Search results for "Signaling pathway"

showing 10 items of 284 documents

Serine- and Threonine/Valine-Dependent Activation of PDK and Tor Orthologs Converge on Sch9 to Promote Aging

2014

Dietary restriction extends longevity in organisms ranging from bacteria to mice and protects primates from a variety of diseases, but the contribution of each dietary component to aging is poorly understood. Here we demonstrate that glucose and specific amino acids promote stress sensitization and aging through the differential activation of the Ras/cAMP/PKA, PKH1/2 and Tor/S6K pathways. Whereas glucose sensitized cells through a Ras-dependent mechanism, threonine and valine promoted cellular sensitization and aging primarily by activating the Tor/S6K pathway and serine promoted sensitization via PDK1 orthologs Pkh1/2. Serine, threonine and valine activated a signaling network in which Sch…

ThreonineCancer ResearchAgingSerineMice0302 clinical medicineSettore BIO/13 - Biologia ApplicataGene Expression Regulation FungalMolecular Cell BiologySerineSignaling in Cellular ProcessesThreonineGenetics (clinical)Cellular Stress Responses0303 health sciencesageing longevity Sch9 Tor Pkhs nutrients amino acidssurvival stress resistanceMechanisms of Signal TransductionValineCell biologyBiochemistryPhosphorylationSignal transductionResearch ArticleSignal TransductionSaccharomyces cerevisiae Proteinslcsh:QH426-470Adenylyl Cyclase Signaling PathwayLongevityP70-S6 Kinase 1Ras SignalingSaccharomyces cerevisiaeBiologyMicrobiologySignaling Pathways3-Phosphoinositide-Dependent Protein Kinases03 medical and health sciencesModel OrganismsStress PhysiologicalGeneticsAnimalsGene NetworksProtein kinase AMolecular BiologyTranscription factorBiologyEcology Evolution Behavior and Systematics030304 developmental biologySerine/threonine-specific protein kinase[SDV.GEN]Life Sciences [q-bio]/GeneticsCyclic AMP-Dependent Protein Kinaseslcsh:GeneticsGlucoseFoodTor SignalingProtein Kinases030217 neurology & neurosurgeryTranscription Factors
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Triclosan induces Fas receptor-dependent apoptosis in mouse neocortical neurons in vitro

2014

Triclosan (TCS) is a commonly used antimicrobial agent in personal care and sanitizing products, as well as in household items. Numerous studies have demonstrated the presence of TCS in various human tissues. Several studies have reported the accumulation of TCS in fish and human brain tissue. The aim of the present study was to investigate the effect of TCS on apoptosis in mouse neocortical neurons after 7 days of culture in vitro following 3, 6 and 24 h of exposure. To explore the mechanism underlying the effects of TCS in neurons, we studied the activation and protein expression of the Fas receptor (FasR) and caspase- 8, caspase-9 and caspase-3, as well as DNA fragmentation in TCS-treate…

Time FactorsExtrinsic apoptotic signaling pathwayApoptosisNeocortexDNA fragmentation.DNA FragmentationCaspase 8caspase-8FasRMicePregnancyAnimalsfas ReceptorFADDEnzyme InhibitorsCells CulturedNeuronsDose-Response Relationship DrugL-Lactate DehydrogenasebiologyGeneral NeurosciencefungiEmbryo MammalianStaurosporineFas receptorApoptotic bodyTriclosanIn vitroCell biologyBiochemistryApoptosisCaspasesbiology.proteinFatty Acid Synthesis InhibitorsDNA fragmentationFemaleNeuroscience
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High miR-196a levels promote the oncogenic phenotype of colorectal cancer cells.

2009

AIM: To analyze the relevance of the microRNA miR-196a for colorectal oncogenesis. METHODS: The impact of miR-196a on the restriction targets HoxA7, HoxB8, HoxC8 and HoxD8 was analyzed by reverse transcription polymerase chain reaction (RT-PCR) after transient transfection of SW480 cancer cells. The miR-196a transcription profile in colorectal cancer samples, mucosa samples and diverse cancer cell lines was quantified by RT-PCR. Transiently miR-196a-transfected colorectal cancer cells were used for diverse functional assays in vitro and for a xenograft lung metastasis model in vivo. RESULTS: HoxA7, HoxB8, HoxC8 and HoxD8 were restricted by miR-196a in a dose-dependent and gene-specific mann…

Transcription GeneticColorectal cancerColonTransplantation HeterologousMouse model of colorectal and intestinal cancerBiologymedicine.disease_causeMiceCell Line TumormicroRNAmedicineCell AdhesionAnimalsHumansProtein kinase BCell ProliferationAkt/PKB signaling pathwayGastroenterologyGeneral MedicineOriginal Articlesmedicine.diseaseReverse transcription polymerase chain reactionMicroRNAsPhenotypeCancer cellCancer researchCarcinogenesisColorectal NeoplasmsNeoplasm TransplantationSignal TransductionWorld journal of gastroenterology
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Hypoxia-inducible factor 1Α may regulate the commitment of mesenchymal stromal cells toward angio-osteogenesis by mirna-675-5P

2017

Abstract Background aims During bone formation, angiogenesis and osteogenesis are regulated by hypoxia, which is able to induce blood vessel formation, as well as recruit and differentiate human mesenchymal stromal cells (hMSCs). The molecular mechanisms involved in HIF-1α response and hMSC differentiation during bone formation are still unclear. This study aimed to investigate the synergistic role of hypoxia and hypoxia-mimetic microRNA miR-675-5p in angiogenesis response and osteo-chondroblast commitment of hMSCs. Methods By using a suitable in vitro cell model of hMSCs (maintained in hypoxia or normoxia), the role of HIF-1α and miR-675-5p in angiogenesis and osteogenesis coupling was inv…

Transcriptional ActivationVascular Endothelial Growth Factor A0301 basic medicineCancer ResearchAngiogenesisCellular differentiationImmunologyNeovascularization PhysiologicBiology03 medical and health scienceschemistry.chemical_compoundOsteogenesisMiR-675-5pmedicineHumansImmunology and AllergyHypoxiaCells Culturedbeta CateninGenetics (clinical)TransplantationOsteoblastsMesenchymal stromal cellMesenchymal stem cellWnt signaling pathwayCell DifferentiationMesenchymal Stem CellsOsteoblastCell BiologyHypoxia-Inducible Factor 1 alpha SubunitCell HypoxiaUp-RegulationCell biologyVascular endothelial growth factorMicroRNAsVascular endothelial growth factor A030104 developmental biologymedicine.anatomical_structureGene Expression RegulationOncologyHypoxia-inducible factorschemistryRegenerative medicineImmunologyOsteoblast commitmentCytotherapy
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Impairing Otp homeodomain function in oral ectoderm cells affects skeletogenesis in sea urchin embryos

2003

AbstractIn the sea urchin embryo skeletogenesis is the result of a complex series of molecular and cellular events that coordinate the morphogenetic process. Past and recent evidence strongly indicate that skeletal initiation and growth are strictly dependent on signals emanating from the oral ectodermal wall. As previously suggested, Orthopedia (Otp), a homeodomain-containing transcription factor specifically expressed in a small subset of oral ectoderm cells, might be implicated in this signalling pathway. In this study, we utilize three different strategies to address the issue of whether Otp is an upstream regulator of sketelogenesis. We describe the effects of microinjection of Otp mor…

Transcriptional Activationanimal structuresMorpholinoOrthopedia homeoboxMolecular Sequence DataEctodermNerve Tissue ProteinsBiologyFusion geneEctodermmedicineSkeletogenesisAnimalsAmino Acid SequenceSea urchin embryoTranscription factorMolecular BiologyMessenger RNAExtracellular Matrix ProteinsBone DevelopmentEmbryoCell BiologyMolecular biologyHedgehog signaling pathwayMorpholino oligonucleotidesCytoskeletal Proteinsmedicine.anatomical_structureProtein BiosynthesisSea Urchinsembryonic structuresHomeoboxDevelopmental BiologyDevelopmental Biology
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Targeting cancer stem cells and the tumor microenvironment

2015

Compelling evidence indicates that the survival and behavior of cancer stem cells (CSCs) are positively regulated by specific stimuli received from the tumor microenvironment, which dictates the maintenance of stemness, invasiveness, and protection against drug-induced apoptotic signals. CSCs are per se endowed with multiple treatment resistance capabilities, thus the eradication of CSC pools offers a precious strategy in achieving a long-term cancer remission. Numerous therapies, aimed at eradicating CSCs, have been elaborated such as: (i) selective targeting of CSCs, (ii) modulating their stemness and (iii) influencing the microenvironment. In this context, markers commonly exploited to i…

Tumor microenvironmentCyclopamineCancer therapymedicine.medical_treatmentCellular differentiationCancer stem cellWnt signaling pathwayContext (language use)BiologyStemness modulator drugCXCR4Targeted therapyTargeted therapychemistry.chemical_compoundchemistryTumor microenvironmentCancer stem cellmedicineCancer research
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Progression of colorectal cancers correlates with overexpression and loss of polarization of expression of the htid-1 tumor suppressor.

2007

Recently, we identified htid-1, the human counterpart of the Drosophila tumor suppressor gene lethal(2)tumorous imaginal discs [l(2)tid], as a direct molecular ligand of the adenomatous polyposis coli (APC) tumor suppressor. The gene encodes three cytosolic (Tid50, Tid48 and Tid46) and three mitochondrial (Tid43, Tid40 and Tid38) proteins. In the colorectal epithelium the cytosolic forms hTid50/hTid48 interact under physiological conditions with the N-terminal region of APC. This complex which associates with additional proteins such as Hsp70, Hsc70, Actin, Dvl and Axin defines a novel physiological state of APC unrelated to beta-catenin degradation. Here we show that the expression of the …

Tumor suppressor geneProtein familyAdenomatous polyposis coliColorectal cancerAntibodies NeoplasmRNA SplicingAdenomatous Polyposis Coli ProteinGeneticsmedicineHumansHSP70 Heat-Shock ProteinsRNA NeoplasmIntestinal MucosaDNA PrimersGeneticsOncogenebiologyTumor Suppressor ProteinsWnt signaling pathwayCell DifferentiationGeneral MedicineCell cycleHSP40 Heat-Shock Proteinsmedicine.diseaseGene Expression Regulation NeoplasticChaperone (protein)biology.proteinCancer researchDisease ProgressionColorectal NeoplasmsInternational journal of molecular medicine
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NOSIP, a novel modulator of endothelial nitric oxide synthase activity.

2001

Production of nitric oxide (NO) in endothelial cells is regulated by direct interactions of endothelial nitric oxide synthase (eNOS) with effector proteins such as Ca2+-calmodulin, by posttranslational modifications such as phosphorylation via protein kinase B, and by translocation of the enzyme from the plasma membrane caveolae to intracellular compartments. Reversible acylation of eNOS is thought to contribute to the intracellular trafficking of the enzyme; however, protein factor(s) that govern the translocation of the enzyme are still unknown. Here we have used the yeast two-hybrid system and identified a novel 34 kDa protein, termed NOSIP (eNOS interacting protein), which avidly binds …

Ubiquitin-Protein LigasesMolecular Sequence DataCHO CellsCaveolaeBiochemistryNitric oxideSubstrate Specificitychemistry.chemical_compoundEnosCaveolaeCricetinaeTwo-Hybrid System TechniquesGeneticsAnimalsHumansAmino Acid SequenceRNA MessengerMolecular BiologyProtein kinase BCalcimycinBinding SitesbiologyAkt/PKB signaling pathwayGene Expression Profilingbiology.organism_classificationImmunohistochemistryPrecipitin TestsTransport proteinCell biologyNitric oxide synthaseProtein TransportchemistryBiochemistrybiology.proteinEndothelium VascularNitric Oxide SynthaseCarrier ProteinsSequence AlignmentIntracellularBiotechnologyProtein BindingFASEB journal : official publication of the Federation of American Societies for Experimental Biology
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Vascular Endothelial Growth Factor, a Key Modulator of the Anti-Tumor Immune Response

2021

During tumor growth, angiogenesis is required to ensure oxygen and nutrient transport to the tumor. Vascular endothelial growth factor (VEGF) is the major inducer of angiogenesis and appears to be a key modulator of the anti-tumor immune response. Indeed, VEGF modulates innate and adaptive immune responses through direct interactions and indirectly by modulating protein expressions on endothelial cells or vascular permeability. The inhibition of the VEGF signaling pathway is clinically approved for the treatment of several cancers. Therapies targeting VEGF can modulate the tumor vasculature and the immune response. In this review, we discuss the roles of VEGF in the anti-tumor immune respon…

Vascular Endothelial Growth Factor AAngiogenesisAngiogenesis InhibitorsVascular permeabilityReviewAdaptive Immunityimmune responsechemistry.chemical_compoundangiogenesisNeoplasmsVEGF Signaling PathwayInducerBiology (General)SpectroscopyNeovascularization Pathologicvascular endothelial growth factorGeneral MedicineSorafenibComputer Science ApplicationsBevacizumabGene Expression Regulation NeoplasticVascular endothelial growth factorVascular endothelial growth factor AChemistrySignal TransductionQH301-705.5Recombinant Fusion ProteinsAntibodies Monoclonal HumanizedCatalysisCapillary PermeabilityInorganic ChemistryImmune systemmedicineHumansImmunologic FactorscancerPhysical and Theoretical ChemistryMolecular BiologyQD1-999Vascular Endothelial Growth Factor Receptor-1business.industryOrganic ChemistryEndothelial CellsCancermedicine.diseaseImmunity InnateReceptors Vascular Endothelial Growth FactorchemistryCancer researchbusinessInternational Journal of Molecular Sciences
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Sonic hedgehog promotes angiogenesis and osteogenesis in a coculture system consisting of primary osteoblasts and outgrowth endothelial cells.

2009

A number of previous studies documented the angiogenic potential of outgrowth endothelial cells in vitro and in vivo and provided evidence that therapeutic success could depend on coculture or coimplantation strategies. Thus, deeper insight into the molecular mechanisms underlying this pro-angiogenic effect of cocultures might provide new translational options for tissue engineering and regenerative medicine. One promising signaling pathway in bone repair involved in neoangiogenesis and bone formation is the sonic hedgehog (Shh) pathway. In this article, we focus on the effect of Shh on the formation of microvessel-like structures and osteoblastic differentiation in cocultures of primary os…

Vascular Endothelial Growth Factor Amedicine.medical_specialtyCyclopamineAngiogenesisCellular differentiationBiomedical EngineeringNeovascularization PhysiologicBioengineeringBiochemistryBiomaterialsAngiopoietin-2chemistry.chemical_compoundOsteogenesisInternal medicinemedicineAngiopoietin-1HumansHedgehog ProteinsRNA MessengerSonic hedgehogBone regenerationDNA PrimersOsteoblastsbiologyBase SequenceTissue EngineeringVeratrum AlkaloidsEndothelial CellsCell DifferentiationOriginal ArticlesHedgehog signaling pathwayCoculture TechniquesRecombinant ProteinsCell biologyCapillariesUp-RegulationVascular endothelial growth factorVascular endothelial growth factor AEndocrinologychemistrybiology.proteinSignal TransductionTissue engineering. Part A
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