Search results for "Structure-Activity Relationship"

showing 10 items of 743 documents

Vanilloid Derivatives as Tyrosinase Inhibitors Driven by Virtual Screening-Based QSAR Models

2010

A number of vanilloids have been tested as tyrosinase inhibitors using Ligand-Based Virtual Screening (LBVS) driven by QSAR (Quantitative Structure-Activity Relationship) models as the multi-agent classification system. A total of 81 models were used to screen this family. Then, a preliminary cluster analysis of the selected chemicals was carried out based on their bioactivity to detect possible similar substructural features among these compounds and the active database used in the QSAR model construction. The compounds identified were tested in vitro to corroborate the results obtained in silico. Among them, two chemicals, isovanillin (K(M) (app) = 1.08 mM) near to kojic acid (reference d…

Quantitative structure–activity relationshipStereochemistryTyrosinaseIn silicoQuantitative Structure-Activity RelationshipPharmaceutical ScienceIsovanillinModels BiologicalSkin DiseasesVanilloidsAnalytical Chemistrychemistry.chemical_compoundCluster AnalysisHumansEnvironmental ChemistryComputer SimulationEnzyme InhibitorsSpectroscopyVirtual screeningMonophenol MonooxygenaseReference drugCombinatorial chemistrychemistryBenzaldehydesDrug DesignKojic acidAlgorithmsDrug Testing and Analysis
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Atom, atom-type, and total nonstochastic and stochastic quadratic fingerprints: a promising approach for modeling of antibacterial activity.

2005

The TOpological MOlecular COMputer Design (TOMOCOMD-CARDD) approach has been introduced for the classification and design of antimicrobial agents using computer-aided molecular design. For this propose, atom, atom-type, and total quadratic indices have been generalized to codify chemical structure information. In this sense, stochastic quadratic indices have been introduced for the description of the molecular structure. These stochastic fingerprints are based on a simple model for the intramolecular movement of all valence-bond electrons. In this work, a complete data set containing 1006 antimicrobial agents is collected and presented. Two structure-based antibacterial activity classificat…

Quantitative structure–activity relationshipStochastic ProcessesMolecular modelDatabases FactualChemistryOrganic ChemistryClinical BiochemistryMolecular ConformationPharmaceutical ScienceAtom (order theory)Quantitative Structure-Activity RelationshipModels TheoreticalLinear discriminant analysisBiochemistryAnti-Bacterial AgentsSet (abstract data type)Quadratic equationSimple (abstract algebra)Drug DiscoveryMolecular MedicineComputer SimulationBiological systemMolecular BiologyAntibacterial agentBioorganicmedicinal chemistry
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Harmonization of QSAR Best Practices and Molecular Docking Provides an Efficient Virtual Screening Tool for Discovering New G-Quadruplex Ligands

2015

Telomeres and telomerase are key players in tumorogenesis. Among the various strategies proposed for telomerase inhibition or telomere uncapping, the stabilization of telomeric G-quadruplex (G4) structures is a very promising one. Additionally, G4 stabilizing ligands also act over tumors mediated by the alternative elongation of telomeres. Accordingly, the discovery of novel compounds able to act on telomeres and/or inhibit the telomerase enzyme by stabilizing DNA telomeric G4 structures as well as the development of approaches efficiently prioritizing such compounds constitute active areas of research in computational medicinal chemistry and anticancer drug discovery. In this direction, we…

Quantitative structure–activity relationshipTelomeraseGeneral Chemical EngineeringDrug Evaluation PreclinicalQuantitative Structure-Activity RelationshipComputational biologyLibrary and Information SciencesBiologyG-quadruplexCrystallography X-RayLigandsMolecular Docking Simulationchemistry.chemical_compoundDrug DiscoveryHumansCell ProliferationGeneticsVirtual screeningMolecular StructureDrug discoveryQSARGeneral ChemistryFibroblastsTelomereComputer Science ApplicationsTelomereG-QuadruplexesMolecular Docking SimulationchemistryAcridinesDNAHeLa Cells
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Virtual darwinian drug design: QSAR inverse problem, virtual combinatorial chemistry, and computational screening.

2001

The generation of diversity and its further selection by an external system is a common mechanism for the evolution of the living species and for the current drug design methods. This assumption allows us to label the methods based on generation and selection of molecular diversity as "Darwinian" ones, and to distinguish them from the structure-based, structure-modulation approaches. An example of a Darwinian method is the inverse QSAR. It consists of the computational generation of candidate chemical structures and their selection according to a previously established QSAR model. New trends in the field of combinatorial chemical syntheses comprise the concepts of virtual combinatorial synt…

Quantitative structure–activity relationshipVirtual screeningCombinatorial Chemistry TechniquesChemistryOrganic ChemistryQuantitative Structure-Activity RelationshipGeneral MedicineInverse problemCombinatorial chemistryBiological EvolutionField (computer science)Computer Science ApplicationsDrug DesignDrug DiscoveryGraph (abstract data type)Combinatorial Chemistry TechniquesComputer SimulationDesign methodsSelection (genetic algorithm)Combinatorial chemistryhigh throughput screening
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Modeling Natural Anti-Inflammatory Compounds by Molecular Topology

2011

One of the main pharmacological problems today in the treatment of chronic inflammation diseases consists of the fact that anti-inflammatory drugs usually exhibit side effects. The natural products offer a great hope in the identification of bioactive lead compounds and their development into drugs for treating inflammatory diseases. Computer-aided drug design has proved to be a very useful tool for discovering new drugs and, specifically, Molecular Topology has become a good technique for such a goal. A topological-mathematical model, obtained by linear discriminant analysis, has been developed for the search of new anti-inflammatory natural compounds. An external validation obtained with …

Quantitative structure–activity relationshiplinear discriminant analysismedicine.drug_classAnti-Inflammatory AgentsQuantitative Structure-Activity RelationshipComputational biologyCatalysisAnti-inflammatoryNatural (archaeology)ArticleModel validationInorganic Chemistrylcsh:ChemistrymedicinePhysical and Theoretical ChemistryMolecular Biologylcsh:QH301-705.5Spectroscopynaturalanti-inflammatoryVirtual screeningBiological ProductsChemistryOrganic ChemistryExternal validationGeneral MedicineMolecular Topologyvirtual screeningCombinatorial chemistryComputer Science Applicationslcsh:Biology (General)lcsh:QD1-999Models ChemicalMolecular Topology; virtual screening; natural; anti-inflammatory; linear discriminant analysisIdentification (biology)Molecular topologyInternational Journal of Molecular Sciences
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Monoamine Oxidase Inhibition by β-Carbolines: A Quantum Chemical Approach

1979

Monoamine oxidase inhibition by beta-carboline derivatives is related to the energy change, delta E, arising from complex formation between the inhibitor and the enzyme. The energy change was expressed in terms of electronic indexes, which were estimated for a set of aromatic beta-carbolines. The electronic indexes were correlated to the experimental activity indexes by a simplified quantum chemical perturbational treatment with a multiple regression procedure. A characteristic structure for the inhibitor-enzyme complex was derived from the correlation. The molecules are linked by two kinds of bond. One involves the lone pyridine nitrogen pair of beta-carbolines; the other is due to a pi-el…

Quantum chemicalIndole testchemistry.chemical_classificationTryptamineIndolesMonoamine Oxidase InhibitorsChemistryMonoamine oxidaseStereochemistryPharmaceutical ScienceModels BiologicalStructure-Activity Relationshipchemistry.chemical_compoundNon-competitive inhibitionEnzymePyridineQuantum TheoryThermodynamicsMoleculeCarbolinesJournal of Pharmaceutical Sciences
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Aza-isoindolo and isoindolo-azaquinoxaline derivatives with antiproliferative activity

2015

Abstract Three new ring systems, pyrido[2′,3′:3,4]pyrrolo[1,2- a ]quinoxalines, pyrido[3′,2′:3,4]pyrrolo[1,2- a ]quinoxalines and pyrido[2′,3′:5,6]pyrazino[2,1- a ]isoindoles, were synthesized through an aza-substitution on the already active isoindolo-quinoxaline system and in particular in the position 7 or 4 of the isoindole moiety and in position 5 of the quinoxaline portion. All new compounds were screened by the National Cancer Institute (Bethesda, MD) against a panel of 60 human tumor cell lines. Biological results of the most active derivatives, with pGI 50 values between 7.09 and 7.27, confirmed the importance of the presence of methoxy substituents for biological activity. The ant…

QuinoxalineIsoindolesAzaisoindolo-quinoxalinesStereochemistryAntiproliferative activity; Apoptosis; Azaisoindolo-quinoxalines; DNA interaction; Isoindolo-azaquinoxalines; Quinoxalines; Antineoplastic Agents; Apoptosis; Aza Compounds; Cell Line Tumor; Cell Proliferation; Dose-Response Relationship Drug; Drug Screening Assays Antitumor; Humans; Isoindoles; Molecular Structure; Quinoxalines; Structure-Activity Relationship; Drug Discovery3003 Pharmaceutical Science; Organic Chemistry; Pharmacology; Medicine (all)ApoptosisAntineoplastic AgentsAntiproliferative activityIsoindolesRing (chemistry)Drug Screening AssaysCell LineDose-Response Relationshipchemistry.chemical_compoundStructure-Activity RelationshipQuinoxalineCell Line TumorQuinoxalinesDrug DiscoverymedicineMoietyHumansAntiproliferative activity; Apoptosis; Azaisoindolo-quinoxalines; DNA interaction; Isoindolo-azaquinoxalines; Quinoxalines; Drug Discovery3003 Pharmaceutical Science; Organic Chemistry; PharmacologyCell ProliferationPharmacologyAza CompoundsAzaisoindolo-quinoxalineTumorDose-Response Relationship DrugMolecular StructureDrug Discovery3003 Pharmaceutical ScienceMedicine (all)Organic ChemistryApoptosiBiological activityGeneral MedicineAntitumorCell cycleSettore CHIM/08 - Chimica FarmaceuticaDNA interactionSettore ING-IND/22 - Scienza E Tecnologia Dei MaterialiMechanism of actionchemistryIsoindolo-azaquinoxalineDrug Screening Assays Antitumormedicine.symptomDrugIsoindoleIsoindolo-azaquinoxalines
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Inhibition of GABA and benzodiazepine receptor binding by penicillins.

1980

Penicillins are thought to be GABA receptor antagonists. In order to determine the affinities of various penicillin derivatives for the GABA receptor, their potencies as inhibitors of specific [3H]GABA binding to rat brain membranes were investigated. All investigated penicillins inhibit specific [3H]GABA binding, with IC50 values ranging from 2 to 60 mM. The results are consistent with the assumption that penicillins are weak GABA receptor antagonists.

Receptors Cell SurfaceFlunitrazepamPenicillinsPharmacologygamma-Aminobutyric acidBenzodiazepinesStructure-Activity RelationshipGABA receptorpolycyclic compoundsmedicineStructure–activity relationshipAnimalsgamma-Aminobutyric AcidBenzodiazepine receptor bindingChemistryGeneral NeuroscienceBrainGABA receptor antagonistReceptors GABA-AAffinitiesRatsPenicillinnervous systemBiochemistryFlunitrazepammedicine.drugNeuroscience letters
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Non-genomic effects of progesterone on the signaling function of G protein-coupled receptors

1999

Progesterone at concentrations between 10 microM and 200 microM affected the calcium signaling evoked by ligand stimulation of G protein-coupled receptors expressed in several cell lines. At 160 microM progesterone the signaling of all receptors was completely abolished. The effect of progesterone was fast, reversible and was not prevented by cycloheximide indicating its non-genomic nature. Overall, the action of progesterone was more cell type-specific than receptor-specific. Our results are in contrast to a recent report [Grazzini, E., Guillon, G., Mouillac, B. and Zingg, H.H. (1998) Nature 392, 509-512] in which a direct high-affinity interaction between the oxytocin receptor and progest…

Receptors Neuropeptidemedicine.medical_specialtyReceptors VasopressinTime FactorsBiophysicsStimulationCHO CellsCycloheximideBiologyNon-genomic effectCalcium signalBiochemistryCell Linechemistry.chemical_compoundStructure-Activity RelationshipSpecies SpecificityStructural BiologyInternal medicineCricetinaeProgesterone receptorGeneticsmedicineTumor Cells CulturedAnimalsHumansG protein-coupled receptorCycloheximideReceptorMolecular BiologyProgesteroneG protein-coupled receptorCalcium signalingProtein Synthesis InhibitorsDose-Response Relationship DrugCell BiologyLigand (biochemistry)Oxytocin receptorKineticsEndocrinologychemistryReceptors OxytocinAnisotropyCalciumReceptors CholecystokininSignal TransductionFEBS Letters
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Structure-activity relationship studies of novel heteroretinoids: induction of apoptosis in the HL-60 cell line by a novel isoxazole-containing heter…

1999

In a search for retinoic acid receptor (RAR and RXR)-selective ligands, a series of isoxazole retinoids was synthesized and evaluated in vitro in transcriptional activation and competition binding assays for RARs and RXRs. In addition, these compounds were evaluated for their differentiating, cytotoxic, and apoptotic activities. In general, these derivatives showed scarcely any binding affinity and were not active in the transcriptional assay. However, among these isoxazole derivatives, the cis-isomer 14b was identified as a potent inducer of apoptosis, and its activity was found to be 6.5 and 4 times superior than that of 13-cis- and 9-cis-retinoic acids, respectively. On the other hand, c…

Receptors Retinoic AcidRetinoic acidCarboxylic AcidsApoptosisHL-60 CellsTretinoinRetinoid X receptorchemistry.chemical_compoundRetinoidsStructure-Activity RelationshipDrug DiscoveryStructure–activity relationshipHumansIsoxazoleIsotretinoinAlitretinoinMolecular StructureBiological activityCell DifferentiationStereoisomerismIsoxazolesLigand (biochemistry)In vitroRetinoic acid receptorchemistryBiochemistryMolecular MedicineGranulocytesJournal of medicinal chemistry
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