Search results for "SVI"

showing 10 items of 4456 documents

The role of the human cytomegalovirus UL111A gene in down-regulating CD4+ T-cell recognition of latently infected cells: implications for virus elimi…

2009

AbstractThe capacity of human cytomegalovirus (HCMV) to establish and maintain a latent infection from which it can later reactivate ensures its widespread distribution in the population, but the mechanisms enabling maintenance of latency in the face of a robust immune system are poorly understood. We examined the role of the HCMV UL111A gene, which encodes homologs of the immunosuppressive cytokine interleukin-10 in the context of latent infection of myeloid progenitor cells. A UL111A deletion virus was able to establish, maintain, and reactivate from experimental latency in a manner comparable with parental virus, but major histocompatibility complex class II levels increased significantl…

Human cytomegalovirusCD4-Positive T-LymphocytesIsoantigensMyeloidGenes Viralmedicine.medical_treatmentImmunologyPopulationCytomegalovirusDown-RegulationBiologyIn Vitro Techniquesmedicine.disease_causeBiochemistryAutoantigensHerpesviridaeVirusImmune systemmedicineHumansProgenitor celleducationMyeloid Progenitor Cellseducation.field_of_studyHistocompatibility Antigens Class IICell BiologyHematologymedicine.diseaseVirologyVirus LatencyCytokinemedicine.anatomical_structureImmunologyCytomegalovirus InfectionsHost-Pathogen InteractionsGene DeletionBlood
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Reconstitution of lymphocyte populations and cytomegalovirus viremia or disease after allogeneic peripheral blood stem cell transplantation

2003

Early reconstitution of lymphoid populations was monitored by immunophenotyping in 57 allogeneic peripheral blood stem cell (allo-PBSC) transplant patients either with or without cytomegalovirus (CMV) viremia or disease. Cell counts for total lymphocytes and CD4(+) T cells above the percentile 60th at day 14 postransplant were associated significantly with CMV viremia-free survival within 120 days after transplant. Recovery of total lymphocyte, CD3(+), and CD8(+) T-cell counts proceeded at a more rapid rate in CMV viremic patients than in nonviremic patients, irrespective of whether preemptive treatment with ganciclovir had been prescribed. Significant expansion of CD8(+) and CD8(+) CD57(+)…

Human cytomegalovirusGanciclovirLymphocyteCongenital cytomegalovirus infectionvirus diseasesViremiaBiologymedicine.diseasebiology.organism_classificationVirologyInfectious Diseasesmedicine.anatomical_structureImmunophenotypingBetaherpesvirinaeVirologyImmunologymedicineStem cellmedicine.drugJournal of Medical Virology
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Presence of Epstein-Barr virus, cytomegalovirus and human papillomavirus in normal oral mucosa of HIV-infected and renal transplant patients

2001

OBJECTIVES: To determine the prevalence of EBV-DNA,CMV-DNA and HPV-DNA in oral healthy mucosa of HIV-infected and renal transplant patients. To associate the detection of viral genomes with laboratory parameters of immunodeficiency, gender, antiretroviral and immunosuppressive therapy. DESIGN: A cross-sectional analysis of lingual and buccal cytobrushings from HIV-infected and renal transplant patients. SUBJECTS AND METHODS: Lingual and buccal cytobrushings were obtained from clinically normal oral mucosa of 57 HIV+, 40 renal transplant patients and 30 healthy uninfected controls, all matched for age at baseline of examination. Presence of EBV-, CMV- and HPV-DNA was assessed by polymerase c…

Human cytomegalovirusKidneybiologybusiness.industryvirus diseasesmedicine.diseasebiology.organism_classificationmedicine.disease_causeHerpesviridaeTransplantationmedicine.anatomical_structureOtorhinolaryngologyAcquired immunodeficiency syndrome (AIDS)ImmunologyLentivirusmedicineViral diseasebusinessGeneral DentistryImmunodeficiencyOral Diseases
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Impact of infectious burden on extent and long-term prognosis of atherosclerosis.

2002

Background — Recent findings suggest a causative role of infections in the pathogenesis of atherosclerosis. In hypothesizing an association between infectious agents and the development of atherosclerosis, we would expect a correlation to the extent of atherosclerosis. Moreover, this effect could be multiplied by the number of pathogens to which an individual had been exposed. Methods and Results — In 572 patients, IgG or IgA antibodies to herpes simplex virus 1 and 2, cytomegalovirus, Epstein-Barr virus, Hemophilus influenzae , Chlamydia pneumoniae , Mycoplasma pneumoniae , and Helicobacter pylori were measured. The extent of atherosclerosis was determined by coronary angiography, carotid…

Human cytomegalovirusMalemedicine.medical_specialtyHerpesvirus 4 HumanTime FactorsArteriosclerosisCarotid arteriesCytomegalovirusCoronary diseasemedicine.disease_causeAntibodies ViralHerpesviridaePathogenesisRisk FactorsPhysiology (medical)medicineHumansSimplexvirusIntensive care medicineAgedHelicobacter pyloribusiness.industryBacterial InfectionsChlamydophila pneumoniaeMiddle Agedmedicine.diseasePrognosisAntibodies BacterialHaemophilus influenzaeSurvival AnalysisDNA Virus InfectionsImmunoglobulin AMycoplasma pneumoniaeSurvival RateC-Reactive ProteinLogistic ModelsHerpesvirus hominisImmunoglobulin GImmunologyMultivariate AnalysisVirusesFemaleCardiology and Cardiovascular MedicinebusinessCirculation
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Frequent coinfection of cells explains functional in vivo complementation between cytomegalovirus variants in the multiply infected host.

2005

In contrast to many other virus infections, primary cytomegalovirus (CMV) infection does not fully protect against reinfection. Accordingly, clinical data have revealed a coexistence of multiple human CMV variants/ strains in individual patients. Notably, the phenomenon of multiple infection was found to correlate with increased virus load and severity of CMV disease. Although of obvious medical relevance, the mechanism underlying this correlation is unknown. A weak immune response in an individual could be responsible for a more severe disease and for multiple infections. Alternatively, synergistic contributions of variants that differ in their biological properties can lead to qualitative…

Human cytomegalovirusMuromegalovirusImmunologyPopulationGreen Fluorescent ProteinsBiologymedicine.disease_causeMicrobiologyHerpesviridaeVirusMiceViral ProteinsBetaherpesvirinaeVirologymedicineAnimalseducationLungeducation.field_of_studyMice Inbred BALB CIntegrasesVirulenceGenetic VariationInborn immunodeficiencyCytomegalovirusmedicine.diseasebiology.organism_classificationVirologyGenetic Diversity and EvolutionInsect ScienceImmunologyCytomegalovirus InfectionsCoinfectionNIH 3T3 CellsFemaleSpleenJournal of virology
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Experimental Preemptive Immunotherapy of Murine Cytomegalovirus Disease with CD8 T-Cell Lines Specific for ppM83 and pM84, the Two Homologs of Human …

2001

ABSTRACTCD8 T cells are the principal antiviral effectors controlling cytomegalovirus (CMV) infection. For human CMV, the virion tegument protein ppUL83 (pp65) has been identified as a source of immunodominant peptides and is regarded as a candidate for cytoimmunotherapy and vaccination. Two sequence homologs of ppUL83 are known for murine CMV, namely the virion protein ppM83 (pp105) expressed late in the viral replication cycle and the nonstructural protein pM84 (p65) expressed in the early phase. Here we show that ppM83, unlike ppUL83, is not delivered into the antigen presentation pathway after virus penetration before or in absence of viral gene expression, while other virion proteins o…

Human cytomegalovirusMuromegalovirusmedicine.medical_treatmentImmunologyImmunodominanceCD8-Positive T-LymphocytesBiologyMicrobiologyCell LineViral Matrix ProteinsInterferon-gammaMiceImmune systemAntigenVirologyVaccines and Antiviral AgentsmedicineAnimalsCytotoxic T cellMice Inbred BALB CHerpesviridae InfectionsImmunotherapyPhosphoproteinsmedicine.diseaseAdoptive TransferVirologyPeptide FragmentsDisease Models AnimalViral replicationInsect ScienceImmunologyFemaleCytokine secretionImmunologic MemoryJournal of Virology
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Human cytomegalovirus glycoprotein B genotypes in immunocompetent, immunocompromised, and congenitally infected Italian populations

2003

Human cytomegalovirus (HCMV) strains, obtained from immunocompetent and immunocompromised Italian hosts, were typed with glycoprotein B (gB) gene restriction analysis. A predominant circulation of HCMV strains with gB type 2 and 3 was detected in both the immunocompetent host with a primary HCMV infection and the immunocompromised host with or without HCMV disease. No association between gB types and subjects with different risks of developing HCMV disease was found. All four gB genotypes were capable of causing congenital infection in Italian babies, with gB type 1 accounting for 50% of the strains examined in symptomatic infants and a remarkable incidence of gB type 4 viruses.

Human cytomegalovirusSettore MED/07 - Microbiologia E Microbiologia Clinicamedicine.medical_specialtyGenotypevirusesRestriction MappingCongenital cytomegalovirus infectionCytomegalovirusHIV Infectionsmedicine.disease_causePolymerase Chain ReactionHerpesviridaeVirusImmunocompromised HostMedical microbiologyViral Envelope ProteinsBetaherpesvirinaeVirologyGenotypemedicineHumansBone Marrow TransplantationbiologyInfant Newbornvirus diseasesGeneral Medicinebiology.organism_classificationmedicine.diseaseKidney TransplantationVirologyHuman cytomegalovirus immunocompromised gB genotypes ItalyCytomegalovirus InfectionsViral diseaseImmunocompetenceArchives of Virology
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The viral chemokine MCK-2 of murine cytomegalovirus promotes infection as part of a gH/gL/MCK-2 complex.

2013

Human cytomegalovirus (HCMV) forms two gH/gL glycoprotein complexes, gH/gL/gO and gH/gL/pUL(128,130,131A), which determine the tropism, the entry pathways and the mode of spread of the virus. For murine cytomegalovirus (MCMV), which serves as a model for HCMV, a gH/gL/gO complex functionally homologous to the HCMV gH/gL/gO complex has been described. Knock-out of MCMV gO does impair, but not abolish, virus spread indicating that also MCMV might form an alternative gH/gL complex. Here, we show that the MCMV CC chemokine MCK-2 forms a complex with the glycoprotein gH, a complex which is incorporated into the virion. We could additionally show that mutants lacking both, gO and MCK-2 are not ab…

Human cytomegalovirusViral DiseasesMuromegalovirusChemokinevirusesMurine Cytomegalovirus ; viral chemokine MCK-2 ; gH/gL/MCK-2 complexMiceViral Envelope ProteinsBiology (General)Cells Culturedchemistry.chemical_classificationMice Inbred BALB Cvirus diseasesHerpesviridae InfectionsRecombinant ProteinsSpecific Pathogen-Free OrganismsInfectious DiseasesLiverChemokines CCMedicineFemaleResearch ArticleQH301-705.5ImmunologyBiologyMicrobiologyVirusCell LineViral ProteinsMuromegalovirusGlycoprotein complexVirologyGeneticsmedicineAnimalsBiologyMolecular BiologyTropismMacrophagesVirionVirus InternalizationRC581-607medicine.diseasebiology.organism_classificationVirologyImmunity InnatechemistryCell cultureMutationMacrophages Peritonealbiology.proteinParasitologyProtein MultimerizationImmunologic diseases. AllergyGlycoprotein
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Longitudinal analysis of human cytomegalovirus glycoprotein B (gB)-specific and neutralizing antibodies in AIDS patients either with or without cytom…

2001

Serum neutralizing and glycoprotein B (gB)-specific antibody levels were monitored prospectively in AIDS patients who either did or did not develop human cytomegalovirus (HCMV) end-organ disease, to delineate further the role of antibodies in protecting against HCMV disease. Antibody levels declined substantially (at least 4-fold) only in patients who developed HCMV disease; this decline in turn occurred concurrently with antigenemia. Nevertheless, AIDS patients who remained free of HCMV disease and did not become antigenemic during the follow-up period maintained stable levels of serum antibodies, with only minor fluctuations. The impact of HAART on the levels of functional anti-HCMV antib…

Human cytomegalovirusbiologybusiness.industryvirusesAntibody titerCongenital cytomegalovirus infectionvirus diseasesmedicine.disease_causemedicine.diseasebiology.organism_classificationVirologyHerpesviridaeInfectious DiseasesBetaherpesvirinaeVirologyImmunopathologyImmunologymedicinebiology.proteinViral diseaseAntibodybusinessJournal of Medical Virology
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Identification of a Conserved HLA-A2-Restricted Decapeptide from the IE1 Protein (pUL123) of Human Cytomegalovirus

2002

Abstract Control of human cytomegalovirus (HCMV) infection is predominantly mediated by cytolytic CD8 + T lymphocytes (CTL). Among the roughly 200 HCMV-encoded polypeptides, the tegument protein pp65 (ppUL83) and the nonstructural IE1 protein are considered to be dominant CTL targets. Yet the importance of CTL against IE1 for protective immunity against HCMV reactivation and disease has remained elusive. Analyses have been difficult, as all MHC class I presented peptides of IE1 defined so far are located in parts of the protein that are variable between viral strains. In this study a conserved decameric peptide from IE1 (P6, IE1 354–363 ) that bound to HLA-A2 was identified. Using peptide-p…

Human cytomegalovirusherpesvirusesViral proteinvirusesMolecular Sequence DataIE1CytomegalovirusEpitopes T-Lymphocytecytotoxic T lymphocytesmedicine.disease_causeImmediate early proteinCell LineImmediate-Early ProteinsViral Proteinsconserved CTL epitopesVirologyHLA-A2 AntigenMHC class ImedicineHumansCytotoxic T cellAmino Acid SequenceConserved SequencebiologyELISPOTvirus diseasesHLA-A2biochemical phenomena metabolism and nutritionCytotoxicity Tests Immunologicmedicine.diseaseVirologyPeptide FragmentsVirus LatencyCTL*human cytomegalovirusCytomegalovirus InfectionsImmunologybiology.proteinPeptidesCD8T-Lymphocytes CytotoxicVirology
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