Search results for "Selectin"

showing 10 items of 148 documents

In Vitro Expression of the Endothelial Phenotype: Comparative Study of Primary Isolated Cells and Cell Lines, Including the Novel Cell Line HPMEC-ST1…

2002

Endothelial cell lines are commonly used in in vitro studies to avoid problems associated with the use of primary endothelial cells such as the presence of contaminating cells, the difficulty in obtaining larger numbers of cells, as well as the progressive loss of cell viability and expression of endothelial markers in the course of in vitro propagation. We have analyzed the characteristics defining distinctive endothelial phenotypes in the cell lines EA.hy926, ECV304, EVLC2, HAEND, HMEC-1, ISO-HAS-1 and a cell line recently generated in our laboratory, HPMEC-ST1.6R, and have compared these phenotypes with those found in primary human endothelial cells isolated from umbilical vein (HUVEC), …

LipopolysaccharidesCD31Cell SurvivalAngiogenesisCD34Vascular Cell Adhesion Molecule-1Antigens CD34Enzyme-Linked Immunosorbent AssayBiologyPolymerase Chain ReactionBiochemistryCell Linevon Willebrand FactorCell AdhesionHumansMicroscopy Phase-ContrastViability assayLungCells CulturedChemokine CCL2SkinMatrigelNeovascularization PathologicInterleukin-6Reverse Transcriptase Polymerase Chain ReactionTumor Necrosis Factor-alphaCell adhesion moleculeInterleukin-8TemperatureGranulocyte-Macrophage Colony-Stimulating FactorCell BiologyIntercellular Adhesion Molecule-1ImmunohistochemistryCell biologyLipoproteins LDLPlatelet Endothelial Cell Adhesion Molecule-1Endothelial stem cellDrug CombinationsPhenotypeCell cultureImmunologyProteoglycansCollagenEndothelium VascularLamininE-SelectinCardiology and Cardiovascular MedicineInterleukin-1Microvascular Research
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Time Response of Oxidative/Nitrosative Stress and Inflammation in LPS-Induced Endotoxaemia—A Comparative Study of Mice and Rats

2017

Sepsis is a severe and multifactorial disease with a high mortality rate. It represents a strong inflammatory response to an infection and is associated with vascular inflammation and oxidative/nitrosative stress. Here, we studied the underlying time responses in the widely used lipopolysaccharide (LPS)-induced endotoxaemia model in mice and rats. LPS (10 mg/kg; from Salmonella Typhosa) was intraperitoneally injected into mice and rats. Animals of every species were divided into five groups and sacrificed at specific points in time (0, 3, 6, 9, 12 h). White blood cells (WBC) decreased significantly in both species after 3 h and partially recovered with time, whereas platelet decrease did no…

LipopolysaccharidesMale0301 basic medicinesepsis; time response; inflammation; oxidative stress; endotoxaemia; mouse; ratLipopolysaccharideNitric Oxide Synthase Type IIBacteremia030204 cardiovascular system & hematologymedicine.disease_causelcsh:ChemistrysepsisendotoxaemiaHemoglobinsLeukocyte CountMicechemistry.chemical_compound0302 clinical medicineoxidative stressratPlateletlcsh:QH301-705.5SpectroscopyGeneral MedicineComputer Science ApplicationsRespiratory burstP-SelectinSalmonella Infectionsmedicine.symptommedicine.medical_specialtyVascular Cell Adhesion Molecule-1InflammationOxidative phosphorylationArticleCatalysisInorganic ChemistrySepsis03 medical and health sciencesSpecies Specificitytime responseInternal medicineReaction TimemedicineAnimalsRats WistarPhysical and Theoretical ChemistryMolecular BiologymouseInterleukin-6Platelet CountTumor Necrosis Factor-alphabusiness.industryOrganic Chemistrymedicine.diseaseRatsMice Inbred C57BL030104 developmental biologyEndocrinologylcsh:Biology (General)lcsh:QD1-999chemistryinflammationImmunologyHemoglobinbusinessOxidative stressInternational Journal of Molecular Sciences
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A role for Toll-like receptor mediated signals in neutrophils in the pathogenesis of the anti-phospholipid syndrome.

2012

The anti-phospholipid syndrome (APS) is characterized by recurrent thrombosis and occurrence of anti-phospholipid antibodies (aPL). aPL are necessary, but not sufficient for the clinical manifestations of APS. Growing evidence suggests a role of innate immune cells, in particular polymorphonuclear neutrophils (PMN) and Toll-like receptors (TLR) to be additionally involved. aPL activate endothelial cells and monocytes through a TLR4-dependent signalling pathway. Whether this is also relevant for PMN in a similar way is currently not known. To address this issue, we used purified PMN from healthy donors and stimulated them in the presence or absence of human monoclonal aPL and the TLR4 agonis…

LipopolysaccharidesNeutrophilsImmunology610 MedizinImmunoglobulinslcsh:MedicineInflammationApoptosisImmunopathologyBiologyNeutrophil ActivationAutoimmune DiseasesPhagocytosisimmune system diseases610 Medical sciencesmedicineHumansInterleukin 8L-SelectinReceptorlcsh:ScienceBiologyImmune ResponseneoplasmsRespiratory BurstInflammationToll-like receptorMultidisciplinaryInnate immune systemCD11b AntigenCoagulation DisordersEffectorInterleukin-8lcsh:RImmunityHematologyAntiphospholipid SyndromeFlow CytometryInnate ImmunityRespiratory burstToll-Like Receptor 4ImmunologyTLR4MedicineClinical Immunologylcsh:Qmedicine.symptomResearch ArticleSignal TransductionPLoS ONE
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Effect of Pro-inflammatory Stimuli on Tumor Cell-Mediated Induction of Endothelial Cell Adhesion Molecules in Vitro

2002

The object of our study was the question about the relevance of the tumor surrounding inflammatory cells with respect to the metastatic potential of the tumor cells. To imitate the role of inflammatory cells, three colon carcinoma (HT-29, HRT-18, and SW-620), one breast carcinoma (MCF-7), and one melanoma (ST-ML-12) cell lines were treated with pro-inflammatory stimuli, LPS, TNF-alpha, or IL-1beta. HUVEC monolayers were then stimulated by the collected supernatants (SN) of the tumor cells, following washing out of the applied stimuli. Analysis of CAM expression on HUVEC was performed using cell enzyme immunoassay. E-selectin, VCAM-1, and, in part, ICAM-1 were significantly up-regulated on H…

LipopolysaccharidesPathologymedicine.medical_specialtyEndotheliummedicine.medical_treatmentClinical BiochemistryCellVascular Cell Adhesion Molecule-1Breast NeoplasmsBiologyPathology and Forensic MedicineImmunoenzyme TechniquesNeoplasmsE-selectinTumor Cells CulturedmedicineHumansMelanomaMolecular BiologyCells CulturedInflammationTumor Necrosis Factor-alphaCell adhesion moleculeCarcinomaIntercellular Adhesion Molecule-1Up-Regulationmedicine.anatomical_structureCytokineTumor progressionCell cultureCulture Media ConditionedColonic Neoplasmsbiology.proteinCancer researchFemaleTumor necrosis factor alphaEndothelium VascularE-SelectinCell Adhesion MoleculesInterleukin-1Experimental and Molecular Pathology
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Endothelialization and Anticoagulation Potential of Surface-Modified PET Intended for Vascular Applications.

2018

In vascular tissue engineering, great attention is paid to the immobilization of biomolecules onto synthetic grafts to increase bio- and hemocompatibility-two critical milestones in the field. The surface modification field of poly(ethylene terephthalate) (PET), a well-known vascular-graft material, is matured and oversaturated. Nevertheless, most developed methods are laborious multistep procedures generally accompanied by coating instability or toxicity issues. Herein, a straightforward surface modification procedure is presented engineered to simultaneously promote surface endothelialization and anticoagulation properties via the covalent immobilization of gelatin through a photoactivate…

LipopolysaccharidesPolymers and PlasticsPoly(ethylene terephthalate)Gene ExpressionBiocompatible Materials02 engineering and technology01 natural sciencesGelatinendothelializationchemistry.chemical_compoundCoatingPolyethylene terephthalateMaterials Chemistrychemistry.chemical_classificationPolyethylene TerephthalatesSurface modifiedhemocompatibility021001 nanoscience & nanotechnologyPlatelet Endothelial Cell Adhesion Molecule-10210 nano-technologyE-Selectinbiotechnologyendotoxin contentazide photograftingAzidesfood.ingredientMaterials scienceBiocompatibilityCell SurvivalSurface PropertiesBioengineeringengineering.material010402 general chemistryBiomaterialsfoodvon Willebrand FactorHuman Umbilical Vein Endothelial CellsHumansTissue EngineeringBiomoleculeAnticoagulants0104 chemical sciencesBlood Vessel ProsthesischemistryengineeringSurface modificationBlood VesselsGelatinAzideBiomarkersBiomedical engineeringMacromolecular bioscience
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Application of X-ray microanalysis to study of the expression of endothelial adhesion molecules on human umbilical vein endothelial cells in vitro

1994

A semi-quantitative procedure is described, which allows the evaluation of expression levels of endothelial adhesion molecules on cultured human umbilical vein endothelial cells (HUVEC) using energy dispersive X-ray microanalysis (EDX). As a model two adhesion molecules, E-selection (CD62E; ELAM-1/endothelial leukocyte adhesion molecule-1) and ICAM-1 (intercellular adhesion molecule-1; CD54), were localized by the use of the silver-enhancement colloidal gold method after stimulation of HUVEC with endotoxin lipopolysaccharide (LPS), tumour necrosis factor (TNF) or a phorbol ester (PMA). The analysis was performed in a scanning electron microscope (SEM) at an accelerating voltage of 15 kV wit…

LipopolysaccharidesUmbilical VeinsHistologyEndotheliumEnzyme-Linked Immunosorbent AssayIn Vitro TechniquesUmbilical veinE-selectinmedicineHumansMolecular BiologyCells CulturedbiologyTumor Necrosis Factor-alphaChemistryCell adhesion moleculeCell BiologyGeneral MedicineImmunogold labellingAdhesionIntercellular Adhesion Molecule-1ImmunohistochemistryMolecular biologyStimulation ChemicalIn vitroMedical Laboratory Technologymedicine.anatomical_structurebiology.proteinTetradecanoylphorbol AcetateTumor necrosis factor alphaEndothelium VascularAnatomyE-SelectinGeneral Agricultural and Biological SciencesCell Adhesion MoleculesElectron Probe MicroanalysisHistochemistry
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Early high-dosage atorvastatin treatment improved serum immune-inflammatory markers and functional outcome in acute ischemic strokes classified as la…

2016

Statins have beneficial effects on cerebral circulation and brain parenchyma during ischemic stroke and reperfusion. The primary hypothesis of this randomized parallel trial was that treatment with 80 mg/day of atorvastatin administered early at admission after acute atherosclerotic ischemic stroke could reduce serum levels of markers of immune-inflammatory activation of the acute phase and that this immune-inflammatory modulation could have a possible effect on prognosis of ischemic stroke evaluated by some outcome indicators. We enrolled 42 patients with acute ischemic stroke classified as large arteries atherosclerosis stroke (LAAS) randomly assigned in a randomized parallel trial to the…

Male3400P-selectinAtorvastatinInterleukin-1beta030204 cardiovascular system & hematologylaw.inventionBrain IschemiaBrain ischemiaCerebral circulation0302 clinical medicineRandomized controlled triallawAtorvastatinIntracranial ArteriosclerosiStrokeInflammation MediatorbiologyClinical Trial/Experimental StudyGeneral MedicineMiddle AgedIntracranial ArteriosclerosisIntercellular Adhesion Molecule-1Interleukin-10StrokeP-SelectinAcute DiseaseCardiologyFemaleInflammation Mediatorsmedicine.symptomE-SelectinResearch Articlemedicine.drugHumanmedicine.medical_specialtyVascular Cell Adhesion Molecule-1Inflammation03 medical and health sciencesInternal medicinemedicineHumanscardiovascular diseasesInterleukin 6AgedInflammationbusiness.industryInterleukin-6Tumor Necrosis Factor-alphaBiomarkermedicine.diseasePhysical therapybiology.proteinbusinessBiomarkers030217 neurology & neurosurgery
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Angiotensin II-Induced Mononuclear Leukocyte Interactions with Arteriolar and Venular Endothelium Are Mediated by the Release of Different CC Chemoki…

2006

Abstract Angiotensin II (Ang-II) is associated with atherogenesis and arterial subendothelial mononuclear leukocyte infiltration. We have demonstrated that Ang-II causes the initial attachment of mononuclear cells to the arteriolar endothelium. We now report on the contribution of CC chemokines to this response. Intraperitoneal administration of 1 nM Ang-II induced MCP-1, RANTES, and MIP-1α generation, maximal at 4 h, followed by mononuclear leukocyte recruitment at 8 and 24 h. Using intravital microscopy within the rat mesenteric microcirculation 4 h after exposure to 1 nM Ang-II, arteriolar mononuclear cell adhesion was 80–90% inhibited by pretreatment with Met-RANTES, a CCR1 and CCR5 ant…

MaleCCR1EndotheliumImmunologyVascular Cell Adhesion Molecule-1Peripheral blood mononuclear cellUmbilical CordRats Sprague-DawleyLeukocyte CountCell MovementCell AdhesionLeukocytesmedicineAnimalsHumansImmunology and AllergyEndotheliumChemokine CCL5Cells CulturedChemokine CCL2Angiotensin II receptor type 1Chemokine CCL26business.industryAngiotensin IIMonocyteEpithelial Cellsmedicine.diseaseAngiotensin IIMolecular biologyRatsP-Selectinmedicine.anatomical_structureChemokines CCImmunologycardiovascular systembusinessInfiltration (medical)Intravital microscopyThe Journal of Immunology
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Role of S128R polymorphism of E-selectin in colon metastasis formation.

2007

The extravasation of cancer cells is a key step of the metastatic cascade. Polymorphisms in genes encoding adhesion molecules can facilitate metastasis by increasing the strength of interaction between tumor and endothelial cells as well as impacting other properties of cancer cells. We investigated the Ser128Arg (a561c at the nucleotide level) polymorphism in the E-selectin gene in patients with metastatic colon cancer and its functional significance. Genotyping for a561c polymorphism was performed on 172 cancer patients and on an age-matched control population. The colon cancer group was divided into groups with (M+) and without observable metastasis (M−). For in vitro functional assays, …

MaleCancer ResearchColorectal cancerBiologyArginineTransfectionMetastasise-SELECTIN; COLON CANCER METASTASISSettore BIO/13 - Biologia ApplicataCell MovementE-selectinmedicineCell AdhesionSerineTumor Cells CulturedHumansNeoplasm MetastasisPolymorphism GeneticCell adhesion moleculeCancerTransfectionMiddle Agedmedicine.diseaseExtravasationColon Carcinoma E-Selectin Metastasis PolymorphismPhenotypeOncologyImmunologyCancer cellColonic NeoplasmsCancer researchbiology.proteinFemaleE-SelectinSignal TransductionInternational journal of cancer
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A novel class of potent nonglycosidic and nonpeptidic pan-selectin inhibitors.

2006

An early step of the inflammatory response, the rolling of leukocytes on activated endothelial cells, is mediated by selectin/carbohydrate interactions. The tetrasaccharide sialy Lewisx is a ligand for E-, P-, and L-selectin and therefore serves as a lead structure for the development of analogues. A combination of synthesis and structure-based design allowed rapid optimization. The current lead 2a was evaluated in our E-selectin cell flow chamber assay where it proved to inhibit rolling and adhesion with an IC50 of 28+/-7 microM. The assays used are predictive for the in vivo efficacy of test compounds as shown for 2a in a proteose peptone induced peritonitis model of acute inflammation in…

MaleModels MolecularInflammationEnzyme-Linked Immunosorbent AssayIn Vitro TechniquesPeritonitisLigandsMiceStructure-Activity RelationshipIn vivoDrug DiscoverymedicineCell AdhesionLeukocytespara-AminobenzoatesTetrasaccharideAnimalsIC50Binding SitesChemistryCell adhesion moleculeAnti-Inflammatory Agents Non-SteroidalCaseinsEndothelial CellsLigand (biochemistry)In vitroPeptide FragmentsMice Inbred C57BLBiochemistryAcute DiseaseSelectinsMolecular Medicinemedicine.symptomE-Selectin4-Aminobenzoic AcidSelectinAlgorithmsProtein BindingJournal of medicinal chemistry
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