Search results for "SECR"

showing 10 items of 1169 documents

Arthropod-induced neoplastic formations on trees change photosynthetic pigment levels and oxidative enzyme activities

2005

Abstract In order to test the hypothesis that arthropod-induced neoplastic formations on trees affect biochemical characteristics of both the newly formed galls and host plant tissues, biochemical characteristics with a possible adaptive role were determined in nine gall-former–host tree combinations. Photosynthetic pigments, extractable protein content, and oxidative enzyme activities were determined in gall tissues, leaf tissues of galled leaves, and leaves on ungalled tree branches. Neoplastic tissues were characterized by a low content of photosynthetic pigments, decreased chlorophyll a/b ratio, lower extractable protein content, and decreased activities of peroxidase and polyphenol oxi…

biologyHost (biology)digestive oral and skin physiologyfood and beveragesPlant SciencePhotosynthetic pigmentPhotosynthesisdigestive systemPolyphenol oxidasedigestive system diseaseschemistry.chemical_compoundPigmentfluids and secretionschemistryvisual_artOxidative enzymeBotanybiology.proteinvisual_art.visual_art_mediumGallEcology Evolution Behavior and SystematicsPeroxidaseJournal of Plant Interactions
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Initiation and Modulation of ECF Generation and Secretion from Human Polymorphonuclear NeutrophilsX.

1978

The primary mediators which are involved in inflammatory processes are histamine, the slow reacting substance of anaphylaxis (SRS-A), the platelet aggregating factor (PAF) and the eosinophil chemotactic factor (ECF). These mediators are primarily localized within mast cells and basophil leucocytes, which represent the target cells of allergic reaction. Binding of IgE to mast cells and basophil leucocytes and addition of the appropriate antigen then leads to the activation of the target cells with a subsequent release of their mediators. SRS and ECF have also been found in a non mast cell source, e.g. in human PMNs. ECF can be generated and released from human PMNs by different stimuli, such…

biologyInflammationBasophilEosinophilMast cellImmunoglobulin ECell biologychemistry.chemical_compoundmedicine.anatomical_structurechemistrymedicinebiology.proteinSecretionmedicine.symptomSlow-reacting substance of anaphylaxisHistamine
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Source of the host marking pheromone in the egg parasitoid Trissolcus basalis (Hymenoptera: Scelionidae).

2001

After oviposition, Trissolcus basalis females always mark the host's surface, depositing host marking substances for herself and to warn other ovipositing females. The perception of these host marking substances, probably through the antennae, can induce the female to leave and seek healthy hosts. Parasitoid females exposed to conspecific parasitized egg masses left the host egg masses significantly more often than when exposed to non-parasitized egg masses. More egg mass leaving behavior also was observed when the egg masses were treated with Dufour's gland secretion but not when treated with secretion from the common oviducts. The common oviduct has a secretory epithelium that produces el…

biologyPhysiologyHost (biology)fungiZoologyHymenopterabiology.organism_classificationParasitoidInsect ScienceBotanyPheromoneOviductSecretionDufour's glandScelionidaeJournal of insect physiology
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Pathogenesis of Atherosclerosis: The Alternative Hypothesis

1998

The concept that oxidation is the major single event underlying the transformation of LDL to a proinflammatory molecule dominates the world literature. An alternative hypothesis on the pathogenesis of atherosclerosis will be presented here. We have found that nonoxidative, enzymatic modification of LDL with ubiquitous enzymes also transforms the molecule to an atherogenic moiety. Enzymatically altered LDL (E-LDL) shares major properties in common with lipoproteins that have been isolated from atherosclerotic lesions. It activates complement and is recognized by a scavenger receptor on human macrophages, thus inducing foam cell formation. Uptake of E-LDL is accompanied by induction of MCP−1 …

biologybusiness.industryComplement systemCell biologyProinflammatory cytokinePathogenesisImmunologybiology.proteinExtracellularMedicinelipids (amino acids peptides and proteins)Radiology Nuclear Medicine and imagingSecretionScavenger receptorAntibodyCardiology and Cardiovascular MedicinebusinessFoam cellJournal of Interventional Cardiology
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Additional file 8: Figure S4. of Establishing gene models from the Pinus pinaster genome using gene capture and BAC sequencing

2016

Alignment of the PAT gene promoter [GenBank:HE866755], to the gene capture PAT gene 5´upstream region. (TIF 332 kb)

body regionsfluids and secretionsanimal structuresanimal diseasescardiovascular system
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Additional file 8: Figure S4. of Establishing gene models from the Pinus pinaster genome using gene capture and BAC sequencing

2016

Alignment of the PAT gene promoter [GenBank:HE866755], to the gene capture PAT gene 5´upstream region. (TIF 332 kb)

body regionsfluids and secretionsanimal structuresanimal diseasescardiovascular system
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Toll-like receptors play a crucial part in the pathophysiological activity of antiphospholipid antibodies

2011

The antiphospholipid syndrome (APS) is a systemic autoimmune disease characterized by thrombosis, recurrent fetal loss and the presence of a variety of antiphospholipid antibodies (aPL), directed to phospholipids like Cardiolipin and phospholipid binding proteins like β2-glycoprotein I. Till date, the pathophysiological processes underlying these thrombotic events were still not fully understood. Recent data support the idea that the aPL might act via enhanced cytokine release due to activation of certain Toll-like receptors. The investigation of some of those mechanisms in more detail enlightens the involvement of the intracellular receptors TLR7 and TLR8 in a central point. Using patients…

business.industryAntiphospholipid antibodiesmedicine.medical_treatmentImmunologyStimulationReview ArticleTLR7medicine.diseaseMonocytesTLR2CytokineRheumatologyAntiphospholipid syndromeImmunologyTLR2MedicineTumor necrosis factor alphaSecretionTLR4businessReceptorTLR8TLR7Autoimmunity Highlights
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Autoantikörper gegen die Komplementkomponente C1q beim Systemischen Lupus Erythematodes

1990

Autoantibodies against C1q, a subcomponent of the first complement component C1, could be detected in 49.4% of sera from patients with systemic lupus erythematosus (SLE). They are directed against the collagen-like portion of the C1q molecule and recognize only bound, but not fluid-phase C1q. The appearance of these autoantibodies in the course of SLE correlates with the detection of IgG in the C1q-Solid-Phase-Bindingassay, with high titres of dsDNA-antibodies and with depressed total complement activity (CH50) and C1q-values. Our investigations show that autoantibodies against the collagen-like portion of bound C1q but not immune complexes are the main constituent of C1q-binding IgG in SLE.

business.industryAutoantibodychemical and pharmacologic phenomenaGeneral Medicineurologic and male genital diseasesComplement activityfluids and secretionsImmune systemimmune system diseasesDrug DiscoveryImmunologyMolecular MedicineMedicineskin and connective tissue diseasesbusinessGenetics (clinical)Anti-SSA/Ro autoantibodiesKlinische Wochenschrift
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FoSBaS: A bi-directional secrecy and collusion resilience key management scheme for BANs

2012

Body Area Network (BAN) consists of various types of small physiological sensors, transmission modules and low computational components and can thus form an E-health solution for continuous all-day and any-place health monitoring. To protect confidentiality of collected data, a shared group key is usually deployed in a BAN, and consequently a secure communication group is generated. In this paper, we propose a bi-directional security and collusion resilience key management scheme for BAN, referred to as FoSBaS. Detailed analysis shows that the scheme can provide both forward security and backward security and resist against collusion attacks. Furthermore, the FoSBaS is implemented on a Sun …

business.industryComputer scienceNode (networking)TestbedCryptographyEnergy consumptionSun SPOTComputer securitycomputer.software_genreSecure communicationForward secrecyBody area networkSecrecyResilience (network)businessKey managementcomputerWireless sensor networkGroup keyComputer network2012 IEEE Wireless Communications and Networking Conference (WCNC)
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SCARKER: A sensor capture resistance and key refreshing scheme for mobile WSNs

2011

How to discover a captured node and to resist node capture attack is a challenging task in Wireless Sensor Networks (WSNs). In this paper, we propose a node capture resistance and key refreshing scheme for mobile WSNs which is based on the Chinese remainder theorem. The scheme is able of providing forward secrecy, backward secrecy and collusion resistance for diminishing the effects of capture attacks. By implementing our scheme on a Sun SPOT based sensor network testbed, we demonstrate that the time for updating a new group key varies from 56 ms to 546 ms and the energy consumption is limited to 16.5–225 mJ, depending on the length of secret keys and the number of sensors in a group.

business.industryComputer scienceNode (networking)TestbedMobile computingEnergy consumptionSun SPOTKey distribution in wireless sensor networksForward secrecyMobile telephonybusinessWireless sensor networkComputer networkGroup key2011 IEEE 36th Conference on Local Computer Networks
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