Search results for "TAGL"

showing 10 items of 296 documents

Protection against 2,4,6-trinitrobenzenesulphonic acid-induced colonic inflammation in mice by the marine products bolinaquinone and petrosaspongioli…

2005

Proinflammatory mediators, namely eicosanoids, reactive oxygen and nitrogen species and cytokines, are clearly involved in the pathogenesis of intestinal bowel disease. bolinaquinone (BQ) and petrosaspongiolide M (PT), two marine products with potent anti-inflammatory action, have been shown to control the production of mediators in acute and chronic inflammatory processes. Hence, we have tested here the hypothesis that BQ and PT could ameliorate inflammation and oxidative stress parameters in 2,4,6-trinitrobenzenesulphonic acid (TNBS)-induced colitis in Balb/c mice. BQ and PT were given orally in doses of 10 or 20mg/kg/day. Treatment of the animals with BQ or PT at the highest dose signifi…

Malemedicine.medical_treatmentAnti-Inflammatory AgentsNitric Oxide Synthase Type IIInflammationNerve Tissue ProteinsPharmacologymedicine.disease_causeBiochemistryProinflammatory cytokinechemistry.chemical_compoundMiceSynaptotagminsDysideamedicineAnimalsOleanolic AcidPharmacologyMice Inbred BALB CMembrane GlycoproteinsbiologySuperoxideNitrotyrosineCalcium-Binding ProteinsInterleukinMembrane ProteinsColitisInflammatory Bowel DiseasesImmunohistochemistryNitric oxide synthasechemistryBiochemistryTrinitrobenzenesulfonic AcidCyclooxygenase 2Prostaglandin-Endoperoxide SynthasesSynaptotagmin IHeme Oxygenase (Decyclizing)biology.proteinmedicine.symptomNitric Oxide SynthaseSesquiterpenesOxidative stressHeme Oxygenase-1Prostaglandin EInterleukin-1Biochemical pharmacology
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Toll-like receptor 2 mediates prostaglandin E2 production in murine peritoneal macrophages and splenocytes in response to Candida albicans

2004

The involvement of Toll-like receptor 2 (TLR2) and TLR4 in triggering signal transduction pathways leading to prostaglandin E(2) (PGE(2)) production in response to Candida albicans has been studied in cells from wild-type, TLR2-/- and TLR4-/- knockout mice. In vitro PGE(2) production by macrophages challenged with zymosan, yeast or hypha cells was strongly inhibited in TLR2-deficient cells, but not in TLR4-/- cells, as compared to macrophages from wild-type mice. PGE(2) production was dependent on de novo cyclooxygenase-2 (Cox2) synthesis, since unchallenged cells failed to produce PGE(2) and specific Cox2 inhibition during challenge totally blocked PGE(2) production. Similar results were o…

Malemedicine.medical_treatmentReceptors Cell SurfaceBiologyMicrobiologyDinoprostoneMicechemistry.chemical_compoundCandida albicansmedicineAnimalsProstaglandin E2Candida albicansMolecular BiologyCells CulturedMice KnockoutToll-like receptorZymosanGeneral Medicinebiology.organism_classificationMolecular biologyToll-Like Receptor 2Corpus albicansToll-Like Receptor 4TLR2chemistryCyclooxygenase 2Prostaglandin-Endoperoxide SynthasesImmunologyMacrophages PeritonealTLR4Femalelipids (amino acids peptides and proteins)Signal Transductionmedicine.drugProstaglandin EResearch in Microbiology
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Cytotoxicity of Root Canal Filling Materials to Three Different Human Cell Lines

2001

The aim of this study was to investigate the biological compatibility of five root canal sealers (Sealapex, Endion, Super-EBA, Ketac-Endo, and AH Plus) and regular and calcium hydroxide-based gutta-percha in three different human cell lines. Cultures without root canal sealers were used as controls. Cell growth, cell morphology, cell viability, protein content of the cells, and prostaglandin E 2 (PGE 2 ) release were used as parameters to determine the cytotoxicity of the materials. The protein content of the three cell lines—nasal fibroblasts, gingival fibroblasts, and epithelial tumor cells—was significantly reduced (p ≤ 0.001) by all materials tested. Determinations of PGE 2 release show…

Materials scienceCell SurvivalRoot canalmedicine.medical_treatmentStatistics as TopicCellGingivaDentistryBiocompatible MaterialsCell morphologyDinoprostoneCell LineCalcium HydroxideRoot Canal Filling Materialschemistry.chemical_compoundTumor Cells CulturedmedicineHumansNeoplasms Glandular and EpithelialViability assayCytotoxicityGeneral DentistryCalcium hydroxideEpoxy ResinsCell growthbusiness.industryProteinsFibroblastsMolecular biologySalicylatesNasal Mucosamedicine.anatomical_structurechemistryGlass Ionomer CementsDentin-Bonding AgentsGutta-PerchabusinessBiomarkersCell DivisionProstaglandin EJournal of Endodontics
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Immobilization and controlled release of prostaglandin E2 from poly-L-lactide-co-glycolide microspheres.

2009

Prostaglandin E(2) (PGE(2)) is an arachidonic acid metabolite involved in physiological homeostasis and numerous pathophysiological conditions. Furthermore, it has been demonstrated that prostaglandins have a stimulating effect not only on angiogenesis in situ and in vitro but also on chondrocyte proliferation in vitro. Thus, PGE(2) represents an interesting signaling molecule for various tissue engineering strategies. However, under physiological conditions, PGE(2) has a half-life time of only 10 min, which limits its use in biomedical applications. In the present study, we investigated if the incorporation of PGE(2) into biodegradable poly-L-lactide-co-glycolide microspheres results in a …

Materials scienceMetabolitemedicine.medical_treatmentKineticsBiomedical EngineeringProstaglandinDinoprostoneBiomaterialschemistry.chemical_compoundmedicineProstaglandin E2Particle SizePolyglactin 910ChromatographyMetals and AlloysControlled releaseIn vitroMicrospheresKineticschemistryBiochemistryDelayed-Action PreparationsCeramics and Compositeslipids (amino acids peptides and proteins)Arachidonic acidProstaglandin Emedicine.drugJournal of biomedical materials research. Part A
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Influence of heme oxygenase 1 modulation on the progression of murine collagen-induced arthritis.

2005

Contains fulltext : 48023.pdf (Publisher’s version ) (Closed access) OBJECTIVE: Heme oxygenase 1 (HO-1) can be induced by inflammatory mediators as an adaptive response. The objective of the present study was to determine the consequences of HO-1 modulation in the murine collagen-induced arthritis (CIA) model. METHODS: DBA/1J mice were treated with an inhibitor of HO-1, tin protoporphyrin IX (SnPP), or with an inducer of HO-1, cobalt protoporphyrin IX (CoPP), from day 22 to day 29 after CIA induction. The clinical evolution of disease was monitored visually. At the end of the experiment, joints were examined for histopathologic changes. Cytokine levels in paws were measured by enzyme-linked…

Metalloporphyrinsmedicine.medical_treatmentImmunologyArthritisProtoporphyrinsInflammationPharmacologyAuto-immunity transplantation and immunotherapy [N4i 4]MiceRheumatologyFibrosismedicinePerception and Action [DCN 1]Immunology and AllergyAnimalsPharmacology (medical)Enzyme InhibitorsChronic inflammation and autoimmunity [UMCN 4.2]biologybusiness.industryMembrane Proteinsmedicine.diseaseCOPPArthritis ExperimentalHeme oxygenaseEnzyme ActivationPathogenesis and modulation of inflammation [N4i 1]Disease Models AnimalCytokineCyclooxygenase 2Mice Inbred DBAProstaglandin-Endoperoxide SynthasesImmunologyChronic DiseaseHeme Oxygenase (Decyclizing)biology.proteinDisease ProgressionTumor necrosis factor alphaJointsCyclooxygenasemedicine.symptombusinessInfection and autoimmunity [NCMLS 1]Heme Oxygenase-1
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Cytokine-mediated regulation of monocyte/macrophage cytotoxicity in human immunodeficiency virus-1 infection.

1992

Monocyte/macrophage-mediated tumor cytotoxicity was studied in patients infected with human immunodeficiency virus-1 (HIV-1) at various stages [Center for disease control (CDC) classification] of the disease. using the P-815 tumor cell line as target cells, the results demonstrated reduced monocyte/macrophage cytotoxicity early in HIV-1-related disease (CDCIII, P0.01). This cellular dysfunction sustained during the progression of the disease. Evidence could be presented that neither exogenous application of macrophage-stimulating cytokines (e.g. interferons) nor their endogenous induction in vitro restored monocyte/macrophage cytotoxicity. However, enhanced tumor necrosis factor (TNF)-alpha…

Microbiology (medical)AdultCytotoxicity Immunologicmedicine.medical_treatmentImmunologyHIV InfectionsBiologyVirusMonocytesmedicineTumor Cells CulturedImmunology and AllergyMacrophageHumansProstaglandin E2CytotoxicityCells CulturedTumor Necrosis Factor-alphaMonocyteInterleukinsMacrophagesGeneral MedicineMiddle AgedIn vitroCytokinemedicine.anatomical_structureImmunologyHIV-1CytokinesTumor necrosis factor alphaInterferonsmedicine.drugMedical microbiology and immunology
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LAAE-14, a new anti-inflammatory drug, increases the survival ofCandida albicans-inoculated mice

2003

LAAE-14, a lipidic acid-amido ether derivative, has been recently described as a new anti-inflammatory drug. We have studied the effect of treatment with this compound on the susceptibility of mice to in vivo experimental Candida albicans infection. ICR mice orally treated with LAAE-14 (25 mg kg(-1)) and experimentally intravenously infected showed a significantly increased survival as compared to control mice. In vitro, the compound did not inhibit the growth of C. albicans yeast cells or the yeast-to-hyphal transition. The in vitro production of prostaglandin E2 by peritoneal macrophages in response to the yeasts and hyphae of C. albicans was significantly decreased upon treatment with LA…

Microbiology (medical)Ratónmedicine.drug_classmedicine.medical_treatmentImmunologyHyphaeMicrobiologyDinoprostoneAnti-inflammatoryMicrobiologyGlutaratesMiceIn vivoCandida albicansmedicineAnimalsImmunology and AllergyProstaglandin E2Candida albicansCells CulturedMice Inbred ICRbiologyAnti-Inflammatory Agents Non-SteroidalCandidiasisGeneral Medicinebiology.organism_classificationSurvival AnalysisCorpus albicansIn vitroDisease Models AnimalInfectious DiseasesMacrophages PeritonealProstaglandin Emedicine.drugFEMS Immunology & Medical Microbiology
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Effect of apical root-end filling materials on gingival fibroblasts

2003

AIM To determine the influence of root-end filling materials on specific cellular responses of gingival fibroblasts (GF). METHODOLOGY The reactions of cells in contact with mineral trioxide aggregate (MTA), amalgam and a chemically inert titanium alloy were determined based on the assessment of prostaglandin (PGE2) release with and without arachidonic acid stimulation, protein and lactate synthesis, and cell proliferation. Cells cultured without test materials served as controls (100%). RESULTS The fibroblasts showed a highly significant decrease in protein synthesis when in contact with amalgam (61.8 +/- 13.6%); MTA (91.2 +/- 5.9%) and titanium (92.4 +/- 4.7%) had little effect on this par…

Mineral trioxide aggregateMaterials scienceCell growthbusiness.industrytechnology industry and agriculturechemistry.chemical_elementProstaglandinTitanium alloyDentistryMolecular biologychemistry.chemical_compoundchemistryArachidonic acidAmalgam (chemistry)businessGeneral DentistryIncubationTitaniumInternational Endodontic Journal
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Characterization of monoclonal antibodies generated against bovine and porcine prostacyclin synthase and quantitation of bovine prostacyclin synthase

1994

AbstractMonoclonal antibodies were raised against prostacyclin synthases purified from bovine and porcine aortae, respectively. Two monoclonal antibodies, RS1 and RS2, were purified and characterized. As shown by enzyme activity precipitation and Western blot analysis, in solubilized bovine and porcine aortae microsomes the monoclonal antibodies reacted only with prostacyclin synthase. The monoclonal antibody RS1 cross-reacts with partially purified prostacyclin synthase from human umbilical veins in an ELISA-based assay. None of the antibodies inhibited the enzyme activity. By combination of the monoclonal antibody RS2 with a polyclonal antibody we established an enzyme-linked immunosorben…

Monoclonal antibodyUmbilical VeinsSwinemedicine.drug_classProstaglandinBlotting WesternBiophysicsProstaglandinEnzyme-Linked Immunosorbent AssayProstacyclinMonoclonal antibodySensitivity and SpecificityBiochemistryProstacyclin synthasechemistry.chemical_compoundCytochrome P-450 Enzyme SystemWestern blotAntibody SpecificityStructural BiologyMicrosomesGeneticsmedicineAnimalsHumansTissue DistributionIsomerasesMolecular BiologyAortaImmunosorbent Techniquesbiologymedicine.diagnostic_testAntibodies MonoclonalCell BiologyMolecular biologyImmunohistochemistryIntramolecular OxidoreductasesBiochemistrychemistryPolyclonal antibodiesImmunoquantitationProstacyclin synthasebiology.proteinImmunohistochemistryCattleAntibodymedicine.drugFEBS Letters
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Relaxant effects of flavonoids on the mouse isolated stomach: structure-activity relationships.

2008

Flavonoids are a large heterogeneous group of benzo-gamma-pyrone derivatives, which are abundantly present in our diet. In this study we investigated the effects of six flavonoids (apigenin, genistein, quercetin, rutin, naringenin and catechin) on the gastric tone in mouse isolated stomach. The mechanical activity was recorded as changes of intraluminal pressure. All flavonoids tested produced a concentration-dependent relaxation, which was reversible after washout. The relative order of potency of the flavonoids was apigenin> or =genistein>quercetin>naringenin> or =rutin>catechin. Analysis of the chemical structure showed that the relaxant activity was progressively diminished by the prese…

NaringeninMalePotassium ChannelsFlavonoidGenisteinAction PotentialsIn Vitro TechniquesNitric OxideSettore BIO/09 - FisiologiaNitric oxidechemistry.chemical_compoundRutinMiceStructure-Activity RelationshipFlavonoids gastric relaxation smooth muscle potassium channels nitric oxideAnimalsheterocyclic compoundsPharmacologychemistry.chemical_classificationFlavonoidsNeuronsDose-Response Relationship DrugStomachfood and beveragesCatechinMuscle SmoothMice Inbred C57BLchemistryBiochemistryGastric MucosaApigeninProstaglandinsQuercetinMuscle ContractionEuropean journal of pharmacology
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