Search results for "TOXICITY"

showing 10 items of 2261 documents

Obesogen effect of bisphenol S alters mRNA expression and DNA methylation profiling in male mouse liver

2020

International audience; Environmental pollution is increasingly considered an important factor involved in the obesity incidence. Endocrine disruptors (EDs) are important actors in the concept of DOHaD (Developmental Origins of Health and Disease), where epigenetic mechanisms play crucial roles. Bisphenol A (BPA), a monomer used in the manufacture of plastics and resins is one of the most studied obesogenic endocrine disruptor. Bisphenol S (BPS), a BPA substitute, has the same obesogenic properties, acting at low doses with a sex-specific effect following perinatal exposure. Since the liver is a major organ in regulating body lipid homeostasis, we investigated gene expression and DNA methyl…

Malemedicine.medical_specialtyEnvironmental EngineeringHealth Toxicology and Mutagenesis[SDV]Life Sciences [q-bio]0208 environmental biotechnologyEnvironmental pollution02 engineering and technologyEndocrine Disruptors010501 environmental sciencesBiology01 natural sciencesEpigenesis GeneticPhenolsPregnancyInternal medicineToxicity TestsGene expressionmedicineAnimalsHumansEnvironmental ChemistryObesityRNA MessengerSulfonesEpigeneticsGene0105 earth and related environmental sciencesDose-Response Relationship DrugPublic Health Environmental and Occupational HealthGeneral MedicineGeneral ChemistryDNA MethylationLipid MetabolismPollution3. Good health020801 environmental engineeringMice Inbred C57BLEndocrinologyGene Expression RegulationLiverEndocrine disruptorPrenatal Exposure Delayed EffectsDNA methylationFemaleObesogenhormones hormone substitutes and hormone antagonistsDNA hypomethylation
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NMDA receptor antagonist felbamate reduces behavioral deficits and blood-brain barrier permeability changes after experimental subarachnoid hemorrhag…

2007

Increased levels of glutamate and aspartate have been detected after subarachnoid hemorrhage (SAH) that correlate with neurological status. The NMDA receptor antagonist felbamate (FBM; 2-phenyl-1,3-propanediol dicarbamate) is an anti-epileptic drug that elicits neuroprotective effects in different experimental models of hypoxia-ischemia. The aim of this dose-response study was to evaluate the effect of FBM after experimental SAH in rats on (1) behavioral deficits (employing a battery of assessment tasks days 1-5 post-injury) and (2) blood-brain barrier (BBB) permeability changes (quantifying microvascular alterations according to the extravasation of protein-bound Evans Blue by a spectropho…

Malemedicine.medical_specialtyExcitotoxicityPhenylcarbamatesBehavioral deficitsmedicine.disease_causeCisterna magnaBlood–brain barrierNeuroprotectionReceptors N-Methyl-D-AspartateFelbamateRats Sprague-Dawleychemistry.chemical_compoundInternal medicinemedicineAnimalsAnimals; Blood-Brain Barrier; Rats; Subarachnoid Hemorrhage; Evans Blue; Behavioral deficits; Cognitive deficits; NMDA receptor; FelbamatePostural BalanceEvans BlueBehavior AnimalDose-Response Relationship Drugbusiness.industryCognitive deficitsMicrocirculationBody WeightGlutamate receptorSubarachnoid HemorrhageNMDA receptorFelbamateRatsEndocrinologymedicine.anatomical_structureNeuroprotective AgentsSpectrometry FluorescencechemistryBlood-Brain BarrierPropylene GlycolsAnesthesiaCerebrovascular CirculationNMDA receptorNeurology (clinical)businessmedicine.drugEvans BlueJournal of neurotrauma
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Maternal Treatment of Spontaneously Hypertensive Rats With Pentaerythritol Tetranitrate Reduces Blood Pressure in Female Offspring

2014

Pentaerythritol tetranitrate is devoid of nitrate tolerance and shows no reproductive or developmental toxicity in animal studies. Recently, pentaerythritol tetranitrate has been demonstrated to reduce the risk of intrauterine growth restriction and the risk of preterm birth in women with abnormal placental perfusion. This study was conducted to test the perinatal programming effect of pentaerythritol tetranitrate in spontaneously hypertensive rats, a rat model of genetic hypertension. Parental spontaneously hypertensive rats were treated with pentaerythritol tetranitrate (50 mg/kg per day) during pregnancy and lactation periods; the offspring received standard chow without pentaerythritol …

Malemedicine.medical_specialtyGPX1Nitric Oxide Synthase Type IIIOffspringVasodilator AgentsDevelopmental toxicityBlood PressureVasodilationPentaerythritol tetranitratePentaerythritolchemistry.chemical_compoundPregnancyRats Inbred SHRInternal medicineInternal MedicinemedicineAnimalsPentaerythritol Tetranitratebusiness.industryGene Expression Regulation DevelopmentalDNARatsVasodilationHeme oxygenaseEndocrinologyBlood pressureAnimals NewbornchemistryMaternal ExposureHypertensionPregnancy AnimalFemaleEndothelium VascularbusinessHypertension
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Selective induction of bilirbuin UDP-glucuronosyl-transferase by perfluorodecanoic acid

1991

Differential effects of perfluorodecanoic acid (PFDA) on rat liver UDP-glucuronosyltransferase isoenzymes have been observed after a single i.p. administration of the compound to young male Sprague-Dawley rats. (1) Bilirubin glucuronidation was induced 2-fold. The induced state was stable for at least 3 weeks. (2) Glucuronidation of 1-naphthol, morphine and testosterone was decreased to half of the control values. These decreases were maximal after 12 days but all three activities returned to normal levels after 3 weeks. (3) Immunoblotting experiments indicated that the differential effects of PFDA on UDP-glucuronosyltransferase activities were due to modulation of enzyme protein concentrat…

Malemedicine.medical_specialtyGlucuronosyltransferaseBilirubinImmunoblottingGlucuronidationToxicologyIsozymechemistry.chemical_compoundInternal medicinemedicineAnimalsInducerGlucuronosyltransferaseEnzyme inducerTestosteroneFluorocarbonsDose-Response Relationship DrugbiologyRats Inbred StrainsGeneral MedicineRatsIsoenzymesEndocrinologychemistryEnzyme InductionToxicitybiology.proteinDecanoic AcidsChemico-Biological Interactions
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Endogenous nitric oxide is responsible for the early loss of P450 in cultured rat hepatocytes

1998

AbstractLoss of P450 during the early hours of monolayer formation is known to be the more serious limitation of primary cultured hepatocytes as an adequate model for the study of drug metabolism, toxicity and P450 induction. This study reports that endogenous nitric oxide (NO) formation is activated shortly after isolation by the classical collagenase-based liver perfusion methods. Both rapid P450 loss and aerobic mitochondrial energy metabolism impairment – with subsequent changes on glucose metabolism – are directly related to the high local generation of the radical at this stage. These effects can be reverted by the sole addition of NO biosynthesis inhibitors during liver perfusion and…

Malemedicine.medical_specialtyGlycogenolysisGlycogenolysisBiophysicsMitochondria LiverCarbohydrate metabolismHepatocyte primary cultureBiochemistryNitric oxideRats Sprague-DawleyP450 contentchemistry.chemical_compoundCytochrome P-450 Enzyme SystemBiosynthesisStructural BiologyInternal medicineGeneticsmedicineAnimalsGlycolysisMolecular BiologyCells CulturedAMPDrug metabolismAdenine NucleotidesNitric oxideCell BiologyLiver GlycogenRatsKineticsNG-Nitroarginine Methyl EsterEndocrinologyLiverchemistryToxicityMicrosomes LiverCollagenaseGlycolysisDrug metabolismmedicine.drugFEBS Letters
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Studies on the disposition, metabolism and hepatotoxicity of coumarin in the rat and Syrian hamster.

2002

The hepatotoxicity, metabolism and disposition of coumarin has been compared in male Sprague-Dawley rats and Syrian hamsters. The treatment of rats for 12, 24 and 42 weeks with diets containing 0.2 and 0.5% coumarin resulted in hepatotoxicity and increased relative liver weights. While levels of cytochrome P450 (CYP) and CYP-dependent enzymes were decreased, levels of reduced glutathione (GSH) and activities of UDP glucuronosyltransferase, gamma-glutamyltransferase and GSH S-transferase were increased. In contrast, coumarin produced few hepatic changes in the Syrian hamster. Following a single oral dose of 25 mg/kg [3-14C]coumarin, radioactivity was rapidly excreted by the rat and Syrian ha…

Malemedicine.medical_specialtyHamsterToxicologyRats Sprague-Dawleychemistry.chemical_compoundCytosolPharmacokineticsLiver Function TestsSpecies SpecificityOral administrationCoumarinsInternal medicineCricetinaemedicineToxicokineticsAnimalsheterocyclic compoundsTissue DistributionNutritionbiologyMesocricetusBody WeightCytochrome P450AnticoagulantsGeneral MedicineGlutathioneOrgan SizeCoumarinGlutathioneDietRatsEndocrinologychemistryLiverToxicitybiology.proteinChemical and Drug Induced Liver InjuryOxidation-ReductionFood ScienceFood and chemical toxicology : an international journal published for the British Industrial Biological Research Association
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Effect of acidosis on lipid peroxidation in brain slices.

1991

Acidification of the incubation medium markedly increased lipid peroxidation of cortical brain slices. Lactic acidosis caused a more extensive lipid peroxidation than did phosphoric acidosis (+35% at pH 6 and +81% at pH 5), probably due to the rapid diffusion of the protonated form of lactic acid across cell membranes. These results support the hypothesis that free radical mechanisms may be involved in the cytotoxicity of acidosis.

Malemedicine.medical_specialtyIn Vitro TechniquesPhosphatesLipid peroxidationchemistry.chemical_compoundSlice preparationInternal medicinemedicineAnimalsCytotoxicityMolecular BiologyIncubationAcidosisCerebral CortexChemistryGeneral NeuroscienceBrainRats Inbred StrainsMetabolismHydrogen-Ion Concentrationmedicine.diseaseLactic acidRatsKineticsEndocrinologyBiochemistryLactic acidosisLactatesNeurology (clinical)Lipid Peroxidationmedicine.symptomAcidosisDevelopmental BiologyBrain research
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Paradoxically, iron overload does not potentiate doxorubicin-induced cardiotoxicity in vitro in cardiomyocytes and in vivo in mice

2015

Doxorubicin (DOX) is known to induce serious cardiotoxicity, which is believed to be mediated by oxidative stress and complex interactions with iron. However, the relationship between iron and DOX-induced cardiotoxicity remains controversial and the role of iron chelation therapy to prevent cardiotoxicity is called into question. Firstly, we evaluated in vitro the effects of DOX in combination with dextran-iron on cell viability in cultured H9c2 cardiomyocytes and EMT-6 cancer cells. Secondly, we used an in vivo murine model of iron overloading (IO) in which male C57BL/6 mice received a daily intra-peritoneal injection of dextran-iron (15mg/kg) for 3weeks (D0-D20) and then (D21) a single su…

Malemedicine.medical_specialtyIron OverloadCell SurvivalHeart VentriclesIronCardiomegaly030204 cardiovascular system & hematologyToxicologymedicine.disease_causeCell LineVentricular MyosinsMice03 medical and health sciences0302 clinical medicine[SDV.MHEP.CSC]Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemAtrial natriuretic peptideIn vivoCell Line TumorInternal medicineNatriuretic Peptide Brainpolycyclic compoundsmedicineAnimalsMyocytes CardiacDoxorubicinViability assay030304 developmental biologyPharmacology0303 health sciencesCardiotoxicityCell growthChemistryDextransBrain natriuretic peptideCardiotoxicity[SDV.MHEP.CSC] Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemUp-Regulation3. Good healthMice Inbred C57BLOxidative Stresscell proliferationEndocrinologyDoxorubicincardiovascular systemOxidative stressmedicine.drugToxicology and Applied Pharmacology
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Sex differences in renal damage induced in the mouse by Amanita virosa.

1977

Summary The sensitivity of male and female mice to Amanita virosa was compared. Dried, homogenizedmushroom was given orally by stomach tubing at doses of 100, 200, 400 and 800 mg dried mushroom/kg body weight. Both in males and in females, the kidneys were the only organs showing macroscopical changes. The dose of 100 mg/kg caused renal damage in females, whereas in males the first signs of kidney damage were seen at the dose of 400 mg/kg. The renal lesions observed in the males were located in the cortex, while in the females they were limited to the outer stripe of the outer medullary zone. Testectomy diminished the nephrotoxicity of A. virosa in male mice and caused changes in the locali…

Malemedicine.medical_specialtyKidneyMedullary cavitybiologyRenal damageStomachAmanitaGeneral MedicineAmanita virosaMushroom Poisoningbiology.organism_classificationKidneyNephrotoxicityMicemedicine.anatomical_structureEndocrinologySex FactorsCortex (anatomy)Internal medicinemedicineAnimalsAmanita phalloidesFemaleCastrationExperimentelle Pathologie
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Absence of lipid peroxidation as determined by ethane exhalation in rats treated with 2, 3, 7, 8-tetrachlorodibenzo-p-dioxin (TCDD).

1985

The exhalation of ethane is widely used as an indicator of in vivo lipid peroxidation. To test the hypothesis that lipid peroxidative events are involved in the toxicity of 2, 3, 7, 8-tetrachlorodibenzo-p-dioxin (TCDD), we administered a lethal dose of TCDD (60 μg/kg), IP to male Sprague Dawley rats (160–180 g) and measured by gas chromatography the exhalation of ethane into the atmosphere of a closed all-glass exposure chamber. TCDD-treated rats exhaled only slightly more ethane than control rats at a single time point 7 days following TCDD administration. Since the exhalation of ethane is the net result of the endogenous production of the gas and its metabolic degradation, the latter was …

Malemedicine.medical_specialtyLipid PeroxidesPolychlorinated DibenzodioxinsHealth Toxicology and MutagenesisEndogenyToxicologyDioxinsLipid peroxidationchemistry.chemical_compoundCytochrome P-450 Enzyme SystemIn vivoInternal medicinemedicineAnimalsheterocyclic compoundsEthaneChemistryLethal doseExhalationRats Inbred StrainsGeneral MedicineMetabolismTetrachlorodibenzo-p-dioxinRatsstomatognathic diseasesEndocrinologyBiochemistryToxicityArchives of toxicology
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