Search results for "TOXICITY"

showing 10 items of 2261 documents

When the functionalisation comes in useful: ionic liquids with a “sweet” appended moiety demonstrate drastically reduced toxicological effects

2020

The growing number of applications of ionic liquids (ILs) in industry have brought attention to the green credentials of synthesis, as well as their cytotoxicities and ecotoxicities both for their use and accidental leakage into the environment. With the abovementioned properties in mind, we designed a class of ILs with either cations bearing a gluconamide motif and aliphatic side chains or the anion incorporating a gluconic acid (derived from food waste) moiety. An IL with an imidazolium cation with an appended gluconic amide (bearing five hydroxyl groups) moiety was also synthesized for a useful comparison. Different structural features were considered, placing emphasis on the nature and …

CytotoxicityGeneral Chemical Engineeringphysicochemical properties02 engineering and technology010402 general chemistry01 natural sciencesionic liquidschemistry.chemical_compoundSDG 3 - Good Health and Well-beinggluconic acidEnvironmental ChemistryMoietyecotoxicityRenewable Energy Sustainability and the EnvironmentChemistrySettore CHIM/06 - Chimica OrganicaGeneral Chemistryionic liquids gluconic acid cytotoxicity ecotoxicity physicochemical properties021001 nanoscience & nanotechnologyCombinatorial chemistry0104 chemical sciencesCytotoxicity; ecotoxicity; gluconic acid; ionic liquids; physicochemical propertiesIonic liquidGluconic acidSurface modification/dk/atira/pure/sustainabledevelopmentgoals/good_health_and_well_beingEcotoxicity0210 nano-technology
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IMPAIRMENT OF DIASTOLIC FUNCTION DURING SHORT-TERM ANTHRACYCLINE CHEMOTHERAPY

1995

International audience; Abstract: Objective-To assess the early changes in left ventricular diastolic and systolic function due to anthracycline treatment. Design-A prospective study of cardiac function by radionuclide angiography in adults before and one month after the end of anthracycline treatment. Patients-60 patients without cardiac disease treated with chemotherapy containing anthracycline. Methods-Cardiac function was assessed by radionuclide measurement throughout treatment. Ejection fraction, peak ejection rate, time to peak ejection rate, filling rate, and time to peak filling rate were measured before and after treatment, To normalise radionuclide measurements of the left ventri…

DIASTOLIC FUNCTIONanimal structures[ INFO.INFO-IM ] Computer Science [cs]/Medical ImagingRADIONUCLIDE ANGIOGRAPHY[INFO.INFO-IM] Computer Science [cs]/Medical Imaging[INFO.INFO-IM]Computer Science [cs]/Medical ImagingANTHRACYCLINE CARDIOTOXICITY
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On the relevance of genotoxicity for fish populations II: genotoxic effects in zebrafish (Danio rerio) exposed to 4-nitroquinoline-1-oxide in a compl…

2003

In order to characterize the impact of genotoxic potentials on populations of aquatic organisms in surface waters, zebrafish (Danio rerio) were exposed to the model genotoxicant 4-nitroquinoline-1-oxide (NQO) in a complete life-cycle test. Fish exposed to mean NQO concentrations of 0, 0.1, 0.3, 1.1, and 2.9 microg/l were examined by several genotoxicity assays with different endpoints. Assays included the unscheduled DNA synthesis (UDS) test, the comet assay, the alkaline filter elution, and the micronucleus test. The genotoxicity assays revealed an increasing genotoxicity, ranging from induction of DNA repair (even at the lowest concentration tested) to primary and secondary DNA alteration…

DNA ReplicationDNA RepairDNA repairHealth Toxicology and Mutagenesis4-Nitroquinoline 1-oxideDanioAquatic ScienceBiologymedicine.disease_causechemistry.chemical_compoundmedicineEcotoxicologyAnimalsToxicity Tests ChronicZebrafishGeneticsMicronucleus TestsDose-Response Relationship DrugMutagenicity Testsbiology.organism_classificationMolecular biology4-Nitroquinoline-1-oxideComet assaychemistryMicronucleus testToxicityComet AssayGenotoxicityMutagensAquatic toxicology (Amsterdam, Netherlands)
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The autoradiographic test for unscheduled DNA synthesis: a sensitive assay for the detection of DNA repair in the HepG2 cell line

2004

International audience; We assessed the DNA-repair capacity of HepG2 cells, which were derived from a human hepatoma, by the unscheduled DNA synthesis assay, using the autoradiography protocol (UDS-AR). We evaluated DNA repair following exposure to direct mutagens (4-nitroquinoline-N-oxide (4-NQO), methyl methanesulfonate (MMS)), to mutagens requiring metabolic activation (benzo[a]pyrene (B[a]P), 2-acetylaminofluorene (2-AAF), N-dimethylnitrosoamine (NDMA)) or to structurally related non-mutagens such as pyrene and 4-acetylaminofluorene (4-AAF). All positive compounds tested induced UDS in HepG2 cells. With 4-NQO and MMS, a concentration-dependent increase in net nuclear grains per cell was…

DNA ReplicationDNA RepairEndpoint DeterminationDNA damageDNA repairHealth Toxicology and Mutagenesis[SDV]Life Sciences [q-bio]Mutagen[SDV.TOX.TCA]Life Sciences [q-bio]/Toxicology/Toxicology and food chainBiologymedicine.disease_causeHEPG203 medical and health scienceschemistry.chemical_compound0302 clinical medicineTumor Cells CulturedGeneticsmedicineHumansComputingMilieux_MISCELLANEOUS030304 developmental biologyGeneticsAnalysis of Variance0303 health sciencesfungiMolecular biologyMethyl methanesulfonate[SDV] Life Sciences [q-bio]chemistryCell culture030220 oncology & carcinogenesisAutoradiographyRegression AnalysisPyreneGenotoxicityDNAMutagens
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A microplate version of the DNA-synthesis inhibition test for rapid detection of DNA-alteration potentials.

1990

A microplate version of the DNA-synthesis inhibition test (DIT) for fast detection of DNA-alteration potentials has been developed. The DIT is based on the concept that DNA damage causes inhibition of DNA synthesis that becomes detectable some time after replicating cells have been in contact with genotoxic agents. In this test procedure human tissue culture cells (HeLa S3), prelabeled with [14C]thymidine, arfe exposed for 90 min to the substances in question. After the cells are rinsed, they are allowed to recover for 2 1/2 h in fresh culture medium, thereby unspecific interactions interfering with DNA replication are practically eliminated. Next, [3H]thymidine is added for 30 min, and the…

DNA ReplicationDNA damageBiophysicsBiologymedicine.disease_causeBiochemistryDNA Synthesis Inhibitionchemistry.chemical_compoundmedicineBenzo(a)pyreneHumansMolecular BiologyChromatographyAutoanalysisDNA synthesisMutagenicity TestsDNA replicationNitroquinolinesCell BiologyDNAMolecular biologychemistryCell cultureMutationThymidineDNAGenotoxicityDNA DamageHeLa CellsMutagensAnalytical biochemistry
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Ras-Related GTPase RhoB Forces Alkylation-Induced Apoptotic Cell Death

2000

rhoB encoding a Ras-related GTPase is immediate-early inducible by genotoxic treatments. To address the question of the physiological role of RhoB in cellular defense, cells stably overexpressing wild-type RhoB protein were generated. Overexpression of RhoB renders cells hypersensitive to the killing effect of alkylating agents including antineoplastic drugs but not to UV-light and doxorubicin. As compared to control cells, RhoB overexpressing cells revealed an increase in the frequency of alkylation-induced apoptotic cell death. This indicates that RhoB is involved in modulating apoptotic signaling. Furthermore, overexpression of RhoB resulted in a prolonged transient block to DNA replicat…

DNA ReplicationDNA ComplementaryAlkylationDNA RepairUltraviolet RaysRHOBBiophysicsApoptosisGTPaseBiologyTransfectionBiochemistryMicechemistry.chemical_compoundRhoB GTP-Binding ProteinmedicineAnimalsDoxorubicinrhoB GTP-Binding ProteinCytotoxicityAntineoplastic Agents AlkylatingMolecular BiologyDNA replication3T3 CellsCell BiologyMethyl MethanesulfonateRatsCell biologychemistryApoptosisCancer researchDNADNA Damagemedicine.drugBiochemical and Biophysical Research Communications
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Genotoxicity of the fungicide dichlofluanid in seven assays

1991

Seven different endpoints for detection of genotoxicity have been used to demonstrate the DNA-altering properties of Dichlofluanid, a fungicide commonly used in viticulture pest control. Each endpoint (DNA synthesis inhibition test, alkaline viscosimetry, umu-test, alkaline filter elution, FADU-test, 32P-postlabeling, and electron microscopy) shows clear evidence of genotoxicity. These data indicate that application of the fungicide dichlofluanid may be mutagenic and/or carcinogenic for exposed humans.

DNA ReplicationSalmonella typhimuriumDNA AlterationEpidemiologyHealth Toxicology and MutagenesisDichlofluanidmedicine.disease_causeCell LineMicechemistry.chemical_compoundmedicineAnimalsHumansBioassayGenetics (clinical)CaptanCarcinogenAniline CompoundsMutagenicity TestsFishesDNAPesticideFungicides IndustrialFungicideBiochemistrychemistryGenotoxicityDNA DamageHeLa CellsMutagensEnvironmental and Molecular Mutagenesis
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In Vitro Assessment of the Genotoxic Hazard of Novel Hydroxamic Acid- and Benzamide-Type Histone Deacetylase Inhibitors (HDACi)

2020

Histone deacetylase inhibitors (HDACi) are already approved for the therapy of leukemias. Since they are also emerging candidate compounds for the treatment of non-malignant diseases, HDACi with a wide therapeutic window and low hazard potential are desirable. Here, we investigated a panel of 12 novel hydroxamic acid- and benzamide-type HDACi employing non-malignant V79 hamster cells as toxicology guideline-conform in vitro model. HDACi causing a &ge

DNA damageApoptosisHydroxamic AcidsDNA damage responseArticleCatalysisCell LineHistonesInorganic Chemistrylcsh:Chemistrychemistry.chemical_compoundHDAC inhibitorsCricetinaeDNA strand breaksmedicineAnimalsHumansDNA Breaks Double-StrandedDNA Breaks Single-StrandedPhosphorylationPhysical and Theoretical Chemistrynormal tissue toxicityMolecular BiologyVorinostatlcsh:QH301-705.5SpectroscopyVorinostatMicronucleus TestsHydroxamic acidMutagenicity TestsEntinostatOrganic ChemistryHistone H2AXgenetic instabilityGeneral MedicineComputer Science ApplicationsHistone Deacetylase Inhibitorschemistrylcsh:Biology (General)lcsh:QD1-999BenzamidesCancer researchComet AssayHistone deacetylasegenotoxic hazardDNAMutagensNucleotide excision repairmedicine.drugInternational Journal of Molecular Sciences
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Detection of DNA damage in stimulated human lymphocytes after enflurane exposure in vitro

1992

DNA damage was detected by nucleoid sedimentation in human lymphocytes stimulated with pokeweed mitogen after exposure to enflurane. Enflurane induces DNA damage at an exposure concentration of 0.2 vol%. Higher enflurane concentrations increase the rate of DNA damage. The DNA damage seen after exposure to enflurane concentrations of 0.2 and 3.0% vol is comparable to damage after X-radiation of 0.1 and 0.7 Gy. DNA single-strand breaks can be demonstrated by nucleoid sedimentation and can indicate damage before DNA repair begins. Therefore, detected DNA single-strand breaks may be reversible. However, DNA repair is not always successful and an increased number of DNA single-strand breaks coul…

DNA damageDNA repairLymphocytePokeweed mitogenEnfluraneDNA Single-StrandedBiologyLymphocyte ActivationBiochemistryMolecular biologyEnfluranechemistry.chemical_compoundmedicine.anatomical_structurechemistryBiochemistryToxicitymedicineHumansNucleoidLymphocytesCells CulturedDNADNA DamageGeneral Environmental Sciencemedicine.drugEnvironmental Research
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Photoinduced DNA damage efficiency and cytotoxicity of novel viologen linked pyrene conjugates.

2010

Novel viologen linked pyrene conjugates permeate cells efficiently and exhibit spacer length dependent DNA damage and cytotoxicity upon photoexcitation.

DNA damagePhotochemistryUltraviolet RaysPhotochemistryCatalysisViologenschemistry.chemical_compoundMiceMaterials ChemistrymedicineAnimalsCytotoxicityPyrenesChemistryMetals and AlloysViologenGeneral ChemistryPermeationSurfaces Coatings and FilmsElectronic Optical and Magnetic MaterialsPhotoexcitationCeramics and CompositesPyrenemedicine.drugConjugateDNA DamageChemical communications (Cambridge, England)
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