Search results for "Signal Transduction"

showing 10 items of 2020 documents

Transcriptome profiling of citrus fruit response to huanglongbing disease.

2010

Huanglongbing (HLB) or "citrus greening" is the most destructive citrus disease worldwide. In this work, we studied host responses of citrus to infection with Candidatus Liberibacter asiaticus (CaLas) using next-generation sequencing technologies. A deep mRNA profile was obtained from peel of healthy and HLB- affected fruit. It was followed by pathway and protein-protein network analysis and quantitative real time PCR analysis of highly regulated genes. We identified differentially regulated pathways and constructed networks that provide a deep insight into the metabolism of affected fruit. Data mining revealed that HLB enhanced transcription of genes involved in the light reactions of phot…

CitrusProtein FoldingGene Identification and Analysislcsh:MedicinePlant ScienceTranscriptomechemistry.chemical_compoundRNA interferencePlant Growth RegulatorsGene Expression Regulation PlantModelsGene expressionPlant Genomics2.1 Biological and endogenous factorsPhotosynthesisAetiologylcsh:SciencePlant Growth and DevelopmentPlant PestsMultidisciplinaryProtein StabilityJasmonic acidfood and beveragesHigh-Throughput Nucleotide SequencingAgriculturePlantsCell biologyCarbohydrate MetabolismResearch ArticleSignal TransductionGeneral Science & TechnologyPlant PathogensProtein degradationBiologyModels BiologicalFruitsMolecular GeneticsRhizobiaceaeSettore AGR/07 - Genetica AgrariaHeat shock proteinBotanyGeneticsGene RegulationGene NetworksBiologyTranscription factorPlant DiseasesAnalysis of VarianceGene Expression Profilinglcsh:RCitrus HLB next-generation sequencing candidatus liberibacterComputational BiologyPlantPlant PathologyBiologicalWRKY protein domainGene expression profilingchemistryGene Expression Regulationlcsh:QGene expressionGene FunctionTranscriptomeTranscription Factors
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Resistin: Insulin resistance to malignancy

2014

Adipose tissue is recognized as an endocrine organ that secretes bioactive substances known as adipokines. Excess adipose tissue and adipose tissue dysfunction lead to dysregulated adipokine production that can contribute to the development of obesity-related co-morbidities. Among the various adipokines, resistin, which was initially considered as a determinant of the emergence of insulin resistance in obesity, has appeared as an important link between obesity and inflammatory processes. Several experimental and clinical studies have suggested an association between increased resistin levels and severe conditions associated with obesity such as cardiovascular disease and malignancies. In th…

Clinical BiochemistryAdipokineAdipose tissueInflammationDiseaseBiochemistryImmune systemInsulin resistanceNeoplasmsHumansMedicineResistinObesityInflammationbusiness.industryBiochemistry (medical)General MedicineAtherosclerosismedicine.diseaseImmunologyResistinTumor necrosis factor alphaInsulin Resistancemedicine.symptombusinessSignal TransductionClinica Chimica Acta
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SMART: Unique splitting-while-merging framework for gene clustering

2014

© 2014 Fa et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Successful clustering algorithms are highly dependent on parameter settings. The clustering performance degrades significantly unless parameters are properly set, and yet, it is difficult to set these parameters a priori. To address this issue, in this paper, we propose a unique splitting-while-merging clustering framework, named "splitting merging awareness tactics" (SMART), which does not require any a priori knowledge of either the number …

Clustering algorithmsMicroarrayslcsh:MedicineGene ExpressionBioinformaticscomputer.software_genreCell SignalingData MiningCluster Analysislcsh:ScienceFinite mixture modelOligonucleotide Array Sequence AnalysisPhysicsMultidisciplinarySMART frameworkConstrained clusteringCompetitive learning modelBioassays and Physiological AnalysisMultigene FamilyCanopy clustering algorithmEngineering and TechnologyData miningInformation TechnologyGenomic Signal ProcessingAlgorithmsResearch ArticleSignal TransductionComputer and Information SciencesFuzzy clusteringCorrelation clusteringResearch and Analysis MethodsClusteringMolecular GeneticsCURE data clustering algorithmGeneticsGene RegulationCluster analysista113Gene Expression Profilinglcsh:RBiology and Life SciencesComputational BiologyCell BiologyDetermining the number of clusters in a data setComputingMethodologies_PATTERNRECOGNITIONSplitting-merging awareness tactics (SMART)Signal ProcessingAffinity propagationlcsh:QGene expressionClustering frameworkcomputer
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Heat shock and Cd2+ exposure regulate PML and Daxx release from ND10 by independent mechanisms that modify the induction of heat-shock proteins 70 an…

2003

Nuclear domains called ND10 or PML bodies might function as nuclear depots by recruiting or releasing certain proteins. Although recruitment of proteins through interferon-induced upregulation and SUMO-1 modification level of PML had been defined, it is not known whether release of proteins is regulated and has physiological consequences. Exposure to sublethal environmental stress revealed a sequential release of ND10-associated proteins. Upon heat shock Daxx and Sp100 were released but PML remained, whereas exposure to subtoxic concentrations of CdCl2 induced the release of ND10-associated proteins, including PML, with Sp100 remaining in a few sites. In both cases,recovery times were simil…

Co-Repressor ProteinsMAP Kinase Signaling SystemMacromolecular SubstancesSUMO-1 ProteinPromyelocytic Leukemia ProteinMicePromyelocytic leukemia proteinDeath-associated protein 6Stress PhysiologicalHeat shock proteinEndopeptidasesAnimalsHSP70 Heat-Shock ProteinsEnzyme InhibitorsHeat shockTranscription factorCells CulturedHeat-Shock ProteinsbiologyTumor Suppressor ProteinsIntracellular Signaling Peptides and ProteinsNuclear ProteinsCell BiologyCell Nucleus StructuresNeoplasm ProteinsCell biologyHsp70Cysteine EndopeptidasesEukaryotic CellsGene Expression RegulationImmunologybiology.proteinSignal transductionCarrier ProteinsCo-Repressor ProteinsHeat-Shock ResponseCadmiumMolecular ChaperonesTranscription FactorsJournal of Cell Science
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Sorting signals in the cytosolic tail of plant p24 proteins involved in the interaction with the COPII coat.

2004

The ability of the cytosolic tail of a plant p24 protein to bind COPI and COPII subunits from plant and animal sources in vitro has been examined. We have found that a dihydrophobic motif in the -7,-8 position (relative to the cytosolic carboxy-terminus), which strongly cooperates with a dilysine motif in the -3,-4 position for COPI binding, is required for COPII binding. In addition, we show that COPI and COPII coat proteins from plant cytosol compete for binding to the sorting motifs in these tails. Only in the absence of the dilysine motif in the -3,-4 position or after COPI depletion could we observe COPII binding to the p24 tail. This competition is not observed when using rat liver cy…

CoatPhysiologyAmino Acid MotifsArabidopsisReceptors Cytoplasmic and NuclearPlant ScienceBiologyCoat Protein Complex ICytosolAnimalsCOPIIBinding SitesVesicular-tubular clusterArabidopsis ProteinsCell BiologyGeneral MedicineCOPIPlant cellIn vitroPeptide FragmentsCell biologyRatsCytosolProtein TransportRat liverCOP-Coated VesiclesProtein BindingSignal TransductionPlantcell physiology
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A Mutually Stimulating Loop Involving Emx2 and Canonical Wnt Signalling Specifically Promotes Expansion of Occipital Cortex and Hippocampus

2005

The correct size of the different areas composing the mature cerebral cortex depends on the proper early allocation of cortical progenitors to their distinctive areal fates, as well as on appropriate subsequent tuning of their area-specific proliferation--differentiation profiles. Whereas much is known about the genetics of the former process, the molecular mechanisms regulating proliferation and differentiation rates within distinctive cortical proto-areas are still largely obscure. Here we show that a mutual stimulating loop, involving Emx2 and canonical Wnt signalling, specifically promotes expansion of the occipito-hippocampal anlage. Collapse of this loop occurring in Emx2 2/2 mutants …

Cognitive NeuroscienceEMX2HippocampusSettore BIO/11 - Biologia MolecolareProneural genescell cycle genesBiologyHippocampusMiceCellular and Molecular NeuroscienceCortex (anatomy)medicineAnimalsWnt signallingHomeodomain ProteinsNeuronsproneural genesStem CellsGene Expression Regulation DevelopmentalCell DifferentiationCell cycleareal sizingCell Cycle GeneMice Mutant StrainsWnt Proteinsmedicine.anatomical_structureCerebral cortexEmx2Occipital LobeOccipital lobeareal sizing; Emx2; Wnt signalling; cell cycle genes; proneural genesNeuroscienceCell DivisionSignal TransductionTranscription FactorsCerebral Cortex
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Epithelial NEMO links innate immunity to chronic intestinal inflammation

2007

Deregulation of intestinal immune responses seems to have a principal function in the pathogenesis of inflammatory bowel disease(1-4). The gut epithelium is critically involved in the maintenance of intestinal immune homeostasis-acting as a physical barrier separating luminal bacteria and immune cells, and also expressing antimicrobial peptides(3,5,6). However, the molecular mechanisms that control this function of gut epithelial cells are poorly understood. Here we show that the transcription factor NF kappa B, a master regulator of pro-inflammatory responses(7,8), functions in gut epithelial cells to control epithelial integrity and the interaction between the mucosal immune system and gu…

ColonAntimicrobial peptidesApoptosisBiologyPathogenesisInterleukin 22MiceImmune systemAnimalsHomeostasisMultidisciplinaryInnate immune systemNF-kappa BEpithelial CellsColitisImmunity InnateI-kappa B KinaseGut EpitheliumCell biologyIntestinesReceptors Tumor Necrosis Factor Type IChronic DiseaseMyeloid Differentiation Factor 88Tumor Necrosis FactorsImmunologyChronic inflammatory responseTumor necrosis factor alphaSignal TransductionNature
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Dynamic regulation of the cancer stem cell compartment by Cripto-1 in colorectal cancer.

2015

Stemness was recently depicted as a dynamic condition in normal and tumor cells. We found that the embryonic protein Cripto-1 (CR1) was expressed by normal stem cells at the bottom of colonic crypts and by cancer stem cells (CSCs) in colorectal tumor tissues. CR1-positive populations isolated from patient-derived tumor spheroids exhibited increased clonogenic capacity and expression of stem-cell-related genes. CR1 expression in tumor spheroids was variable over time, being subject to a complex regulation of the intracellular, surface and secreted protein, which was related to changes of the clonogenic capacity at the population level. CR1 silencing induced CSC growth arrest in vitro with a …

Colorectal cancerColorectal NeoplasmCriptoMiceIntercellular Signaling Peptides and ProteinTumor Cells CulturedRegulation of gene expressionCulturedstem cell; CRIPTO 1GPI-Linked ProteinCell biologyNeoplasm ProteinsTumor CellsGene Expression Regulation NeoplasticGenes srcNeoplastic Stem CellsIntercellular Signaling Peptides and ProteinsFemaleStem cellColorectal NeoplasmsHumanSignal Transductioncolorectal cancerBiologyGPI-Linked ProteinsAnimals; Colorectal Neoplasms; Female; GPI-Linked Proteins; Gene Expression Regulation Neoplastic; Genes src; Humans; Intercellular Signaling Peptides and Proteins; Mice; Neoplasm Proteins; Neoplastic Stem Cells; Proto-Oncogene Proteins c-akt; Signal Transduction; Spheroids Cellular; Tumor Cells Cultured; Cell Biology; Molecular BiologyNeoplasm ProteinCancer stem cellSettore MED/04 - PATOLOGIA GENERALESpheroids CellularmedicineGene silencingAnimalsHumansClonogenic assayProtein kinase BMolecular BiologysrcOriginal PaperNeoplasticAnimalCell Biologymedicine.diseaseGene Expression RegulationGenesNeoplastic Stem CellCellularSpheroidsanimals; colorectal neoplasms; female; GPI-linked proteins; gene expression regulation; neoplastic; genes src; humans; intercellular signaling peptides and proteins; mice; neoplasm proteins; neoplastic stem cells; proto-oncogene proteins c-akt; signal transduction; spheroids; cellular; tumor cells; culturedAnimals; Colorectal Neoplasms; Female; GPI-Linked Proteins; Gene Expression Regulation Neoplastic; Genes src; Humans; Intercellular Signaling Peptides and Proteins; Mice; Neoplasm Proteins; Neoplastic Stem Cells; Proto-Oncogene Proteins c-akt; Signal Transduction; Spheroids Cellular; Tumor Cells Cultured; Molecular Biology; Cell BiologyProto-Oncogene Proteins c-akt
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A variant of Smurf2 protects mice against colitis-associated colon cancer by inducing transforming growth factor β signaling.

2011

Background & Aims Transforming growth factor (TGF)-β signaling, which is down-regulated by the E3 ubiquitin ligase Smad ubiquitin regulating factor 2 (Smurf2), promotes development of cancer. We identified a splice variant of Smurf2 (ΔE2Smurf2) and investigated its role in colon carcinogenesis in mice. Methods Colitis-associated colon cancer was induced in mice by administration of azoxymethane, followed by 3 cycles of oral administration of dextran sodium sulfate. Messenger RNA levels of Smurf2 in colon tumors and control tissue were measured by quantitative polymerase chain reaction; lymphocyte and cytokine levels were measured in tumor and tissue samples. Results Tumor-infiltrating CD4 +…

Colorectal cancermedicine.medical_treatmentLymphocyteUbiquitin-Protein LigasesSMADBiologyProinflammatory cytokineReceptors G-Protein-Coupledchemistry.chemical_compoundMiceTransforming Growth Factor betamedicineAnimalsCells CulturedHepatologyAzoxymethaneGene Expression ProfilingGastroenterologymedicine.diseaseColitisMolecular biologyUbiquitin ligaseMice Inbred C57BLProto-Oncogene Proteins c-kitCytokinemedicine.anatomical_structureHyaluronan ReceptorschemistryColonic Neoplasmsbiology.proteinCancer researchTransforming growth factorSignal TransductionGastroenterology
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The Study of Carbamoyl Phosphate Synthetase 1 Deficiency Sheds Light on the Mechanism for Switching On/Off the Urea Cycle

2015

12 páginas, 4 figuras, 2 tablas.

Conformational changeCarbamoyl-Phosphate Synthase I Deficiency DiseaseAllosteric regulationCarbamoyl-Phosphate Synthase (Ammonia)Urea cycle diseases610 Medicine & healthBiologyMolecular Dynamics Simulationurologic and male genital diseases03 medical and health sciences0302 clinical medicineGlutamates1311 GeneticsAmmoniaEnzyme StabilityGeneticsmedicine1312 Molecular BiologyHumansUreaHyperammonemiaSite-directed mutagenesisMolecular Biology030304 developmental biologychemistry.chemical_classification0303 health sciencesSite-directed mutagenesisurogenital systemMutagenesisCarbamoyl phosphate synthetase 1HyperammonemiaCarbamoyl phosphate synthetasemedicine.diseaseAllosteric regulation3. Good healthProtein Structure TertiaryRestrained molecular dynamicsKineticsEnzymeBiochemistrychemistry10036 Medical ClinicEnzymeUrea cycleMutationInborn errors030217 neurology & neurosurgerySignal Transduction
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