Search results for "STING"

showing 10 items of 3756 documents

Fasciola hepatica development in the experimentally infected black rat Rattus rattus

1998

The finding of natural infection of Rattus rattus by Fasciola hepatica on Corsica has stimulated further research into the role of the black rat in the epidemiology of fascioliasis. Corsican black rats were experimentally individually infected with 20 metacercariae from cattle and murine isolates obtained from naturally infected bovines and black rats. The following results were obtained: (a) in R. rattus infected with the cattle isolate, normal adult fluke development took place and infection persisted for a long period, with emission of eggs showing embryogenic capacity; (b) the development of F. hepatica adults paralleled the ontogenetic trajectories observed in other rodent-F. hepatica …

MaleFascioliasisOntogenyHelminthiasisZoologyHepaticaparasitic diseasesmedicineAnimalsParasite hostingFasciola hepaticaDisease ReservoirsInfectivityGeneral VeterinarybiologyEcologyGeneral MedicineFasciola hepaticamedicine.diseasebiology.organism_classificationRatsMuridaestomatognathic diseasesInfectious DiseasesBlack ratInsect ScienceCattleFemaleParasitologyDisease SusceptibilityTrematodaParasitology Research
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Correlation between egg-shedding and uterus development in Fasciola hepatica human and animal isolates: applied implications

2010

The emission of Fasciola hepatica eggs in faeces is usually subject to oscillations along time in animals as well as humans. Thus, looking for alternative biological markers reflecting eggs shed per gram of faeces (epg) with lower oscillations may be useful. This study analyzes the possible relationship between liver-fluke uterus area and epg. Uterus area (UA) development of adult F. hepatica obtained at different days post infection (dpi) in a Wistar rat model with isolates obtained from cattle, sheep, pigs and humans from the endemic human fascioliasis zone of the Northern Bolivian Altiplano was analyzed and compared with the number of eggs shed per gram of faeces as obtained through the …

MaleFascioliasisVeterinary medicineSwineSnailsUterusHost-Parasite InteractionsFecesHepaticaparasitic diseasesmedicineParasite Egg CountAnimalsHumansParasite hostingFasciola hepaticaRats WistarParasite Egg CountFecesOvumSheepGeneral VeterinarybiologyHost (biology)UterusGeneral MedicineFasciola hepaticaLiver flukebiology.organism_classificationRatsSpecific Pathogen-Free Organismsmedicine.anatomical_structureImmunologyRegression AnalysisCattleFemaleParasitologyBiomarkersVeterinary Parasitology
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Global, regional, and national comparative risk assessment of 79 behavioural, environmental and occupational, and metabolic risks or clusters of risk…

2015

Summary Background The Global Burden of Disease, Injuries, and Risk Factor study 2013 (GBD 2013) is the first of a series of annual updates of the GBD. Risk factor quantification, particularly of modifiable risk factors, can help to identify emerging threats to population health and opportunities for prevention. The GBD 2013 provides a timely opportunity to update the comparative risk assessment with new data for exposure, relative risks, and evidence on the appropriate counterfactual risk distribution. Methods Attributable deaths, years of life lost, years lived with disability, and disability-adjusted life-years (DALYs) have been estimated for 79 risks or clusters of risks using the GBD 2…

MaleFine particulate matterNutrition and DiseaseMESH : SanitationHealth BehaviorDiseasesMESH: Metabolic DiseasesMESH: Global Health030204 cardiovascular system & hematologyMESH: Risk AssessmentGlobal HealthMESH : Nutritional StatusMESH: Occupational Exposure0302 clinical medicineUnsafe SexMESH: Risk FactorsRisk FactorsVoeding en ZiekteMedicineAir-pollutionMESH : Female030212 general & internal medicineMESH : Risk AssessmentSanitationWasting2. Zero hungerFactors de risc en les malaltiesMedicine (all)[ SDV.SPEE ] Life Sciences [q-bio]/Santé publique et épidémiologiePublic Health Global Health Social Medicine and EpidemiologyGeneral MedicineMESH : Occupational DiseasesMESH: Nutritional StatusAll-cause mortalityMESH : Risk FactorshumanitiesEnvironmental Exposure; Female; Global Health; Health Behavior; Humans; Male; Metabolic Diseases; Nutritional Status; Occupational Diseases; Occupational Exposure; Risk Assessment; Risk Factors; Sanitation; Medicine (all)Tobacco smoking3. Good healthOccupational DiseasesNutritional StatuMESH : Occupational ExposureMESH : Metabolic DiseasesCohortFemalemedicine.symptomRisk assessmentBlood-pressureHumanMESH: Occupational DiseasesRisk factors in diseasesCoronary-heart-diseaseMESH : MaleMESH: Health BehaviorMESH: Environmental ExposureNutritional StatusPopulation healthBody-mass indexRisk Assessment03 medical and health sciencesHousehold cookingMetabolic DiseasesCardiovascular-diseaseEnvironmental healthGeneral & Internal MedicineOccupational Exposureparasitic diseasesLife ScienceMESH: SanitationHumansRisk factorMESH : Health BehaviorVLAGGBD2013MESH: Humansbusiness.industryRisk FactorGlobal Burden of Disease Study; 79 behavioural environmental and occupational and metabolic risksLong-term exposureMESH : HumansCAUSE-SPECIFIC MORTALITYEnvironmental ExposureMESH: MaleMetabolic DiseaseOccupational DiseaseFolkhälsovetenskap global hälsa socialmedicin och epidemiologiMALE BRITISH DOCTORSYears of potential life lostRelative riskMalaltiesMESH : Global HealthOUTDOOR AIR-POLLUTION[SDV.SPEE]Life Sciences [q-bio]/Santé publique et épidémiologiebusinessMESH : Environmental ExposureMESH: FemaleLancet
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Transforming RNA-Seq Data to Improve the Performance of Prognostic Gene Signatures

2014

Gene expression measurements have successfully been used for building prognostic signatures, i.e for identifying a short list of important genes that can predict patient outcome. Mostly microarray measurements have been considered, and there is little advice available for building multivariable risk prediction models from RNA-Seq data. We specifically consider penalized regression techniques, such as the lasso and componentwise boosting, which can simultaneously consider all measurements and provide both, multivariable regression models for prediction and automated variable selection. However, they might be affected by the typical skewness, mean-variance-dependency or extreme values of RNA-…

MaleGene Expressionlcsh:Medicinecomputer.software_genreBioinformaticslcsh:ScienceExtreme value theoryMultidisciplinaryMultivariable calculusStatisticsRegression analysisGenomicsPrognosisKidney NeoplasmsNeoplasm ProteinsLeukemia Myeloid AcuteMedicineProbability distributionFemaleSequence AnalysisAlgorithmsResearch ArticleStatistical DistributionsRiskBoosting (machine learning)Clinical Research DesignFeature selectionBiostatisticsBiologyMachine learningMolecular GeneticsGenome Analysis ToolsCovariateHumansStatistical MethodsGene PredictionBiologyCarcinoma Renal CellProbabilityClinical GeneticsSequence Analysis RNAbusiness.industrylcsh:RPersonalized MedicineModelingComputational BiologyProbability TheorySurvival AnalysisSkewnessMultivariate AnalysisRNAlcsh:QArtificial intelligenceGenome Expression AnalysisTranscriptomebusinesscomputerMathematicsPLoS ONE
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A novel DFNB1 deletion allele supports the existence of a distant cis-regulatory region that controls GJB2 and GJB6 expression

2010

Contains fulltext : 87760_1.pdf (author's version ) (Open Access) Contains fulltext : 87760_2.pdf (Publisher’s version ) (Closed access) Eleven affected members of a large German-American family segregating recessively inherited, congenital, non-syndromic sensorineural hearing loss (SNHL) were found to be homozygous for the common 35delG mutation of GJB2, the gene encoding the gap junction protein Connexin 26. Surprisingly, four additional family members with bilateral profound SNHL carried only a single 35delG mutation. Previously, we demonstrated reduced expression of both GJB2 and GJB6 mRNA from the allele carried in trans with that bearing the 35delG mutation in these four persons. Usin…

MaleGenetics and epigenetic pathways of disease [NCMLS 6][SDV]Life Sciences [q-bio]PenetranceMESH: Base SequenceRegulatory Sequences Nucleic Acidsensorineural hearing lossConnexinsMESH: GenotypeMESH: Hearing Loss Sensorineural/diagnosisMESH: PenetranceGenotypeCopy-number variationGenetics (clinical)Sequence DeletionGeneticsComparative Genomic Hybridization0303 health sciencesMESH: Genetic TestingMESH: Gene Expression Regulation*030305 genetics & heredityPenetranceGJB2PedigreeConnexin 26MESH: Sequence Deletion*MESH: Hearing Loss Sensorineural/geneticsFemaleChromosome DeletionFunctional Neurogenomics [DCN 2]GJB6GenotypeMESH: PedigreeMESH: Chromosome DeletionHearing Loss SensorineuralMolecular Sequence Dataconnexin 26connexin 30DFNB1gene expression regulationGJB2GJB6sensorineural hearing losssequence deletionBiologyMESH: Connexin 30MESH: Connexins/genetics*MESH: Sequence Homology Nucleic AcidArticleGenomic disorders and inherited multi-system disorders [IGMD 3]03 medical and health sciencesMonoallelic MutationGJB6MESH: Connexin 26Sequence Homology Nucleic AcidConnexin 30otorhinolaryngologic diseasesGeneticsHumansGenetic TestingAlleleGeneMESH: Regulatory Sequences Nucleic Acid/genetics*AllelesDFNB1030304 developmental biologyFamily HealthMESH: HumansMESH: Molecular Sequence DataBase SequenceChromosomes Human Pair 13MESH: AllelesBreakpointMESH: MaleMESH: Comparative Genomic HybridizationGene Expression RegulationMESH: Family Healthbiology.proteinHuman medicineMESH: Chromosomes Human Pair 13/geneticsMESH: FemaleClinical Genetics
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Bayesian model to detect phenotype-specific genes for copy number data

2012

Abstract Background An important question in genetic studies is to determine those genetic variants, in particular CNVs, that are specific to different groups of individuals. This could help in elucidating differences in disease predisposition and response to pharmaceutical treatments. We propose a Bayesian model designed to analyze thousands of copy number variants (CNVs) where only few of them are expected to be associated with a specific phenotype. Results The model is illustrated by analyzing three major human groups belonging to HapMap data. We also show how the model can be used to determine specific CNVs related to response to treatment in patients diagnosed with ovarian cancer. The …

MaleGenotypeGene DosageHapMap ProjectBiologylcsh:Computer applications to medicine. Medical informaticsPopulation stratificationBayesian inferencePolymorphism Single NucleotideBiochemistry03 medical and health sciencesBayes' theorem0302 clinical medicineStructural BiologymedicineHumansComputer SimulationGenetic Predisposition to DiseaseGenetic TestingCopy-number variationInternational HapMap Projectlcsh:QH301-705.5Molecular Biology030304 developmental biologyGenetic testingGenetics0303 health sciencesModels StatisticalModels Geneticmedicine.diagnostic_testMethodology ArticleApplied MathematicsConfoundingBayes Theorem3. Good healthComputer Science ApplicationsPhenotypelcsh:Biology (General)030220 oncology & carcinogenesislcsh:R858-859.7FemaleDNA microarrayAlgorithmsBMC Bioinformatics
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Assessment of Susceptibility Risk Factors for ADHD in Imaging Genetic Studies

2019

Objective: ADHD consists of a count of symptoms that often presents heterogeneity due to overdispersion and excess of zeros. Statistical inference is usually based on a dichotomous outcome that is underpowered. The main goal of this study was to determine a suited probability distribution to analyze ADHD symptoms in Imaging Genetic studies. Method: We used two independent population samples of children to evaluate the consistency of the standard probability distributions based on count data for describing ADHD symptoms. Results: We showed that the zero-inflated negative binomial (ZINB) distribution provided the best power for modeling ADHD symptoms. ZINB reveals a genetic variant, rs273342…

MaleGenotypeImaging geneticsPopulationNegative binomial distributionPolymorphism Single NucleotideADHD symptomsImaging Genetics03 medical and health sciencesImaging Three-Dimensional0302 clinical medicineOverdispersionRisk FactorsStatisticsmental disordersDevelopmental and Educational PsychologyStatistical inferenceHumansGenetic Predisposition to Disease0501 psychology and cognitive sciencesGenetic TestingLongitudinal StudiesPoisson DistributionProspective Studiesp-valueMAPRE2Childeducationchildhoodzero-inflated negative binomialeducation.field_of_studyModels Statisticalbasal ganglia perivascular volumes05 social sciencesMagnetic Resonance Imagingcount dataVirchow-Robin spaceBinomial DistributionClinical PsychologyAttention Deficit Disorder with HyperactivityChild PreschoolProbability distributionFemalePsychology030217 neurology & neurosurgery050104 developmental & child psychologyCount data
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A novel KCNQ3 mutation in familial epilepsy with focal seizures and intellectual disability

2015

Mutations in the KCNQ2 gene encoding for voltage-gated potassium channel subunits have been found in patients affected with early onset epilepsies with wide phenotypic heterogeneity, ranging from benign familial neonatal seizures (BFNS) to epileptic encephalopathy with cognitive impairment, drug resistance, and characteristic electroencephalography (EEG) and neuroradiologic features. By contrast, only few KCNQ3 mutations have been rarely described, mostly in patients with typical BFNS. We report clinical, genetic, and functional data from a family in which early onset epilepsy and neurocognitive deficits segregated with a novel mutation in KCNQ3 (c.989G>T; p.R330L). Electrophysiological stu…

MaleGenotype-phenotype correlationmedicine.medical_specialtyNeurologyBenign familial neonatal seizuresMutantGenotype-phenotype correlationsmedicine.disease_causeMutagenesiKCNQ3 Potassium ChannelEpilepsyKCNQBenign Familial Neonatal Seizures KCNQ cognitive impairment voltage-gated potassium channels epilepsy mutagenesis genotype-phenotype correlationsSeizuresSettore M-PSI/08 - Psicologia ClinicaIntellectual DisabilityIntellectual disabilitymedicineHumansKCNQ2 Potassium ChannelVoltage-gated potassium channelBenign familial neonatal seizuresGenetic Predisposition to DiseaseGenetic TestingChildGenetic testingGeneticsMutationEpilepsymedicine.diagnostic_testGenetic heterogeneitybusiness.industryMedicine (all)Benign familial neonatal seizures; Cognitive impairment; Epilepsy; Genotype-phenotype correlations; KCNQ; Mutagenesis; Voltage-gated potassium channels; Child; Female; Genetic Testing; Humans; Intellectual Disability; KCNQ2 Potassium Channel; KCNQ3 Potassium Channel; Male; Mutation; Pedigree; Seizures; Genetic Predisposition to Disease; Neurology (clinical); Neurology; Medicine (all)Benign familial neonatal seizuremedicine.diseaseSeizureSettore MED/39 - Neuropsichiatria InfantilePedigreeCognitive impairmentNeurologyMutagenesisMutationFemaleNeurology (clinical)businessVoltage-gated potassium channelsHuman
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The effect of the acanthocephalan parasite Pomphorhynchus laevis on the lipid and glycogen content of its intermediate host Gammarus pulex

2001

Besides conspicuous changes in behaviour, manipulative parasites may also induce subtle physiological effects in the host that may also be favourable to the parasite. In particular, parasites may be able to influence the re-allocation of resources in their own favour. We studied the association between the presence of the acanthocephalan parasite, Pomphorhynchus laevis, and inter-individual variation in the lipid and glycogen content of its crustacean host, Gammarus pulex (Amphipoda). Infected gravid females had significantly lower lipid contents than uninfected females, but there was no difference in the lipid contents of non-gravid females and males that were infected with P. laevis. In c…

MaleHelminthiasisZoologyParasitismStatistics NonparametricAcanthocephalaHost-Parasite Interactionschemistry.chemical_compoundSex FactorsGammarus roeseliCrustaceaPrevalenceAnimalsParasite hostingbiologyGlycogenHost (biology)EcologyIntermediate hostLipid Metabolismbiology.organism_classificationGammarus pulexInfectious DiseaseschemistryFemaleParasitologyPomphorhynchus laevisGlycogenInternational Journal for Parasitology
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Hypothalamic Astroglial Connexins are Required for Brain Glucose Sensing-Induced Insulin Secretion

2014

Supplementary Information accompanies the paper on the Journal of Cerebral Blood Flow & Metabolism website; Hypothalamic glucose detection participates in maintaining glycemic balance, food intake, and thermogenesis. Although hypothalamic neurons are the executive cells involved in these responses, there is increasing evidence that astrocytes participate in glucose sensing (GS); however, it is unknown whether astroglial networking is required for glucose sensitivity. Astroglial connexins 30 and 43 (Cx30 and Cx43) form hexameric channels, which are apposed in gap junctions, allowing for the intercellular transfer of small molecules such as glucose throughout the astroglial networks. Here, we…

MaleINVOLVEMENTHOMEOSTASISmedicine.medical_specialtymedicine.medical_treatmentNerve Tissue ProteinsCarbohydrate metabolismBiologyASTROCYTESConnexinsconnexin 43RATSastrocyteInternal medicineInsulin SecretionmedicineAnimalsInsulinTANYCYTESRats WistarhypothalamusIN-VIVOHEMICHANNELSglucose sensingInsulinARCUATE NUCLEUSGap junctionFasting[ SDV.MHEP.EM ] Life Sciences [q-bio]/Human health and pathology/Endocrinology and metabolism[SDV.MHEP.EM]Life Sciences [q-bio]/Human health and pathology/Endocrinology and metabolismBARRIERconnexin 30NETWORKSGlucoseEndocrinologymedicine.anatomical_structureNeurologyHypothalamus[ SDV.NEU ] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]RNA InterferenceOriginal Article[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]sense organsNeurology (clinical)Cardiology and Cardiovascular MedicineThermogenesisIntracellularHomeostasisAstrocyteJournal of Cerebral Blood Flow & Metabolism
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