Search results for "SYNAPTOGENESIS"

showing 10 items of 30 documents

Role of nitric oxide and nitric oxide synthases in experimental models of denervation and reinnervation.

2001

Nitric oxide (NO) is a short-living free molecule synthesized by three different isoforms of nitric oxide synthases (NOS)—neuronal NOS, endothelial NOS, and inducible NOS—associated with neuromuscular transmission, muscle contractility, mitochondrial respiration, and carbohydrate metabolism in skeletal muscle. Neuronal NOS is constitutively expressed at the muscle fiber sarcolemma linked to the dystrophin-glycoprotein complex and concentrated at the neuromuscular endplate. There is increasing evidence that altered expression of neuronal NOS plays a role in muscle fiber damage in neuromuscular diseases such as dystrophinopathies and denervating disorders. Although there have been some previo…

DenervationHistologySarcolemmaNeuromuscular transmissionSynaptogenesisSkeletal muscleBiologyEndothelial NOSNitric OxideMuscle DenervationNitric oxideCell biologyMedical Laboratory Technologychemistry.chemical_compoundmedicine.anatomical_structureBiochemistrychemistrymedicineAnimalsHumansAnatomyNitric Oxide SynthaseMuscle SkeletalInstrumentationReinnervationMicroscopy research and technique
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Alterations in the organization of the isocortical layer I in trisomy 22.

1999

The isocortical layer I of human fetal brains obtained from different cases of chromosomal abnormalities (trisomy 18, 21, 22) and controls without pathological disturbances were investigated histologically and immunohistochemically by using the antibodies SMI 311, SMI 35 and SMI 81 (SNAP 25) as well as antibodies against GAP 43 and calretinin. In cases of trisomy 22 the Cajal-Retzius cells in Nissl-sections and in SMI 311-immunopreparations do not reveal any alterations regarding their location or morphology. However, the axonal plexus, selectively labelled with SMI 35, normally located in layer Ib, is malpositioned in Ia. Likewise, SNAP 25- and GAP 43-immunoreactive structures, which were …

Down syndromePathologymedicine.medical_specialtyChromosomes Human Pair 21Chromosomes Human Pair 22SynaptogenesisChromosome DisordersNerve Tissue ProteinsTrisomyTrisomy 22FetusGAP-43 ProteinS100 Calcium Binding Protein GmedicineHumansGap-43 proteinChromosome AberrationsPlexusbiologyGeneral NeuroscienceSnapBrainGeneral MedicineAnatomymedicine.diseasenervous systemCalbindin 2biology.proteinCalretininDown SyndromeTrisomyChromosomes Human Pair 18Neuroscience research
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Stick around: Cell-Cell Adhesion Molecules during Neocortical Development

2020

The neocortex is an exquisitely organized structure achieved through complex cellular processes from the generation of neural cells to their integration into cortical circuits after complex migration processes. During this long journey, neural cells need to stablish and release adhesive interactions through cell surface receptors known as cell adhesion molecules (CAMs). Several types of CAMs have been described regulating different aspects of neurodevelopment. Whereas some of them mediate interactions with the extracellular matrix, others allow contacts with additional cells. In this review, we will focus on the role of two important families of cell-cell adhesion molecules (C-CAMs), classi…

Extracellular matrixCorticogenesisNeocortexmedicine.anatomical_structureChemistryCadherinNectinCell adhesion moleculeCell surface receptorSynaptogenesismedicineanatomy_morphologyNeuroscience
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Functional annotation of genes overlapping copy number variants in autistic patients: focus on axon pathfinding.

2010

We have used Gene Ontology (GO) and pathway analyses to uncover the common functions associated to the genes overlapping Copy Number Variants (CNVs) in autistic patients. Our source of data were four published studies [1- 4]. We first applied a two-step enrichment strategy for autism-specific genes. We fished out from the four mentioned studies a list of 2928 genes overall overlapping 328 CNVs in patients and we first selected a sub-group of 2044 genes after excluding those ones that are also involved in CNVs reported in the Database of Genomic Variants (enrichment step 1). We then selected from the step 1-enriched list a sub-group of 514 genes each of which was found to be deleted or dupli…

GeneticsCandidate geneneurodevelopmentAutism Spectrum Disorders Copy Number Variants Gene Ontology axon guidance signalling neurodevelopment candidate genes.media_common.quotation_subjectSynaptogenesisBiologymedicine.diseaseCopy Number VariantsArticleAutism Spectrum Disordersaxon guidance signallingIngenuityGene OntologySettore BIO/13 - Biologia ApplicataGeneticsmedicineAutismAxon guidanceCopy-number variationcandidate genes.GeneGenetics (clinical)Function (biology)media_commonCurrent genomics
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Neurotrophic Properties of Silexan, an Essential Oil from the Flowers of Lavender-Preclinical Evidence for Antidepressant-Like Properties.

2020

Abstract Background Silexan, a special essential oil from flowering tops of lavandula angustifolia, is used to treat subsyndromal anxiety disorders. In a recent clinical trial, Silexan also showed antidepressant effects in patients suffering from mixed anxiety-depression (ICD-10 F41.2). Since preclinical data explaining antidepressant properties of Silexan are missing, we decided to investigate if Silexan also shows antidepressant-like effects in vitro as well as in vivo models. Methods We used the forced swimming test (FST) in rats as a simple behavioral test indicative of antidepressant activity in vivo. As environmental events and other risk factors contribute to depression through conve…

Imipraminemedicine.drug_classSynaptogenesisCell Culture TechniquesPregabalinTricyclic antidepressantPharmacologyCREBImipramineRats Sprague-Dawley03 medical and health sciences0302 clinical medicinemedicineOils VolatileAnimalsPlant OilsPharmacology (medical)030304 developmental biology0303 health sciencesbiologyDose-Response Relationship Drugbusiness.industryGeneral MedicineCREB-Binding ProteinCyclic AMP-Dependent Protein KinasesAntidepressive AgentsRatsPsychiatry and Mental healthLavandulaSynaptic plasticitybiology.proteinAntidepressantbusiness030217 neurology & neurosurgerymedicine.drugBehavioural despair testNeurotrophinPharmacopsychiatry
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Microglial and BDNF impact on the induction of the post ischemic neuroplasticity

2011

Evidences showing that under certain circumstances, inflammatory response could be neuroprotective and could also promote adult neurogenesis are growing. In this context, the objective of this work was to investigate the impact of microglial cells in the neuroplastic events. Rats were subjected to photothrombotic ischemia and microglial cells activation was blocked by the mean of poly(ADP-ribose)polymérase-1 (PARP-1) inhibition using 3- aminobenzamide (3-AB) since this protein has been shown to play a major role in this activation. Our results show that PARP-1 activity reduction was associated with a strong repression of the acute microglial activation. Beside, 3-AB treated animals exhibite…

Inflammation[SDV.SA]Life Sciences [q-bio]/Agricultural sciences[SDV.SA] Life Sciences [q-bio]/Agricultural sciences[SDV.MHEP] Life Sciences [q-bio]/Human health and pathologyExperimental strokeNeuroplasticitéAVC expérimentalAxonal growthSynaptogenèseBDNFnervous system[ SDV.MHEP ] Life Sciences [q-bio]/Human health and pathologyNeuroplasticityMicrogliaSynaptogenesis[ SDV.SA ] Life Sciences [q-bio]/Agricultural sciencesMicroglieCroissance axonale[SDV.MHEP]Life Sciences [q-bio]/Human health and pathology
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PSA-NCAM expression in the piriform cortex of the adult rat. Modulation by NMDA receptor antagonist administration.

2002

Administration of NMDA receptor antagonists upregulates the expression of the polysialylated form of the neural cell adhesion molecule (PSA-NCAM) in the adult hippocampus. Since the piriform cortex is also populated by PSA-NCAM immunoreactive neurons during adulthood, we sought to characterize them in detail and to test whether NMDA receptor antagonists also modulate PSA-NCAM in this cortical region. PSA-NCAM immunoreactivity is located mainly in layer II, where many neurogliaform and some pyramidal-semilunar transitional neurons are labeled. Many large neurons in layer III and endopiriform nucleus also express PSA-NCAM. Interestingly, some small labeled cells resembling migratory neuroblas…

MaleDoublecortin ProteinSynaptogenesisHippocampusNeural Cell Adhesion Molecule L1Receptors N-Methyl-D-AspartateRats Sprague-DawleyNeuroblastCell MovementPiriform cortexmedicineAnimalsMolecular BiologyNeural Cell Adhesion MoleculesNeuronsbiologyGeneral NeuroscienceOlfactory PathwaysDoublecortinRatsmedicine.anatomical_structurenervous systembiology.proteinSialic AcidsNeural cell adhesion moleculeNeurology (clinical)NeuNNeuroscienceNucleusExcitatory Amino Acid AntagonistsInjections IntraperitonealDevelopmental BiologyBrain research
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PSA-NCAM expression in the rat medial prefrontal cortex

2005

The rat medial prefrontal cortex, an area considered homologous to the human prefrontal cortex, is a region in which neuronal structural plasticity has been described during adulthood. Some plastic processes such as neurite outgrowth and synaptogenesis are known to be regulated by the polysialylated form of the neural cell adhesion molecule (PSA-NCAM). Since PSA-NCAM is present in regions of the adult CNS which are undergoing structural remodeling, such as the hypothalamus or the hippocampus, we have analyzed the expression of this molecule in the medial prefrontal cortex of adult rats using immunohistochemistry. PSA-NCAM immunoreactivity was found both in cell bodies and in the neuropil of…

MaleNeuropilNeuriteInterneuronAntimetabolitesCell SurvivalSynaptophysinSynaptogenesisPrefrontal CortexHippocampusNeural Cell Adhesion Molecule L1BiologyRats Sprague-DawleyNeuroplasticityNeuropilmedicineAnimalsFluorescent Antibody Technique IndirectPrefrontal cortexNeuronsNeuronal PlasticityGlutamate DecarboxylasePyramidal CellsGeneral NeuroscienceImmunohistochemistryRatsPhenotypemedicine.anatomical_structureBromodeoxyuridinenervous systemSialic AcidsNeural cell adhesion moleculeNeuroscienceNeuroscience
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Proneurotrophin Binding to P75 Neurotrophin Receptor (P75ntr) Is Essential for Brain Lesion Formation and Functional Impairment after Experimental Tr…

2015

Traumatic brain injury (TBI) initiates an excessive mediator release of e.g. neurotrophins, which promote neuronal survival, differentiation, and modulate synaptic plasticity. Paradoxically, mature forms of neurotrophins promote neuronal survival, whereas unprocessed forms of neurotrophins induce cell death through p75 neurotrophin receptor (p75NTR) signaling. p75NTR is widely expressed during synaptogenesis and is subsequently downregulated in adulthood. Repair mechanisms after acute cerebral insults can reactivate its expression. Therefore, the influence of p75NTR on secondary brain damage was addressed. mRNA levels of p75NTR and its ligands were quantified in brain tissue up to 7 days af…

MaleProgrammed cell deathmedicine.medical_specialtyTraumatic brain injurySynaptogenesisReceptors Nerve Growth FactorBrain damageMiceInternal medicineAnimalsMedicineLow-affinity nerve growth factor receptorRNA MessengerMice KnockoutBehavior AnimalCell Deathbiologybusiness.industrymedicine.diseaseMice Inbred C57BLDisease Models AnimalEndocrinologyBrain InjuriesSynaptic plasticitybiology.proteinFemalesense organsNeurology (clinical)medicine.symptomSignal transductionbusinessNeuroscienceProtein BindingSignal TransductionNeurotrophinJournal of Neurotrauma
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Changes in the expression of cation-Cl- cotransporters, NKCC1 and KCC2, during cortical malformation induced by neonatal freeze-lesion.

2007

Focal cortical malformations comprise a heterogeneous group of disturbances in brain development, often associated with intractable epilepsy. A focal freeze-lesion of cerebral cortex in newborn rat produces a cortical malformation that resembles human polymicrogyria, clinical conditions that results from abnormal neuronal migration. The change in GABAergic functions that occurs during early brain development is induced by an alteration in Cl(-) homeostasis and plays important roles in neocortical development by modulating such events as laminar organization and synaptogenesis. We therefore investigated the relationship between pathogenesis of polymicrogyria and ontogeny of Cl(-) homeostasis…

MaleSodium-Potassium-Chloride SymportersSynaptogenesisDown-RegulationBiologyNervous System MalformationsLaminar organizationChloridesCell MovementChloride ChannelsCortex (anatomy)Parietal LobeGlial Fibrillary Acidic ProteinmedicinePolymicrogyriaAnimalsSolute Carrier Family 12 Member 2RNA MessengerRats Wistargamma-Aminobutyric AcidCerebral CortexSymportersGeneral NeuroscienceColocalizationCell DifferentiationGeneral Medicinemedicine.diseaseDenervationImmunohistochemistryMicrogyrusRatsUp-RegulationCold Temperaturemedicine.anatomical_structureNeuronal migration disorderBromodeoxyuridineCerebral cortexPhosphopyruvate HydrataseNeuroscienceBiomarkersNeuroscience research
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