Search results for "Superoxide dismutase"

showing 10 items of 296 documents

Two‐dimensional analysis of myocardial protein expression following myocardial ischemia and reperfusion in rabbits

2002

Myocardial ischemia and reperfusion injury (MI/R) can be related to leukocyte activation with subsequent release of cytokines and oxygen derived free radicals. Activation of the complement system has been implicated in the pathogenesis of myocardial ischemia and reperfusion injury. Inflammatory injury will subsequently result in cellular activation and protein synthesis. In the present study we analyzed the myocardial protein expression and its pattern following myocardial ischemia and reperfusion, with and without complement inhibition with the synthetic serine protease inhibitor Futhan/nafamstat mesilate (FUT-175) known to inhibit classical and alternative complement pathway in a rabbit m…

MaleSerine Proteinase InhibitorsNecrosisProteomeNeutrophilsMyocardial IschemiaIschemiaMyocardial Reperfusion InjuryPharmacologyGuanidinesBiochemistrySuperoxide dismutaseNecrosisRandom AllocationComplement inhibitormedicineAnimalsElectrophoresis Gel Two-DimensionalCreatine KinaseMolecular BiologybiologySuperoxide DismutaseChemistryGene Expression ProfilingMyocardiumHemodynamicsProteinsalpha-Crystallin B Chainmedicine.diseaseBenzamidinesComplement systemBiochemistrybiology.proteinAlternative complement pathwayCreatine kinaseRabbitsmedicine.symptomReperfusion injuryPROTEOMICS
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Sex-dependent changes in the pulmonary vasoconstriction potential of newborn rats following short-term oxygen exposure

2012

Chronic exposure to supplemental oxygen (O(2)) induces lung damage and mortality in a sex-dependent manner. The effect of short-term hyperoxia on the newborn pulmonary vasculature is unknown but is, however, of clinical significance in the neonatal resuscitation context. We hypothesize that short-term hyperoxia has a sex-dependent effect on the pulmonary vasculature.Following 1-h 100% O(2) exposure, the pulmonary arteries and lung tissues of newborn rats were evaluated.Superoxide dismutase 3 (SOD3) expression in female pups' lungs was increased as compared with that in the lungs of male pups. As compared with air-treated pups, the response of male pups to thromboxane was increased by O(2), …

MaleTime FactorsHypertension PulmonaryHyperoxiaPulmonary ArteryRats Sprague-DawleySex FactorsSuperoxidesPeroxynitrous AcidHypoxic pulmonary vasoconstrictionAnimalsVasoconstrictor AgentsMedicineFamilial Primary Pulmonary Hypertensionskin and connective tissue diseasesOXYGEN EXPOSURELungrho-Associated KinasesDose-Response Relationship DrugSuperoxide Dismutasebusiness.industryFree Radical ScavengersHydrogen PeroxideRatsUp-RegulationEnzyme ActivationDisease Models AnimalOxidative StressAnimals NewbornVasoconstrictionAnesthesiaPediatrics Perinatology and Child HealthFemalesense organsbusinessPediatric Research
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Effect of a two-year supplementation with low doses of antioxidant vitamins and/or minerals in elderly subjects on levels of nutrients and antioxidan…

1997

Eighty-one elderly hospitalized subjects (65 years) were recruited for a double-blind placebo-controlled study to examine low dose supplementation of antioxidant vitamins and minerals on biological and functional parameters of free radical metabolism. Subjects were randomly assigned to one of the four treatment groups, daily receiving for 2 years: placebo group; mineral group: 20 mg zinc, 100 micrograms selenium; vitamin group: 120 mg vitamin C (Vit C), 6 mg beta-carotene (beta CA), 15 mg vitamin E (Vit E); mineral and vitamin group: Zn 20 mg, Se 100 micrograms, Vit C 120 mg, beta CA 6 mg, Vit E 15 mg.Fifty-seven subjects completed the study. A large frequency of Vit C, Zn and Se deficienci…

MaleVitaminmedicine.medical_specialtyErythrocytesAntioxidantmedicine.medical_treatmentNutritional StatusMedicine (miscellaneous)chemistry.chemical_elementAscorbic AcidThiobarbituric Acid Reactive SubstancesAntioxidantsSeleniumchemistry.chemical_compoundOral administrationInternal medicinemedicineHumansVitamin ENutritional Physiological PhenomenaAgedAged 80 and overGlutathione PeroxidaseMineralsNutrition and DieteticsVitamin CSuperoxide DismutaseVitamin EVitaminsMetabolismbeta CaroteneAscorbic acidGlutathioneZincEndocrinologychemistryBiochemistryFemaleSeleniumJournal of the American College of Nutrition
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Effect of xanthine oxidase-generated extracellular superoxide on skeletal muscle force generation

2009

Skeletal muscle contractions increase superoxide anion in skeletal muscle extracellular space. We tested the hypotheses that 1) after an isometric contraction protocol, xanthine oxidase (XO) activity is a source of superoxide anion in the extracellular space of skeletal muscle and 2) the increase in XO-derived extracellular superoxide anion during contractions affects skeletal muscle contractile function. Superoxide anion was monitored in the extracellular space of mouse gastrocnemius muscles by following the reduction of cytochrome c in muscle microdialysates. A 15-min protocol of nondamaging isometric contractions increased the reduction of cytochrome c in microdialysates, indicating an …

MaleXanthine Oxidasemedicine.medical_specialtyPhysiologyOxypurinolfree radicalsSuperoxide dismutaseExtensor digitorum longus muscleMice03 medical and health sciencesGastrocnemius musclechemistry.chemical_compound0302 clinical medicineSuperoxidescontractile functionIsometric ContractionPhysiology (medical)Internal medicinemedicineAnimalsMuscle Skeletal030304 developmental biologySoleus muscle0303 health sciencesexercisebiologyMuscle fatigueSuperoxide DismutaseChemistrySuperoxideCytochromes cSkeletal muscleArticlesmusculoskeletal systemElectric StimulationMice Inbred C57BLmedicine.anatomical_structureEndocrinologyBiochemistryModels AnimalMuscle Fatiguebiology.proteinmedicine.symptomExtracellular Space030217 neurology & neurosurgeryMuscle ContractionMuscle contractionAmerican Journal of Physiology-Regulatory, Integrative and Comparative Physiology
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TORC1 Inhibition by Rapamycin Promotes Antioxidant Defences in a Drosophila Model of Friedreich’s Ataxia

2015

Friedreich's ataxia (FRDA), the most common inherited ataxia in the Caucasian population, is a multisystemic disease caused by a significant decrease in the frataxin level. To identify genes capable of modifying the severity of the symptoms of frataxin depletion, we performed a candidate genetic screen in a Drosophila RNAi-based model of FRDA. We found that genetic reduction in TOR Complex 1 (TORC1) signalling improves the impaired motor performance phenotype of FRDA model flies. Pharmacologic inhibition of TORC1 signalling by rapamycin also restored this phenotype and increased the lifespan and ATP levels. Furthermore, rapamycin reduced the altered levels of malondialdehyde + 4-hydroxyalke…

Malelcsh:MedicineGene Expressionmedicine.disease_causeAntioxidantsAnimals Genetically ModifiedAdenosine Triphosphate0302 clinical medicineRNA interferenceIron-Binding ProteinsMalondialdehydeDrosophila Proteinslcsh:ScienceAconitate HydrataseGenetics0303 health sciencesMultidisciplinaryReverse Transcriptase Polymerase Chain ReactionGlutathione3. Good healthCell biologyDrosophila melanogasterRNA Interferencemedicine.symptomImmunosuppressive AgentsDrosophila ProteinResearch ArticleAtaxiaLongevityMotor ActivityBiologyAconitase03 medical and health sciencesmedicineAnimalsHumans030304 developmental biologySirolimusAldehydesSuperoxide Dismutaselcsh:RAutophagyRepressor ProteinsDisease Models AnimalOxidative StressFriedreich AtaxiaFrataxinbiology.proteinlcsh:Q030217 neurology & neurosurgeryOxidative stressTranscription FactorsGenetic screenPLOS ONE
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Iridoid extracts from Ajuga iva increase the antioxidant enzyme activities in red blood cells of rats fed a cholesterol-rich diet.

2010

The lyophilized aqueous extract of Ajuga iva (Ai) is able to reduce oxidative stress, which may prevent lipid peroxidation in hypercholesterolemic rats. Iridoids (I) were isolated from Ai. We hypothesized that the antioxidant defense status in red blood cells (RBC) and tissues in rats fed a cholesterol-rich diet and treated with Ai may be correlated to these compounds. Male Wistar rats (n = 32) weighing 120 +/- 5 g were fed a diet containing 1% cholesterol for 15 days. After this phase, hypercholesterolemic (HC) rats were divided into groups, fed the same diet, and received either the same or different doses (5, 10, or 15 mg/kg body weight by intraperitoneal injection) of I for 15 days. Com…

Malemedicine.medical_specialtyAntioxidantErythrocytesEndocrinology Diabetes and Metabolismmedicine.medical_treatmentGlutathione reductaseHypercholesterolemiaAjugamedicine.disease_causeThiobarbituric Acid Reactive SubstancesAntioxidantsLipid peroxidationSuperoxide dismutaseCholesterol Dietarychemistry.chemical_compoundEndocrinologyInternal medicinemedicineTBARSAnimalsIridoidsRats Wistarchemistry.chemical_classificationNutrition and DieteticsbiologyDose-Response Relationship DrugPlant ExtractsGlutathione peroxidaseAnticholesteremic AgentsMusclesMyocardiumGlutathioneRatsOxidative StressEndocrinologychemistryLiverbiology.proteinOxidative stressPhytotherapyNutrition research (New York, N.Y.)
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Mitochondria from females exhibit higher antioxidant gene expression and lower oxidative damage than males

2003

We have investigated the differential mitochondrial oxidative stress between males and females to understand the molecular mechanisms enabling females to live longer than males. Mitochondria are a major source of free radicals in cells. Those from female rats generate half the amount of peroxides than those of males. This does not occur in ovariectomized animals. Estrogen replacement therapy prevents the effect of ovariectomy. Mitochondria from females have higher levels of reduced glutathione than those from males. Those from ovariectomized rats have similar levels to males, and estrogen therapy prevents the fall in glutathione levels that occurs in ovariectomized animals. Oxidative damage…

Malemedicine.medical_specialtyAntioxidantOvariectomymedicine.medical_treatmentMitochondria LiverMitochondrionBiologymedicine.disease_causeDNA MitochondrialBiochemistryAntioxidantsGene Expression Regulation EnzymologicSuperoxide dismutasechemistry.chemical_compoundRNA Ribosomal 16SPhysiology (medical)Internal medicinemedicineAnimalsRats WistarDNA Primerschemistry.chemical_classificationGlutathione PeroxidaseReverse Transcriptase Polymerase Chain ReactionSuperoxide DismutaseGlutathione peroxidaseEstrogensGlutathioneGlutathionePeroxidesRatsOxygenOxidative StressEndocrinologychemistryOvariectomized ratbiology.proteinRNAFemaleDismutaseReactive Oxygen SpeciesOxidation-ReductionOxidative stressFree Radical Biology and Medicine
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Antioxidant enzyme activities and the production of MDA and 8-oxo-dG in chronic lymphocytic leukemia.

2001

Abstract Chronic lymphocytic leukemia (CLL) is a neoplastic disease susceptible to antioxidant enzyme alterations and oxidative stress. We have examined the activities of superoxide dismutase (SOD), catalase (CAT), and glutathione peroxidase (GPx), and the oxidized/reduced glutathione (GSSG/GSH) ratio together with the levels of malondialdehyde (MDA) and 8-oxo-2′-deoxyguanosine (8-oxo-dG) in lymphocytes of CLL patients and compared them with those of normal subjects of the same age. SOD and CAT activity decreased in CLL lymphocytes while GPx activity increased. GSH content of CLL lymphocytes also increased, and GSSG concentration remained constant. Thus, a reduced GSSG/GSH ratio was obtaine…

Malemedicine.medical_specialtyAntioxidantmedicine.medical_treatmentChronic lymphocytic leukemiamedicine.disease_causeBiochemistryAntioxidantsSuperoxide dismutasechemistry.chemical_compoundhemic and lymphatic diseasesPhysiology (medical)Internal medicineMalondialdehydemedicineHumansLymphocytesAgedchemistry.chemical_classificationGlutathione PeroxidasebiologySuperoxide DismutaseGlutathione peroxidaseDeoxyguanosineGlutathioneDNA NeoplasmMiddle AgedMalondialdehydemedicine.diseaseCatalaseGlutathioneLeukemia Lymphocytic Chronic B-CellEndocrinologychemistryBiochemistryCatalase8-Hydroxy-2'-Deoxyguanosinebiology.proteinFemaleLipid PeroxidationOxidoreductasesOxidative stressDNA DamageFree radical biologymedicine
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Impact of cardiovascular risk factors on oxidative stress and DNA damage in a high risk Mediterranean population

2009

The impact of classic cardiovascular risk factors on oxidative stress status in a high-risk cardiovascular Mediterranean population of 527 subjects was estimated. Oxidative stress markers (malondialdehyde, 8-oxo-7′8′-dihydro-2′-deoxyguanosine, oxidized/reduced glutathione ratio) together with the activity of antioxidant enzyme triad (superoxide dismutase, catalase, glutathione peroxidase) were analysed in circulating mononuclear blood cells. Malondialdehyde, oxidized glutathione and the ratio of oxidized to reduced glutathione were signifi cantly higher while catalase and glutathione peroxidase activities were signifi cantly lower in high cardiovascular risk participants than in controls. S…

Malemedicine.medical_specialtyAntioxidantmedicine.medical_treatmentPopulationmedicine.disease_causeBiochemistrySuperoxide dismutasechemistry.chemical_compoundSex FactorsRisk FactorsInternal medicineDiabetes mellitusmedicineHumanseducationAgedAged 80 and overchemistry.chemical_classificationeducation.field_of_studybiologyGlutathione peroxidaseAge FactorsGeneral MedicineGlutathioneMiddle AgedMalondialdehydemedicine.diseaseOxidative StressEndocrinologychemistryBiochemistryCardiovascular DiseasesSpainbiology.proteinFemaleOxidative stressDNA DamageFree Radical Research
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Decreased cell proliferation and higher oxidative stress in fibroblasts from Down Syndrome fetuses. Preliminary study

2013

Abstract Down Syndrome is the most common chromosomal disease and is also known for its decreased incidence of solid tumors and its progeroid phenotype. Cellular and systemic oxidative stress has been considered as one of the Down Syndrome phenotype causes. We correlated, in a preliminary study, the fibroblast proliferation rate and different cell proliferation key regulators, like Rcan1 and the telomere length from Down Syndrome fetuses, with their oxidative stress profile and the Ribonucleic acid and protein expression of the main antioxidant enzymes together with their activity. Increased oxidized glutathione/glutathione ratio and high peroxide production were found in our cell model. Th…

Malemedicine.medical_specialtyAntioxidantmedicine.medical_treatmentPrimary Cell CultureSuperoxide dismutasemedicine.disease_causeSuperoxide dismutasechemistry.chemical_compoundFetusSuperoxide Dismutase-1ThioredoxinsInternal medicineGlutaredoxinmedicineHumansThioredoxinMolecular BiologyGlutaredoxinsCell ProliferationSkinchemistry.chemical_classificationReactive oxygen speciesGlutathione PeroxidaseTelomere lengthbiologyGlutathione peroxidaseTelomere HomeostasisGlutathioneRcan1FibroblastsTelomereCatalaseGlutathioneProgeroidOxidative StressEndocrinologychemistryBiochemistryGene Expression Regulationbiology.proteinMolecular MedicineFemaleThioredoxinDown SyndromeOxidative stressSignal TransductionBiochimica et Biophysica Acta (BBA) - Molecular Basis of Disease
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