Search results for "Skeletal"

showing 10 items of 3025 documents

Effect of different ergonomic supports on muscle activity of dentists during posterior composite restoration

2019

BackgroundThe aim of this study was to evaluate the effect of different ergonomic supports on the muscle activity of two trunk muscles while a group of dentists performed a common dental procedure on a phantom head, divided into three tasks.MethodsA one-way repeated measures study (ANOVA) was conducted on a group of 36 dentists. The middle trapezius and lumbar erector spinae muscles were measured with and without the use of different ergonomic supports (ergonomic stool, magnification lenses and both) using a portable surface electromyography (sEMG) device. Intraclass correlation coefficients (ICCs) and the absolute standard error of measurement (SEM) were used to establish the reliability o…

Anatomy and PhysiologyIntraclass correlationDentistslcsh:MedicineMagnificationElectromyographyMusculoskeletal disordersGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciences0302 clinical medicineLumbarErector spinae musclesMedicine0501 psychology and cognitive sciencesMuscle activity050107 human factorsOrthodonticsmedicine.diagnostic_testElectromyographyPhysical therapy modalitiesbusiness.industryGeneral Neurosciencelcsh:R05 social sciencesRepeated measures design030206 dentistryGeneral MedicineKinesiologyOrthopedicsDentistryPublic HealthErgonomicsGeneral Agricultural and Biological SciencesTrunk musclebusinessPeerJ
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Receptor Activator of NF-kB (RANK) Expression in Primary Tumors Associates with Bone Metastasis Occurrence in Breast Cancer Patients

2011

Background\ud Receptor activator of NFkB (RANK), its ligand (RANKL) and the decoy receptor of RANKL (osteoprotegerin, OPG) play a pivotal role in bone remodeling by regulating osteoclasts formation and activity. RANKL stimulates migration of RANK-expressing tumor cells in vitro, conversely inhibited by OPG.\ud \ud Materials and Methods\ud We examined mRNA expression levels of RANKL/RANK/OPG in a publicly available microarray dataset of 295 primary breast cancer patients. We next analyzed RANK expression by immunohistochemistry in an independent series of 93 primary breast cancer specimens and investigated a possible association with clinicopathological parameters, bone recurrence and surviv…

Anatomy and PhysiologyMicroarraysSettore MED/06 - Oncologia MedicaCancer TreatmentLigandsMetastasisBone remodelingMetastasisBasic Cancer ResearchBreast TumorsBone and Soft Tissue SarcomasNeoplasm MetastasisMusculoskeletal SystemOligonucleotide Array Sequence AnalysisMultidisciplinaryPredictive markerReceptor Activator of Nuclear Factor-kappa BQRBone metastasisMiddle AgedImmunohistochemistryGene Expression Regulation NeoplasticOncologyRANKLMedicineFemaleResearch Articlemusculoskeletal diseasesmedicine.medical_specialtyHistologyScienceBone NeoplasmsBreast NeoplasmsBiologyBreast cancerAntibody TherapySDG 3 - Good Health and Well-beingOsteoprotegerinInternal medicinemedicineHumansRNA MessengerBoneBiologyAgedBreast cancer bone metastasis RANK-RANKLRANK LigandOsteoprotegerinComputational BiologyCancers and NeoplasmsRANK Ligandmedicine.diseaseEndocrinologyCancer researchbiology.protein
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Toxicity assessment and biodegradation potential of water-soluble sludge containing 2,4,6-trinitrotoluen

2013

The water-soluble phase of trinitrotoluene-containing sludge (SLP) was characterized with regard to trinitrotoluene (TNT) concentration, ecotoxicity, and a model biodegradation experiment as evaluation criteria for further development of appropriate treatment technologies. SLP contained 67.8 mg TNT/l. The results of germination and root-elongation tests indicated that SLP had a species-specific phytotoxic effect. The results of a 21 day degradation experiment demonstrated TNT conversion to 4-amino-2,6-DNT and 2-amino-4,6-DNT, with a simultaneous reduction in the total concentration of nitroaromatics. Addition of inoculum stimulated the TNT degradation process. The presence of the sludge sol…

Aniline CompoundsEnvironmental EngineeringSewagebiodegradation; enzyme activity; indigenous microorganisms; phytotoxicity; solubility; TNTChemistryGerminationBiodegradationEcotoxicologymusculoskeletal systemPlant RootsBiodegradation EnvironmentalMicrobial population biologyGerminationEnvironmental chemistryVattenbehandlingToxicity TestsToxicityWater TreatmentTrinitrotolueneDegradation (geology)Water treatmentEcotoxicityTrinitrotolueneWater Science and Technology
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Glycogen synthase 2 is a novel target gene of peroxisome proliferator-activated receptors.

2007

International audience; Glycogen synthase 2 (Gys-2) is the ratelimiting enzyme in the storage of glycogen in liver and adipose tissue, yet little is known about regulation of Gys-2 transcription. The peroxisome proliferator-activated receptors (PPARs) are transcription factors involved in the regulation of lipid and glucose metabolism and might be hypothesized to govern glycogen synthesis as well. Here, we show that Gys-2 is a direct target gene of PPARalpha, PPARbeta/delta and PPARgamma. Expression of Gys-2 is significantly reduced in adipose tissue of PPARalpha-/-, PPARbeta/delta-/- and PPARgamma+/- mice. Furthermore, synthetic PPARbeta/delta, and gamma agonists markedly up-regulate Gys-2…

Animals; Chromatin/ultrastructure; DNA Primers; Gene Expression Regulation Enzymologic; Glycogen Synthase/genetics; Hepatocytes/enzymology; Hepatocytes/physiology; Mice; Mice Knockout; Peroxisome Proliferator-Activated Receptors/deficiency; Peroxisome Proliferator-Activated Receptors/genetics; Polymerase Chain Reaction; RNA/genetics; RNA/isolation & purification; Rats; Transcription GeneticTranscription GeneticPeroxisome proliferator-activated receptorMESH : HepatocytesPPREPolymerase Chain Reactionadipose-tissuePPARMESH: HepatocytesMice0302 clinical medicineMESH: Animals610 Medicine & healthchemistry.chemical_classificationRegulation of gene expression0303 health sciencesGlycogenglycogen-synthaseChromatinGlycogen Synthase030220 oncology & carcinogenesisMESH : DNA PrimersmicroarrayMESH: DNA Primersmedicine.medical_specialtyHealth aging / healthy living [IGMD 5]fatty-acid oxidationliverGene Expression Regulation EnzymologicMESH: Chromatin03 medical and health sciencesskeletal-muscleGlycogen synthaseMolecular Biology[ SDV.BBM ] Life Sciences [q-bio]/Biochemistry Molecular BiologyHNF4αVLAGPharmacologybeta/deltaMESH: Polymerase Chain Reactionresponse elementsMESH : Peroxisome Proliferator-Activated ReceptorsEndocrinologychemistryMicrobial pathogenesis and host defense [UMCN 4.1]Response elementPeroxisome Proliferator-Activated ReceptorsAdipose tissueMESH: Peroxisome Proliferator-Activated Receptorsin-vivoMESH: Mice KnockoutTransactivationchemistry.chemical_compoundVoeding Metabolisme en GenomicaMESH : RNAMESH : Polymerase Chain ReactionMice KnockoutMESH : ChromatinMESH : RatsMESH: Gene Expression Regulation EnzymologicMetabolism and Genomicsadipose tissueMetabolisme en GenomicaMolecular MedicineNutrition Metabolism and GenomicsMESH : Glycogen SynthaseResearch ArticleMESH: Ratsglycogen synthase 2610 Medicine & healthBiologyMESH : Gene Expression Regulation EnzymologicCellular and Molecular NeuroscienceVoedingMESH: RNAInternal medicineMESH : MicemedicineAnimals[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular BiologyTranscription factorMESH: Micealpha ppar-alpha030304 developmental biologyNutritionDNA PrimersMESH: Glycogen SynthaseMESH: Transcription GeneticMESH : Transcription GeneticCell BiologyRatsgene transcriptionbiology.proteinHepatocytesRNAMESH : Mice KnockoutgammaMESH : Animalsmetabolism
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Interstitial Lung Disease during ANCA-Associated Vasculitis: A Poor-Prognosis Factor

2018

IF 7.873; International audience

Anti-fibrosing agents[SDV.MHEP.RSOA] Life Sciences [q-bio]/Human health and pathology/Rhumatology and musculoskeletal system[SDV.MHEP.RSOA]Life Sciences [q-bio]/Human health and pathology/Rhumatology and musculoskeletal system[SDV]Life Sciences [q-bio]ANCA-associated vasculitis (AAV)Interstitial lung disease (ILD)ComputingMilieux_MISCELLANEOUS
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Moderate exercise is an antioxidant: Upregulation of antioxidant genes by training

2006

Exercise causes oxidative stress only when exhaustive. Strenuous exercise causes oxidation of glutathione, release of cytosolic enzymes, and other signs of cell damage. However, there is increasing evidence that reactive oxygen species (ROS) not only are toxic but also play an important role in cell signaling and in the regulation of gene expression. Xanthine oxidase is involved in the generation of superoxide associated with exhaustive exercise. Allopurinol (an inhibitor of this enzyme) prevents muscle damage after exhaustive exercise, but also modifies cell signaling pathways associated with both moderate and exhaustive exercise in rats and humans. In gastrocnemius muscle from rats, exerc…

AntioxidantFree Radicalsmedicine.medical_treatmentBiologyPharmacologymedicine.disease_causeModels BiologicalBiochemistryAntioxidantsGene Expression Regulation EnzymologicSuperoxide dismutasechemistry.chemical_compoundDownregulation and upregulationPhysical Conditioning AnimalPhysiology (medical)medicineAnimalsHumansMuscle SkeletalXanthine oxidaseExerciseCell damagechemistry.chemical_classificationReactive oxygen speciesSuperoxidemedicine.diseaseAdaptation PhysiologicalUp-RegulationchemistryBiochemistrybiology.proteinOxidative stressFree Radical Biology and Medicine
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Glutathione metabolism in skeletal muscle derived cells of the L6 line

1993

Skeletal muscle derived L6 myoblasts possess a considerably high resting total glutathione (TGSH) pool. Exposure to L-buthionine-[S,R]-sulphoximine resulted in a 90% depletion of the intracellular TGSH pool. All the key enzymes of glutathione metabolism, especially glutathione S-transferase, were observed to be considerably active in the undifferentiated cells. Se-dependent glutathione peroxidase activity appeared to account for most of the total GSH peroxidase activity of the cells. A significant contribution of gamma-glutamyl transpeptidase-independent (5 mM acivicin insensitive) mechanism to the extracellular GSH uptake capacity of the muscle cells was evident. Efflux of oxidized glutath…

AntioxidantGPX3AntimetabolitesPhysiologymedicine.medical_treatmentGlutathione reductaseBiologyCell Linechemistry.chemical_compoundtert-ButylhydroperoxideMethionine SulfoximinemedicineAnimalsMyocyteInhibinsButhionine SulfoximineAcivicinGlutathione TransferaseMusclesSkeletal muscleGlutathioneMetabolismGlutathioneActivinsPeroxidesRatsmedicine.anatomical_structureBiochemistrychemistryEnergy MetabolismActa Physiologica Scandinavica
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mHealth for the Monitoring of Brace Compliance and Wellbeing in Adolescents with Idiopathic Scoliosis: Study Protocol for a Feasibility Study

2021

Escoliosis idiopática del adolescente; Aplicación móvil; Eficacia del tratamiento Escoliosi idiopàtica de l'adolescent; Aplicació mòbil; Eficàcia del tractament Adolescent idiopathic scoliosis; Mobile application; Treatment effectiveness Attempts to optimize monitoring of brace adherence prescribed to adolescents with idiopathic scoliosis (IS) have generally relied on sensors. Sensors, however, are intrusive and do not allow the assessment of psychological and physical consequences of brace use that might underlie poor adherence. Mobile applications have emerged as alternatives to monitor brace compliance. However, the feasibility and utility of these app-based systems to assess key psychol…

Aparells ortopèdicsFerides i lesions TractamentHealth Toxicology and Mutagenesis:Health Services Administration::Patient Care Management::Delivery of Health Care::Telemedicine [HEALTH CARE]Adolescents0302 clinical medicineMedicine030212 general & internal medicinemHealthEscoliosiRbrace adherenceTelemedicinaInformation and Communication TechnologiesTelemedicineTest (assessment)DistressScoliosistreatment effectiveness:Musculoskeletal Diseases::Bone Diseases::Spinal Diseases::Spinal Curvatures::Scoliosis [DISEASES]adolescent idiopathic scoliosisMedicinemusculoskeletal diseasesmedicine.medical_specialtyAdolescentTelefonia mòbilArticle:administración de los servicios de salud::gestión de la atención al paciente::prestación sanitaria::telemedicina [ATENCIÓN DE SALUD]03 medical and health sciencesQuality of life (healthcare)HumansComunicació i tecnologiaProtocol (science)adolescent idiopathic scoliosis; Information and Communication Technologies; mobile application; brace adherence; treatment effectivenessBracesbusiness.industry:enfermedades musculoesqueléticas::enfermedades óseas::enfermedades de la columna vertebral::desviaciones de la columna vertebral::escoliosis [ENFERMEDADES]:equipos y suministros::equipo quirúrgico::equipos ortopédicos::aparatos ortésicos::tirantes [TÉCNICAS Y EQUIPOS ANALÍTICOS DIAGNÓSTICOS Y TERAPÉUTICOS]Public Health Environmental and Occupational HealthUsabilitymobile applicationBraceMood:Equipment and Supplies::Surgical Equipment::Orthopedic Equipment::Orthotic Devices::Braces [ANALYTICAL DIAGNOSTIC AND THERAPEUTIC TECHNIQUES AND EQUIPMENT]Physical therapyQuality of LifeFeasibility StudiesPatient Compliancebusiness030217 neurology & neurosurgeryInternational Journal of Environmental Research and Public Health
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Lack of Dystrophin Affects Bronchial Epithelium in mdx Mice

2016

Mild exercise training may positively affect the course of Duchenne Muscular Dystrophy (DMD). Training causes mild bronchial epithelial injury in both humans and mice, but no study assessed the effects of exercise in mdx mice, a well known model of DMD. The airway epithelium was examined in mdx (C57BL/10ScSn-Dmdmdx) mice, and in wild type (WT, C57BL/10ScSc) mice either under sedentary conditions (mdx-SD, WT-SD) or during mild exercise training (mdx-EX, WT-EX). At baseline, and after 30 and 45 days of training (5 d/wk for 6 weeks), epithelial morphology and markers of regeneration, apoptosis, and cellular stress were assessed. The number of goblet cells in bronchial epithelium was much lower…

Apoptosis; Chaperonin 60 (HSP60); Dystrophin; Goblet cells; Proliferation; Clinical Biochemistry; Cell Biology; PhysiologyPhysiologyProliferationClinical BiochemistryGene ExpressionApoptosiBronchiCell BiologyEpitheliumMice Inbred C57BLMuscular Dystrophy DuchenneDystrophinMice Inbred mdxAnimalsRegenerationChaperonin 60 (HSP60)Muscle SkeletalGoblet cell
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Infantile intranuclear rod myopathy.

1997

This report concerns three unrelated floppy infants, two girls and one boy, each biopsied at the age of 1 month. They were hypotonic since birth and required artificial ventilation. The two girls died at the ages of 4 and 3½ months, respectively, the boy is still alive at the age of 2 years, but requires assisted ventilation. Each of the three infants showed, by muscle biopsy, abundant intranuclear rods, the boy and one girl also had sarcoplasmic rods, which were not present in the other girl's muscle. Absence of sarcoplasmic rods, but the presence of intranuclear rods could also be documented in her autopsied muscle. Using an antibody against α-actinin, immunoelectron microscopy showed re…

Artificial ventilationMalePediatricsmedicine.medical_specialtygenetic structuresmedicine.medical_treatmentBiopsyMyosinsMyopathies Nemaline03 medical and health sciences0302 clinical medicineFatal Outcome030225 pediatricsmedicineHumansEndotheliumFloppy InfantMicroscopy ImmunoelectronMuscle SkeletalCell Nucleusbusiness.industryInfant NewbornPediatrics Perinatology and Child HealthFemalesense organsNeurology (clinical)Intranuclear Rod Myopathybusiness030217 neurology & neurosurgeryJournal of child neurology
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