Search results for "Signal"

showing 10 items of 6924 documents

3D modul for syntetisk kalkulator

2006

Masteroppgave i informasjons- og kommunikasjonsteknologi 2006 - Høgskolen i Agder, Grimstad ActionScript er et språk som har blitt benyttet mer og mer de senere årene og er en del av verktøyet Flash. Dette er et design verktøy som har gode metoder for animasjoner i 2D og genererer små filer som gjør det svært egnet for internett. Denne rapporten ser på mulighetene for å generere, håndtere og visualisere tredimensjonale objekter i Flash ved bruk av ActionScript. Det blir diskutert roteringsmatriser opp mot Quaternion rotasjon og isometrisk projeksjon opp mot perspektivisk projeksjon i forhold til 3D objekter. Rapporten tar for seg ulike kurver og flater og hvilke begrensninger ActionScript h…

QuaternionIKT590FlaterActionScriptVDP::Matematikk og naturvitenskap: 400::Informasjons- og kommunikasjonsvitenskap: 420::Simulering visualisering signalbehandling bildeanalyse: 4293DVDP::Matematikk og naturvitenskap: 400::Informasjons- og kommunikasjonsvitenskap: 420::Systemutvikling og -arbeid: 426
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Rho prevents apoptosis through Bcl-2 expression: Implications for interleukin-2 receptor signal transduction

1997

Here we describe a Rho-mediated apoptosis suppression pathway driven by Bcl-2 expression in the interleukin (IL)-4- or IL-2-dependent murine T cell line TS1 alpha beta. IL-2, but not IL-4, induces Bcl-2 expression through RhoA activation which is inhibited by the specific Rho family inhibitor, Clostridium difficile Toxin B, as well as by a dominant negative RhoA mutant. Using transient transfections of RhoA mutants tagged with the vesicular stomatitis virus glycoprotein, we show that a constitutively active RhoA mutant induces Bcl-2 expression and prevents apoptosis upon IL-4 withdrawal. Finally, we have identified the signaling pathway involved together with RhoA in Bcl-2 induction and sho…

RHOAImmunologyDown-RegulationClostridium difficile toxin AApoptosisClostridium difficile toxin BTransfectionCell LineMicePhosphatidylinositol 3-Kinaseschemistry.chemical_compoundGTP-Binding ProteinsAnimalsHumansImmunology and AllergyPhosphatidylinositolProtein kinase AProtein Kinase CbiologyKinaseInterleukinReceptors Interleukin-2Molecular biologyCell biologyProto-Oncogene Proteins c-bcl-2chemistrybiology.proteinInterleukin-2Signal transductionrhoA GTP-Binding ProteinSignal TransductionEuropean Journal of Immunology
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Clathrin-independent entry of baculovirus triggers uptake of E. coli in non-phagocytic human cells

2008

The prototype baculovirus, Autographa californica multiple nucleopolyhedrovirus, an insect pathogen, holds great potential as a gene therapy vector. To develop transductional targeting and gene delivery by baculovirus, we focused on characterizing the nature and regulation of its uptake in human cancer cells. Baculovirus entered the cells along fluid-phase markers from the raft areas into smooth-surfaced vesicles devoid of clathrin. Notably, regulators associated with macropinocytosis, namely EIPA, Pak1, Rab34, and Rac1, had no significant effect on viral transduction, and the virus did not induce fluid-phase uptake. The internalization and nuclear uptake was, however, affected by mutants o…

RHOASciencevirusesmedia_common.quotation_subjecteducationGene deliveryClathrinCell LineMembrane Lipids03 medical and health sciencesPhagocytosisCell Biology/Membranes and SortingViral entryVirologyEscherichia coliBiochemistry/Cell Signaling and Trafficking StructuresHumansInternalization030304 developmental biologymedia_commonAdenosine Triphosphatases0303 health sciencesMultidisciplinaryBase SequencebiologyADP-Ribosylation FactorsQ030302 biochemistry & molecular biologyHEK 293 cellsRTransfectionMolecular biologyClathrinEndocytosisNucleopolyhedroviruses3. Good healthCell biologyvirologiaADP-Ribosylation Factor 6Cell culturebiology.proteinMedicineRNA InterferenceResearch Article
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The ras-related small GTP-binding protein RhoB is immediate-early inducible by DNA damaging treatments.

1995

The low molecular weight GTP-binding proteins RhoA, RhoB, and RhoC are characterized as specific substrates for the ADP-ribosyltransferase C3 from Clostridium botulinum and are supposed to be involved in the organization of the microfilamental network and transformation. rhoB is known to be immediate-early inducible by growth factors and protein-tyrosine kinases. Since increasing evidence indicates overlapping of growth factor- and UV-induced signal pathways, we studied the effect of UV light and other genotoxic agents on early rhoB transcription. Within 30 min after UV irradiation of NIH3T3 cells, the amount of rhoB mRNA increased 3-4-fold. Elevated rhoB mRNA was accompanied by an increase…

RHOAUltraviolet RaysRHOBRetinoic acidCycloheximideBiologyBiochemistrychemistry.chemical_compoundMiceGTP-Binding ProteinsRhoB GTP-Binding ProteinAnimalsRNA MessengerProtein kinase ArhoB GTP-Binding ProteinMolecular BiologyGenes Immediate-EarlyAdenosine Diphosphate RiboseKinaseMembrane ProteinsCell Biology3T3 CellsDNAMolecular biologychemistryGene Expression Regulationbiology.proteinDactinomycinTetradecanoylphorbol AcetateSignal transductionDNA DamageThe Journal of biological chemistry
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Protein kinase inhibitor β enhances the constitutive activity of G-protein-coupled zinc receptor GPR39.

2014

GPR39 is a G-protein-coupled zinc receptor that protects against diverse effectors of cell death. Its protective activity is mediated via constitutive activation of Gα13 and the RhoA pathway, leading to increased SRE (serum-response element)-dependent transcription; the zinc-dependent immediate activation of GPR39 involves Gq-mediated increases in cytosolic Ca2+ and Gs coupling leading to increased cAMP levels. We used the cytosolic and soluble C-terminus of GPR39 in a Y2H (yeast-2-hybrid) screen for interacting proteins, thus identifying PKIB (protein kinase A inhibitor β). Co-expression of GPR39 with PKIB increased the protective activity of GPR39 via the constitutive, but not the ligand-…

RHOAmedicine.drug_classG proteinCHO CellsBiochemistryCell LineReceptors G-Protein-CoupledMiceCricetulusTwo-Hybrid System TechniquesmedicineAnimalsHumansProtein kinase AReceptorMolecular BiologyProtein Kinase InhibitorsbiologyEffectorCell MembraneIntracellular Signaling Peptides and ProteinsCell BiologyProtein kinase inhibitorCyclic AMP-Dependent Protein KinasesProtein Kinase A InhibitorCytosolZincBiochemistrybiology.proteinHeLa CellsThe Biochemical journal
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IAPs and Resistance to Death Receptors in Cancer

2017

Since their identification in mammal cells, IAPs emerged have as potent regulators of death receptor signalling pathways, determining the cell fate in response to receptor stimulation. Among IAPs, cIAP1 and cIAP2 are active components of receptor-associated signalling complexes able to promote the activation of ubiquitin-dependent survival signalling pathways. For its part, XIAP is an important regulator of caspase activity, determining the apoptotic signalling pathway engaged after death receptor stimulation. The use of IAP antagonists is a promising strategy in order to overcome the resistance of tumor cells to death receptor stimulation.

RIPK1chemistry.chemical_compoundSignallingchemistryApoptosisNecroptosisRegulatorCancer researchNF-κBBiologyCell fate determinationXIAP
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EBV-Induced Gene 3 Transcription Is Induced by TLR Signaling in Primary Dendritic Cells via NF-κB Activation

2005

Abstract The EBV-induced gene 3 (EBI3) is expressed in dendritic cells (DCs) and part of the cytokine IL-27 that controls Th cell development. However, its regulated expression in DCs is poorly understood. In the present study we demonstrate that EBI3 is expressed in splenic CD8−, CD8+, and plasmacytoid DC subsets and is induced upon TLR signaling. Cloning and functional analysis of the EBI3 promoter using in vivo footprinting and mutagenesis showed that stimulation via TLR2, TLR4, and TLR9 transactivated the promoter in primary DCs via NF-κB and Ets binding sites at −90 and −73 bp upstream of the transcriptional start site, respectively. Furthermore, we observed that NF-κB p50/p65 and PU.1…

RNA Capsmedicine.medical_treatmentDNA Mutational AnalysisMolecular Sequence DataImmunologyAntigen-Presenting CellsReceptors Cell SurfaceBiologyCell LineMinor Histocompatibility AntigensJurkat CellsMiceCell Line TumorGene expressionmedicineAnimalsHumansImmunology and AllergyReceptors CytokinePromoter Regions GeneticGlycoproteinsMice KnockoutMembrane GlycoproteinsInnate immune systemBase SequenceToll-Like ReceptorsHEK 293 cellsNF-kappa BTLR9hemic and immune systemsEBI3Dendritic CellsMolecular biologyToll-Like Receptor 2Up-RegulationMice Inbred C57BLToll-Like Receptor 4Protein SubunitsTLR2CytokineGene Expression RegulationToll-Like Receptor 9NIH 3T3 CellsTLR4Protein BindingSignal TransductionThe Journal of Immunology
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Abscisic Acid Connects Phytohormone Signaling with RNA Metabolic Pathways and Promotes an Antiviral Response that Is Evaded by a Self-Controlled RNA …

2020

© 2020 The Authors.

RNA StabilityvirusesPotyvirusArabidopsisPlant Scienceantiviral immune evasionBiochemistryArticleTranscriptomeAbscisic acidPlant Growth RegulatorsPlant virusTobaccoPlant ImmunityMolecular BiologyImmune EvasionPlant DiseasesRNA metabolismbiologyfungimathematical modelingPotyvirusfood and beveragesRNARNA virusTranslation (biology)viral polyprotein processingCell BiologyViral polyprotein processingbiology.organism_classificationCell biologyViral replicationRNA PlantAntiviral immune evasionMathematical modelingMetabolic Networks and PathwaysAbscisic AcidSignal TransductionBiotechnologyPlant Communications
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Age-dependent regulation of antioxidant genes by p38α MAPK in the liver

2018

p38α is a redox sensitive MAPK activated by pro-inflammatory cytokines and environmental, genotoxic and endoplasmic reticulum stresses. The aim of this work was to assess whether p38α controls the antioxidant defense in the liver, and if so, to elucidate the mechanism(s) involved and the age-related changes. For this purpose, we used liver-specific p38α-deficient mice at two different ages: young-mice (4 months-old) and old-mice (24 months-old). The liver of young p38α knock-out mice exhibited a decrease in GSH levels and an increase in GSSG/GSH ratio and malondialdehyde levels. However, old mice deficient in p38α had higher hepatic GSH levels and lower GSSG/GSH ratio than young p38α knock-…

ROS Reactive oxygen species;RSK1 Ribosomal S6 kinase10301 basic medicineMAPK/ERK pathwayAgingHPLC High-performance liquid chromatographyAntioxidantmedicine.medical_treatmentTBP TATA-binding proteinClinical BiochemistryDEN Diethyl nitrosamine;MKP-1 MAPK phosphatase-1IκB kinaseGCLc Glutamate cysteine ligase catalytic subunitp38 Mitogen-Activated Protein KinasesG6PDH Glucose-6-phosphate dehydrogenaseBiochemistryAntioxidantsMicechemistry.chemical_compoundSuperoxide Dismutase-1Akt Protein kinase B0302 clinical medicineNrf2 Nuclear factor erythroid 2-related factor-2IL InterleukinSOD1 Cu/Zn-superoxide dismutaselcsh:QH301-705.5Mice KnockoutMK2 MAP-activated protein kinase 2;PGC-1α Peroxisome proliferator-activated receptor gamma coactivator 1-alphachemistry.chemical_classificationlcsh:R5-920Trx ThioredoxinGlutathione DisulfideTNF-α Tumor necrosis factor-alphabiologyLPS Lipopolysaccharide;GSSG Oxidized glutathione;MEF Mouse embryonic fibroblastsNF-kappa BGstm1 Glutathione S-transferase mu 1CatalaseEndoplasmic Reticulum StressGlutathioneLiverGSH Reduced glutathione;Catalase030220 oncology & carcinogenesisJNK c-Jun N-terminal kinaselcsh:Medicine (General)Research Papermedicine.medical_specialtyNF-E2-Related Factor 2Glutamate-Cysteine LigaseMKK MAPK kinaseAP-1 Activator protein-1IKK IƙB KinaseGene Expression Regulation EnzymologicSuperoxide dismutase03 medical and health sciencesInternal medicineGlutamate cysteine ligaseEGFR Epidermal growth factor receptormedicineAnimalsNuclear factor ƙBAnd catalaseChIP Chromatin immunoprecipitation;Protein kinase BNF-ƙB Nuclear factor kappa BSuperoxide DismutaseSuperoxide dismutase 1Superoxide dismutase 2Organic ChemistryGlutathioneASK1 Apoptosis signal-regulating kinase 1ATF2 activating transcription factor 2;030104 developmental biologyEndocrinologyEnzymeHsp Heat shock proteinlcsh:Biology (General)chemistrybiology.proteinSOD2 Mn-superoxide dismutaseMAPK mitogen activated protein kinaseNEM N-ethyl maleimide;Redox Biology
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Where to Look

2010

SETI scientists are convinced that, of all possible types of radiation, any signal coming to us from another galactic civilization will have the form of electromagnetic waves; therefore, the search centers exclusively on these types of radiation. Why electromagnetic waves?

Radio telescopePhysicsPhysics::Popular PhysicsRadio galaxyAstronomyRadiationSignalElectromagnetic radiationSearch for extraterrestrial intelligenceRadio spectrumRadio wave
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