Search results for "STRESS"

showing 10 items of 6278 documents

Beneficial Role of Exercise in the Modulation of

2021

Duchenne muscular dystrophy (DMD) is an X-linked recessive progressive lethal disorder caused by the lack of dystrophin, which determines myofibers mechanical instability, oxidative stress, inflammation, and susceptibility to contraction-induced injuries. Unfortunately, at present, there is no efficient therapy for DMD. Beyond several promising gene- and stem cells-based strategies under investigation, physical activity may represent a valid noninvasive therapeutic approach to slow down the progression of the pathology. However, ethical issues, the limited number of studies in humans and the lack of consistency of the investigated training interventions generate loss of consensus regarding …

0301 basic medicineDuchenne muscular dystrophyPhysiologyDuchenne muscular dystrophyClinical BiochemistryInflammationReviewBioinformaticsmedicine.disease_causeBiochemistrySettore BIO/09 - FisiologiaMuscle hypertrophy03 medical and health sciencesTherapeutic approach0302 clinical medicineFibrosismedicineTrainingMuscle inflammationVoluntary exerciseMolecular BiologySwimmingbiologybusiness.industrylcsh:RM1-950ROSCell Biologymedicine.diseaselcsh:Therapeutics. Pharmacology030104 developmental biologyantioxidantsTreadmill runningbiology.proteinmedicine.symptomAntioxidantDystrophinExercise prescriptionbusiness030217 neurology & neurosurgeryOxidative stressAntioxidants (Basel, Switzerland)
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Effects of the LPA1 Receptor Deficiency and Stress on the Hippocampal LPA Species in Mice

2019

Lysophosphatidic acid (LPA) is an important bioactive lipid species that functions in intracellular signaling through six characterized G protein-coupled receptors (LPA1-6). Among these receptors, LPA1 is a strong candidate to mediate the central effects of LPA on emotion and may be involved in promoting normal emotional behaviors. Alterations in this receptor may induce vulnerability to stress and predispose an individual to a psychopathological disease. In fact, mice lacking the LPA1 receptor exhibit emotional dysregulation and cognitive alterations in hippocampus-dependent tasks. Moreover, the loss of this receptor results in a phenotype of low resilience with dysfunctional coping in res…

0301 basic medicineElevated plus mazemedicine.medical_specialtyMALDI-TOFF mass spectrometry:Medicina Básica [Ciências Médicas]BiologyHippocampal formationemotionslcsh:RC321-57103 medical and health scienceschemistry.chemical_compoundstressCellular and Molecular Neuroscience0302 clinical medicineInternal medicineLysophosphatidic acidmedicineReceptorlcsh:Neurosciences. Biological psychiatry. NeuropsychiatryMolecular BiologyScience & TechnologyEmotional dysregulationmedicine.diseasePhenotypeLPA species030104 developmental biologyEndocrinologychemistryMood disordersCiências Médicas::Medicina Básicalipids (amino acids peptides and proteins)LPA receptor 1LPA1 receptorbiological phenomena cell phenomena and immunity030217 neurology & neurosurgeryIntracellularLPA(1) receptorFrontiers in Molecular Neuroscience
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Hsp40 Is Involved in Cilia Regeneration in Sea Urchin Embryos

2003

In a previous paper we demonstrated that, in Paracentrotus lividus embryos, deciliation represents a specific kind of stress that induces an increase in the levels of an acidic protein of about 40 kD (p40). Here we report that deciliation also induces an increase in Hsp40 chaperone levels and enhancement of its ectodermal localization. We suggest that Hsp40 might play a chaperoning role in cilia regeneration.

0301 basic medicineEmbryo NonmammalianHistologyParacentrotus lividus03 medical and health sciences0302 clinical medicineStress PhysiologicalCulture Techniquesbiology.animalEctodermBotanyAnimalsRegenerationElectrophoresis Gel Two-DimensionalCiliaSettore BIO/06 - Anatomia Comparata E CitologiaSea urchinHeat-Shock ProteinsCentrosomebiologyCiliumEmbryoHSP40 Heat-Shock ProteinsSea urchin embryobiology.organism_classificationHsp40 deciliation sea urchinCell biology030104 developmental biologySea UrchinsAnatomy030217 neurology & neurosurgeryMolecular Chaperones
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Nickel toxicity in P. lividus embryos: Dose dependent effects and gene expression analysis.

2018

Abstract Many industrial activities release Nickel (Ni) in the environment with harmful effects for terrestrial and marine organisms. Despite many studies on the mechanisms of Ni toxicity are available, the understanding about its toxic effects on marine organisms is more limited. We used Paracentrotus lividus as a model to analyze the effects on the stress pathways in embryos continuously exposed to different Ni doses, ranging from 0.03 to 0.5 mM. We deeply examined the altered embryonic morphologies at 24 and 48 h after Ni exposure. Some different phenotypes have been classified, showing alterations at the expenses of the dorso-ventral axis as well as the skeleton and/or the pigment cells…

0301 basic medicineEmbryo NonmammalianPigment cellmRNASettore BIO/05 - ZoologiaEmbryonic DevelopmentGene ExpressionDevelopmentAquatic ScienceOceanographyParacentrotus lividus03 medical and health sciencesNickelGene expressionAnimalsInvertebrateProtein kinase AGeneSkeletonEchinodermbiologyAnimalChemistryStress responseEmbryoGeneral Medicinebiology.organism_classificationPollutionPhenotypeCell biologyHeavy metal030104 developmental biologyToxicityUnfolded protein responseParacentrotusParacentrotuWater Pollutants ChemicalMarine environmental research
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Response to metals treatment of Fra1, a member of the AP-1 transcription factor family, in P. lividus sea urchin embryos

2018

Abstract Lithium (Li), Nickel (Ni), and Zinc (Zn) are metals normally present in the seawater, although they can have adverse effects on the marine ecosystem at high concentrations by interfering with many biological processes. These metals are toxic for sea urchin embryos, affecting their morphology and developmental pathways. In particular, they perturb differently the correct organization of the embryonic axes (animal-vegetal, dorso-ventral): Li is a vegetalizing agent and Ni disrupts the dorso-ventral axis, while Zn has an animalizing effect. To deeply address the response of Paracentrotus lividus embryos to these metals, we studied the expression profiling of Pl-Fra transcription facto…

0301 basic medicineEmbryo NonmammalianProto-oncogeneSea UrchinSettore BIO/05 - ZoologiaAquatic ScienceOceanographyParacentrotus lividus03 medical and health sciencesAnimalsMetallothioneinTranscription factorbiologyCell growthChemistryAnimalMetalStress responseEmbryoGeneral MedicineLeucin zipperBlastulabiology.organism_classificationPollutionCell biologyGene expression profilingTranscription Factor AP-1AP-1 transcription factor030104 developmental biologyHeavy metalGene Expression RegulationMetalsSea UrchinsParacentrotusParacentrotuMetallothioneinWater Pollutants Chemical
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Coexposure to sulfamethoxazole and cadmium impairs development and attenuates transcriptional response in sea urchin embryo

2017

Abstract Among sulfonamides, sulfamethoxazole represents one of the most widely employed. A considerable amount of sulfamethoxazole is introduced into the marine environment after utilization in aquaculture. The cytotoxicity of sulfamethoxazole relies mainly on arylhydroxylamine metabolites and it is associated with the production of reactive oxygen species. Cadmium represents a metal largely employed in several anthropic activities and it is toxic for all living organisms even at low concentrations. Since it is not degraded, cadmium irreversibly accumulates into cells. In order to understand the mechanisms of response to changes in the chemical environment, we investigated by light microsc…

0301 basic medicineEmbryo NonmammalianSulfamethoxazoleHealth Toxicology and Mutagenesis; Defense mechanisms;Gene ExpressionAquaculture010501 environmental sciencesmedicine.disease_cause01 natural sciencesCoexposureToxicologychemistry.chemical_compoundCadmium ChlorideGene expression profile; Stress responsechemistry.chemical_classificationCadmiumeducation.field_of_studyEchinodermSulfamethoxazoleChemistry (all)General MedicinePollutionCadmiumDefense mechanismEchinodermsmedicine.drugProgrammed cell deathEnvironmental EngineeringPopulationchemistry.chemical_elementSettore BIO/11 - Biologia MolecolareCadmium chlorideBiologyMicrobiologyCoexposure; Defense mechanisms; Echinoderms; Gene expression profile; Stress response; Chemistry (all); Environmental Chemistry03 medical and health sciencesDetoxificationmedicineAnimalsEnvironmental Chemistryeducation0105 earth and related environmental sciencesReactive oxygen speciesStress responsePublic Health Environmental and Occupational HealthGeneral ChemistryGene expression profile030104 developmental biologychemistrySea UrchinsWater Pollutants ChemicalOxidative stress
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ATR expands embryonic stem cell fate potential in response to replication stress

2020

Fondazione Italiana per la Ricerca sul Cancro FIRC 18112 Sina Atashpaz.Fondazione Umberto Veronesi Sina Atashpaz Associazione Italiana per la Ricerca sul Cancro AIRC 5xmille METAMECH program Vincenzo Costanzo Giovanni Armenise-Harvard Foundation Vincenzo Costanzo European Research Council Consolidator grant 614541 Vincenzo Costanzo Associazione Italiana per la Ricerca sul Cancro Fellowship 23961 Negar ArghavanifarDanish Cancer Society KBVU-2014 Andres Joaquin Lopez-Contreras Danish Council for Independent Research Sapere Aude, DFF Starting Grant 2014 Andres Joaquin Lopez-Contreras European Research Council ERC-2015-STG-679068 Andres Joaquin Lopez-Contreras Danish National Research Foundatio…

0301 basic medicineEndogenyAtaxia Telangiectasia Mutated ProteinsMice0302 clinical medicineTandem Mass SpectrometryTranscription (biology)GENE ATRcell biologyCloning MolecularBiology (General)Cells Cultured0303 health sciencesGeneral NeuroscienceQRTotipotentCell DifferentiationEmbryoGeneral MedicineCell biologyMedicinebiological phenomena cell phenomena and immunityResearch ArticleQH301-705.5replication stressDNA damageScienceSettore MED/08 - Anatomia PatologicaBiologyGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciencesAnimalsRNA MessengerGeneEmbryonic Stem CellsmouseCell Proliferation030304 developmental biologyMessenger RNAGeneral Immunology and MicrobiologyChimeraSequence Analysis RNAEmbryogenesisTELOMERE ELONGATIONEPIGENETIC RESTRICTIONembryonic stem cellEmbryonic stem cellATR030104 developmental biologyGene Expression RegulationDNA-DAMAGECheckpoint Kinase 1GENOMIC STABILITY030217 neurology & neurosurgeryChromatography LiquidDNA DamageeLife
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Red fruit (Pandanus conoideus Lam) oil stimulates nitric oxide production and reduces oxidative stress in endothelial cells

2018

Abstract Red fruit oil (RFO) is used in traditional medicine for the treatment of a number of diseases. However, evidence for the biological effects and action mechanisms is still lacking. In the present study, we show for the first time that RFO stimulated the phosphorylation of the endothelial nitric oxide synthase (eNOS) and enhanced the NO production in human endothelial cells. In isolated mouse aorta, RFO induced a vasodilation, with a significant effect evident at a concentration as low as 1:100,000 dilution. The RFO-induced vasodilation could be completely prevented by eNOS inhibition, indicating that RFO contains highly potent substances stimulating eNOS activity. In addition, RFO r…

0301 basic medicineEndothelial cellsMedicine (miscellaneous)VasodilationMouse aorta030204 cardiovascular system & hematologyPharmacologymedicine.disease_causeNitric oxide03 medical and health scienceschemistry.chemical_compoundPandanus conoideus Lam0302 clinical medicineEnosmedicineTX341-641Nutrition and DieteticsbiologyNutrition. Foods and food supplyNitric oxidePandanus conoideusbiology.organism_classificationComet assay030104 developmental biologychemistryOxidative stressPhosphorylationEndothelial nitric oxide synthaseReactive oxygen speciesOxidative stressFood ScienceJournal of Functional Foods
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Paraoxonase-2 regulates coagulation activation through endothelial tissue factor

2017

Oxidative stress and inflammation of the vessel wall contribute to prothrombotic states. The antioxidative protein paraoxonase-2 (PON2) shows reduced expression in human atherosclerotic plaques and endothelial cells in particular. Supporting a direct role for PON2 in cardiovascular diseases, Pon2 deficiency in mice promotes atherogenesis through incompletely understood mechanisms. Here, we show that deregulated redox regulation in Pon2 deficiency causes vascular inflammation and abnormalities in blood coagulation. In unchallenged Pon2-/- mice, we find increased oxidative stress and endothelial dysfunction. Bone marrow transplantation experiments and studies with endothelial cells provide ev…

0301 basic medicineEndotheliumImmunologyInflammation030204 cardiovascular system & hematologymedicine.disease_causeModels BiologicalBiochemistryThromboplastinMice03 medical and health sciencesTissue factor0302 clinical medicinemedicineAnimalsHumansThromboplastinPlateletEndothelial dysfunctionBlood CoagulationInflammationMice KnockoutAryldialkylphosphataseChemistryEndothelial CellsCell BiologyHematologymedicine.diseaseEndothelial stem cellOxidative Stress030104 developmental biologymedicine.anatomical_structureCancer researchCytokinesInflammation Mediatorsmedicine.symptomOxidation-ReductionOxidative stressBlood
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Effects of UV filter 4-methylbenzylidene camphor during early development of Solea senegalensis Kaup, 1858

2018

Abstract The inclusion of organic UV filters in personal care products (PCPs) has increased in recent years. 4-Methylbenzylidene camphor (4MBC) is one of the most used UV filters, and thus it is commonly found in aquatic ecosystems, with proved negative effects on aquatic organisms. Effects on early life stages of marine vertebrates are largely unknown. Therefore, the main goal of this work was to evaluate 4MBC effects on Senegalese sole (Solea Senegalensis Kaup, 1858) larvae at different levels of biological organization. S. senegalensis were exposed to increasing concentrations of 4MBC from egg stage until 96 h. Mortality, growth, malformations, behaviour and biochemical responses, includ…

0301 basic medicineEnvironmental EngineeringZoologyUV filter010501 environmental sciencesmedicine.disease_cause01 natural sciencesLipid peroxidation03 medical and health scienceschemistry.chemical_compoundLactate dehydrogenaseToxicity TestsmedicineAnimalsEnvironmental ChemistryWaste Management and Disposal0105 earth and related environmental sciencesCholinesterasebiologyPollutionAcetylcholinesteraseCamphor030104 developmental biologychemistryCatalaseFlatfishesbiology.proteinSunscreening AgentsAnaerobic exerciseWater Pollutants ChemicalOxidative stressScience of The Total Environment
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