Search results for "Steatosi"

showing 10 items of 261 documents

Foxa1 reduces lipid accumulation in human hepatocytes and is down-regulated in nonalcoholic fatty liver.

2012

Triglyceride accumulation in nonalcoholic fatty liver (NAFL) results from unbalanced lipid metabolism which, in the liver, is controlled by several transcription factors. The Foxa subfamily of winged helix/forkhead box (Fox) transcription factors comprises three members which play important roles in controlling both metabolism and homeostasis through the regulation of multiple target genes in the liver, pancreas and adipose tissue. In the mouse liver, Foxa2 is repressed by insulin and mediates fasting responses. Unlike Foxa2 however, the role of Foxa1 in the liver has not yet been investigated in detail. In this study, we evaluate the role of Foxa1 in two human liver cell models, primary cu…

MaleGene Expressionlcsh:MedicineBiochemistrychemistry.chemical_compoundNon-alcoholic Fatty Liver DiseaseMolecular Cell Biologylcsh:ScienceCells Culturedchemistry.chemical_classificationMultidisciplinaryLiver DiseasesFatty liverAnimal ModelsHep G2 CellsPeroxisomeMiddle AgedLipidsMedicineFemaleResearch ArticleAdultHepatocyte Nuclear Factor 3-alphamedicine.medical_specialtyPrimary Cell CultureDown-RegulationGastroenterology and HepatologyBiologyYoung AdultInsulin resistanceModel OrganismsInternal medicinemedicineAnimalsHumansBiologyAgedTriglyceridelcsh:RFatty acidProteinsLipid metabolismmedicine.diseaseLipid MetabolismRatsFatty LiverEndocrinologyMetabolismchemistryHepatocyteslcsh:QFOXA2SteatosisPLoS ONE
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Hyperuricaemia: another metabolic feature affecting the severity of chronic hepatitis because of HCV infection.

2011

Background Several works observed a link between uric acid serum levels and clinical and histological features of nonalcoholic fatty liver disease. An association between chronic hepatitis C (CHC) and uric acid levels has been poorly investigated. Aims To assess the potential association between uric acid serum levels and both histological features of liver damage and sustained virological response (SVR) in a homogeneous cohort of CHC patients. Methods Consecutive biopsy-proven CHC patients were included. Hyperuricaemia was diagnosed with uric acid serum levels >7 mg/dl in men, and >6 mg/dl in women. Patients underwent therapy with pegylated interferon plus ribavirin. Results Hyperuricaemia…

MaleHCV STEATOSIS HYPERURICEMIAmedicine.medical_specialtyBiopsyRenal functionHepacivirusHyperuricemiaSettore MED/08 - Anatomia PatologicaGastroenterologychemistry.chemical_compoundPegylated interferonInternal medicineNonalcoholic fatty liver diseasemedicineHumansSettore MED/12 - GastroenterologiaHepatologybusiness.industryRibavirinHepatitis CHepatitis C ChronicMiddle Agedmedicine.diseasePrognosisUric AcidFatty LiverEndocrinologychemistryUric acidFemaleSteatosisbusinessBody mass indexBiomarkersmedicine.drug
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Risk of chemotherapy-associated liver injury (CALI) in PNPLA3 p.148M allele carriers: Preliminary results of a transient elastography-based study

2019

Liver steatosis is one of the side effects of chemotherapy. The PNPLA3 p.I148M, TM6SF2 p.E167K and MBOAT7 p.G17E variants represent genetic determinants for progressive liver diseases. Here, we investigate their association with chemotherapy-associated steatosis.Prospectively, we recruited 87 patients undergoing systemic chemotherapy for gastrointestinal cancers. Hepatic fat (controlled attenuation parameter, CAP) and liver stiffness (LSM) were measured non-invasively before the initiation of chemotherapy (T0) and after at least two (T1) and four cycles (T2). Genetic variants were genotyped using allelic discrimination assays.In the final dataset (n = 60) patients demonstrated the following…

MaleHeterozygotemedicine.medical_specialtymedicine.medical_treatmentAntineoplastic AgentsGastroenterology03 medical and health sciences0302 clinical medicineFat accumulationInternal medicinemedicineHumansGenetic Predisposition to DiseaseAdiponutrinProspective StudiesAlleleeducationAllelesTriglyceridesAgedLiver injuryChemotherapyeducation.field_of_studyPolymorphism GeneticHepatologybusiness.industryGastroenterologyMembrane ProteinsLipaseMiddle Agedmedicine.diseaseFatty LiverLiver030220 oncology & carcinogenesisElasticity Imaging TechniquesFemale030211 gastroenterology & hepatologySteatosisTransient elastographybusinessAcyltransferasesTM6SF2Digestive and Liver Disease
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Grape seed and skin extract reduces pancreas lipotoxicity, oxidative stress and inflammation in high fat diet fed rats.

2016

IF 2.326; International audience; Obesity is related to an elevated risk of diabetes and the mechanisms whereby fat adversely affects the pancreas are poorly understood. We studied the effect of a high fat diet (HFD) on pancreas steatosis, oxidative stress and inflammation as well as the putative protection afforded by grape seed and skin extract (GSSE). HFD induced body weight gain, without affecting insulinemia, nor glycemia and dropped adiponectemia. HFD also provoked the ectopic deposition of cholesterol and triglyceride, and an oxidative stress characterized by increased lipoperoxidation and carbonylation, inhibition of antioxidant enzyme activities such as CAT, GPx and SOD, depletion …

MaleMESH : Oxidative Stress0301 basic medicineMESH: InflammationAntioxidantmedicine.medical_treatmentMESH: Grape Seed Extractmedicine.disease_causeAntioxidantschemistry.chemical_compound0302 clinical medicineMESH: ObesityVitisMESH: AnimalsMESH : Pancreas2. Zero hungerMESH: Oxidative StressMESH : Grape Seed ExtractMESH : RatsMESH : Diet High-Fatfood and beveragesGeneral MedicineMESH: Pancreas3. Good healthLipotoxicity030220 oncology & carcinogenesisSeedsMESH : AntioxidantsMESH : Obesitymedicine.symptommedicine.medical_specialtyGrape seed and skin extractMESH: RatsMESH : MaleInflammationBiologyDiet High-FatMESH : Rats WistarMESH: VitisMESH : Vitis03 medical and health sciencesDiabetes mellitusInternal medicinemedicineAnimalsObesityRats WistarPancreasPharmacologyInflammationMESH : InflammationGrape Seed ExtractTriglycerideCholesterolMESH: Antioxidants[ SDV.SP.PHARMA ] Life Sciences [q-bio]/Pharmaceutical sciences/PharmacologyMESH: Rats Wistarmedicine.diseaseMESH : SeedsMESH: MaleRatsMESH: Diet High-Fat030104 developmental biologyEndocrinologychemistryMESH: SeedsOxidative stress[SDV.SP.PHARMA]Life Sciences [q-bio]/Pharmaceutical sciences/PharmacologyMESH : AnimalsSteatosisOxidative stress
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Metabolic syndrome triggered by high-fructose diet favors choroidal neovascularization and impairs retinal light sensitivity in the rat

2014

Diabetic retinopathy and age-related macular degeneration are the leading causes of blindness in Western populations. Although it is a matter of controversy, large-scale population-based studies have reported increased prevalence of age-related macular degeneration in patients with diabetes or diabetic retinopathy. We hypothesized that metabolic syndrome, one of the major risk factors for type 2 diabetes, would represent a favorable environment for the development of choroidal neovascularization, the main complication of age-related macular degeneration. The fructose-fed rat was used as a model for metabolic syndrome in which choroidal neovascularization was induced by laser photocoagulatio…

MaleOrganes des sensmedicine.medical_treatment[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionVisual Acuitylcsh:MedicineGene ExpressionType 2 diabetesinduced insulin-resistanceanimal-modelscholesterol homeostasis0302 clinical medicineRetinal Rod Photoreceptor CellsRats Inbred BNHyperinsulinemiaMedicine and Health Sciencesanimal modèleratlcsh:Science2. Zero hungerMetabolic Syndrome0303 health scienceseducation.field_of_studyMultidisciplinaryLaser Coagulationsyndrome métaboliqueReverse Transcriptase Polymerase Chain Reactionhepatic steatosisFatty AcidsAngiographyDiabetic retinopathyChoroidal neovascularizationAdipose Tissue[ SDV.MHEP.OS ] Life Sciences [q-bio]/Human health and pathology/Sensory OrgansAlimentation et NutritionRetinal Disordersmedicine.symptomLaser coagulationResearch Articlediabètemedicine.medical_specialtymacular degenerationPopulationSensory Organselectroretinographic oscillatory potentials;induced insulin-resistance;fatty-acid profile;macular degeneration;diabetic-retinopathy;animal-models;cholesterol homeostasis;hepatic steatosis;mouse;associationAntigens Differentiation MyelomonocyticMédecine humaine et pathologieFructoseBiologyRetina03 medical and health sciencesAntigens CDDiabetes mellitusInternal medicine[ SDV.MHEP ] Life Sciences [q-bio]/Human health and pathologymedicineElectroretinographyelectroretinographic oscillatory potentialsAnimalsHumansFood and Nutrition[SDV.MHEP.OS]Life Sciences [q-bio]/Human health and pathology/Sensory OrganseducationRetinopathymouse030304 developmental biologyNutritiondiabetic-retinopathylcsh:RassociationBiology and Life Sciencesdégénérescence maculaireMacular degenerationmedicine.diseaseChoroidal Neovascularizationeye diseasesDietFatty LiverOphthalmologyEndocrinologyMetabolic Disordersfatty-acid profile030221 ophthalmology & optometrylcsh:QInsulinomaHuman health and pathologysense organs[SDV.AEN]Life Sciences [q-bio]/Food and Nutrition[SDV.MHEP]Life Sciences [q-bio]/Human health and pathology
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Immunopositivity for histone macroH2A1 isoforms marks steatosisassociated hepatocellular carcinoma.

2012

BackgroundHepatocellular carcinoma (HCC) is one of the most common cancers worldwide. Prevention and risk reduction are important and the identification of specific biomarkers for early diagnosis of HCC represents an active field of research. Increasing evidence indicates that fat accumulation in the liver, defined as hepatosteatosis, is an independent and strong risk factor for developing an HCC. MacroH2A1, a histone protein generally associated with the repressed regions of chromosomes, is involved in hepatic lipid metabolism and is present in two alternative spliced isoforms, macroH2A1.1 and macroH2A1.2. These isoforms have been shown to predict lung and colon cancer recurrence but to ou…

MalePathologyMouseBiological Markers/metabolismEpidemiologyTumor Microenvironment/geneticsColorectal cancerGene ExpressionHepatocytes/metabolism/pathologyNonalcoholic SteatohepatitisHistonesFatty Liver/chemically induced/complications/genetics/metabolismMice0302 clinical medicineGastrointestinal CancersTumor MicroenvironmentPathologyProtein IsoformsDiethylnitrosamineSettore MED/49 - Scienze Tecniche Dietetiche ApplicateMice KnockoutRegulation of gene expression0303 health sciencesMultidisciplinaryProtein Isoforms/genetics/metabolismbiologyLiver DiseasesPTEN Phosphohydrolase/deficiency/geneticshepatocellular carcinoma biomarker histone variant steatosis epigeneticsLiver NeoplasmsQFatty liverRHistone ModificationAnimal ModelsImmunohistochemistry3. Good healthHistoneOncology030220 oncology & carcinogenesisHepatocellular carcinomaMedicineEpigeneticsCarcinoma Hepatocellular/etiology/genetics/metabolism/pathologyResearch ArticleGene isoformmedicine.medical_specialtyCarcinoma HepatocellularHistologyClinical Research DesignScienceGastroenterology and HepatologyDiet High-Fat03 medical and health sciencesModel OrganismsDiagnostic MedicineGastrointestinal TumorsGeneticsCancer GeneticsCancer Detection and DiagnosisEarly DetectionmedicineAnimalsHumansAnimal Models of DiseaseObesityddc:612BiologyHistones/genetics/metabolismNutrition030304 developmental biologyCell NucleusCell Nucleus/genetics/metabolism/pathologyTumor microenvironmentbusiness.industryPTEN PhosphohydrolaseCancers and NeoplasmsHepatocellular Carcinomamedicine.diseasedigestive system diseasesFatty LiverBiomarker EpidemiologyGene Expression RegulationHepatocytesbiology.proteinLiver Neoplasms/etiology/genetics/metabolism/pathologySteatosisbusinessBiomarkersGeneral Pathology
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Hyperferritinemia is a risk factor for steatosis in chronic liver disease.

2009

AIM: To investigate the relationship between ferritin and steatosis in patients with chronically abnormal liver function tests (LFTs) and high ferritin level. METHODS: One hundred and twenty-four consecutive patients with hyperferritinemia (male > 300 ng/mL, female > 200 ng/mL) were evaluated; clinical, biochemical and serological data, iron status parameters, HFE gene mutations and homeostasis model assessment score were obtained. Steatosis was graded by ultrasound as absent or present. Histology was available in 53 patients only. RESULTS: Mean level of ferritin was 881 ± 77 ng/mL in men and 549 ± 82 ng/mL in women. The diagnosis was chronic hepatitis C in 53 (42.7%), non-alcoholic fatty l…

MalePathologymedicine.medical_specialtyHyperferritinemia chronic liver disease.Chronic liver diseaseGastroenterologyLiver Function TestsRisk FactorsInternal medicinemedicineHumansmedicine.diagnostic_testbiologybusiness.industryLiver DiseasesFatty liverGastroenterologyGeneral MedicineMiddle Agedmedicine.diseaseFerritinFatty LiverBrief ArticlesLiver biopsyHereditary hemochromatosisChronic DiseaseFerritinsMutationbiology.proteinAbnormal Liver Function TestFemaleSteatosisSteatohepatitisbusinessWorld journal of gastroenterology
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Nonalcoholic fatty liver and metabolic syndrome in Italy: Results from a multicentric study of the Italian Arteriosclerosis society

2013

Nonalcoholic fatty liver disease (NAFLD) is associated with all the components of metabolic syndrome (MS) and might to be considered an additional component of MS itself. The Italian Society for the Study of Atherosclerosis (SISA) in 2005 started a research project aimed to study the NAFLD, using ultrasound (US), in nondiabetic MS subjects matching at least one of the ATP III criteria for HDL-C or triglycerides [TG]. Prevalence of US-NAFLD and its associated risk factors and prevalence of hypertransaminasemia and its possible determinants were evaluated. NAFLD prevalence was 0.78. Men with steatosis compared to men without steatosis were younger (P < 0.05) with higher TG (P < 0.03), homeost…

MaleSettore MED/09 - Medicina InternaEndocrinology Diabetes and MetabolismNonalcoholic fatty liverSex FactorSettore MED/13 - EndocrinologiaBody Mass IndexEndocrinologyNon-alcoholic Fatty Liver DiseaseRisk FactorsNonalcoholic fatty liver diseaseAtherosclerosis; Metabolic Syndrome X; Sex Factors; Humans; Lipids; Aged; Fatty Liver; Non-alcoholic Fatty Liver Disease; Body Mass Index; Italy; Logistic Models; Risk Factors; Liver; Middle Aged; Female; MaleUltrasonographyMulticentric studyMetabolic Syndrome XFatty liverGeneral MedicineArteriosclerosisLipidMiddle AgedLipidsMetabolic syndromeDiabetes and MetabolismItalyLiverAtherosclerosiFemaleHumanmedicine.medical_specialtyLogistic ModelNonalcoholic fatty liver Metabolic syndrome Lipids Multicentric studyInsulin resistanceSex FactorsDiabetes mellitusInternal medicinemedicineInternal MedicineHumansLipids; Metabolic syndrome; Multicentric study; Nonalcoholic fatty liver; Aged; Body Mass Index; Fatty Liver; Female; Humans; Italy; Lipids; Liver; Logistic Models; Male; Metabolic Syndrome X; Middle Aged; Non-alcoholic Fatty Liver Disease; Risk Factors; Sex Factors; Atherosclerosis; Endocrinology; Internal Medicine; Endocrinology Diabetes and MetabolismAgedbusiness.industryRisk Factornutritional and metabolic diseasesAmbientalemedicine.diseaseAtherosclerosisFatty LiverEndocrinologyLogistic ModelsMetabolic syndromeSteatosisbusinessBody mass index
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Response to antiviral therapy and hepatic expression of cyclooxygenases in chronic hepatitis C

2007

OBJECTIVES: The aims of this study were to investigate the expression of cyclooxygenase-1 and cyclooxygenase-2 (COX-1 and COX-2) in chronic hepatitis C (CHC) by immunohistochemistry, based on the hypothesis that COXs expression could vary according to genotype, viral load, liver steatosis, BMI and response to therapy and to determine whether the addition of selective COX inhibitors could have a rationale in increasing the efficacy of antiviral therapy. METHODS: We used 35 formalin-fixed, paraffin-embedded liver tissue samples obtained by needle biopsy from patients with CHC (17F/18M) with one of two types of genotype (1b and 3a). The presence of COX-1 and COX-2 in the cytoplasm of hepatocyt…

MaleSteatosisGene ExpressionHepacivirusChronic hepatitis CGastroenterologychemistry.chemical_compoundmedicine.diagnostic_testFatty liverGastroenterologyMiddle AgedImmunohistochemistryRecombinant ProteinsCyclooxygenaseTreatment OutcomeLiverRNA ViralFemaleViral loadmedicine.drugAdultmedicine.medical_specialtyAdolescentGenotypeCombination therapyAlpha interferonInterferon alpha-2Antiviral AgentsInternal medicineRibavirinBiopsymedicineHumansInterferon alfaAgedStaining and LabelingHepatologybusiness.industryRibavirinInterferon-alphaHepatitis C AntibodiesHepatitis C Chronicmedicine.diseasechemistryCyclooxygenase 2Prostaglandin-Endoperoxide SynthasesImmunologyCyclooxygenase 1SteatosisbusinessInterferon-αEuropean Journal of Gastroenterology &amp; Hepatology
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Fatty liver in familial hypobetalipoproteinemia: triglyceride assembly into VLDL particles is affected by the extent of hepatic steatosis

2003

Familial hypobetalipoproteinemia (FHBL) subjects may develop fatty liver. Liver fat was assessed in 21 FHBL with six different apolipoprotein B (apoB) truncations (apoB-4 to apoB-89) and 14 controls by magnetic resonance spectroscopy (MRS). Liver fat percentages were 16.7 +/- 11.5 and 3.3 +/- 2.9 (mean +/- SD) (P = 0.001). Liver fat percentage was positively correlated with body mass index, waist circumference, and areas under the insulin curves of 2 h glucose tolerance tests, suggesting that obesity may affect the severity of liver fat accumulation in both groups. Despite 5-fold differences in liver fat percentage, mean values for obesity and insulin indexes were similar. Thus, for similar…

MaleVery low-density lipoproteinSettore MED/09 - Medicina InternaApolipoprotein Bmedicine.medical_treatmentPalmitic AcidLipoproteins VLDLSeverity of Illness IndexTriglyceridevery low density lipoprotein assemblyBiochemistryBody Mass IndexMagnetic resonence spectroscopy; Nonalcoholic fatty liver; Nonesterified fatty acids; Very low density lipoprotein assembly; Adult; Body Mass Index; Dietary Fats; Fatty Liver; Female; Glucose Tolerance Test; Humans; Hypobetalipoproteinemias; Insulin Resistance; Lipoproteins VLDL; Liver Diseases; Male; Middle Aged; Obesity; Palmitic Acids; Severity of Illness Index; Triglycerides; EndocrinologyHypobetalipoproteinemiaschemistry.chemical_compoundEndocrinologyDietary FatGlucose tolerance testmedicine.diagnostic_testbiologyChemistryLiver DiseasesLiver DiseaseFatty liverMiddle AgedFemalemagnetic resonence spectroscopyHumanAdultmedicine.medical_specialtyPalmitic Acidsnonesterified fatty acidsQD415-436Internal medicinemedicinenonalcoholic fatty liverHumansObesityTriglyceridesTriglycerideInsulinNonesterified fatty acidCell BiologyGlucose Tolerance Testmedicine.diseaseDietary FatsFatty LiverEndocrinologybiology.proteinHypobetalipoproteinemiaInsulin ResistanceSteatosisHypobetalipoproteinemiaJournal of Lipid Research
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