Search results for "Synaptic plasticity"

showing 10 items of 132 documents

Sparse Distributed Representation of Odors in a Large-scale Olfactory Bulb Circuit

2013

In the olfactory bulb, lateral inhibition mediated by granule cells has been suggested to modulate the timing of mitral cell firing, thereby shaping the representation of input odorants. Current experimental techniques, however, do not enable a clear study of how the mitral-granule cell network sculpts odor inputs to represent odor information spatially and temporally. To address this critical step in the neural basis of odor recognition, we built a biophysical network model of mitral and granule cells, corresponding to 1/100th of the real system in the rat, and used direct experimental imaging data of glomeruli activated by various odors. The model allows the systematic investigation and g…

Circuit ModelsMaleNerve net0302 clinical medicineLateral inhibitionOdorlcsh:QH301-705.5NeuronsFeedback PhysiologicalCoding Mechanisms0303 health sciencesNeuronal PlasticityEcologyAnatomyOlfactory BulbSynapseSensory Systemsmedicine.anatomical_structureComputational Theory and MathematicsModeling and SimulationExcitatory postsynaptic potentialResearch ArticleModels NeurologicalBiologyInhibitory postsynaptic potential03 medical and health sciencesCellular and Molecular NeuroscienceGeneticNeuroplasticityGeneticsmedicineAnimalsComputer SimulationBiologyMolecular BiologyEcology Evolution Behavior and Systematics030304 developmental biologyComputational NeuroscienceOlfactory SystemAnimalComputational BiologyNeuronEcology Evolution Behavior and SystematicRatsOlfactory bulbOdorlcsh:Biology (General)OdorantsSynapsesSynaptic plasticityRatNerve NetNeuroscience030217 neurology & neurosurgeryNeuroscience
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The APC/C cofactor Cdh1 prevents replicative stress and p53-dependent cell death in neural progenitors

2013

The E3-ubiquitin ligase APC/C-Cdh1 is essential for endoreduplication but its relevance in the mammalian mitotic cell cycle is still unclear. Here we show that genetic ablation of Cdh1 in the developing nervous system results in hypoplastic brain and hydrocephalus. These defects correlate with enhanced levels of Cdh1 substrates and increased entry into the S phase in neural progenitors. However, cell division is prevented in the absence of Cdh1 due to hyperactivation of cyclin-dependent kinases, replicative stress, induction of p53, G2 arrest and apoptotic death of these progenitor cells. Concomitant ablation of p53 rescues apoptosis but not replicative stress, resulting in the presence of …

DNA ReplicationMaleProgrammed cell deathCell divisionNeurogenesisGeneral Physics and AstronomyApoptosisCell Cycle ProteinsBiologyAnaphase-Promoting Complex-CyclosomeCdh1 ProteinsGeneral Biochemistry Genetics and Molecular BiologyMice03 medical and health sciences0302 clinical medicineNeural Stem CellsAnimalsProgenitor cell030304 developmental biologyProgenitorMice KnockoutNeuronschemistry.chemical_classification0303 health sciencesDNA ligaseMultidisciplinaryCell CycleNeurogenesisBrainOrgan SizeGeneral ChemistryCell cycle3. Good healthCell biologyMice Inbred C57BLchemistrySynaptic plasticityFemaleTumor Suppressor Protein p53Cell Division030217 neurology & neurosurgeryNature Communications
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Proteomic signature of the Dravet syndrome in the genetic Scn1a-A1783V mouse model

2021

AbstractBackgroundDravet syndrome is a rare, severe pediatric epileptic encephalopathy associated with intellectual and motor disabilities. Proteomic profiling in a mouse model of Dravet syndrome can provide information about the molecular consequences of the genetic deficiency and about pathophysiological mechanisms developing during the disease course.MethodsA knock-in mouse model of Dravet syndrome with Scn1a haploinsufficiency was used for whole proteome, seizure and behavioral analysis. Hippocampal tissue was dissected from two-(prior to epilepsy manifestation) and four-(following epilepsy manifestation) week-old male mice and analyzed using LC-MS/MS with label-free quantification. Pro…

EpilepsyDravet syndromeProteomic ProfilingProteomeSynaptic plasticitymedicineHippocampusBiologymedicine.diseaseHaploinsufficiencyNeuroscienceAstrogliosis
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Role of polysialic acid (PSA) in the control of food intake and body weight

2013

Hypothalamus plays a major role in the regulation of energy homeostasis by the presence of neural circuits controlling food intake. These circuits are plastic and can be rewired during adulthood. We hypothesized that synaptic plasticity can occur during physiological conditions. We have shown that synaptic contact on hypothalamic anorexigen POMC neurons are rewired in mouse upon high fat diet (HFD). This synaptic process is mandatory to adjust energy intake and requires the glycan PSA (polysialic acid). PSA promotes synaptic plasticity in the brain by the weakening of cell-to-cell interaction by addition on NCAM (neural cell adhesion molecule). We hypothesized that a defect in brain synapti…

Food intakePlasticité synaptique[SDV.MHEP.PHY] Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]HypothalamusPrise alimentaireEnergy homeostasisHoméostasie énergétique[SDV.NEU] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Synaptic plasticity
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Expression of synapsin I gene in primary cultures of differentiating rat cortical neurons

1995

Synapsin I is a neuron-specific protein which is present in two isoforms, Ia and Ib. In the last few years this protein has been demonstrated to play a central role in the regulation of neurotransmitter release and synaptic plasticity. In this paper the developmental expression of this protein has been investigated in primary neuronal cultures from fetal rat brain cortices. The presence of thyroid hormone in the culture medium stimulates an early expression of the protein without exerting any effect at the level of mRNA transcription and accumulation. These observations implicate a T3-dependent regulation of this neuron-specific gene at the level of mRNA translation. © 1995 Plenum Publishin…

Gene isoformmedicine.medical_specialtySynapsin ITime FactorsTranscription GeneticBlotting Westernsynapsin IGene ExpressionBiologyBiochemistryCellular and Molecular Neurosciencechemistry.chemical_compoundFetusInternal medicineSettore BIO/10 - BiochimicaGene expressionmedicineAnimalsRNA MessengerNeurotransmitterCells CulturedCell NucleusCerebral CortexNeuronsMessenger RNANeuroscience (all)Cell DifferentiationGeneral MedicineSynapsinBlotting NorthernSynapsinsthyroid hormoneRatsCell biologyKineticsmedicine.anatomical_structureEndocrinologynervous systemchemistryNeuronal differentiationSynaptic plasticityTriiodothyronineSettore MED/26 - NeurologiaNeuron
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A Novel 1,4-Dihydropyridine Derivative Improves Spatial Learning and Memory and Modifies Brain Protein Expression in Wild Type and Transgenic APPSweD…

2015

Ca2+ blockers, particularly those capable of crossing the blood-brain barrier (BBB), have been suggested as a possible treatment or disease modifying agents for neurodegenerative disorders, e.g., Alzheimer's disease. The present study investigated the effects of a novel 4-(N-dodecyl) pyridinium group-containing 1,4-dihydropyridine derivative (AP-12) on cognition and synaptic protein expression in the brain. Treatment of AP-12 was investigated in wild type C57BL/6J mice and transgenic Alzheimer's disease model mice (Tg APPSweDI) using behavioral tests and immunohistochemistry, as well as mass spectrometry to assess the blood-brain barrier (BBB) penetration. The data demonstrated the ability …

Genetically modified mouseMalePathologymedicine.medical_specialtyDihydropyridinesTime Factorsmedicine.drug_classTransgeneSpatial Learninglcsh:MedicineMice TransgenicBlood–brain barrierAnxiolyticGyrus CinguliHippocampus03 medical and health sciences0302 clinical medicineHomer Scaffolding ProteinsMemorymedicineAnimalsHumanslcsh:Science030304 developmental biology0303 health sciencesMultidisciplinaryAmyloid beta-PeptidesGlutamate Decarboxylaselcsh:RDihydropyridineWild typeBrainmedicine.disease3. Good healthMice Inbred C57BLmedicine.anatomical_structureAnti-Anxiety AgentsBlood-Brain BarrierSynaptic plasticitylcsh:QAlzheimer's diseaseCarrier ProteinsNeuroscience030217 neurology & neurosurgerymedicine.drugResearch ArticlePloS one
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Mildronate improves cognition and reduces amyloid-β pathology in transgenic Alzheimer's disease mice

2013

Mildronate, a carnitine congener drug, previously has been shown to provide neuroprotection in an azidothymidine-induced mouse model of neurotoxicity and in a Parkinson's disease rat model. The aim of this study was to investigate the effects of mildronate treatment on cognition and pathology in Alzheimer's disease (AD) model mice (APP(SweDI)). Mildronate was administered i.p. daily at 50 or 100 mg/kg for 28 days. At the end of treatment, the animals were behaviorally and cognitively tested, and brains were assessed for AD-related pathology, inflammation, synaptic markers, and acetylcholinesterase (AChE). The data show that mildronate treatment significantly improved animal performance in w…

Genetically modified mousePathologymedicine.medical_specialtybiologyNeurotoxicityHippocampusWater mazemedicine.diseaseAcetylcholinesteraseNeuroprotectionCellular and Molecular Neurosciencechemistry.chemical_compoundchemistrySynaptic plasticitymedicineSynaptophysinbiology.proteinPsychologyJournal of Neuroscience Research
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Naturally occurring autoantibodies interfere with β-amyloid metabolism and improve cognition in a transgenic mouse model of Alzheimer's disease 24 h …

2013

There is evidence that naturally occurring antibodies directed against Aβ (nAbs-Aβ) have a role in Aβ-metabolism and Aβ-clearance. The presence of nAbs-Aβ leads to a reduction in amyloid fibrillation and thus a reduction in their toxicity. We investigated the effects of nAbs-Aβ in respect to oligomerization and used the Tg2576 transgenic mouse model in order to investigate the rapid effect with a single-dose (24 h) on oligomer breakdown and cytokine secretion along with immunohistochemical characterization of synaptic plasticity. nAbs-Aβ were able to reduce toxic oligomer concentration with an increase in Aβ-monomers. Cytokine secretion was significantly reduced. Synaptic plasticity was als…

Genetically modified mousemedicine.medical_specialtytoxic oligomersAmyloidBlotting WesternEnzyme-Linked Immunosorbent AssayMice TransgenicBiologyAnimals Genetically ModifiedCellular and Molecular NeuroscienceMiceCognitionAlzheimer DiseaseInternal medicinemedicineAnimalsBiological PsychiatryAutoantibodiesAmyloid beta-Peptidesβ-amyloidbehaviorAutoantibodyAlzheimer's diseasemedicine.diseasenatural occurring autoantibodiesCell biologyPsychiatry and Mental healthDisease Models AnimalEndocrinologyinflammationSynaptic plasticityToxicitybiology.proteinCytokinesCytokine secretionOriginal ArticleFemaleAlzheimer's diseaseAntibodyTranslational Psychiatry
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Neuroplasticity and environment: A pharmacotherapeutic approach toward preclinical and clinical understanding

2021

International audience; Emerging research in the field of behavioral neuroscience has demonstrated the fundamental role of the cerebral organization. For brain development, various important functions are required for instance synaptic plasticity and neurogenesis. This property is described in terms of neuroplasticity. Neuroplasticity denotes the extraordinary ability of the brain for the recognition of its structure and function after an experience with some external stimuli and involves in the various behavioral processes of the brain such as cognitive functions. It has multiple factors that can range from gene alteration to environmental factors. Environmental factors exert both negative…

Health Toxicology and Mutagenesis0208 environmental biotechnology02 engineering and technologyEnvironmental stress010501 environmental sciencesBehavioral neuroscience01 natural sciencesNeuroplasticitymedicineEnvironmental Chemistry0105 earth and related environmental sciencesEnvironmental enrichmentMechanism (biology)NeurogenesisPublic Health Environmental and Occupational HealthBrainCognition020801 environmental engineeringSleep deprivationSynaptic plasticity[SDE]Environmental SciencesNeuroplasticitymedicine.symptomCNSPsychologyNeuroscience
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Neurotrophic Properties of Silexan, an Essential Oil from the Flowers of Lavender-Preclinical Evidence for Antidepressant-Like Properties.

2020

Abstract Background Silexan, a special essential oil from flowering tops of lavandula angustifolia, is used to treat subsyndromal anxiety disorders. In a recent clinical trial, Silexan also showed antidepressant effects in patients suffering from mixed anxiety-depression (ICD-10 F41.2). Since preclinical data explaining antidepressant properties of Silexan are missing, we decided to investigate if Silexan also shows antidepressant-like effects in vitro as well as in vivo models. Methods We used the forced swimming test (FST) in rats as a simple behavioral test indicative of antidepressant activity in vivo. As environmental events and other risk factors contribute to depression through conve…

Imipraminemedicine.drug_classSynaptogenesisCell Culture TechniquesPregabalinTricyclic antidepressantPharmacologyCREBImipramineRats Sprague-Dawley03 medical and health sciences0302 clinical medicinemedicineOils VolatileAnimalsPlant OilsPharmacology (medical)030304 developmental biology0303 health sciencesbiologyDose-Response Relationship Drugbusiness.industryGeneral MedicineCREB-Binding ProteinCyclic AMP-Dependent Protein KinasesAntidepressive AgentsRatsPsychiatry and Mental healthLavandulaSynaptic plasticitybiology.proteinAntidepressantbusiness030217 neurology & neurosurgerymedicine.drugBehavioural despair testNeurotrophinPharmacopsychiatry
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