Search results for "Rheumatoid arthriti"

showing 10 items of 211 documents

Leflunomide (HWA 486), a novel immunomodulating compound for the treatment of autoimmune disorders and reactions leading to transplantation rejection.

1991

Leflunomide has been shown to be very effective in preventing and curing several autoimmune animal diseases. Further, this agent is as effective as cyclosporin A in preventing the rejection of skin and kidney transplants in rats. Preliminary results from patients suffering from severe cases of rheumatoid arthritis demonstrated that clinical and immunological parameters could be improved with leflunomide therapy. Mode of action studies revealed that this substance antagonizes the proliferation inducing activity of several cytokines and is cytostatic for certain cell types. In this light, we could show that tyrosine phosphorylation of the RR-SRC peptide substrate and the autophosphorylation o…

Graft RejectionImmunologyMolecular Sequence DataGraft vs Host DiseasePharmacologyToxicologyAutoimmune Diseaseschemistry.chemical_compoundEpidermal growth factorCyclosporin amedicineAnimalsHumansPharmacology (medical)Amino Acid SequenceMode of actionLeflunomidePharmacologybusiness.industryAnti-Inflammatory Agents Non-SteroidalTyrosine phosphorylationIsoxazolesmedicine.diseaseTransplantationDisease Models AnimalchemistryRheumatoid arthritisImmunologybusinessTyrosine kinaseImmunosuppressive AgentsLeflunomidemedicine.drugAgents and actions
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Immunological alterations in hepatitis C virus infection

2013

A higher prevalence of immunological processes has recently been reported in patients with hepatitis C virus (HCV) infection, focusing the attention of physicians and researchers on the close association between HCV and immune disorders. HCV lymphotropism represents the most important step in the pathogenesis of virus-related immunological diseases and experimental, virologic, and clinical evidence has demonstrated a trigger role for HCV both in systemic autoimmune diseases, such as rheumatoid arthritis, Sjogren syndrome, hemolytic anemia and severe thrombocytopenia, and in organ-specific autoimmune diseases, such as autoimmune hepatitis, thyroid disorders and diabetes. This review will out…

Hemolytic anemiaHepacivirusHepatitis C virusAutoimmunityAutoimmune hepatitisHepacivirusmedicine.disease_causeAutoimmunityAutoimmune DiseasesImmune systemmedicineAnimalsHumansTopic Highlightbiologybusiness.industryGastroenterologyAutoantibodyvirus diseasesGeneral MedicineHepatitis C Chronicmedicine.diseasebiology.organism_classificationVirologyHepatitis Cdigestive system diseasesRheumatoid arthritisImmunologybusiness
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Interleukin 9 neutralisation reduces collagen-induced arthritis severity in mouse models.

2021

Objective Interleukin 9 (IL-9) is a mediator of tissue damage in several inflammatory diseases. In this study we aimed to evaluate the effects of in vivo IL-9 neutralisation in mice developing collagen induced arthritis (CIA). Methods DBA/1 were immunised with collagen in Freund’s complete adjuvant (CFA) to induce arthritis. Anti-IL-9 mAb was injected in mice after the onset of arthritis (Group A) or on the same day as sensitisation and again on the day of the challenge (Group B). Histological analysis was performed in joints of mice and spleen cells were also analysed by flow cytometry. A geneset analysis was carried out on whole tarsal joint tissue transcriptomes. Results IL-9 was over-ex…

IL-9 collagen-induced arthritis rheumatoid arthritis IL-9 blockade geneset analysis.RheumatologyImmunologyImmunology and AllergyClinical and experimental rheumatology
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The collagen-like component of the complement system, C1q, is recognized by 7 S autoantibodies and is functionally impaired in synovial fluids of pat…

1996

Cross-reactivity between type II collagen (CII) and C1q, the collagen-like subunit of the first component of complement, has been demonstrated in synovial fluid (SF) from rheumatoid arthritis (RA) patients. Many authors have studied autoimmunity to CII in RA, but little work has been done on autoimmunity to C1q in RA. In the data presented here, we have been able to show that in addition to native C1q, an altered form of C1q is present in SF from RA patients. Furthermore, a low molecular weight form of C1q is present in RA SF, although its role, if any, in the pathogenesis of RA is unclear. The presence in these RA SF of C1q-specific antibodies (IgG and IgM) has been studied and we have par…

ImmunologyMolecular Sequence DataType II collagenArthritischemical and pharmacologic phenomenamedicine.disease_causeurologic and male genital diseasesAutoimmunityArthritis Rheumatoidfluids and secretionsimmune system diseasesSynovial FluidmedicineImmunology and AllergySynovial fluidHumansAmino Acid Sequenceskin and connective tissue diseasesAutoantibodiesbiologybusiness.industryComplement C1qAutoantibodyAntibodies Monoclonalmedicine.diseaseComplement systemMolecular WeightRheumatoid arthritisImmunoglobulin GImmunologybiology.proteinCollagenAntibodybusinessResearch ArticleImmunology
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Heat Shock Proteins Alterations in Rheumatoid Arthritis

2022

Rheumatoid arthritis (RA) is a chronic inflammatory and autoimmune disease characterized by the attack of the immune system on the body’s healthy joint lining and degeneration of articular structures. This disease involves an increased release of inflammatory mediators in the affected joint that sensitize sensory neurons and create a positive feedback loop to further enhance their release. Among these mediators, the cytokines and neuropeptides are responsible for the crippling pain and the persistent neurogenic inflammation associated with RA. More importantly, specific proteins released either centrally or peripherally have been shown to play opposing roles in the pathogenesis of this dise…

InflammationHeat shock proteins Hsp therapy Inflammation Neurogenic inflammation Rheumatoid arthritis VaccineOrganic ChemistryGeneral MedicineChaperonin 60CatalysisComputer Science ApplicationsInorganic ChemistryArthritis RheumatoidHumansHSP70 Heat-Shock ProteinsHSP90 Heat-Shock ProteinsPhysical and Theoretical ChemistryMolecular BiologySpectroscopyHeat-Shock Proteins
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Anti-inflammatory Therapies for Cardiovascular Disease: Signaling Pathways and Mechanisms

2019

Cardiovascular diseases (CVD) are the clinical manifestation of atherosclerosis, a chronic inflammatory disease promoted by several risk factors such as dyslipidemia, type 2 diabetes mellitus, hypertension, and smoking. Acute CVD events are the result of an unresolved inflammatory chronic state that promotes the rupture of unstable plaque lesions. Of note, the existing intensive therapies modify risk factors but do not prevent life-threatening recurrent ischemic events in high-risk patients, who have a residual inflammatory risk displayed by increased C-reactive protein (CRP) levels. Better understanding of the role of innate and adaptive immunity in plaque development and rupture has led t…

Inflammationbusiness.industrymedicine.medical_treatmentAnti-Inflammatory AgentsType 2 Diabetes MellitusGeneral MedicineDisease030204 cardiovascular system & hematologymedicine.diseaseBioinformaticsAcquired immune systemClinical trial03 medical and health sciencesC-Reactive Protein0302 clinical medicineCytokineCardiovascular DiseasesPsoriasisRheumatoid arthritismedicineHumansbusinessBiomarkersDyslipidemiaSignal TransductionRevista Española de Cardiología (English Edition)
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Targeting IL-6 signalling in early rheumatoid arthritis is followed by Th1 and Th17 suppression and Th2 expansion.

2014

OBJECTIVES: To investigate the in vitro and ex-vivo effect of IL-6 inhibition on the balance between Th1, Th2, Th17 and Treg cells. METHODS: Ten consecutive adult patients with active early rheumatoid arthritis (ERA) and ten healthy volunteers were included in the study. The percentages of Th1, Th2, Th17 and Treg cells were analysed by flow cytometry in the peripheral blood mononuclear cells obtained from controls and from RA patients at the time of first evaluation and just before the third TCZ infusion. The in vitro effect of TCZ on the different subsets of CD4+ T cells and the expression levels of Th1, Th2, Th17 and Treg-related cytokines was also assessed. RESULTS: Treatment with TCZ, b…

Interleukin 6; Tocilizumab; Early Rheumatoid ArthritisSettore MED/16 - ReumatologiaEarly Rheumatoid Arthritischemical and pharmacologic phenomenahemic and immune systemsInterleukin 6Tocilizumab
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Unterschiede im Erhaltungszustand zwischen vorderem und hinterem Kreuzband bei rheumatoider Arthritis

2008

In rheumatoid arthritis (RA) we examined the cruciate ligaments of 16 patients by the use of light- and transmission electron microscopy. Considerable differences between the anterior (ACL) and the posterior cruciate ligament (PCL) were observed. The ACL showed dramatic ultrastructural changes, against that the PCL was compact and well conserved. The preservation of PCL in joint surgery can morphologically be recommended. Perhaps the greater inflammatory process on the surface of ACL is responsible for the observed differences.

Joint surgerybusiness.industryAnterior cruciate ligamenttechnology industry and agriculturemacromolecular substancesAnatomyequipment and suppliesmusculoskeletal systemmedicine.diseaseLower limbmedicine.anatomical_structurePosterior cruciate ligamentRheumatoid arthritismedicineOrthopedics and Sports MedicineSurgeryNuclear medicinebusinesshuman activitiesZeitschrift für Orthopädie und ihre Grenzgebiete
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T Cell Large Granular Lymphocytic Leukemia in Association with Sjögren’s Syndrome

2009

T cell large granular lymphocytic (LGL) leukemia is a rare condition accounting for 2–3% of all mature lymphoid leukemias. Here, we present the case of a 73-year-old woman presenting with neutropenia and anemia (hemoglobin 9.9 g/dl). Hematological assessment revealed the presence of a T cell LGL leukemia. At the time of T cell LGL leukemia diagnosis, the patient developed xerophthalmia and xerostomia, and a diagnosis of Sjögren’s syndrome was made following salivary gland biopsy. The finding of large granular lymphocytes in the context of autoimmune disorders is well-known, though it often occurs with rheumatoid arthritis or in association with a positive autoantibody titer in the absence o…

LGL leukemiaNeutropeniaT cellchemical and pharmacologic phenomenaContext (language use)Settore MED/08 - Anatomia PatologicaNeutropeniamedicine.disease_causeAutoimmune DiseasesSettore MED/15 - Malattie Del SangueAutoimmunityhemic and lymphatic diseasesmedicineHumansAgedbusiness.industryAutoantibodyAnemiaHematologyGeneral Medicinemedicine.diseaseLymphomaLeukemia Large Granular LymphocyticLeukemiaSjogren's Syndromemedicine.anatomical_structureRheumatoid arthritisImmunologyFemalebusinessImmunosuppressive AgentsActa Haematologica
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Porphyromonas gingivalis, Periodontal and Systemic Implications: A Systematic Review

2019

In recent scientific literature, oral infections and systemic manifestations, or correlations between oral health and systemic diseases are a topic of discussion. Porphyromonas gingivalis is one of the bacteria implicated in the biofilm formation of bacterial plaque, and plays an important role in the progression of periodontal disease. In this systematic review authors have evaluated the literature of the last 10 years on P. gingivalis and all the systemic implications proven. This study therefore evaluates all the districts of the organism in which this bacterium may have implications. From the results it emerges that P. gingivalis has implications in the onset of different systemic patho…

LPSOral healthbrain diseasesArthritisBacteremiaReviewOral healthPorphyromonas gingivaliNeurodegenerative disease03 medical and health sciences0302 clinical medicinePeriodontal diseasemedicineneurodegenerative diseasesperiodontitisGeneral DentistryPorphyromonas gingivalisOrganismBrain disease030304 developmental biologyPeriodontitis0303 health sciencesbiologybusiness.industryPeriodontiti030206 dentistryCardiovascular diseasemedicine.diseasebiology.organism_classificationcardiovascular diseasesarthritisRheumatoid arthritisImmunologybusinessPorphyromonas gingivalisArthritiDentistry Journal
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