Search results for "Splicing"

showing 10 items of 235 documents

Molecular modularity and asymmetry of the molluscan mantle revealed by a gene expression atlas

2018

15 pages; International audience; Background: Conchiferan molluscs construct a biocalcified shell that likely supported much of their evolutionary success.However, beyond broad proteomic and transcriptomic surveys of molluscan shells and the shell-forming mantle tissue,little is known of the spatial and ontogenetic regulation of shell fabrication. In addition, most efforts have been focused onspecies that deposit nacre, which is at odds with the majority of conchiferan species that fabricate shells using acrossed-lamellar microstructure, sensu lato. Results: By combining proteomic and transcriptomic sequencing with in situhybridization we have identified a suite of gene products associated …

Proteomics0301 basic medicineGlycosylationProteomematrix proteinHealth InformaticsLymnaea stagnalisProteomicsalternative splicing03 medical and health sciencesmolluscAnimal Shells[SDV.BBM.GTP]Life Sciences [q-bio]/Biochemistry Molecular Biology/Genomics [q-bio.GN]evolutionAnimalsMantle (mollusc)GeneGenetic Association Studiesmodularitymollusc; biomineralizationRegulation of gene expressionMineralsbiologyPhylumResearchGene Expression ProfilingGene Expression Regulation Developmentalbiomineralizationbiology.organism_classificationComputer Science ApplicationsGene expression profilingEvolvability030104 developmental biologyMolluscashellEvolutionary biologygene expressiontranscriptomeasymmetryGigaScience
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SANS (USH1G) regulates pre-mRNA splicing by mediating the intra-nuclear transfer of tri-snRNP complexes

2021

Abstract Splicing is catalyzed by the spliceosome, a compositionally dynamic complex assembled stepwise on pre-mRNA. We reveal links between splicing machinery components and the intrinsically disordered ciliopathy protein SANS. Pathogenic mutations in SANS/USH1G lead to Usher syndrome—the most common cause of deaf-blindness. Previously, SANS was shown to function only in the cytosol and primary cilia. Here, we have uncovered molecular links between SANS and pre-mRNA splicing catalyzed by the spliceosome in the nucleus. We show that SANS is found in Cajal bodies and nuclear speckles, where it interacts with components of spliceosomal sub-complexes such as SF3B1 and the large splicing cofact…

ProteomicsAcademicSubjects/SCI00010Ribonucleoprotein U4-U6 Small NuclearSF3B1 GeneMass Spectrometry0302 clinical medicineRNA Small NuclearRNA PrecursorsIn Situ Hybridization FluorescenceRibonucleoprotein0303 health sciencesChemistryRibonucleoproteins Small NuclearImmunohistochemistryCell biologyDNA-Binding Proteinsmedicine.anatomical_structureGene Knockdown TechniquesRNA splicingRNA Splicing FactorsUsher SyndromesSpliceosomeCoiled BodiesNerve Tissue ProteinsBiologyMinor Histocompatibility Antigens03 medical and health sciencesMicroscopy Electron TransmissionRNA and RNA-protein complexesGeneticsmedicineHumanssnRNPEye ProteinsGeneCell Proliferation030304 developmental biologyCell NucleusRNAmedicine.diseasePhosphoproteinsCiliopathyAlternative SplicingCell nucleusHEK293 CellsCajal bodyCytoplasmSpliceosomesNucleus030217 neurology & neurosurgeryTranscription FactorsNucleic Acids Research
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Classes of non-conventional tetraspanins defined by alternative splicing

2019

AbstractTetraspanins emerge as a family of membrane proteins mediating an exceptional broad diversity of functions. The naming refers to their four transmembrane segments, which define the tetraspanins‘ typical membrane topology. In this study, we analyzed alternative splicing of tetraspanins. Besides isoforms with four transmembrane segments, most mRNA sequences are coding for isoforms with one, two or three transmembrane segments, representing structurally mono-, di- and trispanins. Moreover, alternative splicing may alter transmembrane topology, delete parts of the large extracellular loop, or generate alternative N- or C-termini. As a result, we define structure-based classes of non-con…

ProteomicsGene isoformRNA splicingTetraspaninslcsh:MedicineComputational biologyBiologyEndoplasmic ReticulumArticleStructure-Activity Relationship03 medical and health sciences0302 clinical medicineIsomerismHumanslcsh:ScienceGene030304 developmental biology0303 health sciencesMultidisciplinarylcsh:RAlternative splicingLipid microdomainMembrane ProteinsTransmembrane proteinAlternative SplicingMembrane protein030220 oncology & carcinogenesisMembrane topologyembryonic structureslcsh:QStructural biologyFunction (biology)Scientific Reports
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THE “SALT-TASTING” NEWBORN

2021

Pseudohypoaldosteronism type 1 (PHA1) is a rare genetic disease due to the peripheral resistance to aldosterone. Clinical spectrum with neonatal onset includes salt loss, hyponatremia, hypochloraemia, hyperkalaemia, metabolic acidosis and increased plasmatic levels of aldosterone. Two forms of the disease - renal and systemic – have been described, which are genetically distinct and with wide clinical expressivity. The most severe generalized PHA1 is caused by mutations in the genes encoding for the subunits of the epithelial sodium channels (ENaC). The paper reports the case of a newborn of the first pregnancy of healthy and consanguineous Sicilian parents, with a clinical and hormonal pic…

Pseudohypoaldosteronism ENaC SCNN1A gene New splicing mutation Next generation sequencing
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Chronological expression of Ciliated Bronchial Epithelium 1 during pulmonary development

2009

Ciliated Bronchial Epithelium (CBE) 1 is a novel gene, which is expressed in ciliated cells. As cilia are important during embryogenesis, the present authors characterised the murine homologue of CBE1 (Cbe1) and compared its temporal expression during murine and human lung development. Cbe1 cDNA was cloned and characterised using sequencing, standard PCR and Western blotting. Mouse and human embryonic/fetal lungs (HELs) were harvested for mRNA analysis and protein localisation in vivo and in vitro using RT-PCR and immunohistochemistry. The Cbe1 amino acid sequence was >75% identical with CBE1 and its alternative splicing and tissue distribution were highly conserved. Pulmonary expression of…

Pulmonary and Respiratory MedicinePathologymedicine.medical_specialtyDNA ComplementaryTime FactorsBlotting WesternDNA Mutational AnalysisBiologyTransfectionStatistics NonparametricImmunoenzyme TechniquesMiceOpen Reading FramesCiliogenesisGene expressionmedicineAnimalsHumansAmino Acid SequenceRNA MessengerCloning MolecularLungDNA PrimersFetusMessenger RNALungReverse Transcriptase Polymerase Chain ReactionEmbryogenesisAlternative splicingNuclear ProteinsCell DifferentiationMolecular biologyEpitheliumDNA-Binding Proteinsmedicine.anatomical_structureBronchial epithelium Asthma DevelopmentTranscription FactorsEuropean Respiratory Journal
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A characterization of regular circular languages generated by marked splicing systems

2009

AbstractSplicing systems are generative devices of formal languages, introduced by Head in 1987 to model biological phenomena on linear and circular DNA molecules. A splicing system is defined by giving an initial set I and a set R of rules. Some unanswered questions are related to the computational power of circular splicing systems. In particular, a still open question is to find a characterization of circular languages generated by finite circular splicing systems (i.e., circular splicing systems with both I and R finite sets). In this paper we introduce a special class of the latter systems named marked systems. We prove that a marked system S generates a regular circular language if an…

Pure mathematicsGeneral Computer ScienceMolecular computing Splicing systems Circular words Formal languages Automata theoryMolecular computingQuantitative Biology::GenomicsDecidabilityTheoretical Computer ScienceSet (abstract data type)Formal languagesRegular languageFormal languageRNA splicingAutomata theorySplicing systemsCircular wordsFinite setAlgorithmWord (computer architecture)Automata theoryMathematicsComputer Science(all)
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The mitochondrial genome of Schizosaccharomyces pombe. Stimulation of intra-chromosomal recombination in Escherichia coli by the gene product of the …

1991

The open reading frame of the first intron of the mitochondrial cox1 gene (cox1I1) was expressed in Escherichia coli. The putative intron-encoded protein stimulated the formation of intra-chromosomal lac +-recombinants about threefold. No stimulation was found when the reading frame was inserted in the opposite direction, or when it was interrupted by a deletion. The intronic open reading frame did not complement recA − or recB − mutants of E. coli. In S. pombe, elimination of this intron did not abolish homologous recombination in mitochondria. A possible role of the recombinase activity in yeast mitochondria will be discussed.

RNA SplicingGenes FungalMolecular Sequence DataSaccharomyces cerevisiaeBiologymedicine.disease_causeDNA MitochondrialElectron Transport Complex IVFungal ProteinsRecombinasesOpen Reading FramesSequence Homology Nucleic AcidEndoribonucleasesSchizosaccharomycesGeneticsmedicineRecombinaseEscherichia coliAmino Acid SequenceDNA FungalEscherichia coliRecBCDRecombination GeneticRecombinase activityBase SequenceIntegrasesIntronGeneral Medicinebiology.organism_classificationMolecular biologyNucleotidyltransferasesIntronsOpen reading frameSchizosaccharomyces pombeDNA NucleotidyltransferasesbacteriaHomologous recombination
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The polypyrimidine tract-binding protein (PTB) is involved in the post-transcriptional regulation of human inducible nitric oxide synthase expression.

2006

Human inducible nitric oxide synthase (iNOS) expression is regulated by transcriptional and post-transcriptional mechanisms. We have recently shown that the multifunctional RNA-binding proteins KH-type splicing regulatory protein and tristetraprolin are critically involved in the post-transcriptional regulation of human iNOS expression. Several reports have shown that KH-type splicing regulatory protein colocalizes with the polypyrimidine tract-binding protein (PTB), and both RNA-binding proteins seem to interact with the same mRNAs. Therefore we analyzed the involvement of PTB in human iNOS expression. In human DLD-1 cells, cytokine incubation necessary to induce iNOS expression did not ch…

Recombinant Fusion ProteinsTristetraprolinGreen Fluorescent ProteinsNitric Oxide Synthase Type IImacromolecular substancesBiologyIn Vitro TechniquesTransfectionenvironment and public healthBiochemistryGene Expression Regulation EnzymologicCell LineCell Line TumorHumansPolypyrimidine tract-binding proteinRNA MessengerEnzyme InhibitorsPromoter Regions GeneticMolecular BiologyPost-transcriptional regulationRegulation of gene expressionMessenger RNAintegumentary systemCarcinomaEpithelial CellsCell BiologyTransfectionMolecular biologyNitric oxide synthaseRNA splicingColonic Neoplasmsbiology.proteinCytokinesRNA InterferenceProtein Processing Post-TranslationalDichlororibofuranosylbenzimidazolePolypyrimidine Tract-Binding ProteinThe Journal of biological chemistry
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Anything but Ordinary - Emerging Splicing Mechanisms in Eukaryotic Gene Regulation.

2021

Splicing of precursor mRNAs (pre-mRNA) is an important step during eukaryotic gene expression. The identification of the actual splice sites and the proper removal of introns are essential for the production of the desired mRNA isoforms and their encoded proteins. While the basic mechanisms of splicing regulation are well understood, recent work has uncovered a growing number of noncanonical splicing mechanisms that play key roles in the regulation of gene expression. In this review, we summarize the current principles of splicing regulation, including the impact of cis and trans regulatory elements, as well as the influence of chromatin structure, transcription, and RNA modifications. We f…

Regulation of gene expression0303 health sciencesRNA SplicingIntronRNAEukaryotaRNA-binding proteinComputational biologyBiologyIntronsChromatin03 medical and health sciencesAlternative Splicing0302 clinical medicineCircular RNARNA splicingGene expressionGeneticsRNA PrecursorsHumansRNA Messenger030217 neurology & neurosurgery030304 developmental biologyTrends in genetics : TIG
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Molecular genetics of autosomal dominant retinitis pigmentosa (ADRP): a comprehensive study of 43 Italian families

2005

Retinitis pigmentosa is the most common form of retinal degeneration and is heterogeneous both clinically and genetically. The autosomal dominant forms ( ADRP) can be caused by mutations in 12 different genes. This report describes the first simultaneous mutation analysis of all the known ADRP genes in the same population, represented by 43 Italian families. This analysis allowed the identification of causative mutations in 12 of the families (28% of the total). Seven different mutations were identified, two of which are novel (458delC and 6901C --> T (P2301S), in the CRX and PRPF8 genes, respectively). Several novel polymorphisms leading to amino acid changes in the FSCN2, NRL, IMPDH1, and…

Retinal degenerationDNA Mutational Analysismedicine.disease_causeGene FrequencyPrevalenceAge of OnsetSPLICING-FACTOR GENESChildGenetics (clinical)Genes DominantGeneticsMutationeducation.field_of_studyRNA-Binding ProteinsMiddle AgedDNA-Binding ProteinsBasic-Leucine Zipper Transcription FactorsItalyChild PreschoolMESSENGER-RNAMicrotubule-Associated ProteinsRetinitis PigmentosaFORMAdultRhodopsinmedicine.medical_specialtycongenital hereditary and neonatal diseases and abnormalitiesAdolescentPopulationRHODOPSIN GENEBiologyMolecular geneticsRetinitis pigmentosaGeneticsmedicineHumansFamilyEye ProteinseducationGeneAllele frequencyHomeodomain ProteinsMUTATIONSmedicine.diseaseeye diseasesMutationTrans-ActivatorsMutation testingOnline Mutation ReportCarrier Proteins
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