Search results for "Sympathomimetics"

showing 2 items of 22 documents

Day-night differences in the sensitivity of adrenoceptors in the Syrian hamster pineal gland: an in vivo iontophoretic study.

1989

Abstract Investigations on the regulation of pineal melatonin synthesis in the Syrian hamster revealed distinct differences compared to this well-understood mechanism in rat. E.g., a circadian profile of pineal norepinephrine (NE) is absent, there is no β-adrenoceptor sensitivity during daytime and adrenergic receptor supersensitivity is not easily achieved. To elucidate the action of NE on pineal receptor sites, the effects of iontophoretic application of adrenergic compounds on spontaneous electrical discharge rates of pinealocytes were investigated during day- and nighttime. Following application of either NE, isoproterenol or clonidine, cells were activated, inhibited or not affected. W…

SympathomimeticsMaleendocrine systemmedicine.medical_specialtyAdrenergic receptorHamsterAdrenergicAction PotentialsBiologyPineal GlandClonidinePinealocytePineal glandNorepinephrineInternal medicineCricetinaemedicineAnimalsCircadian rhythmReceptorMolecular BiologyMesocricetusGeneral NeuroscienceIsoproterenolCircadian RhythmReceptors Adrenergicmedicine.anatomical_structureEndocrinologyNeurology (clinical)Developmental BiologyBrain research
researchProduct

Adrenergic Stimulation of Cyclic GMP Formation Requires NO-Dependent Activation of Cytosolic Guanylate Cyclase in Rat Pinealocytes

1993

Cyclic GMP (cGMP) formation in rat pinealocytes is regulated through a synergistic dual receptor mechanism involving beta- and alpha 1-adrenergic receptors. The effects of NG-monomethyl-L-arginine (NMMA), which inhibits nitric oxide (NO) synthase and NO-mediated activation of cytosolic guanylate cyclase, and methylene blue (MB), which inhibits cytosolic guanylate cyclase, were investigated in an attempt to understand the role of NO in adrenergic cGMP formation. Both NMMA and MB inhibited beta-adrenergic stimulation of cGMP formation as well as alpha 1-adrenergic potentiation of beta-adrenergic stimulation of cGMP formation, whereas they had no effect in unstimulated pinealocytes. The inhibi…

medicine.medical_specialtyGUCY1B3AdrenergicStimulationArginineNitric OxidePineal GlandBiochemistryPinealocyteNitric oxideCellular and Molecular Neurosciencechemistry.chemical_compoundCytosolInternal medicinemedicineAnimalsSympathomimeticsCyclic GMPCells Culturedomega-N-MethylargininebiologyChemistryGUCY1A3Guanylate cyclase 2CRatsEnzyme ActivationMethylene BlueNitric oxide synthaseEndocrinologyGuanylate Cyclasebiology.proteinJournal of Neurochemistry
researchProduct