Search results for "Rna-Binding proteins"

showing 10 items of 149 documents

Cloning of a rat-specific long PCP4/PEP19 isoform

2007

We report the identification of a cDNA that encodes a putative protein of 94 amino acids and expected molecular weight of 10.7 kDa, the C-terminal half of which is identical to that of PEP19, a small, brain-specific protein involved in Ca++/calmodulin signaling. The novel rat-specific protein, tentatively named long PEP19 isoform (LPI), is the product of alternative splicing of the rat PCP4 gene encoding PEP19. We found that antibodies raised against the first 13 N-terminal amino acids of LPI, not present in PEP19, recognize a protein enriched in the developing rat brain.

Cell ExtractsGene isoformProtein isoformDNA ComplementaryCalmodulinMolecular Sequence DataNerve Tissue ProteinsAntibodiesRats Sprague-DawleyMiceExonComplementary DNAGeneticsAnimalsHumansProtein IsoformsAmino Acid SequenceRNA MessengerCloning MolecularPeptide sequencechemistry.chemical_classificationBase SequencebiologyGene Expression ProfilingAlternative splicingBrainGene Expression Regulation DevelopmentalRNA-Binding ProteinsExonsGeneral MedicineMolecular biologyIntronsRatsAmino acidchemistryBiochemistrybiology.proteinCalmodulin-Binding ProteinsPeptidesInternational Journal of Molecular Medicine
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Nucleo-cytoplasmic shuttling of RNA-binding factors: mRNA buffering and beyond.

2022

Gene expression is a highly regulated process that adapts RNAs and proteins content to the cellular context. Under steady-state conditions, mRNA homeostasis is robustly maintained by tight controls that act on both nuclear transcription and cytoplasmic mRNA stability. In recent years, it has been revealed that several RNA-binding proteins (RBPs) that perform functions in mRNA decay can move to the nucleus and regulate transcription. The RBPs involved in transcription can also travel to the cytoplasm and regulate mRNA degradation and/or translation. The multifaceted functions of these shuttling nucleo-cytoplasm RBPs have raised the possibility that they can act as mRNA metabolism coordinator…

Cell NucleusCytoplasmRNA StabilityBiophysicsRNA-Binding ProteinsRNA-binding proteinsBiochemistryTranscripció genèticaShuttlingmRNA decayStructural BiologyGeneticsRNARNA MessengerMolecular BiologyCrosstalkTranscriptionInteraccions RNA-proteïna
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Nuclear-mitochondrial interaction.

2007

The biogenesis of mitochondria depends on the coordinated expression of nuclear and mitochondrial genomes. Consequently, the control of mitochondrial biogenesis and function depends on extremely complex processes requiring a variety of well orchestrated regulatory mechanisms. It is clear that the interplay of transcription factors and coactivators contributes to the expression of both nuclear and mitochondrial respiratory genes. In addition, the regulation of mitochondria biogenesis depends on proteins that, interacting with messenger RNAs for mitochondrial proteins, influence their metabolism and expression. Moreover, a tight regulation of the import and final assembly of mitochondrial pro…

Cell NucleusRNA-binding proteinRNA-binding proteinsCell BiologyCell CommunicationBiologyMitochondrionCell biologyEpigenesis GeneticMitochondriamitochondrial fusionMitochondrial biogenesisNeoplasmsMolecular MedicineAnimalsHumansMitochondrial fissionMolecular BiologyTranscription factorPost-transcriptional regulationBiogenesistranscriptional factorpost-transcriptional regulationTranscription FactorsMitochondrion
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The effect of cadmium on brain cells in culture

2009

Cadmium is a long-living heavy metal, abundantly present in the environment, which accumulates in the body. In this study, we investigated the effects of cadmium on the expression of molecular chaperones, and of certain cell-specific proteins, in a variety of brain cell types in culture, namely primary cultures of rat cortical neurons and astrocytes, a brain capillary endothelial cell line (RB4E.B cells), and pheochromocytoma cells (PC12), induced or not to differentiate by NGF treatment. The metal induces a dose-dependent increase of Hsp70 in all cell types. Responses to the metal are cell-specific in the case of Hsc70 and Hsp90: i) in astrocytes, as well as in PC12 cells, cadmium has no s…

Cell typecadmium brain cells molecular chaperones PIPPinCell SurvivalCellBlotting Westernchemistry.chemical_elementNerve Tissue ProteinsBiologyPC12 CellsSettore BIO/10 - BiochimicaNerve Growth FactorGeneticsmedicineAnimalsCytoskeletonCell ShapeCells CulturedFluorescent DyesCerebral CortexNeuronsCadmiumBrainEndothelial CellsRNA-Binding ProteinsCell DifferentiationGeneral MedicineCell cycleMolecular biologyHsp70Cell biologyRatsEndothelial stem cellmedicine.anatomical_structurechemistryApoptosisAstrocytesCadmiumMolecular Chaperones
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Identification of an Antigen Related to the Sea Urchin RNA-Binding Protein LP54 in Mammalian Central Nervous System

2001

LP54 is an RNA-binding protein involved in localization of maternal messengers in sea urchin egg and embryos. Using a polyclonal antibody directed against Paracentrotus lividus LP54 we detected a 66-kDa cross-reacting antigen in undifferentiated and differentiated SH-SY5Y human neuroblastoma cells. After treatment of undifferentiated cells with detergent, the 66-kDa antigen was found to be enriched in the cytoskeletal fraction. By Western blot the expression of this antigen was also analyzed in regions of the CNS and in tissues of the adult rat and its exclusive presence in the hippocampus and thalamus was revealed. The immunoreactivity with P. lividus antibody against LP54 in hippocampal l…

Central Nervous SystemRNA localizationOctoxynolBlotting WesternDetergentsRNA-binding proteinBinding CompetitiveHippocampusParacentrotus lividusThalamusWestern blotAntigenbiology.animalTumor Cells CulturedmedicineAnimalsHumansRNA MessengerMolecular BiologySea urchinCytoskeletonbiologymedicine.diagnostic_testRNA-Binding ProteinsCell Differentiationbiology.organism_classificationMolecular biologyRatsMicroscopy FluorescencePolyclonal antibodiesSea Urchinsbiology.proteinElectrophoresis Polyacrylamide GelAntibodyMicrotubule-Associated ProteinsMolecular Cell Biology Research Communications
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Two Enhancers Control Transcription of Drosophila muscleblind in the Embryonic Somatic Musculature and in the Central Nervous System

2014

The phylogenetically conserved family of Muscleblind proteins are RNA-binding factors involved in a variety of gene expression processes including alternative splicing regulation, RNA stability and subcellular localization, and miRNA biogenesis, which typically contribute to cell-type specific differentiation. In humans, sequestration of Muscleblind-like proteins MBNL1 and MBNL2 has been implicated in degenerative disorders, particularly expansion diseases such as myotonic dystrophy type 1 and 2. Drosophila muscleblind was previously shown to be expressed in embryonic somatic and visceral muscle subtypes, and in the central nervous system, and to depend on Mef2 for transcriptional activatio…

Central Nervous SystemTranscription Geneticlcsh:MedicineEnhancer RNAsMechanical Treatment of SpecimensExonGenes ReporterMolecular Cell BiologyMorphogenesisPattern Formationlcsh:SciencePromoter Regions GeneticConserved SequenceGeneticsRegulation of gene expressionMultidisciplinaryMusclesDrosophila MelanogasterGene Expression Regulation DevelopmentalRNA-Binding ProteinsCell DifferentiationGenomicsAnimal ModelsInsectsEnhancer Elements GeneticElectroporationSpecimen DisruptionOrgan SpecificityRegulatory sequenceDrosophilaResearch ArticleMef2ArthropodaMolecular Sequence DataDNA transcriptionBiologyResearch and Analysis MethodsGenètica molecularModel OrganismsGeneticsAnimalsHumansEnhancerTranscription factorBase SequenceBiology and life scienceslcsh:ROrganismsPromoterCell BiologyInvertebratesSpecimen Preparation and Treatmentlcsh:QGene expressionAnimal GeneticsDevelopmental BiologyNeurosciencePLoS ONE
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Mechanisms of RNA loading into exosomes

2015

AbstractUpon fusion of multivesicular bodies (MVBs) with the plasma membrane, intraluminal vesicles (ILVs) are released into the extracellular space as exosomes. Since the lipid composition of the exosomal membrane resembles that of raft microdomains, the inward budding process involves the raft-like region of the MVB limiting membrane. Although published research suggests that cellular RNAs may be selectively sorted into exosomes, the molecular mechanisms remain elusive. In this review, we suggest that there is a continuous interaction of cellular RNAs with the outer (cytoplasmic) surface of MVBs and that the selection for incorporation of these RNAs into ILVs is based on their affinity to…

CeramideBiophysicsBiologyExosomesModels BiologicalBiochemistryIntraluminal vesiclesCeramideMembrane Lipidschemistry.chemical_compoundRaftsMembrane MicrodomainsStructural BiologymicroRNAGeneticsExtracellularAnimalsHumansMolecular BiologyVesicleCell MembraneMembraneMultivesicular BodiesRNA-Binding ProteinsRNAMicroRNACell BiologyRaftMicrovesiclesCell biologychemistryCytoplasmRNAlipids (amino acids peptides and proteins)FEBS Letters
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An exon junction complex‐independent function of Barentsz in neuromuscular synapse growth

2021

The exon junction complex controls the translation, degradation, and localization of spliced mRNAs, and three of its core subunits also play a role in splicing. Here, we show that a fourth subunit, Barentsz, has distinct functions within and separate from the exon junction complex in Drosophila neuromuscular development. The distribution of mitochondria in larval muscles requires Barentsz as well as other exon junction complex subunits and is not rescued by a Barentsz transgene in which residues required for binding to the core subunit eIF4AIII are mutated. In contrast, interactions with the exon junction complex are not required for Barentsz to promote the growth of neuromuscular synapses.…

ChemistryTransgeneProtein subunitMutantRNA-Binding ProteinsTranslation (biology)ExonsBiochemistryNeuromuscular junctionCell biologySynapsemedicine.anatomical_structureRNA splicingEukaryotic Initiation Factor-4ASynapsesGeneticsmedicineExon junction complexAnimalsDrosophila ProteinsDrosophilaMolecular BiologyReports
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RIP-Chip analysis supports different roles for AGO2 and GW182 proteins in recruiting and processing microRNA targets.

2019

Background MicroRNAs (miRNAs) are small non-coding RNA molecules mediating the translational repression and degradation of target mRNAs in the cell. Mature miRNAs are used as a template by the RNA-induced silencing complex (RISC) to recognize the complementary mRNAs to be regulated. To discern further RISC functions, we analyzed the activities of two RISC proteins, AGO2 and GW182, in the MCF-7 human breast cancer cell line. Methods We performed three RIP-Chip experiments using either anti-AGO2 or anti-GW182 antibodies and compiled a data set made up of the miRNA and mRNA expression profiles of three samples for each experiment. Specifically, we analyzed the input sample, the immunoprecipita…

Chromatin ImmunoprecipitationSupport Vector MachineRIP-Chip data analysisMiRNA bindingComputational biologyBiologylcsh:Computer applications to medicine. Medical informaticsBiochemistryAutoantigens03 medical and health sciencesOpen Reading Frames0302 clinical medicineStructural BiologymicroRNARIP-Chip data analysiCoding regionGene silencingHumansRNA MessengerMolecular BiologyGenelcsh:QH301-705.5030304 developmental biology0303 health sciencesBinding SitesApplied MathematicsGene Expression ProfilingResearchRNARNA-Binding ProteinsmicroRNA target predictionRISC proteins AGO2 and GW182Computer Science ApplicationsSettore BIO/18 - GeneticaMicroRNAslcsh:Biology (General)Gene Expression Regulation030220 oncology & carcinogenesismicroRNA regulatory activityArgonaute ProteinsMCF-7 Cellslcsh:R858-859.7DNA microarrayRIP-ChipBMC bioinformatics
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MicroRNA-Based Therapeutic Perspectives in Myotonic Dystrophy

2019

Myotonic dystrophy involves two types of chronically debilitating rare neuromuscular diseases: type 1 (DM1) and type 2 (DM2). Both share similarities in molecular cause, clinical signs, and symptoms with DM2 patients usually displaying milder phenotypes. It is well documented that key clinical symptoms in DM are associated with a strong mis-regulation of RNA metabolism observed in patient’s cells. This mis-regulation is triggered by two leading DM-linked events: the sequestration of Muscleblind-like proteins (MBNL) and the mis-regulation of the CUGBP RNA-Binding Protein Elav-Like Family Member 1 (CELF1) that cause significant alterations to their important functions in RNA processing. It ha…

Context (language use)miRNA-based drugReviewBioinformaticsMyotonic dystrophyCatalysislcsh:ChemistryInorganic ChemistryMBNL proteinsCELF1microRNADrug DiscoveryMedicineAnimalsHumansPhysical and Theoretical Chemistrylcsh:QH301-705.5Molecular BiologySpectroscopyCELF1 ProteinRna processingmyotonic dystrophymicroRNAbusiness.industryOrganic ChemistryAlternative splicingmiRNA-targeting drugRNA-Binding ProteinsGeneral MedicineGenetic Therapymedicine.diseasePhenotypeComputer Science ApplicationsAlternative SplicingMicroRNAslcsh:Biology (General)lcsh:QD1-999Drug developmentGene Expression Regulationantisense oligonucleotidesbusinessFunction (biology)International Journal of Molecular Sciences
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