Search results for "SENSITIZATION"

showing 10 items of 237 documents

Contact sensitivity as a model for T-cell activation in skin.

1995

Contact sensitivity has served as a useful model for the primary activation of T cells in skin and skin-associated lymphoid tissue. We have been interested in the early signals necessary for the induction of an allergen-specific T-cell response, as well as the factors controlling the intensity and extent of such an immune reaction. Because cytokines qualified as possible candidate molecules involved in directing primary immune responses in skin, we studied the early changes in the cytokine pattern of the epidermis. Apart from defining a cytokine pattern specifically induced only after application of allergen, we also identified Langerhans-cell-derived interleukin (IL)-1β as the first cytoki…

Langerhans cellmedicine.medical_treatmentT cellT-LymphocytesInterleukinCell BiologyDermatologyBiologyDermatitis ContactLymphocyte ActivationBiochemistryMicemedicine.anatomical_structureImmune systemCytokineImmunologymedicineAnimalsAntigen-presenting cellMolecular BiologyImmunologic ToleranceSensitizationSkinThe Journal of investigative dermatology
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Decreased release of histamine and sulfidoleukotrienes by human peripheral blood leukocytes after wasp venom immunotherapy is partially due to induct…

1999

Abstract Background: Recent studies provide evidence that venom immunotherapy (VIT) alters the pattern of cytokine production by inducing an allergen-specific T-cell shift in cytokine expression from T H2 (IL-4, IL-5) to T H1 (IFN-γ) cytokines and also inducing the production of IL-10. Objective: This study was carried out to analyze whether these changes in cytokine production of T cells already observed 1 week after the initiation of VIT in subjects with wasp venom allergy also influence the reactivity of effector cells, such as mast cells and basophils. Methods: All subjects included in this study had a history of severe systemic allergic reactions to wasp stings and positive skin test r…

LeukotrienesAllergyT-Lymphocytesmedicine.medical_treatmentImmunologyDown-RegulationWasp VenomsImmunoglobulin EHistamine ReleaseInterferon-gammachemistry.chemical_compoundImmune systemLeukocytesmedicineHumansImmunology and AllergyInterferon gammaLeukotriene E4biologyReceptors IgEAntibodies Monoclonalmedicine.diseaseBasophilsInterleukin-10Interleukin 10CytokinechemistryDesensitization ImmunologicImmunologybiology.proteinHistaminemedicine.drugJournal of Allergy and Clinical Immunology
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Importance of selenium status in patients with chronic heart failure

2015

Abstract Overactivity of the immune system may be a worthwhile therapeutic target for implementing prognostic improvements. Therefore the impact of lipopolysaccharide (LPS) desensitization on survival may help in the development of novel therapies. An understanding of the pathophysiology of the trace element selenium may complement such approaches, as recent data suggest that inflammatory responses are selenium-dependent.

Lipopolysaccharidebusiness.industrymedicine.medical_treatmentchemistry.chemical_elementmedicine.diseasePathophysiologychemistry.chemical_compoundImmune systemchemistryHeart failureImmunologyMedicineIn patientlcsh:Qbusinesslcsh:Sciencelcsh:Science (General)SeleniumDesensitization (medicine)lcsh:Q1-390Perspectives in Science
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Maternal Motivation: Exploring the Roles of Prolactin and Pup Stimuli

2020

Motherhood entails increased motivation for pups, which become strong reinforcers and guide maternal behaviours. This depends on steroids and lactogens acting on the brain of females during pregnancy and postpartum. Since virgin female mice exposed to pups are nearly spontaneously maternal, the specific roles of endocrine and pup-derived signals in the induction of maternal motivation remain unclear. This work investigates maternal motivation in dams and virgin female mice, using a novel variant of the pup retrieval paradigm, the motivated pup retrieval test. We also analyse the role of prolactin (PRL) and of stimuli derived from a litter of pups and its mother, in the acquisition of matern…

Litter (animal)medicine.medical_specialtyprolactinEndocrinology Diabetes and Metabolism030209 endocrinology & metabolismBiologyAmygdala030218 nuclear medicine & medical imaging03 medical and health sciencesCellular and Molecular NeuroscienceMice0302 clinical medicineEndocrinologyInternal medicinemedicinesociosexual brain networkEndocrine systemAnimalsMaternal BehaviorSensitizationreproductive and urinary physiologyPregnancyMotivationIncreased motivationBehavior AnimalEndocrine and Autonomic Systemsamygdalamedicine.diseaseProlactinProlactinbehaviourmaternalmedicine.anatomical_structureEndocrinologyAnimals NewbornimmunohistochemistryMedial preoptic nucleusFemale
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Environmental risk factors and lung diseases in children: From guidelines to health effects

2013

Abstract During the last decades research all over the world has highlighted the deleterious effects of outdoor and indoor pollution on respiratory health of adults and children. The World Health Organization (WHO) “ Air quality guidelines for Europe ” played a fundamental role in providing information and guidance to authorities involved in the air pollution field and they are considered the key source on which the European Commission's directive on air quality is based. Children appear to be most vulnerable to the harmful effects of outdoor pollutants, which can cause both acute exacerbations, as well as chronic respiratory symptoms and diseases. Possible mechanisms include the induction …

Lung Diseasesmedicine.medical_specialtyAir pollutionmedicine.disease_causelaw.inventionAllergic sensitizationSettore MED/38 - Pediatria Generale E SpecialisticachildrenRisk FactorslawEnvironmental healthEpidemiologymedicineHumanspollutionChildAir quality indexPollutantAir PollutantsLungbusiness.industryMortality rateInfantObstetrics and Gynecologyhealthpollution health childrenVentilationmedicine.anatomical_structureChild PreschoolPediatrics Perinatology and Child HealthVentilation (architecture)Tobacco Smoke Pollutionbusiness
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Mast cells and the development of allergic airway disease

2008

Murine models have highlighted the importance of T-cells and TH2 cytokines in development of allergen-induced airway disease. In contrast, the role of mast cells for the development of allergic airway disease has been controversial. Recent studies in murine models demonstrate a significant contribution of mast cells during the development of airway hyperresponsiveness and airway inflammation. Furthermore these models have allowed identifying certain mast cell-produced mediators (e.g. histamine and leukotriene B4) to be involved in the recruitment of effector T-cells into the lung. Additionally, mast cell-produced TNF can directly activate TH2 cells and contribute to the development of aller…

Lungbusiness.industryLeukotriene B4EffectorAirway inflammationPublic Health Environmental and Occupational HealthReviewrespiratory systemToxicologyrespiratory tract diseaseschemistry.chemical_compoundmedicine.anatomical_structureAirway diseasechemistryImmunologymedicineTumor necrosis factor alphabusinessSafety ResearchSensitizationHistamineJournal of Occupational Medicine and Toxicology
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Modulation of Contact Sensitivity Responses by Bacterial Superantigen

1995

Superantigens are potent modulators of the immune system, especially T cells. Therefore, we determined the influence of superantigens on the T-cell-mediated immune response, contact sensitivity. We chose the combination of staphylococcal enterotoxin B (SEB) as superantigen and 2,4-dinitrofluorbenzene (DNFB) as the contact sensitizer, because in BALB/c mice SEB reacts almost exclusively with V beta 8+ T cells, and these cells are capable of transferring contact sensitivity to DNFB from sensitized donors to naive syngeneic recipients. Pretreatment with a single intradermal injection of 50 ng SEB 24 h before DNFB exposure at the same site on the lower abdomen enhanced the induction of contact …

Lymphoid Tissue24-dinitrofluorbenzeneReceptors Antigen T-Cell alpha-betaT-LymphocytesDown-Regulationchemical and pharmacologic phenomenaDermatologyEnterotoxinDermatitis Contactcontact sensitivityBacterial superantigenBiochemistrysuperantigenProinflammatory cytokineEnterotoxinsInterferon-gammaMiceImmune systemmedicineSuperantigenAnimalsIntradermal injectionMolecular BiologySensitizationSkinAntigens BacterialMice Inbred BALB CSuperantigensbusiness.industryhemic and immune systemsCell BiologyContact sensitivitybiological factorsStaphylococcal enterotoxin Bmedicine.anatomical_structureImmunologyDinitrofluorobenzeneFemaleImmunizationbusinessJournal of Investigative Dermatology
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Chemotherapy overcomes TRAIL-R4-mediated TRAIL resistance at the DISC level

2011

International audience; TNF-related apoptosis-inducing ligand or Apo2L (Apo2L/TRAIL) is a promising anti-cancer drug owing to its ability to trigger apoptosis by binding to TRAIL-R1 or TRAIL-R2, two membrane-bound receptors that are often expressed by tumor cells. TRAIL can also bind non-functional receptors such as TRAIL-R4, but controversies still exist regarding their potential to inhibit TRAIL-induced apoptosis. We show here that TRAIL-R4, expressed either endogenously or ectopically, inhibits TRAIL-induced apoptosis. Interestingly, the combination of chemotherapeutic drugs with TRAIL restores tumor cell sensitivity to apoptosis in TRAIL-R4-expressing cells. This sensitization, which ma…

MESH: CASP8 and FADD-Like Apoptosis Regulating ProteinMESH : Antineoplastic Combined Chemotherapy ProtocolsCASP8 and FADD-Like Apoptosis Regulating ProteinTRAILApoptosisMESH : Models BiologicalMitochondrionMESH : RNA Small InterferingMESH: Caspase 8TNF-Related Apoptosis-Inducing LigandMESH : TNF-Related Apoptosis-Inducing LigandMESH : Tumor Necrosis Factor Decoy Receptors0302 clinical medicineRNA interferenceNeoplasmsAntineoplastic Combined Chemotherapy ProtocolsMESH: RNA Small InterferingMESH: NeoplasmsRNA Small InterferingReceptorSensitizationCaspase 80303 health sciencesMESH : Caspase 8MESH: Drug Resistance Neoplasm3. Good healthCell biologyMESH: Antineoplastic Combined Chemotherapy ProtocolsMESH : Drug Resistance Neoplasmmedicine.anatomical_structure030220 oncology & carcinogenesisRNA InterferenceMESH : GPI-Linked ProteinsMESH: TNF-Related Apoptosis-Inducing LigandDeath Domain Receptor Signaling Adaptor ProteinsProgrammed cell deathMESH: Cell Line Tumorc-FLIPMESH: RNA InterferenceBiologyGPI-Linked ProteinsCaspase 8Models Biological03 medical and health sciencesCell Line TumorReceptors Tumor Necrosis Factor Member 10cmedicineTRAIL-R4HumanscancerChemotherapy[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular BiologyMESH: Receptors TNF-Related Apoptosis-Inducing LigandMESH : Receptors TNF-Related Apoptosis-Inducing Ligand[ SDV.BBM ] Life Sciences [q-bio]/Biochemistry Molecular BiologyMolecular Biology030304 developmental biologyOriginal PaperMESH: HumansMESH : Cell Line TumorMESH: ApoptosisMESH : HumansMESH: Models BiologicalMESH : CASP8 and FADD-Like Apoptosis Regulating ProteinCell BiologyMESH: Tumor Necrosis Factor Decoy ReceptorsMESH : NeoplasmsReceptors TNF-Related Apoptosis-Inducing LigandTumor Necrosis Factor Decoy ReceptorsDrug Resistance NeoplasmApoptosisMESH : RNA InterferenceMESH: GPI-Linked ProteinsMESH : ApoptosisMESH : Death Domain Receptor Signaling Adaptor ProteinsMESH: Death Domain Receptor Signaling Adaptor ProteinsTumor Necrosis Factor Decoy Receptors
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Increased ethanol consumption after interruption of fat bingeing

2018

There is a marked comorbidity between alcohol abuse and eating disorders, especially in the young population. We have previously reported that bingeing on fat during adolescence increases the rewarding effects of ethanol (EtOH). The aim of the present work was to study if vulnerability to EtOH persists after cessation of binge eating. OF1 mice binged on fat (HFB: high-fat binge) during adolescence (PND 25-43) and were tested for 15 days after the last access to HFB (on PND 59) using the self-administration paradigm, the conditioned place preference (CPP) and locomotor sensitization to ethanol. Our results showed that after 15 days of cessation of fat ingestion, mice increased their consumpt…

Male0301 basic medicinePhysiologySocial Scienceslcsh:MedicineAlcohol abuseDrug AddictionBiochemistryFatsMicechemistry.chemical_compound0302 clinical medicineCocaineMedicine and Health SciencesPsychologyIngestionPublic and Occupational HealthBulimiaOvereatinglcsh:ScienceMultidisciplinaryOrganic CompoundsLipidsBody FluidsChemistryEating disordersBloodBehavioral PharmacologyPhysical SciencesAnatomymedicine.symptomResearch Articlemedicine.medical_specialtyAdolescentAlcohol DrinkingSubstance-Related DisordersAddiction03 medical and health sciencesAlkaloidsRecreational Drug UseInternal medicineMental Health and Psychiatrymental disordersmedicineAnimalsHumansNutritionPharmacologyEthanolEthanolBinge eatingBiological Locomotionbusiness.industryOrganic Chemistrylcsh:RChemical CompoundsBiology and Life Sciencesmedicine.diseaseDietary FatsConditioned place preferenceDietLocomotor sensitization030104 developmental biologyEndocrinologychemistryFoodAlcoholslcsh:Qbusiness030217 neurology & neurosurgeryPLOS ONE
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The Effect of Outdoor Aeroallergens on Asthma Hospitalizations in Children in North-Western Tuscany, Italy.

2022

Few data are currently available on the effects of aeroallergens in triggering respiratory symptoms in children. To evaluate the potential effects of daily outdoor aeroallergens loads on childhood admissions, in this case-crossover study, we analyzed data from 85 children hospitalized at the University Hospital of Pisa, Italy, for asthma or asthma-like symptoms without respiratory infection, between 2010 and 2019. Data were linked to outdoor allergens, temperature, nitrogen dioxide, and relative humidity observed during the same period. A 10-grains/m3 increase in the total aeroallergen concentration was associated with an increased risk of admission at lag 0 (OR = 1.054, 95% CI: 1.011&ndash…

MaleAir PollutantsCross-Over Studiessensitization to outdoor allergens.AllergyHealth Toxicology and MutagenesisNitrogen DioxideAlternaria alternata; allergy; asthma; climate change; pollen; sensitization to outdoor allergens.Public Health Environmental and Occupational HealthAllergensAsthmaHospitalizationAllergy; Alternaria alternata; Asthma; Climate change; Pollen; Sensitization to outdoor allergensAllergy Alternaria alternata Asthma Climate change Pollen Sensitization to outdoor allergensAlternaria alternataClimate changePollenHumansChildSensitization to outdoor allergensInternational journal of environmental research and public health
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