Search results for "Systemic inflammation"

showing 10 items of 93 documents

Oral Unsaturated Fat Load Impairs Postprandial Systemic Inflammation in Primary Hypercholesterolemia Patients.

2021

Context: Primary hypercholesterolemia (PH) is a lipid disorder characterized by elevated levels of cholesterol and low-density lipoprotein (LDL). Low-grade systemic inflammation is associated with PH, which might explain the higher incidence of cardiovascular diseases in this setting.Objective: To evaluate the effect of an oral unsaturated fat load (OUFL) on different immune parameters and functional consequences in patients with PH in postprandial state.Design: A commercial liquid preparation of long-chain triglycerides (Supracal®; ω6/ω3 ratio >20/1, OUFL) was administered to 20 patients and 10 age-matched controls. Whole blood was collected before (fasting state) and 4 h after admi…

0301 basic medicinemedicine.medical_specialtyEndotheliumleukocytesinflammatory mediatorsRM1-950030204 cardiovascular system & hematologySystemic inflammationendothelial dysfunction03 medical and health scienceschemistry.chemical_compound0302 clinical medicineoral unsaturated fat loadInternal medicineprimary hypercholesterolemiamedicineplatelet activationPharmacology (medical)Platelet activationEndothelial dysfunctionOriginal ResearchPharmacologysystemic inflammationCholesterolbusiness.industryUnsaturated fatmedicine.disease030104 developmental biologyEndocrinologymedicine.anatomical_structurePostprandialchemistryTherapeutics. Pharmacologymedicine.symptombusinessLipoproteinFrontiers in pharmacology
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Molecular Pathways Mediating Immunosuppression in Response to Prolonged Intensive Physical Training, Low-Energy Availability, and Intensive Weight Lo…

2019

Exercise and exercise-induced weight loss have a beneficial effect on overall health, including positive effects on molecular pathways associated with immune function, especially in overweight individuals. The main aim of our study was to assess how energy deprivation (i.e., "semi-starvation") leading to substantial fat mass loss affects the immune system and immunosuppression in previously normal weight individuals. Thus, to address this hypothesis, we applied a high-throughput systems biology approach to better characterize potential key pathways associated with immune system modulation during intensive weight loss and subsequent weight regain. We examined 42 healthy female physique athle…

0301 basic medicinemedicine.medical_treatmentPhysiologyliikuntaOverweightSystemic inflammationLeukocyte Countphysical training0302 clinical medicineWeight lossLeukocytesImmunology and AllergyMedicineOXIDATIVE STRESSta315DIETARY RESTRICTIONSport and Fitness SciencesOriginal Research2. Zero hungerimmunosuppressionIdrottsvetenskapbioinformatiikkaImmunosuppressionbioinformaticslow energy availability3. Good healthimmuunivasteIMMUNE FUNCTIONOBESITYChemokine secretionFemalemedicine.symptomfyysinen aktiivisuusAdultlcsh:Immunologic diseases. AllergyImmunologyEXERCISEInflammationYoung Adult03 medical and health sciencesLEPTINImmune systemINFLAMMATIONImmune ToleranceHumansimmunosuppression ; low energy availability ; physical training ; bioinformatics ; weight lossCell Proliferationbusiness.industrylaihdutusCYTOKINESmedicine.diseaseObesityDietenergiansaanti030104 developmental biologyHEMATOPOIETIC STEMImmunoglobulin G3121 General medicine internal medicine and other clinical medicineCELLS3111 Biomedicineweight lossEnergy IntakeTranscriptomelcsh:RC581-607business030215 immunologyFrontiers in Immunology
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SHIP2: A “NEW” Insulin Pathway Target for Aging Research

2014

Strong evidence suggests that systemic inflammation and central adiposity contribute to and perpetuate metabolic syndrome. All of these alterations predispose individuals to type 2 diabetes mellitus (T2DM), cardiovascular disease, as well as Alzheimer's disease (AD), all characterized by chronic inflammatory status. On the other hand, extensive abnormalities in insulin and insulin-like growth factor I (IGF-I) and IGF-II signaling mechanisms in brains with AD have been demonstrated, suggesting that AD could be a third form of diabetes. The Src homology domain-containing inositol 5-phosphatase 2 (SHIP2) has an important role in the insulin pathway because its over-expression causes impairment…

AdultAgingmedicine.medical_specialtymedicine.medical_treatmentDiseaseBiologySystemic inflammationPolymorphism Single Nucleotidepolymorphismchemistry.chemical_compounddomain-containing inositol 5-phosphatase 2 (SHIP2) insulin-like growth factor I (IGF-I) type 2 diabetes mellitus (T2DM)INFLAMMATIONGene FrequencyAlzheimer DiseaseDiabetes mellitusInternal medicinemedicineHumansInsulinSettore MED/05 - Patologia ClinicaSNPInositolAgedSettore MED/04 - Patologia GeneraleALZHEIMER’S DISEASEResearchInsulinInositol Polyphosphate 5-PhosphatasesNEURODEGENERATIONType 2 Diabetes Mellitusmedicine.diseasePhosphoric Monoester HydrolasesEndocrinologyDiabetes Mellitus Type 2chemistryImmunologySettore MED/26 - NeurologiaGeriatrics and Gerontologymedicine.symptomMetabolic syndromeSignal TransductionRejuvenation Research
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Inflammatory capacity of exosomes released in the early stages of acute pancreatitis predicts the severity of the disease

2021

As acute pancreatitis progresses to the severe form, a life-threatening systemic inflammation is triggered. Although the mechanisms involved in this process are not yet well understood, it has been proposed that circulating exosomes may be involved in the progression of inflammation from the pancreas to distant organs. Here, the inflammatory capacity and protein profile of plasma exosomes obtained during the first 24 h of hospitalization of patients diagnosed with acute pancreatitis were characterized and compared with the final severity of the disease. We found that the final severity of the disease strongly correlates with the inflammatory capacity of exosomes in the early stages of acute…

AdultMaleProteomicsInflammationSystemic inflammationExosomesS100A9Pathology and Forensic MedicineS100A8medicineHumansS100A8PancreasS100A9AgedAged 80 and overInflammationbusiness.industryMiddle Agedmedicine.diseaseMicrovesiclesAcute pancreatitisPancreatitisAcute DiseaseImmunologyDisease ProgressionPancreatitisAcute pancreatitisFemaleTumor necrosis factor alphamedicine.symptombusinessSignal TransductionJournal of Pathology
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Interferon- and ribavirin-free therapy with new direct acting antivirals (DAA) for chronic hepatitis C improves vascular endothelial function.

2018

Abstract Introduction Chronic Hepatitis C virus infection (HCV) is associated with extrahepatic manifestations and an increased prevalence in cardiovascular disease. New direct acting antivirals (DAA) have revolutionized HCV treatment with high rates of sustained virological response (SVR). Recently it was demonstrated, that SVR reduces morbidity and overall mortality more than can be solely explained by hepatic effects, suggesting that treatment with DAA also affects cardiovascular disease. The aim of this pilot study was to identify possible underlying mechanisms behind the HCV-associated cardiovascular mortality reported by others. Methods and results 20 HCV patients (10 genotype GT1, 10…

AdultMalemedicine.medical_specialtyEndotheliumInflammationPilot Projects030204 cardiovascular system & hematologymedicine.disease_causeSystemic inflammationGastroenterologyAntiviral AgentsCohort Studies03 medical and health scienceschemistry.chemical_compound0302 clinical medicineInsulin resistanceInterferonInternal medicineRibavirinClinical endpointmedicineHumansbusiness.industryRibavirinHepatitis C ChronicMiddle Agedmedicine.diseasemedicine.anatomical_structureTreatment Outcomechemistry030211 gastroenterology & hepatologyDrug Therapy CombinationFemaleEndothelium VascularInterferonsmedicine.symptomCardiology and Cardiovascular MedicinebusinessOxidative stressmedicine.drugInternational journal of cardiology
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Inflammation, genetics, and longevity: further studies on the protective effects in men of IL-10 -1082 promoter SNP and its interaction with TNF-alph…

2003

Ageing is associated with chronic, low grade inflammatory activity leading to long term tissue damage, and systemic chronic inflammation has been found to be related to mortality risk from all causes in older persons.1 Also, the genetic constitution of the organism interacting with systemic inflammation may cause defined organ specific illnesses. Thus, age related diseases such as Alzheimer’s disease (AD), Parkinson’s disease, atherosclerosis, type 2 diabetes, sarcopenia, and osteoporosis, are initiated or worsened by systemic inflammation, suggesting the critical importance of unregulated systemic inflammation in the shortening of survival in humans.1–3 Accordingly, proinflammatory cytokin…

AdultMalemedicine.medical_specialtyGenotypemedicine.medical_treatmentDNA Mutational AnalysisLongevityInflammationSingle-nucleotide polymorphismBiologySystemic inflammationPolymorphism Single NucleotideProinflammatory cytokineGene FrequencyInternal medicineGeneticsmedicineHumansAllelePromoter Regions GeneticGenetics (clinical)AgedGeneticsAged 80 and overInflammationTumor Necrosis Factor-alphaAge FactorsDNAMiddle AgedInterleukin-10Interleukin 10CytokineEndocrinologyImmunologyFemalemedicine.symptomCentenarianLetter to JMGJournal of medical genetics
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Acute Phase Proteins Are Baseline Predictors of Tuberculosis Treatment Failure

2021

Systemic inflammation is a characteristic feature of pulmonary tuberculosis (PTB). Whether systemic inflammation is associated with treatment failure in PTB is not known. Participants, who were newly diagnosed, sputum smear and culture positive individuals with drug-sensitive PTB, were treated with standard anti-tuberculosis treatment and classified as having treatment failure or microbiological cure. The plasma levels of acute phase proteins were assessed at baseline (pre-treatment). Baseline levels of C-reactive protein (CRP), alpha-2 macroglobulin (a2M), Haptoglobin and serum amyloid P (SAP) were significantly higher in treatment failure compared to cured individuals. ROC curve analysis …

AdultMalemedicine.medical_specialtyTuberculosisImmunologyAntitubercular AgentsSystemic inflammationGastroenterologyCohort StudiesInternal medicinemedicineHumansImmunology and AllergyProspective StudiesTreatment FailureTuberculosis PulmonaryOriginal ResearchHaptoglobinsbiologybusiness.industryHaptoglobinAcute-phase proteinArea under the curveRC581-607Middle Agedmedicine.diseasePregnancy-Associated alpha 2-MacroglobulinsMacroglobulinSerum Amyloid P-ComponentC-Reactive ProteintuberculosisROC CurveinflammationTB treatmentacute phase proteinsbiology.proteinbiomarkerSputumBiomarker (medicine)FemaleImmunologic diseases. Allergymedicine.symptombusinessBiomarkersAcute-Phase ProteinsFrontiers in Immunology
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Effects of acute exercise and allopurinol administration on soluble urokinase plasminogen activator receptor (suPAR).

2013

Background Although physical exercise acutely increases the most widely used inflammatory biomarkers, there is no information on its effect on soluble urokinase plasminogen activating receptor (suPAR), a circulating biomarker increasingly used for the assessment of systemic inflammation. Methods suPAR was assessed with the quantitative suPARnostic Standard ELISA Assay (Virogates, Birkerod, Denmark) in 12 professional football players before and after a football match. The athletes were divided into two experimental groups. An oral dose of 300 mg of allopurinol was administered to one group of six participants four hours before a match; the other six participants received placebo. Results Se…

AdultMalemedicine.medical_specialtyacute exercise; allopurinol; suPARAllopurinolPhysical exerciseEnzyme-Linked Immunosorbent AssayallopurinolPlaceboSystemic inflammationacute exerciseGastroenterologyGeneral Biochemistry Genetics and Molecular BiologySuparnosticReceptors Urokinase Plasminogen ActivatorsuPARPlaceboschemistry.chemical_compoundInternal medicinemedicineHumansEnzyme InhibitorsXanthine oxidaseExerciseUrokinasebusiness.industrychemistrySuPARmedicine.symptombusinessmedicine.drug
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Menopause and ovariectomy cause a low grade of systemic inflammation that may be prevented by chronic treatment with low doses of estrogen or losarta…

2009

Abstract The incidence of cardiovascular diseases in premenopausal women is lower than in men or postmenopausal women. This study reports the discovery of a low grade of systemic inflammation, including monocyte adhesion to arterial endothelium, elicited by menopause or estrogen depletion. Chronic treatment with low dose of 17-β-estradiol or inhibition of the renin-angiotensin system reduced this inflammation. Using an in vitro flow chamber system with human arterial and venous endothelial cells, we found that leukocytes from healthy postmenopausal women were more adhesive to the arterial endothelium than those from premenopausal women regardless of the stimulus used on endothelial cells. I…

Adultmedicine.medical_specialtyEndotheliummedicine.drug_classOvariectomyImmunologyInflammationSystemic inflammationLosartanRats Sprague-DawleyInternal medicinemedicineCell AdhesionLeukocytesImmunology and AllergyAnimalsHumansCells CulturedInflammationCell adhesion moleculebusiness.industryEndothelial CellsEstrogensBenzazepinesMiddle Agedmedicine.diseaseRatsMenopausemedicine.anatomical_structureEndocrinologyLosartanEstrogenCase-Control StudiesOvariectomized ratFemaleEndothelium Vascularmedicine.symptomChemokinesMenopausebusinessAngiotensin II Type 1 Receptor BlockersCell Adhesion Moleculesmedicine.drugJournal of immunology (Baltimore, Md. : 1950)
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Oxidant mechanisms in childhood obesity: the link between inflammation and oxidative stress.

2011

Evidence of obesity-induced oxidative stress in adults has emerged in the past several years, and similar evidence has been demonstrated in children more recently. The reactive species of oxygen or nitrogen can chemically alter all major classes of biomolecules by modifying their structure and function. Organisms have developed mechanisms to protect biomolecules from the deleterious effects of free radicals. These include the enzymes superoxide dismutase, catalase, and glutathione peroxidase, as well as water and lipid-soluble antioxidants, such as glutathione, ascorbate (vitamin C), α-tocopherol (vitamin E), and β-carotene. Obesity creates oxidant conditions that favor the development of c…

Adultmedicine.medical_specialtyOxidative phosphorylationSystemic inflammationmedicine.disease_causeSuperoxide dismutasechemistry.chemical_compoundRisk FactorsPhysiology (medical)Internal medicinemedicineHumansObesityChildchemistry.chemical_classificationInflammationReactive oxygen speciesbiologybusiness.industryGlutathione peroxidaseBiochemistry (medical)Public Health Environmental and Occupational HealthGeneral MedicineGlutathioneMalondialdehydeOxidantsOxidative StressEndocrinologychemistrybiology.proteinmedicine.symptomMorbiditybusinessOxidative stressTranslational research : the journal of laboratory and clinical medicine
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