Search results for "T-Lymphocyte Subsets"

showing 10 items of 172 documents

Genetic evolution of T-cell resistance in the course of melanoma progression

2014

Abstract Purpose: CD8+ T lymphocytes can kill autologous melanoma cells, but their activity is impaired when poorly immunogenic tumor phenotypes evolve in the course of disease progression. Here, we analyzed three consecutive melanoma lesions obtained within one year of developing stage IV disease for their recognition by autologous T cells. Experimental Design: One skin (Ma-Mel-48a) and two lymph node (Ma-Mel-48b, Ma-Mel-48c) metastases were analyzed for T-cell infiltration. Melanoma cell lines established from the respective lesions were characterized, determining the T-cell–stimulatory capacity, expression of surface molecules involved in T-cell activation, and specific genetic alteratio…

Cancer ResearchB7 Antigensmedicine.medical_treatmentMedizinGene ExpressionT-Lymphocyte Subsetshemic and lymphatic diseasesCluster AnalysisLymphocytesNeoplasm MetastasisLymph nodeMelanomaTumorImmunogenicityMelanomaSingle Nucleotidemedicine.anatomical_structurePhenotypeButorphanolOncologyDisease ProgressionCytokinesEvolutionT cellHuman leukocyte antigenBiologyPolymorphism Single NucleotideArticleCell LineEvolution MolecularLymphocytes Tumor-InfiltratingCell Line TumormedicineHumansGenetic Predisposition to DiseaseTumor-InfiltratingAllelePolymorphismneoplasmsAllelesNeoplasm StagingHistocompatibility Antigens Class IMolecularImmunotherapymedicine.diseaseAlleles; B7 Antigens; Butorphanol; Cell Line Tumor; Cluster Analysis; Cytokines; Disease Progression; Gene Expression; Genetic Predisposition to Disease; Histocompatibility Antigens Class I; Humans; Lymphocytes Tumor-Infiltrating; Melanoma; Mutation; Neoplasm Metastasis; Neoplasm Staging; Phenotype; Polymorphism Single Nucleotide; T-Lymphocyte Subsets; beta 2-Microglobulin; Evolution Molecular; Oncology; Cancer ResearchImmunologyMutationbeta 2-MicroglobulinCD8
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Functional TCR Retrieval from Single Antigen-Specific Human T Cells Reveals Multiple Novel Epitopes

2014

Abstract The determination of the epitope specificity of disease-associated T-cell responses is relevant for the development of biomarkers and targeted immunotherapies against cancer, autoimmune, and infectious diseases. The lack of known T-cell epitopes and corresponding T-cell receptors (TCR) for novel antigens hinders the efficient development and monitoring of new therapies. We developed an integrated approach for the systematic retrieval and functional characterization of TCRs from single antigen-reactive T cells that includes the identification of epitope specificity. This is accomplished through the rapid cloning of full-length TCR-α and TCR-β chains directly from single antigen-spec…

Cancer ResearchReceptors Antigen T-Cell/geneticsmedicine.medical_treatmentImmunologyReceptors Antigen T-CellEpitopes T-LymphocyteHistocompatibility Antigens Class I/immunologyComputational biologyBiologyEpitopeCell LineViral Matrix ProteinsMiceHistocompatibility Antigens Class II/immunologyAntigenAntigens NeoplasmT-Lymphocyte SubsetsmedicineAnimalsHumansViral Matrix Proteins/immunologyMembrane Proteins/geneticsCloning MolecularPhosphoproteins/immunologyAntigens Neoplasm/immunologyEpitopes T-Lymphocyte/immunologyHistocompatibility Antigens Class IT-cell receptorHistocompatibility Antigens Class IIPTEN PhosphohydrolasePTEN Phosphohydrolase/geneticsMembrane ProteinsRNAImmunotherapyPhosphoproteinsMolecular biologyT-Lymphocyte Subsets/immunologyIn vitroCell cultureCD8Protein BindingCancer Immunology Research
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A specific CD4 epitope bound by tregalizumab mediates activation of regulatory T cells by a unique signaling pathway

2014

CD4(+)CD25(+) regulatory T cells (Tregs) represent a specialized subpopulation of T cells, which are essential for maintaining peripheral tolerance and preventing autoimmunity. The immunomodulatory effects of Tregs depend on their activation status. Here we show that, in contrast to conventional anti-CD4 monoclonal antibodies (mAbs), the humanized CD4-specific monoclonal antibody tregalizumab (BT-061) is able to selectively activate the suppressive properties of Tregs in vitro. BT-061 activates Tregs by binding to CD4 and activation of signaling downstream pathways. The specific functionality of BT-061 may be explained by the recognition of a unique, conformational epitope on domain 2 of th…

Cell signalingProtein Conformationmedicine.drug_classMolecular Sequence DataImmunologyAntibodies Monoclonal HumanizedCrystallography X-RayLymphocyte ActivationMonoclonal antibodyT-Lymphocytes RegulatoryEpitopeT-Lymphocyte SubsetsTransforming Growth Factor betamedicineHumansImmunology and Allergyddc:610Amino Acid SequenceIL-2 receptorPhosphorylationCells CulturedbiologyInterleukin-2 Receptor alpha SubunitAntibodies MonoclonalPeripheral toleranceCell BiologyTransforming growth factor betaMolecular biologyCell biologyCD4 Antigensbiology.proteinEpitopes B-LymphocyteSignal transductionImmunosuppressive AgentsProtein BindingSignal TransductionConformational epitopeImmunology & Cell Biology
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Th9 cells, new players in adaptive immunity.

2014

Upon antigen-specific stimulation, naive CD4⁺ T cells have the potential to differentiate into various T helper (Th) cell subsets. Earlier models of Th cell differentiation focused on IFN-γ-producing Th1 cells and IL-4-secreting Th2 cells. The discovery of additional CD4⁺ Th cell subsets has extended our understanding of Th cell differentiation beyond this dichotomy. Among these is the recently described Th9 cell subset, which preferentially produces interleukin (IL)-9. Here, we review the latest developments in Th9 cell development and differentiation, focusing on contributing environmental signals, and discuss potential physiological and pathophysiological functions of these cells. We des…

Cellular differentiationImmunologyReceptors Antigen T-CellAdaptive ImmunityMiceT-Lymphocyte SubsetsTransforming Growth Factor betaNeoplasmsmedicineHypersensitivityImmunology and AllergyAnimalsHumansInterleukin 9Interleukin 4biologyCell growthLymphocyte differentiationInterleukin-9Models ImmunologicalReceptors Interleukin-2Transforming growth factor betaT helper cellT-Lymphocytes Helper-InducerAcquired immune systemReceptors Interleukin-4medicine.anatomical_structureImmunologyInterferon Regulatory Factorsbiology.proteinSignal TransductionTrends in immunology
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Understanding the Role of T Cells in CNS Homeostasis.

2015

T cells within the central nervous system (CNS) have been generally considered pathogenic, especially in the context of neuroinflammatory disease. However, recent findings have revealed varied functions for T cells in the healthy CNS, as well as more complex roles for these cells in infection and injury than previously appreciated. Here we review evidence indicating important roles for different T cell subsets in the maintenance of CNS homeostasis. We examine the contribution of T cells in limiting inflammation and damage upon CNS injury, infection, and in neurodegeneration, and discuss the current understanding of the cellular and molecular mechanisms involved. Insight into these processes…

Central Nervous SystemT cellT-LymphocytesImmunologyCentral nervous systemContext (language use)InflammationDiseaseBiologyLymphocyte Depletion03 medical and health sciences0302 clinical medicineT-Lymphocyte SubsetsmedicineImmunology and AllergyAnimalsHomeostasisHumansNeurodegenerationmedicine.diseaseCns injurymedicine.anatomical_structureImmunologymedicine.symptomNeurogenic Inflammation030217 neurology & neurosurgeryHomeostasis030215 immunologyTrends in immunology
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Cytokines in Colitis-Associated Cancer: Potential Drug Targets?

2008

In inflammatory bowel disease (IBD), such as UC and CD, the development of colorectal carcinoma can be initiated through chronic inflammation, depending on the duration and severity of the disease. Growing evidence supports a role for various cytokines, released by epithelial and immune cells, in the pathogenesis of colitis associated cancer (CAC). For instance, TNF-alpha has been recently shown to promote tumor development in experimental colitis. Due to its role in the pathogenesis of IBD, TNF-alpha blockade has become one of the cornerstones of IBD therapy. Thus, anti-TNF-alpha strategies could also provide effective anti-tumor therapies. TGF-beta has been shown to attenuate an anti-tumo…

Colorectal cancerImmunologyInflammationInflammatory bowel diseasePathogenesisImmune systemT-Lymphocyte SubsetsAnimalsHumansImmunology and AllergyMedicineColitisPharmacologyInterleukin-6Tumor Necrosis Factor-alphabusiness.industryCancerGeneral MedicineColitisInflammatory Bowel Diseasesmedicine.diseaseInterleukin-12Interleukin-10ImmunologyCytokinesTumor necrosis factor alphamedicine.symptomColorectal NeoplasmsbusinessInflammation & Allergy - Drug Targets
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γδ T cells as a potential tool in colon cancer immunotherapy

2014

γδ T cells are capable of recognizing tumor cells and exert potent cellular cytotoxicity against a large range of tumors, including colon cancer. However, tumors utilize numerous strategies to escape recognition or killing by patrolling γδ T cells, such a downregulation of NKG2D ligands, MICA/B and ULBPs. Therefore, the combined upregulation of T-cell receptorand NKG2D ligands on tumor cells and induction of NKG2D expression on γδ T cells may greatly enhance tumor killing and unlock the functions of γδ T cells. Here, we briefly review current data on the mechanisms of γδ T-cell recognition and killing of colon cancer cells and propose that γδ T cells may represent a promising target for the…

Cytotoxicity ImmunologicColorectal cancermedicine.medical_treatmentImmunologyNkg2d ligandsLarge rangeLigandsDownregulation and upregulationT-Lymphocyte SubsetsmedicineHumansImmunology and AllergyIn patientCell-mediated cytotoxicitybusiness.industryReceptors Antigen T-Cell gamma-deltaImmunotherapymedicine.diseaseNKG2Dgamma delta cellcolon cancerOncologyColonic NeoplasmsImmunologyNeoplastic Stem CellsImmunotherapybusinessImmunotherapy
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Combining dasatinib with dexamethasone long-term leads to maintenance of antiviral and antileukemia specific cytotoxic T cell responses in vitro

2012

Maintaining graft versus leukemia (GvL) and antivirus responses of cytotoxic T cells (CTLs) while suppressing graft-versus-host disease (GvHD) remains a challenge after allogeneic bone marrow transplantation. Clinical observations indicate that combining glucocorticoids with multi-tyrosine-kinase inhibitors could be a successful therapeutic approach. We and others have shown that the BCR-ABL/SRC kinase inhibitor dasatinib may enhance or suppress T cells in vitro. In this report, we evaluated combination effects of dasatinib and dexamethasone on CD3 + and virus-specific CD8 + T cells directly ex vivo and on antigen-specific leukemia-reactive and alloreactive CD8 + T cell clones. Functional o…

Cytotoxicity ImmunologicHerpesvirus 4 HumanCancer ResearchNaive T cellT cellDasatinibDrug Evaluation PreclinicalReceptors Antigen T-CellCytomegalovirusApoptosisT-Cell Antigen Receptor SpecificityBiologyLymphocyte ActivationCell DegranulationDexamethasoneAntigenHLA AntigensT-Lymphocyte SubsetsGeneticsmedicineHumansCytotoxic T cellAntigens ViralProtein Kinase InhibitorsMolecular BiologyCells CulturedDegranulationDrug SynergismT-Lymphocytes Helper-InducerCell BiologyHematologyDasatinibThiazolesPyrimidinesmedicine.anatomical_structureImmunologyCancer researchCytokinesK562 CellsMemory T cellCell DivisionCD8Signal TransductionT-Lymphocytes Cytotoxicmedicine.drugExperimental Hematology
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Homing and memory patterns of human gammadelta T cells in physiopathological situations.

2004

Vgamma9Vdelta2 are a heterogeneous population of T cells and comprise distinct naive, memory and effector populations that can be distinguished on the basis of surface marker expression and effector functions. We review here these recently studied features of Vgamma9Vdelta2 T lymphocyte biology and the roles they play in infectious and autoimmune diseases.

EffectorImmunologyT lymphocyteBiologymedicine.disease_causeInfectionsLymphocyte ActivationMicrobiologyAutoimmunityAutoimmune DiseasesHeterogeneous populationInfectious DiseasesT-Lymphocyte SubsetsSurface markerImmunologymedicineHumansTuberculosisEffector functionsMalaria FalciparumProtozoal diseaseImmunologic MemoryHoming (hematopoietic)Microbes and infection
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MAPK3 deficiency drives autoimmunity via DC arming.

2010

DC are professional APC that instruct T cells during the inflammatory course of EAE. We have previously shown that MAPK3 (Erk1) is important for the induction of T-cell anergy. Our goal was to determine the influence of MAPK3 on the capacity of DC to arm T-cell responses in autoimmunity. We report that DC from Mapk3(-/-) mice have a significantly higher membrane expression of CD86 and MHC-II and--when loaded with the myelin oligodendrocyte glycoprotein--show a superior capacity to prime naive T cells towards an inflammatory phenotype than Mapk3(+/+) DC. Nonetheless and as previously described, Mapk3(-/-) mice were only slightly but not significantly more susceptible to myelin oligodendrocyt…

Encephalomyelitis Autoimmune ExperimentalMAP Kinase Signaling SystemOvalbuminImmunologyMedizinAutoimmunityMice TransgenicT-Cell Antigen Receptor SpecificityBiologymedicine.disease_causeAutoimmunityMyelinMiceImmune systemT-Lymphocyte SubsetsmedicineImmunology and AllergyAnimalsNeuroinflammationGlycoproteinsCD86Mitogen-Activated Protein Kinase 3KinaseHistocompatibility Antigens Class IIDendritic Cellsmedicine.diseaseOligodendrocytePeptide FragmentsSpecific Pathogen-Free OrganismsMice Inbred C57BLmedicine.anatomical_structureRadiation ChimeraImmunologyCytokinesMyelin-Oligodendrocyte GlycoproteinB7-2 AntigenInfiltration (medical)European journal of immunology
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