerosisnicotinamide ribosidelcsh:QP1-981business.industryNeurodegenerationmedicine.disease030104 developmental biologymedicine.anatomical_structurechemistryNicotinamide ribosideNAD+ kinasebusiness030217 neurology & neurosurgeryOxidative stressFrontiers in Physiology
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Heat Shock Protein 60 in Cardiovascular Physiology and Diseases.

2020

Heat shock protein 60 (HSP60) is a highly conserved protein abundantly expressed in both prokaryotic and eukaryotic cells. In mammals, HSP60 has been primarily considered to reside in the mitochondria, where HSP60 and HSP10 form a complex and facilitate mitochondrial protein folding. However, HSP60 is also observed in the cytoplasm, the plasma membrane, and the extracellular space. HSP60 regulates a broad spectrum of cellular events including protein trafficking, peptide hormone signaling, cell survival, cell proliferation, inflammation, and immunization. In the cardiovascular system, growing evidence indicates that HSP60 could not only play an important role under physiological conditions,…

0301 basic medicineanimal structuresMini Reviewheat shock proteinheart failureInflammationchemical and pharmacologic phenomenacardiomyocyteBiologyMitochondrionBiochemistry Genetics and Molecular Biology (miscellaneous)Biochemistrycomplex mixtures03 medical and health sciences0302 clinical medicineHeat shock proteinmedicineMolecular Bioscienceslcsh:QH301-705.5Molecular BiologyCell growthfungiCardiovascular physiologyCell biology030104 developmental biologylcsh:Biology (General)Cytoplasm030220 oncology & carcinogenesisHSP60medicine.symptomSignal transductionatherosclerosisHSP60Frontiers in molecular biosciences
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Resveratrol in Autism Spectrum Disorders: Behavioral and Molecular Effects

2020

Resveratrol (RSV) is a polyphenolic stillbenoid with significant anti-oxidative and anti-inflammatory properties recently tested in animal models of several neurological diseases. Altered immune alteration and oxidative stress have also been found in patients with autism spectrum disorders (ASD), and these alterations could add to the pathophysiology associated with ASD. We reviewed the current evidence about the effects of RSV administration in animal models and in patients with ASD. RSV administration improves the core-symptoms (social impairment and stereotyped activity) in animal models and it also displays beneficial effects in other behavioral abnormalities such as hyperactivity, anxi…

0301 basic medicineantioxidantPhysiologyClinical BiochemistryReviewResveratrolBioinformaticsmedicine.disease_causeBiochemistrylaw.inventionnatural compound03 medical and health scienceschemistry.chemical_compound0302 clinical medicineImmune systemRandomized controlled triallawMedicinedevelopmental disordersMolecular BiologyRisperidonebusiness.industryanimal modellcsh:RM1-950Cell Biologymedicine.diseasePathophysiologylcsh:Therapeutics. Pharmacology030104 developmental biologychemistryAutismAnxietynutraceuticalmedicine.symptombusiness030217 neurology & neurosurgeryOxidative stressmedicine.drugAntioxidants
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