Search results for "TRANSCRIPTION FACTORS"

showing 10 items of 848 documents

In Vitro Analysis of the Two-Component System MtrB-MtrA from Corynebacterium glutamicum▿ †

2007

ABSTRACT The two-component system MtrBA is involved in the osmostress response of Corynebacterium glutamicum . MtrB was reconstituted in a functionally active form in liposomes and showed autophosphorylation and phosphatase activity. In proteoliposomes, MtrB activity was stimulated by monovalent cations used by many osmosensors for the detection of hypertonicity. Although MtrB was activated by monovalent cations, they lead in vitro to a general stabilization of histidine kinases and do not represent the stimulus for MtrB to sense hyperosmotic stress.

PhosphataseCorynebacteriumEnzyme ActivatorsMicrobiologyCorynebacterium glutamicumEnzyme activatorBacterial ProteinsOsmotic PressurePhosphorylationMolecular BiologyHistidinebiologyAutophosphorylationRNA-Binding ProteinsCations Monovalentbiology.organism_classificationAdaptation PhysiologicalTwo-component regulatory systemPhosphoric Monoester HydrolasesCorynebacterium glutamicumBiochemistryLiposomesPhosphorylationATP-Binding Cassette TransportersSignal TransductionTranscription Factors
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Plant Occurring Flavonoids as Modulators of the Aryl Hydrocarbon Receptor

2021

International audience; The aryl hydrocarbon receptor (AhR) is a transcription factor deeply implicated in health and diseases. Historically identified as a sensor of xenobiotics and mainly toxic substances, AhR has recently become an emerging pharmacological target in cancer, immunology, inflammatory conditions, and aging. Multiple AhR ligands are recognized, with plant occurring flavonoids being the largest group of natural ligands of AhR in the human diet. The biological implications of the modulatory effects of flavonoids on AhR could be highlighted from a toxicological and environmental concern and for the possible pharmacological applicability. Overall, the possible AhR-mediated harmf…

PhytochemicalsPharmaceutical ScienceDioxin ReceptorReview[CHIM.THER]Chemical Sciences/Medicinal ChemistryPharmacologyFlavonesAnalytical Chemistryfunctional food03 medical and health scienceschemistry.chemical_compoundQD241-4410302 clinical medicineFlavonolsDrug DiscoveryAnimalsHumansxenobioticsPhysical and Theoretical ChemistryTranscription factorBeneficial effectstranscription factor030304 developmental biologychemistry.chemical_classificationFlavonoidsbioactive0303 health sciencesphytocompoundsbiologyfungiOrganic Chemistryfood and beveragesPolyphenolsdioxin receptorrespiratory systemIsoflavonesAryl hydrocarbon receptorIsoflavonesrespiratory tract diseases3. Good healthchemistryReceptors Aryl HydrocarbonChemistry (miscellaneous)030220 oncology & carcinogenesisAh receptorbiology.proteinMolecular MedicineXenobioticTranscription FactorsMolecules
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Homeobox genes and sea urchin development.

2000

We describe the expression of three Paracentrotus lividus homeobox-containing genes of the dispersed class during sea urchin embryogenesis and discuss their possible roles in the mechanisms of cell specification and embryo morphogenesis. PIHbox12 represents the first regulator identified in sea urchin that belongs to the zygotic class of transcription factors. Its early and transient expression and the localization of transcripts suggests that PIHbox12 is involved in cell fate specification of the oral or aboral ectodermal territories at the early cleavage stages. PIHbox9 is expressed just after the completion of gastrulation in a narrow stripe of cells at the ectoderm-endoderm boundary. It…

PlOtpSea urchinEmbryo NonmammalianTime FactorsPlHbox9Genes HomeoboxSettore BIO/11 - Biologia MolecolareNerve Tissue ProteinsHomeobox genesBone and BonesPlHbox12Hox genesSea UrchinsAnimalsCloning MolecularIn Situ HybridizationTranscription Factors
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Gata4 Blocks Somatic Cell Reprogramming By Directly Repressing Nanog

2012

Abstract Somatic cells can be reprogrammed to induced pluripotent stem (iPS) cells by ectopic expression of the four factors Oct4, Klf4, Sox2, and Myc. Here, we investigated the role of Gata4 in the reprogramming process and present evidence for a negative role of this family of transcription factors in the induction of pluripotency. Coexpression of Gata4 with Oct4, Klf4, and Sox2 with or without Myc in mouse embryonic fibroblasts greatly impaired reprogramming and endogenous Nanog expression. The lack of Nanog upregulation was associated with a blockade in the transition from the initiation phase of reprogramming to the full pluripotent state characteristic of iPS cells. Addition of Nanog …

Pluripotent Stem CellsTranscriptional ActivationHomeobox protein NANOGChromatin ImmunoprecipitationTranscription GeneticRex1Kruppel-Like Transcription FactorsDown-RegulationElectrophoretic Mobility Shift AssayBiologyCell LineProto-Oncogene Proteins c-mycKruppel-Like Factor 4MiceSOX2AnimalsRNA MessengerRNA Small InterferingInduced pluripotent stem cellEmbryonic Stem Cellsreproductive and urinary physiologyHomeodomain ProteinsSOXB1 Transcription FactorsNanog Homeobox ProteinCell DifferentiationNanog Homeobox ProteinCell BiologyCellular ReprogrammingEmbryonic stem cellGATA4 Transcription FactorKLF4embryonic structuresHepatocyte Nuclear Factor 3-betaCancer researchMolecular MedicineRNA Interferencebiological phenomena cell phenomena and immunityOctamer Transcription Factor-3ReprogrammingDevelopmental BiologyStem Cells
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Multiple Pluripotent Stem Cell Markers in Human Anaplastic Thyroid Cancer: The Putative Upstream Role of SOX2

2013

Anaplastic thyroid carcinoma (ATC) is a rare and aggressive endocrine tumor with highly undifferentiated morphology. It has been suggested that cancer stem cells (CSCs) might play a central role in ATC. The objectives of this study were (i) to characterize CSCs from ex vivo ATC specimens by investigating the expression of several pluripotent stem cell markers, and (ii) to evaluate in vitro drug resistance modifications after specific CSC transcription factor switch-off.In ex vivo experiments, eight formalin-fixed, paraffin-embedded ATC specimens were analyzed by reverse-transcription and real-time quantitative PCR and immunohistochemistry. In in vitro experiments using ATC SW1736 cells, the…

Pluripotent Stem Cellscongenital hereditary and neonatal diseases and abnormalitiesEndocrinology Diabetes and MetabolismDown-RegulationBiologyThyroid Carcinoma AnaplasticAnaplastic thyroid cancer cancer stem cell SOX-2Settore MED/13 - EndocrinologiaThyroid carcinomaKruppel-Like Factor 4EndocrinologySOX2Cancer stem cellCell Line TumormedicineATP Binding Cassette Transporter Subfamily G Member 2HumansNeoplasmThyroid NeoplasmsAnaplastic thyroid cancerInduced pluripotent stem cellHomeodomain ProteinsSOXB1 Transcription FactorsNanog Homeobox ProteinNanog Homeobox ProteinThyroid Cancer and Nodulesmedicine.diseaseNeoplasm ProteinsUp-RegulationDoxorubicinDrug Resistance NeoplasmCell cultureNeoplastic Stem CellsCancer researchATP-Binding Cassette TransportersCisplatinOctamer Transcription Factor-3Thyroid
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The nucleosome-remodeling ATPase ISWI is regulated by poly-ADP-ribosylation.

2008

ATP-dependent nucleosome-remodeling enzymes and covalent modifiers of chromatin set the functional state of chromatin. However, how these enzymatic activities are coordinated in the nucleus is largely unknown. We found that the evolutionary conserved nucleosome-remodeling ATPase ISWI and the poly-ADP-ribose polymerase PARP genetically interact. We present evidence showing that ISWI is target of poly-ADP-ribosylation. Poly-ADP-ribosylation counteracts ISWI function in vitro and in vivo. Our work suggests that ISWI is a physiological target of PARP and that poly-ADP-ribosylation can be a new, important post-translational modification regulating the activity of ATP-dependent nucleosome remodel…

Poly Adenosine Diphosphate RiboseImmunoprecipitationQH301-705.5Poly ADP ribose polymeraseATPaseBlotting WesternBiochemistryChromosomesGeneral Biochemistry Genetics and Molecular BiologySettore BIO/10 - BiochimicaAnimalsDrosophila ProteinsImmunoprecipitationNucleosomeBiology (General)Transcription factorIn Situ Hybridization FluorescencePolymeraseAdenosine TriphosphatasesGeneral Immunology and MicrobiologybiologyGeneral NeuroscienceGenetics and GenomicsPARP ISWI Poly(ADP)ribosylation Chromatin remodellingCell BiologyChromatinISWI PARPNucleosomesChromatinSettore BIO/18 - GeneticaDrosophila melanogasterBiochemistrybiology.proteinPoly(ADP-ribose) PolymerasesGeneral Agricultural and Biological SciencesFunction (biology)Transcription FactorsResearch ArticlePLoS Biology
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Oligodendrocytes in mouse corpus callosum are coupled via gap junction channels formed by connexin47 and connexin32.

2010

According to previously published ultrastructural studies, oligodendrocytes in white matter exhibit gap junctions with astrocytes, but not among each other, while in vitro oligodendrocytes form functional gap junctions. We have studied functional coupling among oligodendrocytes in acute slices of postnatal mouse corpus callosum. By whole-cell patch clamp we dialyzed oligodendrocytes with biocytin, a gap junction-permeable tracer. On average 61 cells were positive for biocytin detected by labeling with streptavidin-Cy3. About 77% of the coupled cells stained positively for the oligodendrocyte marker protein CNPase, 9% for the astrocyte marker GFAP and 14% were negative for both CNPase and GF…

PolydendrocytesPatch-Clamp TechniquesPopulationNerve Tissue ProteinsBiologyIn Vitro TechniquesConnexinsCorpus CallosumOLIG2Cellular and Molecular Neurosciencechemistry.chemical_compoundMyelinMiceBiocytinGlial Fibrillary Acidic ProteinmedicineBasic Helix-Loop-Helix Transcription FactorsConnexin 30AnimalsAntigenseducationMice Knockouteducation.field_of_studyLysineStem CellsGap junctionGap JunctionsCarbocyaninesOligodendrocyte Transcription Factor 2OligodendrocyteCell biologyMice Inbred C57BLOligodendrogliamedicine.anatomical_structureNeurologychemistryAstrocytesProteoglycansStreptavidin2'3'-Cyclic-Nucleotide PhosphodiesterasesNeuroscienceAstrocyteGlia
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Excess of de novo variants in genes involved in chromatin remodelling in patients with marfanoid habitus and intellectual disability.

2020

PurposeMarfanoid habitus (MH) combined with intellectual disability (ID) (MHID) is a clinically and genetically heterogeneous presentation. The combination of array CGH and targeted sequencing of genes responsible for Marfan or Lujan–Fryns syndrome explain no more than 20% of subjects.MethodsTo further decipher the genetic basis of MHID, we performed exome sequencing on a combination of trio-based (33 subjects) or single probands (31 subjects), of which 61 were sporadic.ResultsWe identified eight genes with de novo variants (DNVs) in at least two unrelated individuals (ARID1B, ATP1A1, DLG4, EHMT1, NFIX, NSD1, NUP205 and ZEB2). Using simulation models, we showed that five genes (DLG4, NFIX, …

ProbandMale[SDV]Life Sciences [q-bio]intellectual deficiencyMESH: NFI Transcription Factorschromatin remodelingMarfan SyndromeCraniofacial AbnormalitiesMESH: ChildIntellectual disabilityMESH: Craniofacial AbnormalitiesMESH: Mental Retardation X-LinkedExomeChildde novo variantsGenetics (clinical)Exome sequencingGeneticsMESH: ExomeMESH: Middle AgedbiologyMESH: Genetic Predisposition to DiseaseMiddle AgedNFIXMESH: Young AdultFemaleAdultMESH: MutationAdolescentChromatin remodelingMESH: Intellectual DisabilityMESH: Marfan SyndromeEHMT1Young AdultMESH: Whole Exome SequencingIntellectual DisabilityExome SequencingGeneticsmedicineHumansGenetic Predisposition to Diseasemarfanoid habitusGeneMESH: Neurodevelopmental DisordersMESH: AdolescentMESH: HumansGenetic heterogeneityMESH: Chromatin Assembly and DisassemblyMESH: Histone-Lysine N-MethyltransferaseMESH: AdultHistone-Lysine N-Methyltransferasemedicine.diseaseChromatin Assembly and DisassemblyMESH: MaleNFI Transcription FactorsNeurodevelopmental DisordersMutationbiology.proteinMental Retardation X-LinkedMESH: FemaleJournal of medical genetics
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Cell cycle independent role of Cyclin E during neural cell fate specification in Drosophila is mediated by its regulation of Prospero function

2009

AbstractDuring development, neural progenitor cells or neuroblasts generate a great intra- and inter-segmental diversity of neuronal and glial cell types in the nervous system. In thoracic segments of the embryonic central nervous system of Drosophila, the neuroblast NB6-4t undergoes an asymmetric first division to generate a neuronal and a glial sublineage, while abdominal NB6-4a divides once symmetrically to generate only 2 glial cells. We had earlier reported a critical function for the G1 cyclin, CyclinE (CycE) in regulating asymmetric cell division in NB6-4t. Here we show that (i) this function of CycE is independent of its role in cell cycle regulation and (ii) the two functions are m…

ProsperoNerve Tissue ProteinsStem cellsCyclinEBiologyCell fate determinationNeuroblastNeuroblastsCyclin EAsymmetric cell divisionAnimalsDrosophila ProteinsCell LineageMolecular BiologyNeural cellCell ProliferationSequence DeletionNeuronsCell fate determinationCell CycleNuclear ProteinsCell DifferentiationCell BiologyCell cycleNeural stem cellUp-RegulationCell biologyProtein TransportDrosophila melanogasternervous systemDrosophilaCNSStem cellGanglion mother cellBiomarkersProtein BindingTranscription FactorsDevelopmental BiologyDevelopmental Biology
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The Role of Low Complexity Regions in Protein Interaction Modes: An Illustration in Huntingtin

2021

Low complexity regions (LCRs) are very frequent in protein sequences, generally having a lower propensity to form structured domains and tending to be much less evolutionarily conserved than globular domains. Their higher abundance in eukaryotes and in species with more cellular types agrees with a growing number of reports on their function in protein interactions regulated by post-translational modifications. LCRs facilitate the increase of regulatory and network complexity required with the emergence of organisms with more complex tissue distribution and development. Although the low conservation and structural flexibility of LCRs complicate their study, evolutionary studies of proteins …

Protein Conformation alpha-Helical0301 basic medicineNetwork complexityHuntingtinintrinsically disordered regionsAmino Acid MotifsComputational biologyBiologyprotein interactionsArticlecompositionally biased regionsCatalysisProtein–protein interactionlcsh:ChemistryEvolution MolecularInorganic ChemistryLow complexity03 medical and health sciencesProtein DomainsProtein Interaction MappingAnimalsHumansp300-CBP Transcription FactorsAmino Acid SequenceProtein Interaction MapsHuntingtinTissue distributionPhysical and Theoretical Chemistrylcsh:QH301-705.5Molecular BiologySpectroscopyHuntingtin Protein030102 biochemistry & molecular biologyOrganic ChemistryNuclear Proteinsp120 GTPase Activating ProteinGeneral MedicineMultiple modesSynapsinslow complexity regionsComputer Science ApplicationshomorepeatsMicroscopy Electron030104 developmental biologylcsh:Biology (General)lcsh:QD1-999Sequence AlignmentFunction (biology)Protein BindingInternational Journal of Molecular Sciences
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