Search results for "S1"

showing 10 items of 755 documents

Expression of insulin-like growth factor system components in Ewing's sarcoma and their association with survival.

2010

Abstract Aims The role of IGF system in the pathogenesis of Ewing’s sarcoma (EWS) is well-documented. However, still little information is available about the value of IGF system components as indicators of prognosis. Understanding the clinical role for IGF system in EWS patients may be important because different subtypes of patients have distinct outcome and may require different treatment protocol. We evaluated the expression of insulin-like growth factor (IGF)-receptor (IGF-IR), insulin receptor (IR), IGF-I and some major intracellular mediators (IRS1, p-ERK) in specimens from EWS patients with primary localised untreated tumours. Patients and methods 290 samples were used for immunohis…

MaleCancer Researchmedicine.medical_specialtyAdolescentmedicine.medical_treatmentEnzyme-Linked Immunosorbent AssaySarcoma EwingPathogenesisInsulin-like growth factorInternal medicinemedicineHumansInsulin-Like Growth Factor IReceptorChildbiologyReverse Transcriptase Polymerase Chain ReactionEwing's sarcomaCancermedicine.diseasePrognosisImmunohistochemistryIRS1Gene Expression Regulation NeoplasticInsulin receptorEndocrinologyTreatment OutcomeOncologybiology.proteinCancer researchDisease ProgressionFemaleSarcomaEuropean journal of cancer (Oxford, England : 1990)
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Mutations in a new gene, encoding a zinc-finger protein, cause tricho-rhino-phalangeal syndrome type I

1999

Tricho-rhino-phalangeal syndrome type I (TRPS I, MIM 190350) is a malformation syndrome characterized by craniofacial and skeletal abnormalities and is inherited in an autosomal dominant manner. TRPS I patients have sparse scalp hair, a bulbous tip of the nose, a long flat philtrum, a thin upper vermilion border and protruding ears. Skeletal abnormalities include cone-shaped epiphyses at the phalanges, hip malformations and short stature. We assigned TRPS1 to human chromosome 8q24. It maps proximal of EXT1, which is affected in a subgroup of patients with multiple cartilaginous exostoses and deleted in all patients with TRPS type II (TRPS II, or Langer-Giedion syndrome, MIM 150230; ref.2-5)…

MaleDNA Complementaryanimal structuresLanger-Giedion SyndromeMolecular Sequence DataBiologyLanger–Giedion syndromeOpen Reading FramesTRPS1 geneotorhinolaryngologic diseasesGeneticsmedicineTricho–rhino–phalangeal syndromeHumansGeneZinc fingerGeneticsSyndrome typeChromosome MappingZinc Fingersmedicine.diseaseBlotting NorthernPedigreeTrichorhinophalangeal syndromeMutationTrichorhinophalangeal Syndrome Type IFemaleChromosomes Human Pair 8Nature genetics
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Electrolytic lesion of the nucleus incertus retards extinction of auditory conditioned fear

2013

Fear memory circuits in the brain function to allow animals and humans to recognize putative sources of danger and adopt an appropriate behavioral response; and research on animal models of fear have helped reveal the anatomical and neurochemical nature of these circuits. The nucleus (n.) incertus in the dorsal pontine tegmentum provides a strong GABAergic projection to forebrain ‘fear centers’ and is strongly activated by neurogenic stressors. In this study in adult male rats, we examined the effect of electrolytic lesions of n. incertus on different stages of the fear conditioning-extinction process and correlated the outcomes with anatomical data on the distribution of n. incertus-derive…

MaleElectrolytic lesionConditioning ClassicalNerve Tissue ProteinsAmygdalaExtinction PsychologicalRats Sprague-DawleyBehavioral NeuroscienceS100 Calcium Binding Protein GPonsNeural PathwaysmedicineAnimalsFear conditioningHabituationFear memoryNeuronsFear processing in the brainBehavior AnimalRelaxinExtinctionFearExtinction (psychology)AmygdalaNucleus IncertusRatsFreezing behaviormedicine.anatomical_structureAcoustic StimulationCalbindin 2Relaxin-3PsychologyNucleus incertusNeuroscienceBasolateral amygdalaBehavioural Brain Research
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Synaptosomes: new vesicles for neuronal mitochondrial transplantation

2021

Abstract Background Mitochondrial dysfunction is a critical factor in the onset and progression of neurodegenerative diseases. Recently, mitochondrial transplantation has been advised as an innovative and attractive strategy to transfer and replace damaged mitochondria. Here we propose, for the first time, to use rat brain extracted synaptosomes, a subcellular fraction of isolated synaptic terminal that contains mitochondria, as mitochondrial delivery systems. Results Synaptosome preparation was validated by the presence of Synaptophysin and PSD95. Synaptosomes were characterized in terms of dimension, zeta potential, polydispersity index and number of particles/ml. Nile Red or CTX-FITCH la…

MaleFIS1Mitochondrial DNAlcsh:Medical technologylcsh:BiotechnologyBiomedical EngineeringPharmaceutical ScienceMedicine (miscellaneous)BioengineeringMitochondrionDNA MitochondrialApplied Microbiology and BiotechnologyMembrane Potentials03 medical and health sciencesDrug Delivery Systems0302 clinical medicinelcsh:TP248.13-248.65medicineAnimalsHomeostasisProtein Interaction Domains and MotifsNeurodegenerationDelivery system030304 developmental biologyMitochondrial transplantationSynaptosome0303 health sciencesbiologyChemistryResearchCytochrome cNeurodegenerationSynaptosomes Mitochondria Neurodegeneration Delivery system Mitochondrial transplantationCytochromes cmedicine.diseaseRatsCell biologyMitochondriaTransplantationlcsh:R855-855.5Cytoplasmbiology.proteinMolecular Medicine030217 neurology & neurosurgerySubcellular FractionsSynaptosomes
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Expression of p11 and Heteromeric TASK Channels in Rat Carotid Body Glomus Cells and Nerve Growth Factor–differentiated PC12 Cells

2020

Psychological stress activates the hypothalamus, augments the sympathetic nervous output, and elevates blood pressure via excitation of the ventral medullary cardiovascular regions. However, anatomical and functional connectivity from the hypothalamus to the ventral medullary cardiovascular regions has not been fully elucidated. We investigated this issue by tract-tracing and functional imaging in rats. Retrograde tracing revealed the rostral ventrolateral medulla was innervated by neurons in the ipsilateral dorsomedial hypothalamus (DMH). Anterograde tracing showed DMH neurons projected to the ventral medullary cardiovascular regions with axon terminals in contiguity with tyrosine hydroxyl…

MaleHistologyNerve Tissue ProteinsProximity ligation assayPC12 Cells03 medical and health sciencesPotassium Channels Tandem Pore Domain0302 clinical medicineGlomus cellmedicineAnimalsHomomericRats WistarAnnexin A2030304 developmental biologyCarotid Body0303 health sciencesChemistryEndoplasmic reticulumS100 ProteinsfungiArticlesImmunohistochemistryRatsCell biologymedicine.anatomical_structureNerve growth factorCytoplasmCarotid bodyAnatomySignal transduction030217 neurology & neurosurgeryJournal of Histochemistry & Cytochemistry
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Molecular and morphological evidence for three species of Diplostomum (Digenea: Diplostomidae), parasites of fishes and fish-eating birds in Spain

2014

Background Recent molecular studies have revealed high species diversity of Diplostomum in central and northern Europe. However, our knowledge of the distribution of Diplostomum spp. in the southern distributional range in Europe of the snail intermediate hosts (Lymnaea stagnalis and Radix spp.) is rather limited. This study aims to fill this gap in our knowledge using molecular and morphological evidence. Methods Nineteen fish species and six fish-eating bird species were sampled opportunistically in three regions (Catalonia, Extremadura and Aragon) in Spain. All isolates of Diplostomum spp. were characterised morphologically and molecularly. Partial sequences of the barcode region of the …

MaleLens metacercariaeEntomologyRange (biology)Molecular Sequence DataZoologyDiplostomum pseudospathaceumDigeneaBirdsFish DiseasesSensuAnimalsITS1-5.8S-ITS2CladePhylogenyFreshwater fishbiologyBird DiseasesEcologyResearchFishesSpecies diversityDiplostomum spathaceumCestode Infectionsbiology.organism_classificationGullsInfectious DiseasesPlatyhelminthsSpainFreshwater fishFemaleParasitologySpecies richnessCox1Parasites & Vectors
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Clinical utility of plasma-based digital next-generation sequencing in patients with advance-stage lung adenocarcinomas with insufficient tumor sampl…

2019

[Background] Approximately 30% of tumor biopsies from patients with advanced-stage lung adenocarcinomas yield insufficient tissue for successful molecular subtyping. We have analyzed the clinical utility of next-generation sequencing (NGS) of cell-free circulating tumor DNA (ctDNA) in patients with inadequate tumor samples for tissue genotyping. [Patients and methods] We conducted the study in a multi-institutional prospective cohort of clinically unselected patients with advanced-stage lung adenocarcinomas with insufficient tissue for EGFR, ALK or ROS1 genotyping across 12 Spanish institutions (n = 93). ctDNA NGS was carried out by Guardant Health (Guardant360, Redwood City, CA), using a h…

MaleLung adenocarcinoma0301 basic medicineOncologyLung NeoplasmsCirculating Tumor DNA0302 clinical medicineco-occurring genomic alterationsGenotypeProspective StudiesNeoplasm MetastasisPrecision MedicineStage (cooking)Prospective cohort studyInsufficient tissueAged 80 and overactionable genomic alterationsHazard ratioHigh-Throughput Nucleotide SequencingDNA NeoplasmGenomicsinsufficient tissueHematologyMiddle AgedActionable genomic alterationsPrognosisSurvival Ratemedicine.anatomical_structureOncology030220 oncology & carcinogenesisFemaleAdultmedicine.medical_specialtydigital next-generation sequencingAdenocarcinoma of Lung03 medical and health sciencesProto-Oncogene ProteinsInternal medicineBiomarkers TumormedicineROS1HumansLung cancerGenotypingAgedDigital next-generation sequencingLungGenome Humanbusiness.industryctDNACo-occurring genomic alterationslung adenocarcinomamedicine.disease030104 developmental biologyMutationbusinessFollow-Up StudiesAnnals of Oncology
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Hippocampal overexpression of Nos1ap promotes endophenotypes related to mental disorders

2021

Abstract Background Nitric oxide synthase 1 adaptor protein (NOS1AP; previously named CAPON) is linked to the glutamatergic postsynaptic density through interaction with neuronal nitric oxide synthase (nNOS). NOS1AP and its interaction with nNOS have been associated with several mental disorders. Despite the high levels of NOS1AP expression in the hippocampus and the relevance of this brain region in glutamatergic signalling as well as mental disorders, a potential role of hippocampal NOS1AP in the pathophysiology of these disorders has not been investigated yet. Methods To uncover the function of NOS1AP in hippocampus, we made use of recombinant adeno-associated viruses to overexpress muri…

MaleMedicine (General)Research paperDendritic spineEndophenotypesNOS1APGene ExpressionHippocampusnNOS610 Medicine & healthNitric Oxide Synthase Type IHippocampal formationBiologyHippocampusSpatial memoryGeneral Biochemistry Genetics and Molecular BiologyMiceGlutamatergicR5-920NOS1APnitric oxideCAPONAnimalsNOS-I610 Medicine & healthAdaptor Proteins Signal TransducingMental DisordersRGeneral MedicineGlutamatergic postsynaptic densityNeuropsychopharmacologyDisease Models Animalpsychiatric disordersGene Expression Regulationnervous systemMedicineDisease SusceptibilityDisks Large Homolog 4 ProteinNeuroscienceBiomarkersProtein BindingSignal Transduction
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The Insulin Receptor Substrate 1 (Irs1) in Intestinal Epithelial Differentiation and in Colorectal Cancer

2012

Colorectal cancer (CRC) is associated with lifestyle factors that affect insulin/IGF signaling, of which the insulin receptor substrate 1 (IRS1) is a key transducer. We investigated expression, localization and pathologic correlations of IRS1 in cancer-uninvolved colonic epithelium, primary CRCs with paired liver metastases and in vitro polarizing Caco2 and HT29 cells. IRS1 mRNA and protein resulted higher, relative to paired mucosa, in adenomas of familial adenomatous polyposis patients and in CRCs that overexpressed c-MYC, ß-catenin, InsRß, and IGF1R. Analysis of IRS1 immunostaining in 24 cases of primary CRC with paired colonic epithelium and hepatic metastasis showed that staining inten…

MalePathologyAnatomy and PhysiologySettore MED/06 - Oncologia MedicaMetastasisIntestinal mucosaInsulin Signaling CascadeMolecular Cell BiologyGastrointestinal CancersBasic Cancer ResearchInsulinIntestinal MucosaInsulin-like Growth FactorCOLON-CARCINOMA-CELLS; GROWTH-FACTOR RECEPTOR; BETA-CATENIN; FACTOR-I; IGF-I; NUCLEAR TRANSLOCATION; ADENOMATOUS POLYPOSIS; STEM-CELL; EXPRESSION; MUTATIONSMultidisciplinarybiologyChemistryQLiver NeoplasmsRCell PolarityCell DifferentiationSignaling CascadesGene Expression Regulation NeoplasticProtein Transportmedicine.anatomical_structureOncologyMedicineFemaleColorectal NeoplasmsHT29 CellsResearch ArticleSignal TransductionAdultendocrine systemmedicine.medical_specialtyColonScienceIRS1 IGF1R colorectal cancerEndocrine SystemGastroenterology and HepatologySignaling PathwaysFamilial adenomatous polyposisHT29 CellsmedicineHumansBiologyAgedInsulin-like growth factor 1 receptorEndocrine Physiologymedicine.diseasedigestive system diseasesEpitheliumIRS1Insulin receptorInsulin Receptor Substrate Proteinsbiology.proteinCancer researchCaco-2 CellsImmunostainingInsulin-Dependent Signal Transduction
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Expression of the Tumor Suppressor Gene Product p16INK4 in Benign and Malignant Melanocytic Lesions

1998

The gene MTS1 encodes p16INK4, an inhibitor of cyclin-dependent kinase 4, and is frequently deleted, mutated, or silenced by promoter methylation in melanoma cells and in the germline of familial melanoma patients. Although MTS1 may thus be the candidate melanoma suppressor gene that maps to chromosome 9p21, it is not clear how dysfunction at that locus temporally relates to melanoma progression. To further test its role in sporadic melanoma, the expression of p16INK4-protein and -mRNA was characterized in melanomas and melanocytic nevi by immunocytochemistry and in situ reverse transcriptase-polymerase chain reaction. Histologic tissue sections were immunolabeled with anti-p16INK4 antibody…

MalePathologymedicine.medical_specialtySkin NeoplasmsTumor suppressor geneBlotting WesternImmunocytochemistrydysplastic neviGene ExpressionDermatologyBiologyMelanocytePolymerase Chain ReactionRetinoblastoma ProteinBiochemistryMalignant transformationAntibody SpecificityGene expressionmelanomamedicineHumansMTS1Genes Tumor SuppressorRNA MessengerneoplasmsMolecular BiologyCyclin-Dependent Kinase Inhibitor p16SkinMicroscopy ConfocalMelanomaInfant NewbornAntibodies MonoclonalCell Biologymedicine.diseaseImmunohistochemistrymedicine.anatomical_structureneviDisease ProgressionCancer researchDysplastic nevusMelanocytesImmunohistochemistryJournal of Investigative Dermatology
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