Search results for "Striatum"

showing 10 items of 178 documents

''Comparative Effect of Treadmill Exercise on Mature BDNF Production in Control versus Stroke Rats''

2012

Quirie, Aurore | Hervieu, Marie | Garnier, Philippe | Demougeot, Celine | Mossiat, Claude | Bertrand, Nathalie | Martin, Alain | Marie, Christine | Prigent-Tessier, Anne; International audience; ''Physical exercise constitutes an innovative strategy to treat deficits associated with stroke through the promotion of BDNF-dependent neuroplasticity. However, there is no consensus on the optimal intensity/duration of exercise. In addition, whether previous stroke changes the effect of exercise on the brain is not known. Therefore, the present study compared the effects of a clinically-relevant form of exercise on cerebral BDNF levels and localization in control versus stroke rats. For this purpo…

MaleBEHAVIORAL RECOVERYTropomyosin receptor kinase BBiochemistryHippocampus0302 clinical medicineNerve Growth FactorHippocampus (mythology)StrokeCerebral Cortex0303 health sciencesNeuronal PlasticityMultidisciplinaryMOTOR RECOVERYQRTRKBNeurochemistryStrokemedicine.anatomical_structureNeurologyOrgan SpecificityCerebral cortex[ SCCO.NEUR ] Cognitive science/NeuroscienceMedicineNeurochemicalsmedicine.symptomResearch ArticleEXPRESSIONmedicine.medical_specialtyHIPPOCAMPAL PLASTICITYCORTEXCerebrovascular DiseasesAnimal TypesScienceBlotting WesternSynaptophysinEnzyme-Linked Immunosorbent AssayPhysical exerciseCONTROLLED-TRIALLesion03 medical and health sciencesPhysical Conditioning AnimalInternal medicineNeuroplasticitymedicineAnimalsLaboratory AnimalsSports and Exercise MedicineProtein PrecursorsRats WistarBiologyIschemic Stroke030304 developmental biologyBrain-derived neurotrophic factorbusiness.industry[SCCO.NEUR]Cognitive science/NeuroscienceBrain-Derived Neurotrophic FactorTRKB''AXONAL-TRANSPORTmedicine.diseaseCorpus StriatumRatsDisease Models AnimalEndocrinology''FOCAL BRAIN ISCHEMIAnervous systemFOCAL BRAIN ISCHEMIAExercise TestPhysical therapyBlood VesselsVeterinary ScienceEndothelium Vascularbusiness030217 neurology & neurosurgerySynaptic PlasticityNeuroscienceNEUROTROPHIC FACTOR
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Rewarding effects and reinstatement of MDMA-induced CPP in adolescent mice.

2007

Although the rewarding effects of 3,4-methylenedioxy-metamphetamine (MDMA) have been demonstrated in self-administration and conditioned place preference (CPP) procedures, its addictive potential (ie, the vulnerability to relapse, measured by its ability to induce reinstatement of an extinguished response), remains poorly understood. In this study, the effects of MDMA (5, 10, and 20 mg/kg) on the acquisition, extinction and reinstatement of CPP were evaluated in mice, using two different protocols during acquisition of CPP. In the first experiment, animals were trained using a two-session/day schedule (MDMA and saline for 4 consecutive days), whereas in the second experiment, they were trai…

MaleBiogenic AminesReinforcement ScheduleN-Methyl-34-methylenedioxyamphetamineStriatumPharmacologyExtinction Psychologicalchemistry.chemical_compoundMiceRewardDopaminemental disordersmedicineAnimalsNeurotransmitterPharmacologyBrain ChemistryAnalysis of VarianceBehavior AnimalDose-Response Relationship DrugMDMAExtinction (psychology)Conditioned place preferenceRatsPsychiatry and Mental healthchemistryAnimals NewbornAnesthesiaHallucinogensConditioning OperantSerotoninAnalysis of variancePsychologypsychological phenomena and processesmedicine.drugNeuropsychopharmacology : official publication of the American College of Neuropsychopharmacology
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Mice lacking α-synuclein display functional deficits in the nigrostriatal dopamine system

2000

alpha-Synuclein (alpha-Syn) is a 14 kDa protein of unknown function that has been implicated in the pathophysiology of Parkinson's disease (PD). Here, we show that alpha-Syn-/- mice are viable and fertile, exhibit intact brain architecture, and possess a normal complement of dopaminergic cell bodies, fibers, and synapses. Nigrostriatal terminals of alpha-Syn-/- mice display a standard pattern of dopamine (DA) discharge and reuptake in response to simple electrical stimulation. However, they exhibit an increased release with paired stimuli that can be mimicked by elevated Ca2+. Concurrent with the altered DA release, alpha-Syn-/- mice display a reduction in striatal DA and an attenuation of …

MaleCalbindinsNeuroscience(all)DopamineDopamine AgentsLong-Term PotentiationPresynaptic TerminalsSynucleinsGene ExpressionGlutamic AcidSubstantia nigraNerve Tissue ProteinsNeurotransmissionMotor ActivityHippocampusSynaptic TransmissionReuptakechemistry.chemical_compoundMiceS100 Calcium Binding Protein GDopamineDopaminergic CellmedicineAnimalsAutoreceptorsAlpha-synucleinMice KnockoutNeuronsGeneral NeuroscienceRab3A GTP-Binding ProteinCorpus Striatumrab3A GTP-Binding Proteinnervous system diseasesMice Inbred C57BLSubstantia NigraAmphetaminechemistrynervous systemalpha-SynucleinCalciumFemaleBeta-synucleinNeuroscienceLocomotionmedicine.drug
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Enhanced Functional Activity of the Cannabinoid Type-1 Receptor Mediates Adolescent Behavior.

2015

Adolescence is characterized by drastic behavioral adaptations and comprises a particularly vulnerable period for the emergence of various psychiatric disorders. Growing evidence reveals that the pathophysiology of these disorders might derive from aberrations of normal neurodevelopmental changes in the adolescent brain. Understanding the molecular underpinnings of adolescent behavior is therefore critical for understanding the origin of psychopathology, but the molecular mechanisms that trigger adolescent behavior are unknown. Here, we hypothesize that the cannabinoid type-1 receptor (CB1R) may play a critical role in mediating adolescent behavior because enhanced endocannabinoid (eCB) sig…

MaleCannabinoid receptorAdolescentmedicine.medical_treatmentIn Vitro TechniquesImpulsivityMediatorRisk-TakingCocaineReceptor Cannabinoid CB1Sulfur IsotopesmedicineAnimalsHumansMaze LearningRadionuclide ImagingSocial BehaviorCannabinoid Receptor AntagonistsBehavior AnimalGeneral NeuroscienceNovelty seekingAge FactorsBrainArticlesPhenotypeEndocannabinoid systemCorpus StriatumRats Inbred F344RatsAdolescent BehaviorGuanosine 5'-O-(3-Thiotriphosphate)Models AnimalMutationExploratory BehaviorCannabinoid receptor antagonistCannabinoidmedicine.symptomRats TransgenicPsychologyNeuroscienceEndocannabinoidsThe Journal of neuroscience : the official journal of the Society for Neuroscience
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A restricted population of CB1 cannabinoid receptors with neuroprotective activity.

2014

The CB1 cannabinoid receptor, the main molecular target of endocannabinoids and cannabis active components, is the most abundant G protein-coupled receptor in the mammalian brain. Of note, CB1 receptors are expressed at the synapses of two opposing (i.e., GABAergic/inhibitory and glutamatergic/excitatory) neuronal populations, so the activation of one and/or another receptor population may conceivably evoke different effects. Despite the widely reported neuroprotective activity of the CB1 receptor in animal models, the precise pathophysiological relevance of those two CB1 receptor pools in neurodegenerative processes is unknown. Here, we first induced excitotoxic damage in the mouse brain b…

MaleCannabinoid receptorPopulationNeurotoxinsExcitotoxicityGlutamic AcidBiologymedicine.disease_causeNeuroprotectionGlutamatergicMiceOrgan Culture TechniquesReceptor Cannabinoid CB1medicineAnimalsHumansGABAergic NeuronsReceptoreducationCaenorhabditis elegans ProteinsAgedCerebral CortexMice KnockoutNeuronseducation.field_of_studyMultidisciplinaryIntegrasesmusculoskeletal neural and ocular physiologyNeurodegenerative DiseasesBiological SciencesMiddle AgedReceptors GABA-AEndocannabinoid systemCorpus Striatumnervous systemGABAergiclipids (amino acids peptides and proteins)FemaleNeurosciencepsychological phenomena and processesEndocannabinoidsSynaptosomesProceedings of the National Academy of Sciences of the United States of America
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Lateral differences in GABA binding sites in rat brain.

1988

An asymmetric distribution of GABA binding sites was found in the cerebral cortex, hippocampus, cerebellar hemispheres, striatum, and thalamus. Higher levels of [3H]GABA binding were observed in the left-side of most brain areas and in a greater percentage of adult rats, but the opposite asymmetry was found in the thalamus. A similar left-right difference in cerebral hemispheres was also found in five day-old rats, suggesting the genetic predetermination of asymmetry.

MaleCerebellumThalamusCentral nervous systemHippocampusStriatumBiochemistryHippocampusFunctional LateralityCellular and Molecular NeurosciencemedicineAnimalsBinding siteCerebral CortexBinding SitesChemistryBrainRats Inbred StrainsGeneral MedicineReceptors GABA-AhumanitiesCorpus StriatumRatsmedicine.anatomical_structurenervous systemCerebral cortexCerebral hemisphereNeuroscienceNeurochemical research
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Oxytocin reverses ethanol consumption and neuroinflammation induced by social defeat in male mice

2020

Abstract Oxytocin (OXT) modulates social interactions, attenuates stressful responses and can decrease drug-seeking and taking behaviors. In previous studies, we observed that social defeat (SD) induced a long-lasting increase in ethanol intake and neuroinflammation in male mice. We also know that OXT blocks the increase in cocaine reward induced by SD. Therefore, in the present study we aimed to evaluate the effect of 1 mg/kg of OXT administered 30 min before each episode of SD on ethanol consumption and the neuroinflammatory response in adult male mice. Three weeks after the last SD, mice underwent oral ethanol self-administration (SA) procedure, and striatal levels of the two chemokines …

MaleChemokinemedicine.medical_specialtyAlcohol DrinkingSelf AdministrationOxytocinSocial DefeatSocial defeatMice03 medical and health sciencesBehavioral Neurosciencechemistry.chemical_compound0302 clinical medicineEndocrinologyNeuritisRewardInternal medicineAnimalsMedicineCX3CL1NeuroinflammationSocial stressMotivationEthanolEthanolbiologyChemokine CX3CL1Endocrine and Autonomic Systemsbusiness.industryChemokine CXCL12Corpus Striatum030227 psychiatryEndocrinologyOxytocinchemistrybiology.proteinbusinessSelf-administrationStress Psychologicalhormones hormone substitutes and hormone antagonists030217 neurology & neurosurgerymedicine.drugHormones and Behavior
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Distinct patterns of Fos immunoreactivity in striatum and hippocampus induced by different kinds of novelty in mice.

2010

In this study the immediate-early gene Fos was used to investigate the response to different novel stimuli in a wide array of brain regions including the hippocampus, the rhinal cortex, the frontal cortex and different components of the striatal complex. Independent groups of CD-1 mice were exposed to three different novelty conditions: (1) novel environment (empty open field); (2) complex novel environment (i.e. open field containing objects); and (3) identity-based detection of novel objects. We observed that a complex novel environment and a knowledge-based novelty modulated Fos levels in both the dorsal and the ventral components of the striatum, while Fos immunoreactivity in the medial…

MaleCognitive NeuroscienceRhinal cortexHippocampusExperimental and Cognitive PsychologyCell CountStriatumEnvironmentMotor ActivityHippocampusOpen fieldTemporal lobeBehavioral NeuroscienceMiceBasal gangliaAnimalsPrefrontal cortexmedial temporal lobe; striatum; prefrontal cortex; object novelty; environmental noveltyNeuronsAnalysis of VarianceBehavior AnimalNoveltyImmunohistochemistryCorpus StriatumPsychologyNeuroscienceProto-Oncogene Proteins c-fosNeurobiology of learning and memory
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Effects of scopolamine on dopamine neurons in the substantia nigra : role of the pedunculopontine tegmental nucleus

2009

Previous neurochemical and behavioral studies suggest that muscarinic receptor antagonism has an excitatory effect on the nigrostriatal dopamine (DA) system. Using in vivo extracellular single unit recording, this study examined whether blockade of the muscarinic receptor by scopolamine alters the firing properties of DA neurons in the substantia nigra (SN). Scopolamine was administered either systemically or locally to DA neurons using microiontophoresis. Surprisingly, scopolamine did not cause any significant change in either the firing rate or pattern of the spontaneously active DA neurons. However, systemic injection of scopolamine significantly increased the number of active DA neurons…

MaleDopamineParkinson's diseaseScopolamineAction PotentialsSubstantia nigraMuscarinic AntagonistsStriatumelectrophysiology microiontophoresisSettore BIO/09 - FisiologiaRats Sprague-DawleyCellular and Molecular NeuroscienceDopamineBasal gangliaMuscarinic acetylcholine receptorPedunculopontine Tegmental NucleusmedicineSubstantia nigraAnimalsPedunculopontine Tegmental NucleusNeuronsAnalysis of VarianceDose-Response Relationship DrugChemistryDrug Administration RoutesIontophoresisAcetylcholineRatsSubstantia Nigranervous systemAutoreceptorBasal gangliaNeuroscienceAcetylcholinemedicine.drug
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Alterations in striatal neuropeptide mRNA produced by repeated administration of L-DOPA, ropinirole or bromocriptine correlate with dyskinesia induct…

2002

Chronic administration of L-DOPA to MPTP-treated common marmosets induces marked dyskinesia while repeated administration of equivalent antiparkisonian doses of ropinirole and bromocriptine produces only mild involuntary movements. The occurrence of dyskinesia has been associated with an altered balance between the direct and indirect striatal output pathways. Using in situ hybridisation histochemistry, we now compare the effects of these drug treatments on striatal preproenkephalin-A (PPE-A) and adenosine A(2a) receptor mRNA expression as markers of the indirect pathway and striatal preprotachykinin (PPT) mRNA and preproenkephalin-B (PPE-B, prodynorphin) mRNA expression as markers of the d…

MaleDyskinesia Drug-Inducedmedicine.medical_specialtyIndolesCaudate nucleusStriatumIndirect pathway of movementAntiparkinson AgentsLevodopachemistry.chemical_compoundDopamine Uptake InhibitorsParkinsonian DisordersTachykininsInternal medicineNeural PathwaysmedicineAnimalsheterocyclic compoundsRNA MessengerProtein PrecursorsBromocriptineGeneral NeuroscienceMPTPPutamenNeuropeptidesReceptors Purinergic P1CallithrixEnkephalinsMazindoldopamine agonists peptide mRNAs L-DOPA 1-methyl-4-phenyl-1236-tetrahydropyridine primates dyskinesiaBromocriptinenervous system diseasesNeostriatumRopiniroleEndocrinologynervous systemchemistryDyskinesiaSettore BIO/14 - FarmacologiaFemalemedicine.symptommedicine.drugNeuroscience
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