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showing 10 items of 4285 documents

Beneficial Effect of Docosahexanoic Acid and Lutein on Retinal Structural, Metabolic, and Functional Abnormalities in Diabetic Rats

2009

To assess the effect of docosahexanoic acid (DHA) and lutein (both compounds with anti-inflammatory and antioxidant properties) on experimental diabetic retinopathy.Male Wistar rats were studied: non-diabetic controls, untreated diabetic controls, and diabetic rats were treated with DHA and lutein or the combination of DHA + insulin and lutein + insulin for 12 weeks. Oxidative stress and inflammatory markers, apoptosis, and functional tests were studied to confirm biochemical and functional changes in the retina of diabetic rats. Malondialdehyde (MDA), glutathione concentrations (GSH), and glutathione peroxidase activity (GPx) were measured as oxidative stress markers. TUNEL assay and caspa…

Blood GlucoseMaleLuteingenetic structuresmedicine.medical_treatmentApoptosismedicine.disease_causeAntioxidantschemistry.chemical_compoundMalondialdehydeInsulinFluorescent Antibody Technique Indirectchemistry.chemical_classificationCaspase 3NitrotyrosineGlutathione peroxidaseAnti-Inflammatory Agents Non-Steroidalfood and beveragesMalondialdehydeGlutathioneSensory SystemsDrug Therapy Combinationmedicine.medical_specialtyDocosahexaenoic AcidsEnzyme-Linked Immunosorbent AssayBiologyRetinaDiabetes Mellitus ExperimentalCellular and Molecular NeuroscienceDiabetes mellitusInternal medicineElectroretinographyIn Situ Nick-End LabelingmedicineAnimalsRats WistarGlutathione PeroxidaseDiabetic RetinopathyInsulinLuteinGlutathionemedicine.diseaseeye diseasesRatsOxidative StressOphthalmologyEndocrinologychemistryTyrosinesense organsBiomarkersOxidative stressCurrent Eye Research
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Feeding enhances extracellular lactate of local origin in the rostromedial hypothalamus but not in the cerebellum.

1999

Abstract The use of brain microdialysis together with chronic vascular catheterization allowed us to assay extracellular fluid lactate (ECF L ) in both the ventromedial–paraventricular (VMH–PVN) area of the hypothalamus and the cerebellum, in parallel with measures of plasma levels, and in relation to food intake. A 45 min scheduled meal increased VMH–PVN ECF L by 28%. This increase was not observed in the cerebellum. The prandial increase in plasma glucose (43%, from 4.74 to 6.77 mM) and lactate (84%, from 0.83 to 1.53 mM) showed a different temporal pattern and lasted longer than that of the ECF L . Glucose delivery by reverse dialysis for 45 min into the VMH–PVN area increased ECF L by 4…

Blood GlucoseMaleMicrodialysismedicine.medical_specialtyCerebellumTime FactorsMicrodialysisCentral nervous systemBiologyDeoxyglucoseBlood–brain barrierEatingInternal medicineCerebellumExtracellular fluidmedicineExtracellularAnimalsLactic AcidRats WistarMolecular BiologyGeneral Neurosciencedigestive oral and skin physiologyMetabolismPostprandial PeriodRatsmedicine.anatomical_structureEndocrinologyGlucosenervous systemHypothalamusVentromedial Hypothalamic NucleusNeurology (clinical)Extracellular SpaceDevelopmental BiologyParaventricular Hypothalamic NucleusBrain research
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Antioxidant effect of Ajuga iva aqueous extract in streptozotocin-induced diabetic rats.

2009

The purpose of this study was to investigate the possible antioxidant effect of an aqueous extract of Ajuga iva (Ai) in streptozotocin (STZ)-induced diabetic rats. Twelve diabetic rats were divided into two groups fed a casein diet supplemented or not with Ai (0.5%), for 4 weeks. In vitro, the Ai extract possessed a very high antioxidant effect (1 mg/ml was similar to those of trolox 300 mmol/l). The results indicated that plasma thiobarbituric acid reactive substances (TBARS) values were reduced by 41% in Ai-treated compared with untreated diabetic rats. TBARS concentrations were lower 1.5-fold in liver, 1.8-fold in heart, 1.9-fold in muscle and 2.1-fold in brain in Ai-treated than untreat…

Blood GlucoseMalemedicine.medical_specialtyAntioxidantThiobarbituric acidmedicine.medical_treatmentGlutathione reductasePharmaceutical ScienceEnzyme-Linked Immunosorbent AssayNitric OxideThiobarbituric Acid Reactive SubstancesAntioxidantsStreptozocinLipid peroxidationchemistry.chemical_compoundInternal medicineDrug DiscoverymedicineTBARSAnimalsInsulinRats WistarPharmacologychemistry.chemical_classificationPlant ExtractsGlutathione peroxidaseBody WeightGlutathioneOrgan SizeCarotenoidsLipidsRatsEndocrinologyComplementary and alternative medicinechemistryBiochemistryMolecular MedicineTroloxLipid PeroxidationPhytomedicine : international journal of phytotherapy and phytopharmacology
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Hypoglycaemic effect of Spergularia purpurea in normal and streptozotocin-induced diabetic rats

2000

Single and repeated oral administration of the water extracts of Spergularia purpurea (SP) at a dose of 10 mg/kg were tested on hypoglycaemic activity in normal and streptozotocin-induced diabetic rats. In normal rats, the water extract of SP decreased significantly the plasma glucose levels 4 h after single oral administration (P0.01), and one week after repeated oral administration (P0.05). A significant decrease of plasma glucose levels was observed 6 h after a single oral administration of the water extract of S. purpurea in severe hyperglycaemic rats (n=6) from 22.78+/-0.60 to 11.21+/-0.49 mmol/l (P0.001). On other hand, water extract of S. purpurea normalised plasma glucose levels aft…

Blood GlucoseMalemedicine.medical_specialtyBlood sugarPharmacognosyDiabetes Mellitus Experimentallaw.inventionLethal Dose 50MicelawOral administrationInternal medicineDiabetes mellitusDrug DiscoverymedicineAnimalsHypoglycemic AgentsInsulinRats WistarPharmacologyPlants MedicinalBehavior AnimalPlant Extractsbusiness.industryBody Weightmedicine.diseaseStreptozotocinRatsMoroccoEndocrinologyPhytochemicalToxicityPhytotherapybusinessmedicine.drugJournal of Ethnopharmacology
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Fructose-enriched diet modifies antioxidant status and lipid metabolism in spontaneously hypertensive rats

2005

Abstract Objective High-fructose consumption in industrial countries has been shown to induce metabolic abnormalities or syndrome X. Changes in antioxidant defense are unknown in hypertension associated with metabolic disorders induced by high-fructose feeding. Methods Twenty spontaneously hypertensive rats were assigned to one of two groups; one received a fructose-enriched diet (60% fructose) and the other a starch diet. After a 13-wk diet period, total antioxidant status was assessed in the blood and liver by monitoring the rate of free radical-induced red blood cell hemolysis. Antioxidants (enzymes and vitamins) were determined in blood and liver. Gene expression of antioxidant enzymes …

Blood GlucoseMalemedicine.medical_specialtyErythrocytesAntioxidantEndocrinology Diabetes and Metabolismmedicine.medical_treatmentalpha-TocopherolGene ExpressionAscorbic AcidFructoseThiobarbituric Acid Reactive SubstancesAntioxidantsLipid peroxidationSuperoxide dismutasechemistry.chemical_compoundRats Inbred SHRInternal medicinemedicineAnimalsInsulinRNA MessengerVitamin Achemistry.chemical_classificationGlutathione PeroxidaseNutrition and DieteticsbiologySuperoxide DismutaseGlutathione peroxidaseStarchFructoseLipid MetabolismAscorbic acidDietRatsRed blood cellEndocrinologymedicine.anatomical_structureLiverchemistryHypertensionbiology.proteinLipid PeroxidationPeroxidaseNutrition
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Time course of changes in serum oxidant/antioxidant status in overfed obese rats and their offspring.

2009

The aim of the present study was to determine the time course of changes in oxidant/antioxidant status, as well as serum glucose, insulin, leptin and lipid levels, liver adipose tissue and muscle lipid and protein contents, in cafeteria-diet-fed dams during gestation and lactation, and in their offspring throughout adulthood. Food intake was also evaluated. The cafeteria diet induced a significant increase in maternal body and relative adipose tissue weights, daily energy intake, and plasma glucose, insulin, leptin and lipid levels at parturition (day 0) and at the end of lactation (day 21). Plasma total antioxidant status [ORAC (oxygen radical absorbance capacity)], erythrocyte catalase an…

Blood GlucoseMalemedicine.medical_specialtyErythrocytesOffspringmedicine.medical_treatmentAdipokineAdipose tissueBiologyAntioxidantschemistry.chemical_compoundBlood serumPregnancyInternal medicinemedicineAnimalsInsulinObesityRats WistarPrenatal Nutritional Physiological PhenomenaTriglycerideInsulinLeptinBody WeightProteinsGeneral Medicinemedicine.diseaseOxidantsLipidsRatsPregnancy ComplicationsOxidative StressEndocrinologychemistryPrenatal Exposure Delayed EffectsFemaleMetabolic syndromeEnergy IntakeClinical science (London, England : 1979)
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Vascular Dysfunction in Streptozotocin-Induced Experimental Diabetes Strictly Depends on Insulin Deficiency

2010

<i>Objective:</i> In previous studies we and others have shown that streptozotocin (STZ)-induced diabetes in rats is associated with vascular oxidative stress and dysfunction. In the present study, we sought to determine whether vascular dysfunction and oxidative stress strictly depend on insulin deficiency. <i>Methods:</i> The effects of insulin (2.5 U/day s.c., 2 weeks) therapy on vascular disorders in STZ-induced (60 mg/kg i.v., 8 weeks) diabetes mellitus (type I) were studied in Wistar rats. The contribution of NADPH oxidase to overall oxidative stress was investigated by in vivo (30 mg/kg/day s.c., 4 days) and in vitro treatment with apocynin. <i>Results:&…

Blood GlucoseMalemedicine.medical_specialtyNitric Oxide Synthase Type IIIEndotheliumPhysiologymedicine.medical_treatmentmedicine.disease_causeStreptozocinDiabetes Mellitus Experimentalchemistry.chemical_compoundInternal medicineDiabetes mellitusmedicineAnimalsInsulinRats WistarEndothelial dysfunctionNADPH oxidasebiologybusiness.industryMyocardiumInsulinAcetophenonesNADPH OxidasesStreptozotocinmedicine.diseaseRatsOxidative StressNG-Nitroarginine Methyl EsterEndocrinologymedicine.anatomical_structurechemistryApocyninbiology.proteinEndothelium VascularCardiology and Cardiovascular MedicinebusinessDiabetic AngiopathiesOxidative stressmedicine.drugJournal of Vascular Research
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AT1-receptor blockade by telmisartan upregulates GTP-cyclohydrolase I and protects eNOS in diabetic rats.

2008

Several enzymatic sources of reactive oxygen species (ROS) were described as potential reasons of eNOS uncoupling in diabetes mellitus. In the present study, we investigated the effects of AT1-receptor blockade with chronic telmisartan (25 mg/kg/day, 6.5 weeks) therapy on expression of the BH4-synthesizing enzyme GTP-cyclohydrolase I (GCH-I), eNOS uncoupling, and endothelial dysfunction in streptozotocin (STZ, 60 mg/kg iv, 7 weeks)-induced diabetes mellitus (type I). Telmisartan therapy did not modify blood glucose and body weight. Aortas from diabetic animals had vascular dysfunction as revealed by isometric tension studies (acetylcholine and nitroglycerin potency). Vascular and cardiac RO…

Blood GlucoseMalemedicine.medical_specialtyNitric Oxide Synthase Type IIImedicine.disease_causeBiochemistryBenzoatesReceptor Angiotensin Type 1chemistry.chemical_compoundEnosPhysiology (medical)Internal medicinemedicineDiabetes MellitusAnimalsTelmisartanEndothelial dysfunctionRats WistarXanthine oxidaseGTP CyclohydrolaseNADPH oxidasebiologySuperoxideBody WeightNADPH Oxidasesmedicine.diseaseStreptozotocinbiology.organism_classificationMitochondriaRatsUp-RegulationEnzyme ActivationOxidative StressEndocrinologychemistrybiology.proteinBenzimidazolesTelmisartanAngiotensin II Type 1 Receptor BlockersOxidative stressmedicine.drugFree radical biologymedicine
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Time course of changes in serum glucose, insulin, lipids and tissue lipase activities in macrosomic offspring of rats with streptozotocin-induced dia…

1999

The aim of this investigation was to determine the time course of changes in serum glucose, insulin and lipid levels, as well as lipid and protein content and lipolytic activities in insulin target organs (liver, adipose tissue and muscle), in macrosomic offspring of streptozotocin-induced mildly hyperglycaemic rats. Food intake and nutritional efficiency were also evaluated. Mild hyperglycaemia in pregnant rats was induced by intraperitoneal injection of streptozotocin (40 mg/kg body weight) on day 5 of gestation. Control pregnant rats were injected with citrate buffer. At birth, macrosomic pups (birth weight > 1.7 S.D. greater than the mean value for the control pups) had higher serum …

Blood GlucoseMalemedicine.medical_specialtyTime Factorsmedicine.medical_treatmentLipolysisAdipose tissueBiologyDiabetes Mellitus ExperimentalFetal Macrosomiachemistry.chemical_compoundEatingInsulin resistanceSex FactorsDiabetes mellitusAdipocyteInternal medicinemedicineFetal macrosomiaAnimalsInsulinObesityRats WistarMuscle SkeletalInsulinLipid metabolismGeneral MedicineLipasemedicine.diseaseStreptozotocinLipidsRatsEndocrinologychemistryAdipose TissueLiverFemalemedicine.drugClinical science (London, England : 1979)
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Vascular Dysfunction in Experimental Diabetes Is Improved by Pentaerithrityl Tetranitrate but Not Isosorbide-5-Mononitrate Therapy

2011

OBJECTIVE Diabetes is associated with vascular oxidative stress, activation of NADPH oxidase, and uncoupling of nitric oxide (NO) synthase (endothelial NO synthase [eNOS]). Pentaerithrityl tetranitrate (PETN) is an organic nitrate with potent antioxidant properties via induction of heme oxygenase-1 (HO-1). We tested whether treatment with PETN improves vascular dysfunction in the setting of experimental diabetes. RESEARCH DESIGN AND METHODS After induction of hyperglycemia by streptozotocin (STZ) injection (60 mg/kg i.v.), PETN (15 mg/kg/day p.o.) or isosorbide-5-mononitrate (ISMN; 75 mg/kg/day p.o.) was fed to Wistar rats for 7 weeks. Oxidative stress was assessed by optical methods and o…

Blood GlucoseMalemedicine.medical_specialtyXanthine OxidaseEndocrinology Diabetes and MetabolismVasodilator AgentsOxidative phosphorylationIsosorbide Dinitratemedicine.disease_causeWeight GainNitric oxideDiabetes Mellitus Experimentalchemistry.chemical_compoundEnosInternal medicineInternal MedicinemedicineAnimalsPentaerythritol TetranitrateGene SilencingEndothelial dysfunctionRats WistarXanthine oxidaseGTP CyclohydrolaseNADPH oxidasebiologyNADPH Oxidasesmedicine.diseasebiology.organism_classificationStreptozotocinPharmacology and TherapeuticsRatsOxidative StressEndocrinologychemistryVasoconstrictionbiology.proteinEndothelium VascularReactive Oxygen SpeciesOxidative stressHeme Oxygenase-1medicine.drugDiabetes
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