Search results for "Smooth Muscle"

showing 10 items of 156 documents

PCSK9 Confers Inflammatory Properties to Extracellular Vesicles Released by Vascular Smooth Muscle Cells

2022

Vascular smooth muscle cells (VSMCs) are key participants in both early- and late-stage atherosclerosis and influence neighbouring cells possibly by means of bioactive molecules, some of which are packed into extracellular vesicles (EVs). Proprotein convertase subtilisin/kexin type 9 (PCSK9) is expressed and secreted by VSMCs. This study aimed to unravel the role of PCSK9 on VSMCs-derived EVs in terms of content and functionality. EVs were isolated from human VSMCs overexpressing human PCSK9 (VSMCPCSK9-EVs) and tested on endothelial cells, monocytes, macrophages and in a model of zebrafish embryos. Compared to EVs released from wild-type VSMCs, VSMCPCSK9-EVs caused a rise in the expression …

Myocytes Smooth MusclePCSK9; atherosclerosis; extracellular vesicles; inflammation; vascular smooth muscle cellsPCSK9; atherosclerosis; extracellular vesicles; inflammation; vascular smooth muscle cells.Muscle Smooth VascularCatalysisPCSK9Inorganic ChemistryExtracellular VesiclesSettore BIO/13 - Biologia ApplicataSettore MED/44 - Medicina del Lavorovascular smooth muscle cellsAnimalsHumansPhysical and Theoretical ChemistryMolecular BiologyZebrafishSpectroscopySettore MED/04 - Patologia GeneraleOrganic ChemistryEndothelial CellsGeneral MedicineComputer Science Applicationsinflammationextracellular vesicles; PCSK9; atherosclerosis; inflammation; vascular smooth muscle cellsSettore BIO/14 - FarmacologiaatherosclerosisProprotein Convertase 9International Journal of Molecular Sciences
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Dexamethasone dipropionate loaded nanoparticles of α-elastin-g-PLGA for potential treatment of restenosis.

2013

A graft copolymer of α-elastin with poly(lactic-co-glycolic) acid (PLGA) has been synthesized and successfully employed to produce nanoparticles. Exploiting the known biological activity of α-elastin to promote the maintenance of smooth muscle cells (SMCs) contractile phenotype and the antiproliferative effect of glucocorticoids, the aim of this research was to produce drug-loaded nanoparticles suitable for potential treatment of restenosis. In particular, nanoparticles of α-elastin-g-PLGA with a mean size of 200 nm have been produced and loaded with dexamethasone dipropionate (10% w/w), chosen as a model drug that inhibits proliferation of vascular SMCs. These nanoparticles are able to pro…

Myocytes Smooth MusclePharmaceutical ScienceDexamethasoneMuscle Smooth VascularCoronary Restenosischemistry.chemical_compoundPolylactic Acid-Polyglycolic Acid CopolymerDrug DiscoveryMyocyteAnimalsHumansLactic AcidParticle SizeCells CulturedCell ProliferationDrug CarriersbiologyCell growthElastaseBiological activityCell DifferentiationElastinBlotPLGAchemistryBiochemistryBiophysicsbiology.proteinMolecular MedicineNanoparticlesCattleDrug carrierElastinPolyglycolic AcidMolecular pharmaceutics
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Relaxant effects of flavonoids on the mouse isolated stomach: structure-activity relationships.

2008

Flavonoids are a large heterogeneous group of benzo-gamma-pyrone derivatives, which are abundantly present in our diet. In this study we investigated the effects of six flavonoids (apigenin, genistein, quercetin, rutin, naringenin and catechin) on the gastric tone in mouse isolated stomach. The mechanical activity was recorded as changes of intraluminal pressure. All flavonoids tested produced a concentration-dependent relaxation, which was reversible after washout. The relative order of potency of the flavonoids was apigenin> or =genistein>quercetin>naringenin> or =rutin>catechin. Analysis of the chemical structure showed that the relaxant activity was progressively diminished by the prese…

NaringeninMalePotassium ChannelsFlavonoidGenisteinAction PotentialsIn Vitro TechniquesNitric OxideSettore BIO/09 - FisiologiaNitric oxidechemistry.chemical_compoundRutinMiceStructure-Activity RelationshipFlavonoids gastric relaxation smooth muscle potassium channels nitric oxideAnimalsheterocyclic compoundsPharmacologychemistry.chemical_classificationFlavonoidsNeuronsDose-Response Relationship DrugStomachfood and beveragesCatechinMuscle SmoothMice Inbred C57BLchemistryBiochemistryGastric MucosaApigeninProstaglandinsQuercetinMuscle ContractionEuropean journal of pharmacology
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Expression of the proto-oncogene c-myc in human stenotic aortocoronary bypass grafts.

2002

Summary Proliferation and differentiation of vascular smooth muscle cells (VSMC) are central events in vascular pathobiology and play a major role in the development of stenotic and restenotic lesions [ 15, 27 ] . The proto-oncogene c-myc and other early cell cycle-regulating genes have been implicated in the induction of cell proliferation and differentiation under diverse pathophysiological conditions [ 11, 13 ] . In the present study we analyzed c-myc mRNAexpression by indirect nonradioactive in situ hybridization technique (NISH) in human stenotic venous bypass grafts (n = 32) retrieved during re-do operations of coronary artery disease and compared the results with 28 native veins (ven…

NeointimaAdultMalePathologymedicine.medical_specialtyVascular smooth muscleCellIn situ hybridizationBiologyProto-Oncogene MasPathology and Forensic MedicineCoronary artery diseaseProto-Oncogene Proteins c-mycmedicineHumansSaphenous VeinRNA MessengerCoronary Artery BypassIn Situ HybridizationAgedOncogeneGraft Occlusion VascularCell BiologyMiddle Agedmedicine.diseasePathophysiologyBlood Vessel Prosthesismedicine.anatomical_structureVasa vasorumFemalePathology, research and practice
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Short-term atorvastatin treatment does not modify neointimal morphology but reduces MMP-2 expression in normocholesterolemic rabbit stented arteries.

2006

The aim of our study was to explore some potential pleiotropic effects of atorvastatin, after stenting in the iliac arteries of normocholesterolemic rabbits. On day 0, 27 rabbits underwent stent implantation and were randomized into either the control group (standard chow, CTRL, n = 15) or the atorvastatin group (10 mg/kg/d per os, Ator, n = 12). On day 30, the stented arteries were harvested for histomorphometry and neointimal analysis [macrophages, matrix metalloproteinases (MMP-2), tissue inhibitor of metalloproteinase-2, vascular smooth muscle cells, and collagen]. Atorvastatin did not induce significant histomorphometric and inflammatory modifications but reduced neointimal expression …

NeointimaMalemedicine.medical_specialtyStatinVascular smooth musclemedicine.drug_classAtorvastatinHypercholesterolemiaUrologyMatrix metalloproteinaseIliac ArteryMuscle Smooth VascularRestenosisInternal medicinemedicineAtorvastatinAnimalsPyrrolesPharmacologyTissue Inhibitor of Metalloproteinase-2Cellular densityChemistrymedicine.diseaseImmunohistochemistryHeptanoic AcidsCardiologyMatrix Metalloproteinase 2StentsStatin therapyRabbitsHydroxymethylglutaryl-CoA Reductase InhibitorsCardiology and Cardiovascular MedicineTunica Intimamedicine.drugJournal of cardiovascular pharmacology
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Rapamycin vermindert die Neointimaausbildung nach vaskulärer Schädigung

2003

Background: Proliferation and migration of vascular smooth muscle cells (SMCs) mark the key processes in the development of bypass graft disease and during neointima formation in restenosis after angioplasty. Growth factors are potent SMC mitogens as they are involved in SMC proliferation and in extracellular matrix (ECM) synthesis. Based on these premises, we examined the effect of the proliferation inhibitor rapamycin in human SMC culture and in a rabbit vascular injury model. Materials and methods: Injection of rapamycin or its vehicle was performed with an infusion-balloon catheter directly into the vessel wall during vascular injury. The intima/media ratio was determined histologicall…

NeointimaPathologymedicine.medical_specialtyVascular smooth musclebiologybusiness.industryVascular diseaseCell growthVimentinPharmacologymedicine.diseaseExtracellular matrixRestenosisSirolimuscardiovascular systembiology.proteinMedicineCardiology and Cardiovascular Medicinebusinessmedicine.drugVasa
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Involvement of PKC and NF-κB in Nitric Oxide Induced Apoptosis in Human Coronary Artery Smooth Muscle Cells

2001

Apoptosis of vascular smooth muscle cells is critically involved in progression of atherosclerosis and may prevent intimal hyperplasia in restenosis and vascular remodeling. Nitric oxide (NO) is known to induce apoptosis, but the signaling pathways still remain unclear. We investigated p53 accumulation, protein kinase C (PKC) activation and nuclear transcription factor (NF-kappaB) binding activity as possible signaling mechanisms of NO-induced apoptosis. Apoptosis was induced dose-dependently with the NO-donors sodiumnitroprusside (SNP: 232+/-48%) and SIN-1 (241+/-90% of actinomycin D induced apoptosis; means +/- SEM, *por =0.05 vs. control) in HSMC. Inhibition of PKC significantly attenuat…

Nitroprussidemedicine.medical_specialtyVascular smooth muscleIntimal hyperplasiaPhysiologyApoptosisDNA FragmentationNaphthalenesNitric OxideMuscle Smooth VascularNitric oxidechemistry.chemical_compoundNF-KappaB Inhibitor alphaRestenosisInternal medicinemedicineHumansNitric Oxide DonorsEnzyme InhibitorsCells CulturedProtein Kinase CProtein kinase CCell Nucleusbusiness.industryNF-kappa BNF-κBStaurosporinemedicine.diseaseCoronary VesselsDNA-Binding Proteinsmedicine.anatomical_structurechemistryApoptosisMolsidomineCancer researchCardiologyI-kappa B ProteinsTumor Suppressor Protein p53businessArteryCellular Physiology and Biochemistry
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Hamartoma of the triceps surae muscle.

1997

A 9-year-old, otherwise healthy girl presented with a 5-year history of pain in her right calf with retarded growth and development of an equinus contracture of her right leg. Magnetic resonance imaging showed an irregular mass with heterogeneous enhancement after contrast in her right triceps surae muscles, especially the soleus. Histological studies of this triceps surae muscle tissue revealed a haphazard distribution of adipose and connective tissue, striated and smooth muscle cells, vessels and lymphoid follicles, as well as nerve bundles which, together, were considered components of a hamartoma.

Pathologymedicine.medical_specialtyHamartomaConnective tissueAdipose tissuePainRight triceps suraePathology and Forensic MedicineDiagnosis DifferentialCellular and Molecular NeuroscienceRetarded growthSmooth muscleTriceps surae musclemedicineHamartomaHumansChildMuscle SkeletalLegmedicine.diagnostic_testbusiness.industryMagnetic resonance imagingAnatomymedicine.diseaseMagnetic Resonance Imagingmedicine.anatomical_structureFemaleNeurology (clinical)businessActa neuropathologica
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Current understanding of somatic stem cells in leiomyoma formation.

2014

Objective To provide a detailed summary of current scientific knowledge of somatic stem cells (SSCs) in murine and human myometrium and their putative implication in leiomyoma formation, as well as to establish new therapeutic options. Design Pubmed and Scholar One manuscripts were used to identify the most relevant studies on SSCs and their implications in human myometrium and leiomyomas. Setting University research laboratory–affiliated infertility clinic. Patient(s) Not applicable. Intervention(s) Not applicable. Main Outcome Measure(s) Not applicable. Result(s) Despite numerous publications on SSCs, it was not until 2007 that scientific evidence based on the use of 5-bromo-2′-deoxyuridi…

Pathologymedicine.medical_specialtyMyocytes Smooth MuscleBioinformaticsBenign tumorSide populationMedicineHumansEpigeneticsSide-Population CellsUterine leiomyomaLeiomyomabusiness.industryMyometriumObstetrics and Gynecologymedicine.diseaseLeiomyomaCell Transformation NeoplasticReproductive MedicineUterine NeoplasmsMyometriumNeoplastic Stem CellsFemalebusinessAdult stem cellHormoneFertility and sterility
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Therapeutic targets for overactive bladder other than smooth muscle

2015

For a long time, our concepts of regulation of urinary bladder function in health and disease as well as of the target structures of therapeutics have focused on detrusor smooth muscle cells. However, other structures including urothelium, afferent nerves and bladder blood vessels may also be important in pathophysiology and its treatment.Based on a selective review of literature, we discuss the role of urothelium, afferent nerve fibers and bladder blood vessels in bladder pathophysiology and as targets for treatment.There is solid evidence now that multiple anatomical structures within the urinary bladder contribute to the regulation of its function and hence may be targets for established…

Pathologymedicine.medical_specialtyMyocytes Smooth MuscleUrinary BladderClinical BiochemistryAnatomical structuresDiseaseurologic and male genital diseasesNerve FibersSmooth muscleAfferentDrug DiscoverymedicineAnimalsHumansMolecular Targeted TherapyUrotheliumPharmacologyAfferent PathwaysUrinary bladderUrinary Bladder Overactivebusiness.industrymedicine.diseasefemale genital diseases and pregnancy complicationsPathophysiologymedicine.anatomical_structureOveractive bladderDrug DesignMolecular MedicineUrotheliumbusinessNeuroscienceExpert Opinion on Therapeutic Targets
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