Search results for "Synaptic Transmission"

showing 10 items of 178 documents

Endocannabinoids render exploratory behaviour largely independent of the test aversiveness: role of glutamatergic transmission.

2009

To investigate the impact of averseness, controllability and familiarity of a test situation on the involvement of the endocannabinoid system in the regulation of exploratory behaviour, we tested conventional and conditional cannabinoid receptor type 1 (CB1)-deficient mice in behavioural paradigms with different emotional load, which depended on the strength of illumination and the ability of the animals to avoid the light stimulus. Complete CB1 null-mutant mice (Total-CB1-KO) showed an anxiogenic-like phenotype under circumstances where they were able to avoid the bright light such as the elevated plus-maze and the light/dark avoidance task. Conditional mutant mice lacking CB1 expression s…

MaleCannabinoid receptorGlutamic AcidStimulus (physiology)Neuropsychological TestsSynaptic TransmissionOpen fieldDevelopmental psychologyBehavioral NeuroscienceGlutamatergicMiceReceptor Cannabinoid CB1PhotophobiaCannabinoid receptor type 1Cannabinoid Receptor ModulatorsGeneticsAvoidance LearningAnimalsHabituationMaze LearningBrain ChemistryCerebral CortexMice KnockoutThigmotaxisBehavior AnimalFearEndocannabinoid systemMice Inbred C57BLPhenotypenervous systemNeurologyExploratory Behaviorlipids (amino acids peptides and proteins)PsychologyNeurosciencepsychological phenomena and processesEndocannabinoidsGenes, brain, and behavior
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Anterograde tracing of retinal afferents to the tree shrew hypothalamus and raphe

2000

The anterograde neuronal transport of Cholera toxin B subunit (CTB) was used in this study to label the termination of retinal afferents in the hypothalamus of the tree shrew Tupaia belangeri. Upon pressure-injection of the substance into the vitreous body of one eye, a major projection of the retinohypothalamic tract (RHT) was found to the hypothalamic suprachiasmatic nuclei (SCN). Although the innervation pattern was bilateral, the ipsilateral SCN received a somewhat stronger projection. Labeling was also found in the supraoptic nucleus and its perinuclear zone, respectively, mainly ipsilaterally as well as in the bilateral para- and periventricular hypothalamic regions without lateral pr…

MaleCholera ToxinHypothalamusBiologySynaptic TransmissionRetinaSupraoptic nucleusAnimalsNeurons AfferentMolecular BiologyNeuronal transportRapheSuprachiasmatic nucleusGeneral NeuroscienceTupaiidaeGeniculate BodiesAnatomyAnterograde tracingHypothalamusRaphe NucleiFemaleSuprachiasmatic NucleusNeurology (clinical)Raphe nucleiSupraoptic NucleusNeuroscienceRetinohypothalamic tractDevelopmental BiologyBrain Research
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Dysregulated Expression of Neuregulin-1 by Cortical Pyramidal Neurons Disrupts Synaptic Plasticity

2014

Summary Neuregulin-1 ( NRG1 ) gene variants are associated with increased genetic risk for schizophrenia. It is unclear whether risk haplotypes cause elevated or decreased expression of NRG1 in the brains of schizophrenia patients, given that both findings have been reported from autopsy studies. To study NRG1 functions in vivo, we generated mouse mutants with reduced and elevated NRG1 levels and analyzed the impact on cortical functions. Loss of NRG1 from cortical projection neurons resulted in increased inhibitory neurotransmission, reduced synaptic plasticity, and hypoactivity. Neuronal overexpression of cysteine-rich domain (CRD)-NRG1, the major brain isoform, caused unbalanced excitato…

MaleDendritic SpinesNeuregulin-1Nonsynaptic plasticityGene ExpressionMice TransgenicNeurotransmissionInhibitory postsynaptic potentialSynaptic TransmissionGeneral Biochemistry Genetics and Molecular BiologyCell MovementInterneuronsConditioning Psychologicalmental disordersAnimalsNeuregulin 1lcsh:QH301-705.5CA1 Region HippocampalNeuronal PlasticitybiologyPyramidal CellsAnatomyFearCortex (botany)Synaptic fatiguelcsh:Biology (General)Synaptic plasticitybiology.proteinExcitatory postsynaptic potentialFemaleNerve NetNeuroscience
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Hampered long-term depression and thin spine loss in the nucleus accumbens of ethanol-dependent rats.

2014

Alcoholism involves long-term cognitive deficits, including memory impairment, resulting in substantial cost to society. Neuronal refinement and stabilization are hypothesized to confer resilience to poor decision making and addictive-like behaviors, such as excessive ethanol drinking and dependence. Accordingly, structural abnormalities are likely to contribute to synaptic dysfunctions that occur from suddenly ceasing the use of alcohol after chronic ingestion. Here we show that ethanol-dependent rats display a loss of dendritic spines in medium spiny neurons of the nucleus accumbens (Nacc) shell, accompanied by a reduction of tyrosine hydroxylase immunostaining and postsynaptic density 95…

MaleDendritic spineDendritic SpinesGlutamic AcidNucleus accumbensNeurotransmissionMedium spiny neuronSynaptic TransmissionNucleus AccumbensOrgan Culture TechniquesAnimalsRats WistarLong-term depressionLong-Term Synaptic Depressiondopamine synaptic plasticity Golgi glutamateMultidisciplinaryNeuronal PlasticityEthanolDopaminergic NeuronsLong-Term Synaptic DepressionCentral Nervous System DepressantsRatsAlcoholismPNAS PlusSynaptic plasticitySettore BIO/14 - FarmacologiaPsychologyNeurosciencePostsynaptic densityProceedings of the National Academy of Sciences of the United States of America
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Chronic stress induces changes in the structure of interneurons and in the expression of molecules related to neuronal structural plasticity and inhi…

2011

Chronic stress in experimental animals, one of the most accepted models of chronic anxiety and depression, induces structural remodeling of principal neurons in the amygdala and increases its excitation by reducing inhibitory tone. These changes may be mediated by the polysialylated form of the neural cell adhesion molecule (PSA-NCAM), a molecule related to neuronal structural plasticity and expressed by interneurons in the adult CNS, which is downregulated in the amygdala after chronic stress. We have analyzed the amygdala of adult mice after 21 days of restraint stress, studying with qRT-PCR the expression of genes related to general and inhibitory neurotransmission, and of PSA synthesizi…

MaleDendritic spineInterneuronDendritic SpinesSynaptophysinNeural Cell Adhesion Molecule L1BiologyNeurotransmissionSynaptic TransmissionAmygdalaImmobilizationMiceDevelopmental NeuroscienceInterneuronsmedicineAnimalsChronic stressNeuronal PlasticityGlutamate DecarboxylaseDendritesAmygdalaImmunohistochemistrySialyltransferasesDisease Models Animalmedicine.anatomical_structurenervous systemNeurologySialic AcidsSynaptophysinbiology.proteinNeural cell adhesion moleculeNeuroscienceStress PsychologicalBasolateral amygdalaExperimental Neurology
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alpha-Bungarotoxin, kappa-bungarotoxin, alpha-cobratoxin and erabutoxin-b do not affect [3H]acetylcholine release from the rat isolated left hemidiap…

1995

Endplate preparations of the rat left hemidiaphragm were incubated with [3H]choline to label neuronal transmitter stores. Nerve evoked release of newly-synthesized [3H]acetylcholine was measured in the absence of cholinesterase inhibitors to investigate whether snake venom neurotoxins by blocking presynaptic nicotinic autoreceptors affect evoked transmitter release. Contractions of the indirectly stimulated hemidiaphragm were recorded to characterize the blocking effect of alpha-neurotoxins at the post-synaptic nicotinic receptors. Neither the long chain neurotoxins alpha-cobratoxin (1 microgram ml-1) and alpha-bungarotoxin (5 microgram ml-1) nor the short chain neurotoxin erabutoxin-b (0.1…

MaleDiaphragmNeurotoxinsPharmacologyReceptors NicotinicTritiumSynaptic TransmissionPostsynaptic potentialmedicineAnimalsCobra Neurotoxin ProteinsChromatography High Pressure LiquidCholinesterasePharmacologyErabutoxinsbiologyChemistryMuscle SmoothGeneral MedicineBungarotoxinmusculoskeletal systemBungarotoxinsAcetylcholineRatsPhrenic NerveNicotinic agonistSnake venomIsotope Labelingbiology.proteinAutoreceptorFemaleCobratoxinNeuroscienceAcetylcholinemedicine.drugMuscle ContractionSnake VenomsNaunyn-Schmiedeberg's archives of pharmacology
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Altered electrical activity in colonic smooth muscle cells from dystrophic (mdx) mice

2001

Because the colon from dystrophic (mdx) mice shows an altered motor pattern, probably due to neural disorders, our aim was to examine the electrophysiological properties of muscle cells and the functionality of nitrergic transmission in circular muscle from normal and mdx colon. Normal colonic cells (resting membrane potential [RMP] about -50 mV) showed spontaneous hyperpolarizations (inhibitory junction potentials; IJPs) and cyclic slow depolarizations were sometimes recorded. Mdx colon had a depolarized RMP (about -36 mV) and spontaneous IJPs, but the cyclic activity was never observed. In the normal colon, Nomega-nitro-L-arginine methyl ester (L-NAME) induced depolarization and abolished…

MaleDuchenne muscular dystrophymedicine.medical_specialtyInhibitory junction potentialColonPhysiologyDuchenne muscular dystrophyInhibitory postsynaptic potentialSynaptic TransmissionSettore BIO/09 - FisiologiaProximal colonMembrane PotentialsMiceSmooth muscleInternal medicinemedicineAnimalsMyocyteEnzyme InhibitorsMembrane potentialNeuroscience (all)Endocrine and Autonomic SystemsChemistryGastroenterologyMuscle SmoothNitric oxideDepolarizationMuscular Dystrophy AnimalHyperpolarization (biology)medicine.diseaseElectric StimulationElectrophysiologyMice Inbred C57BLMuscular Dystrophy DuchenneMdx miceElectrophysiologyNG-Nitroarginine Methyl EsterEndocrinologyMice Inbred mdxSodium nitroprussideNeurosciencemedicine.drugNeurogastroenterology and Motility
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Pancuronium improves the neuromuscular transmission defect of human organophosphate intoxication.

1990

Two patients with acute severe organophosphate intoxication showed (1) single evoked compound muscle action potentials (CMAP) with repetitive discharges and (2) prominent decremental responses of CMAP with 20 and 50 Hz supramaximal nerve stimulation. Following the intravenous injection of single small doses of pancuronium, marked improvement in these abnormalities occurred and persisted for several hours. We postulate that the physiologic improvement following low-dose pancuronium results from blockade of acetylcholine receptors, especially those located on the terminal axon responsible for antidromic backfiring.

MaleInsecticidesNeuromuscular transmissionNeuromuscular JunctionAction PotentialsSuicide AttemptedElectromyographyNeurotransmissionIsoindolesOrganophosphate poisoningSynaptic TransmissionNeuromuscular junctionOrganophosphate PoisoningmedicineHumansPancuroniumAxonAcetylcholine receptormedicine.diagnostic_testParathionbusiness.industryMusclesOrganothiophosphatesOrganothiophosphorus Compoundsmedicine.diseaseAntidromicMedian Nervemedicine.anatomical_structureAnesthesiaNeurology (clinical)businessNeurology
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Neural overexcitation and implication of NMDA and AMPA receptors in a mouse model of temporal lobe epilepsy implying zinc chelation.

2006

Summary: Purpose: Zinc chelation with diethyldithiocarbamate (DEDTC) during nondamaging kainic acid administration enhances excitotoxicity to the level of cell damage. The objective of this work was to study the developing of the lesion in this model of temporal lobe epilepsy and the implications of the different types of glutamate receptors. Methods: The antagonist of the N-methyl-d-aspartate (NMDA) receptor MK-801, and the antagonist of the α-amino-3-hydroxy-5-methyl-4-isoxazolepropionate (AMPA) receptor GYKI52466, were used concomitantly with intraperitoneal administration of kainic acid (15 mg/kg) followed by DEDTC (150 mg/kg) in mouse. The animals were killed at different times from 4 …

MaleKainic acidmedicine.medical_specialtyExcitotoxicityHippocampusKainate receptorHSP72 Heat-Shock ProteinsAMPA receptorBiologymedicine.disease_causeHippocampusReceptors N-Methyl-D-AspartateSynaptic Transmissionchemistry.chemical_compoundBenzodiazepinesMiceReceptors Kainic AcidInternal medicinemedicineAnimalsReceptors AMPACell damageChelating AgentsKainic AcidCell DeathGlutamate receptormedicine.diseaseDisease Models AnimalZincEndocrinologyNeuroprotective Agentsnervous systemNeurologychemistryEpilepsy Temporal LobeNMDA receptorNeurology (clinical)Dizocilpine MaleateDitiocarbProto-Oncogene Proteins c-fosEpilepsia
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Local salsolinol modulates dopamine extracellular levels from rat nucleus accumbens: shell/core differences.

2008

Salsolinol (SAL), a condensation product of dopamine and acetaldehyde that appears in the rat and human brain after ethanol ingestion, has been largely implicated in the aetiology of alcoholism. Although the behavioural consequences of systemic or intracerebral SAL administrations have been described, the neurochemical effects of pharmacologically relevant doses of SAL and other tetrahydroisoquinolines (THIQs) in the brain areas involved in alcohol addiction are practically unknown. To gain an insight into this topic, male Wistar rats were stereotaxically implanted with one concentric microdialysis probe in either the shell or the core of the nucleus accumbens (NAc). Treatments involved loc…

MaleMicrodialysisDopamineMicrodialysisDown-RegulationAcetaldehydePharmacologyNucleus accumbensSynaptic TransmissionNucleus AccumbensCellular and Molecular Neurosciencechemistry.chemical_compoundNeurochemicalAlcohol-Induced Disorders Nervous SystemRewardDopamineparasitic diseasesBasal gangliamedicineAnimalsEthanol metabolismRats WistarNeurotransmitterChromatography High Pressure LiquidDose-Response Relationship DrugEthanolChemistryExtracellular FluidCell BiologyIsoquinolinesRatsUp-RegulationAlcoholismCatecholamineNeurosciencemedicine.drugNeurochemistry international
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