Search results for "Smooth"

showing 10 items of 710 documents

Complex I dysfunction and tolerance to nitroglycerin: an approach based on mitochondrial-targeted antioxidants.

2006

Nitroglycerin (GTN) tolerance was induced in vivo (rats) and in vitro (rat and human vessels). Electrochemical detection revealed that the incubation dose of GTN (5×10 −6 mol/L) did not release NO or modify O 2 consumption when administered acutely. However, development of tolerance produced a decrease in both mitochondrial O 2 consumption and the K m for O 2 in animal and human vessels and endothelial cells in a noncompetitive action. GTN tolerance has been associated with impairment of GTN biotransformation through inhibition of aldehyde dehydrogenase (ALDH)-2, and with uncoupling of mitochondrial respiration. Feeding rats with mitochondrial-targeted antioxidants (mitoquinone [MQ]) and i…

MaleantioxidantAntioxidantPhysiologyUbiquinonemedicine.medical_treatmentMuscle RelaxationVasodilator AgentsAldehyde dehydrogenasePharmacologyMitochondrionmedicine.disease_causeAntioxidantsMuscle Smooth VascularRats Sprague-Dawleychemistry.chemical_compoundNitroglycerinDrug toleranceoxidative stressCyclic GMPchemistry.chemical_classificationbiologyAldehyde Dehydrogenase MitochondrialDrug ToleranceGlutathioneMitochondriamitochondriaBiochemistrycardiovascular systemCardiology and Cardiovascular Medicinecirculatory and respiratory physiologyMuscle ContractionendotheliumIn Vitro TechniquesMitochondrial ProteinsOrganophosphorus CompoundsOxygen ConsumptionRespirationmedicineAnimalsHumansReactive oxygen speciesElectron Transport Complex IDose-Response Relationship DrugEndothelial CellsGlutathioneAldehyde DehydrogenasenitroglycerinRatsOxidative Stresschemistrybiology.proteinReactive Oxygen SpeciesOxidative stressCirculation research
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The Janus face of chlorogenic acid on vascular reactivity: A study on rat isolated vessels

2016

Abstract Background Chlorogenic acid (CGA), the main polyphenol contained in coffee, is a major contributor to dietary polyphenol intake. Few studies reported its anti-hypertensive properties but the mechanisms are still indefinite. Purpose The present study assessed the direct effect of CGA in endothelium denuded or intact aortic rings from male Wistar rats and the mechanisms involved. Methods/Results CGA induced a direct endothelium-dependent relaxation that was significantly reduced by L-NAME (10 −4  M), indomethacin (10 −5  M) and combination of apamin (10 −7  M) and charybdotoxin (10 −7  M). Incubation of rings with CGA induced a dual effect on agonist-induced vasorelaxation. At 10 −6 …

Maleendocrine systemCharybdotoxinEndotheliumPharmaceutical ScienceVasodilation030204 cardiovascular system & hematologyPharmacologyApaminMuscle Smooth Vascular03 medical and health scienceschemistry.chemical_compound0302 clinical medicineDrug DiscoverymedicineAnimals[CHIM]Chemical SciencesRats WistarPhenylephrineAntihypertensive AgentsComputingMilieux_MISCELLANEOUSPharmacology[SDV.SP]Life Sciences [q-bio]/Pharmaceutical sciencesRatsVasodilationmedicine.anatomical_structureComplementary and alternative medicineBiochemistrychemistry030220 oncology & carcinogenesisHypertensionMolecular MedicineSodium nitroprussideEndothelium Vascularmedicine.symptomChlorogenic AcidVasoconstrictionAcetylcholinemedicine.drug
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Role of cholinergic neurons in the motor effects of glucagon-like peptide-2 in mouse colon

2010

Glucagon-like peptide-2 (GLP-2) reduces mouse gastric tone and small intestine transit, but its action on large intestine motility is still unknown. The purposes of the present study were 1) to examine the influence of GLP-2 on spontaneous mechanical activity and on neurally evoked responses, by recording intraluminal pressure from mouse isolated colonic segments; 2) to characterize GLP-2 mechanism of action; and 3) to determine the distribution of GLP-2 receptor (GLP-2R) in the mouse colonic muscle coat by immunohistochemistry. Exogenous GLP-2 (0.1–300 nM) induced a concentration-dependent reduction of the spontaneous mechanical activity, which was abolished by the desensitization of GLP-…

Maleendocrine systemmedicine.medical_specialtyCarbacholColonPhysiologymedicine.drug_classBlotting WesternBiologyApaminSettore BIO/09 - FisiologiaMicechemistry.chemical_compoundenteric nervous systemcolonic motilityPhysiology (medical)Internal medicineGlucagon-Like Peptide 2Receptors GlucagonmedicineAnimalsCholinergic neuronNeuronsAnalysis of VarianceDose-Response Relationship DrugHepatologydigestive oral and skin physiologyGastroenterologyMuscle Smoothgastrointestinal hormoneMotor neuronReceptor antagonistImmunohistochemistryCholine acetyltransferaseElectric StimulationacetylcholineEndocrinologymedicine.anatomical_structurechemistryGlucagon-Like Peptide-2 ReceptorCholinergicGastrointestinal Motilityhormones hormone substitutes and hormone antagonistsAcetylcholineMuscle Contractionmedicine.drugAmerican Journal of Physiology-Gastrointestinal and Liver Physiology
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Interplay between PACAP and NO in mouse ileum

2003

Abstract We investigated the possibility that pituitary adenylate cyclase activating peptide (PACAP) has a role in the control of contractility in the mouse ileum. PACAP-(1-27) produced tetrodotoxin (TTX)-insensitive, concentration-dependent reduction of the amplitude of the spontaneous contractions of longitudinal muscle up to their complete disappearance. This effect was inhibited by PACAP-(6-38), PACAP receptor antagonist, and by apamin, blocker of small-conductance Ca2+-activated K+-channels. Nω-nitro- l -arginine methyl ester (L-NAME), nitric oxide (NO) synthase inhibitor, reduced the PACAP-inhibitory response, and the joint application of apamin plus L-NAME produced additive effects. …

Maleendocrine systemmedicine.medical_specialtyMuscle RelaxationMouse ileumStimulationIn Vitro TechniquesApaminSettore BIO/09 - FisiologiaContractilityMiceCellular and Molecular Neurosciencechemistry.chemical_compoundSmooth muscleIleumInternal medicinemedicineAnimalsMyocyteNitric Oxide DonorsPharmacologyDose-Response Relationship DrugNeuropeptidesNitric oxideSmooth muscle contractionMice Inbred C57BLPituitary adenylate cyclase-activating peptideEndocrinologyApaminchemistryTetrodotoxinPituitary Adenylate Cyclase-Activating PolypeptideSodium nitroprussidePACAP (pituitary adenylate cyclase activating peptide)hormones hormone substitutes and hormone antagonistsmedicine.drugNeuropharmacology
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Antagonism of calcium by zinc in guinea-pig isolated taenia caeci and trachealis muscle

1989

1 In guinea-pig isolated taenia caeci and trachealis bathed in a K+-rich, Ca2+-free medium, CaCl2 (0.01-10 mM) produced a concentration-dependent contraction. Zn2+ (0.01-1 mM), Cd2+ (0.01-1 mM), verapamil (0.01-100 microM) and trifluoperazine (1-100 microM) were effective antagonists of CaCl2-induced responses. 2 Zn2+ and Cd2+ in concentrations from 0.01 to 1 mM were without effect on the tone of taenia and trachea in normal Tris solution. Conversely, Zn2+ and Cd2+, in concentrations of 1 mM, caused contraction of these tissues in a K+-rich, Ca2+-free medium. Zn2+ (1 mM)-induced contractions of taenia and trachea were completely inhibited by verapamil (10 microM). 3 In taenia and trachea sk…

Maleinorganic chemicalsmedicine.medical_specialtyContraction (grammar)Guinea Pigschemistry.chemical_elementTrifluoperazineIn Vitro TechniquesCalciumGuinea pigInternal medicinemedicineAnimalsCecumPharmacologyChemistryCell MembraneMuscle SmoothAcetylcholineTrifluoperazineTracheaZincEndocrinologyVerapamilTrachealis muscleVerapamilCalciumFemalemedicine.symptomAcetylcholineCadmiumResearch Articlemedicine.drugMuscle contraction
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Effects of alkylxanthines on contractility of diaphragm fibres isolated from normal and sensitized guinea-pigs.

1993

Abstract This study investigates the effects of alkylxanthines on twitch tension generated by electrical stimulation (supramaximal pulses, 0·2 ms duration, 1 Hz) of diaphragm muscle fibres isolated from normal and actively-sensitized guinea-pigs. Caffeine, theophylline and theobromine increased, in a concentration-dependent manner (50–500 μm), twitch tension in normal and sensitized diaphragm. Caffeine (500 μm) enhanced contractility to a greater extent than theophylline or theobromine. Twitch potentiation by caffeine (500 μm) was significantly greater in sensitized diaphragm. Verapamil (0·1–100 μm) did not alter twitch contractions in the absence or presence of alkylxanthines in normal or …

Malemedicine.medical_specialtyAdenosineDiaphragmGuinea PigsPharmaceutical ScienceIn Vitro TechniquesDantroleneDantroleneContractilitychemistry.chemical_compoundTheophyllineInternal medicineCaffeinemedicineAnimalsTheophyllineRespiratory systemRats WistarPharmacologyMuscle SmoothSerum Albumin Bovinemusculoskeletal systemElectric StimulationDiaphragm (structural system)Bronchodilator AgentsCulture MediaRatsEndocrinologychemistryVerapamilXanthinesEnprofyllineTheobromineCalciumFemaleImmunizationmedicine.symptomCaffeineExtracellular Spacemedicine.drugMuscle contractionMuscle ContractionThe Journal of pharmacy and pharmacology
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A1 receptors mediate adenosine inhibitory effects in mouse ileum via activation of potassium channels.

2008

Abstract Aims We investigated the effects induced by exogenous adenosine on the spontaneous contractile activity of the longitudinal muscle of a mouse ileum, the receptor subtypes activated, the involvement of enteric nerves and whether opening of K + channels was a downstream event leading to the observed effects. Main methods Mechanical responses of the mouse ileal longitudinal muscle to adenosine were examined in vitro as changes in isometric tension. Key findings Adenosine caused a concentration-dependent reduction of the spontaneous contraction amplitude of the ileal longitudinal muscle up to its complete disappearance. This effect induced was markedly reduced by an A 1 receptor antago…

Malemedicine.medical_specialtyAdenosinePotassium ChannelsAdenosine A2 Receptor AgonistsMouse ileumBlotting WesternAdenosine A3 Receptor AntagonistsAdenosine A1 Receptor AntagonistsApaminSettore BIO/09 - FisiologiaGeneral Biochemistry Genetics and Molecular BiologyAdenosine A1 receptorchemistry.chemical_compoundMiceAdenosine A3 Receptor AgonistsIleumInternal medicineNeural PathwaysmedicinePotassium Channel BlockersPurinergic P1 Receptor AgonistsAnimalsGeneral Pharmacology Toxicology and PharmaceuticsP1 purinoceptorDose-Response Relationship DrugChemistryReceptor Adenosine A1Mechanical activityMuscle SmoothGeneral MedicinePurinergic signallingIberiotoxinAdenosine A3 receptorAdenosineAdenosine receptorAdenosine A1 Receptor AgonistsAdenosine A2 Receptor AntagonistsMice Inbred C57BLEndocrinologyPurinergic P1 Receptor AntagonistsAdenosine A2B receptormedicine.drugMuscle ContractionLife sciences
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The effect of S-(+)-boldine on the α1-adrenoceptor of the guinea-pig aorta

1996

1. The cardiovascular activity of S-(+)-boldine, an aporphine alkaloid structurally related to papaverine, was determined. The work includes functional studies on guinea-pig isolated aorta contracted with noradrenaline, caffeine, KCl or Ca2+, and on guinea-pig trachea contracted with acetylcholine or histamine. 2. S-(+)-boldine inhibited in a concentration-dependent manner the contractile response evoked by noradrenaline (10 microM) in guinea-pig aorta (IC50 = 1.4 +/- 0.2 microM) while the KCl depolarizing solution (60 mM)- or the Ca2+ (1 mM)-induced contractions were only partially affected by boldine up to 300 microM. In contrast, papaverine relaxed noradrenaline (NA), KCl or Ca2+ induced…

Malemedicine.medical_specialtyAporphinesContraction (grammar)Phosphodiesterase InhibitorsMuscle RelaxationGuinea PigsAorta ThoracicIn Vitro TechniquesPhosphatidylinositolsMuscle Smooth Vascularchemistry.chemical_compoundCaffeinePapaverineReceptors Adrenergic alpha-1Internal medicinemedicine.arterymedicinePrazosinAnimalsBoldineThoracic aortaAdrenergic alpha-AntagonistsPharmacologyPapaverineParasympatholyticsCalcium Channel BlockersTracheaEndocrinologychemistryCompetitive antagonistAdrenergic alpha-1 Receptor AntagonistsHistamineAcetylcholineResearch Articlemedicine.drugBritish Journal of Pharmacology
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Cholinergic and GABAergic regulation of nitric oxide synthesis in the guinea pig ileum.

1999

Nitric oxide (NO) synthesis was examined in intact longitudinal muscle-myenteric plexus preparations of the guinea pig ileum by determining the formation of [3H]citrulline during incubation with [3H]arginine. Spontaneous [3H]citrulline production after 30 min was 80–90 dpm/mg, which constituted ∼1% of the tissue radioactivity. Electrical stimulation (10 Hz) led to a threefold increase in [3H]citrulline formation. Removal of calcium from the medium or addition of N G-nitro-l-arginine strongly inhibited both spontaneous and electrically induced production of [3H]citrulline. TTX reduced the electrically induced but not spontaneous [3H]citrulline formation. The electrically induced formation o…

Malemedicine.medical_specialtyBaclofenArgininePhysiologyGuinea PigsScopolamineMyenteric PlexusTubocurarineTetrodotoxinBiologyCholinergic AgonistsIn Vitro TechniquesMecamylamineBicucullineNitric OxideNitroarginineCholinergic Antagonistschemistry.chemical_compoundIleumPhysiology (medical)Internal medicineIsometric ContractionMuscarinic acetylcholine receptorMecamylaminemedicineCitrullineAnimalsEgtazic AcidGABA AgonistsMyenteric plexusgamma-Aminobutyric AcidHepatologyGABAA receptorMuscimolOxotremorineGastroenterologyMuscle SmoothBicucullineElectric StimulationEndocrinologychemistryMuscimolCitrullineFemalemedicine.drugThe American journal of physiology
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Gastric relaxation induced by apigenin and quercetin: Analysis of the mechanism of action

2009

Abstract Aims Recently, flavonoids have been shown to cause murine gastric relaxation. In the present study we examined the mechanism of action underlying gastric relaxation induced by apigenin and quercetin in isolated mouse stomach. Main methods The mechanical activity from the whole stomach was detected as changes in the endoluminal pressure and the response to increasing concentrations of both flavonoids were tested before and after different pharmacological treatments. Key findings Apigenin and quercetin-induced a concentration-dependent gastric relaxation, apigenin being more potent than quercetin. The responses were unaffected by 2′5′dideoxyadenosine, an inhibitor of adenylate cyclas…

Malemedicine.medical_specialtyCarbacholNifedipineMuscle Relaxationchemistry.chemical_elementCalcium antagonistIn Vitro TechniquesMuscarinic AgonistsCalciumPharmacologySettore BIO/09 - FisiologiaGeneral Biochemistry Genetics and Molecular BiologyPotassium ChlorideMicechemistry.chemical_compoundSmooth muscleInternal medicineCyclic AMPmedicineAnimalsheterocyclic compoundsApigeninGeneral Pharmacology Toxicology and PharmaceuticsCyclic GMPCyclic nucleotide phosphodiesteraseChemistryRyanodine receptorStomachMuscle SmoothGeneral MedicineCalcium Channel BlockersMice Inbred C57BLEndocrinologyMechanism of actionGastric toneApigeninFlavonoidCalciumCarbacholQuercetinmedicine.symptomQuercetinIntracellularMuscle Contractionmedicine.drugLife Sciences
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