Search results for "TYR"

showing 10 items of 2017 documents

Continuous intra-amygdalar infusion of GABA in the amygdala kindling model of epilepsy in rat.

2003

Objective: To explore the effect of continuous intra-amygdalar infusion of GABA in the amygdala kindling model of epilepsy in rat. Methods: An electrode and cannula complex was implanted in adult male Wistar rats, the electrode being targeted to the left basolateral amygdala. The animals were subjected to a standard kindling procedure. Osmotic minipumps filled with either GABA or mannitol were connected to cannulas and allowed to infuse during 7 days. Kindling experiments measuring after-discharge and seizure thresholds, seizure severity and duration, and behavioral toxicity were performed before, during and after the drug infusion period. Results: Both after-discharge and seizure threshold…

MaleTime FactorsCentral nervous systemPharmacologyAmygdalachemistry.chemical_compoundEpilepsyBasal gangliamedicineKindling NeurologicAnimalsRats WistarNeurotransmittergamma-Aminobutyric AcidEpilepsyDose-Response Relationship Drugbusiness.industryKindlingInfusion Pumps Implantablemedicine.diseaseAmygdalaElectric StimulationRatsDisease Models Animalmedicine.anatomical_structurenervous systemNeurologychemistryAnesthesiaToxicityAnticonvulsantsNeurology (clinical)businessBasolateral amygdalaEpilepsy research
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Inhibition of the epidermal growth factor receptor tyrosine kinase activity by leflunomide.

1993

AbstractThe active metabolite of leflunomide, A77 1726 inhibits the proliferation of a variety of mammalian cell lines in culture. Epidermal growth factor (EGF)-dependent proliferation is inhibited by A77 1726 at an effective dose of 30–40 μM. A77 1726 appears to directly inhibit the EGF receptor tyrosine-specific kinase activity both in intact cells and purified EGF receptors at the same effective dose. These data suggest that leflunomide inhibits cellular proliferation by the inhibition of tyrosine-specific kinase activities.

MaleToluidinesmedicine.medical_treatmentBiophysicsHydroxybutyratesBiochemistryKB CellsCell LineHuman foreskin fibroblast cellStructural BiologyEpidermal growth factorNitrilesGeneticsmedicineTumor Cells CulturedAnimalsHumansEpidermal growth factor receptorKinase activityPhosphorylationReceptorMolecular BiologyCells CulturedSkinAniline CompoundsbiologyCell growthKinaseEpidermal growth factor receptorGrowth factorAnti-Inflammatory Agents Non-SteroidalCell BiologyIsoxazolesFibroblastsTyrosine-specific kinaseCell biologyErbB ReceptorsBiochemistryCrotonatesbiology.proteinCarcinoma Squamous CellPlatelet-derived growth factor receptorLeflunomideFEBS letters
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Phenotypic expression of the fibroblast growth factor receptor 3 (FGFR3) mutation P250R in a large craniosynostosis family.

1997

The craniosynostosis syndromes are a heterogeneous group of sporadic, autosomal dominant disorders with significant clinical overlap. Recently, we described a large family with autosomal dominant craniosynostosis suggestive of Saethre-Chotzen syndrome, in which linkage to the Saethre-Chotzen syndrome loci on 7p had been excluded. We now report the presence of a mutation in the fibroblast growth factor receptor 3 (FGFR3) in this family. The mutation, P250R, had been previously reported in 10 patients with non-syndromic craniosynostosis. Variable expression of this mutation is evident especially in two additional members of this family, one of whom is severely affected with pancraniosynostosi…

MaleTurkish populationGenetic LinkageBiologyMuenke syndromeCraniosynostosisVariable ExpressionCraniosynostosesGenetic linkageGeneticsmedicineHumansReceptor Fibroblast Growth Factor Type 3Genetics (clinical)GeneticsGenetic heterogeneityInfant NewbornInfantProtein-Tyrosine KinasesFibroblast growth factor receptor 3medicine.diseaseReceptors Fibroblast Growth FactorPedigreePhenotypeMutationMutation (genetic algorithm)FemaleResearch ArticleJournal of Medical Genetics
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Characterization of a population of tyrosine hydroxylase-containing interneurons in the external plexiform layer of the rat olfactory bulb

2012

The olfactory bulb (OB) of mammals contains the major endogenous dopamine-producing system in the forebrain. The vast majority of dopaminergic neurons consists of juxtaglomerular cells, which innervate the olfactory glomeruli and modulate the entrance of sensory information to the OB. Although dopaminergic juxtaglomerular cells have been widely investigated, the presence of dopaminergic interneurons other than juxtaglomerular cells has been largely unexplored. In this study, we analyze a population of tyrosine hydroxylase (TH)-containing interneurons located in the external plexiform layer (EPL) of the rat OB. These interneurons are GABAergic and morphologically heterogeneous. They have an …

MaleTyrosine 3-MonooxygenasePopulationOlfactionBiologyInterneuronsPostsynaptic potentialmedicineAnimalsRats WistarAxoneducationgamma-Aminobutyric Acideducation.field_of_studyGeneral NeuroscienceDopaminergicDendritesOlfactory BulbRatsOlfactory bulbParvalbuminsmedicine.anatomical_structurenervous systemSynapsesForebrainGABAergicNeuroscienceNeuroscience
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Acute inactivation of the medial forebrain bundle imposes oscillations in the SNr: a challenge for the 6-OHDA model?

2010

It has been recently shown that the substantia nigra pars reticulata (SNr) of 6-hydroxydopamine (6-OHDA)-lesioned rats, under urethane anaesthesia, manifests a prominent low frequency oscillation (LFO) of around 1Hz, synchronized with cortical slow wave activity (SWA). Nevertheless, it is poorly understood whether these electrophysiological alterations are correlated only with severe dopamine depletion or may also play a relevant pathogenetic role in the early stages of the dopamine denervation. Hence, here we recorded SNr single units and electrocorticogram (ECoG) in two models of dopamine denervation: (i) acute dopamine denervated rats, obtained by injection of tetrodotoxin (TTX), (ii) ch…

MaleTyrosine 3-Monooxygenasebasal ganglia oscillationsDopamineParkinson's diseaseWistarAction PotentialsParkinson's disease; Low frequency oscillation basal ganglia oscillations; Medial forebrain bundle; Tetrodotoxin; ElectrocorticogramTetrodotoxinSettore BIO/09 - FisiologiaParkinson's disease; low frequency oscillation; basal ganglia oscillations; medial forebrain bundle; Tetrodotoxin; electrocorticogramStatistics NonparametricAnimals; Analysis of Variance; Action Potentials; Electrophysiology; Tyrosine 3-Monooxygenase; Cerebral Cortex; Rats; Biological Clocks; Dopamine; Neurons; Rats Wistar; Substantia Nigra; Immunohistochemistry; Medial Forebrain Bundle; Statistics Nonparametric; MaleMedial forebrain bundlechemistry.chemical_compoundDevelopmental NeuroscienceBiological ClocksDopamineBasal gangliamedicineAnimalsNonparametricRats WistarMedial forebrain bundleElectrocorticographyCerebral CortexNeuronsDenervationAnalysis of Variancemedicine.diagnostic_testChemistryStatisticsLow frequency oscillation basal ganglia oscillationElectrocorticogramImmunohistochemistryRatsCortex (botany)ElectrophysiologySubstantia NigraElectrophysiologynervous systemNeurologylow frequency oscillationTetrodotoxinSettore MED/26 - NeurologiaNeurosciencemedicine.drug
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Metabolism of metyrapone by a soluble enzyme system in rat liver.

1970

The formation of a metabolite of metyrapone by an oxygen sensitive soluble enzyme system in rat liver 105,000×g supernatant has been demonstrated. Enzyme activity requires the intact keto function of metyrapone.

MaleUltraviolet RaysMetabolitechemistry.chemical_elementOxygenchemistry.chemical_compoundEnzyme systemmedicineAnimalsBiotransformationPharmacologyMetyraponebiologyChemistryGeneral MedicineMetabolismMetyraponeEnzyme assayRatsBiochemistryLiverSpectrophotometryRat liverbiology.proteinMicrosomes LiverChromatography Thin LayerOxidoreductasesFunction (biology)NADPmedicine.drugNaunyn-Schmiedebergs Archiv fur Pharmakologie
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Identification and phenotypic characterization of a subpopulation of T84 human colon cancer cells, after selection on activated endothelial cells

2004

The extravasation of metastatic cells is regulated by molecular events involving the initial adhesion of tumor cells to the endothelium and subsequently the migration of the cells in the host connective tissue. The differences in metastatic ability could be attributed to properties intrinsic of the various primary tumor types. Thus, the clonal selection of neoplastic cells during cancer progression results in cells better equipped for survival and formation of colonies in secondary sites. A cell line (T84SF) exhibiting an altered phenotypic appearance was selected from a colon cancer cell line (T84) by repetitive plating on TNFα-activated human endothelial cells and subsequent selection for…

MaleUmbilical VeinsPhysiologyCellular differentiationClinical BiochemistryMice NudeApoptosisCell CommunicationMicechemistry.chemical_compoundCancer stem cellCell Line TumorCell AdhesionAnimalsHumansNeoplasm InvasivenessEnzyme InhibitorsNeoplasm MetastasisPhosphorylationCD40biologyCell growthTyrosine phosphorylationCell BiologyCell biologyEndothelial stem cellPhenotypesrc-Family KinaseschemistryCell cultureColonic NeoplasmsMetalloproteasesbiology.proteinTyrosineEndothelium VascularCell DivisionNeoplasm TransplantationProto-oncogene tyrosine-protein kinase SrcJournal of Cellular Physiology
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Engineered microenvironments for synergistic VEGF - Integrin signalling during vascularization

2017

We have engineered polymer-based microenvironments that promote vasculogenesis both in vitro and in vivo through synergistic integrin-growth factor receptor signalling. Poly(ethyl acrylate) (PEA) triggers spontaneous organization of fibronectin (FN) into nanonetworks which provide availability of critical binding domains. Importantly, the growth factor binding (FNIII12-14) and integrin binding (FNIII9-10) regions are simultaneously available on FN fibrils assembled on PEA. This material platform promotes synergistic integrin/VEGF signalling which is highly effective for vascularization events in vitro with low concentrations of VEGF. VEGF specifically binds to FN fibrils on PEA compared to …

MaleVascular Endothelial Growth Factor AIntegrinsBiophysicsNeovascularization PhysiologicBioengineeringpoly(ethyl acrylate)ArticleBiomaterialsHuman Umbilical Vein Endothelial CellsImage Processing Computer-AssistedAnimalsHumansPhosphorylationExtracellular Signal-Regulated MAP KinasesFibronectinTissue EngineeringPhospholipase C gammaProtein assemblyVascularizationVEGFFibronectinsMice Inbred C57BLCellular MicroenvironmentMechanics of MaterialsFocal Adhesion Protein-Tyrosine KinasesFISICA APLICADAMutationCeramics and CompositesINGENIERIA ELECTRICAGrowth factorsProtein BindingSignal Transduction
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Perlecan-Induced Suppression of Smooth Muscle Cell Proliferation Is Mediated Through Increased Activity of the Tumor Suppressor PTEN

2004

We were interested in the elucidation of the interaction between the heparan sulfate proteoglycan, perlecan, and PTEN in the regulation of vascular smooth muscle cell (SMC) growth. We verified serum-stimulated DNA synthesis, and Akt and FAK phosphorylation were significantly reduced in SMCs overexpressing wild-type PTEN. Our previous studies showed perlecan is a potent inhibitor of serum-stimulated SMC growth. We report in the present study, compared with SMCs plated on fibronectin, serum-stimulated SMCs plated on perlecan exhibited increased PTEN activity, decreased FAK and Akt activities, and high levels of p27, consistent with SMC growth arrest. Adenoviral-mediated overexpression of cons…

MaleVascular smooth musclePhysiology:CIENCIAS MÉDICAS ::Farmacodinámica [UNESCO]Aorta ThoracicBasement MembraneCulture Media Serum-FreeMuscle Smooth VascularRats Sprague-DawleyMicePhosphorylationCells CulturedGlycosaminoglycansbiologyProtein-Tyrosine KinasesCell cycle:CIENCIAS MÉDICAS [UNESCO]musculoskeletal systemUNESCO::CIENCIAS MÉDICAS ::FarmacodinámicaUNESCO::CIENCIAS MÉDICAScardiovascular systemPhosphorylationSmooth muscle cell proliferationCardiology and Cardiovascular MedicineCell DivisionDNA ReplicationBasement membraneRecombinant Fusion ProteinsPerlecanProtein Serine-Threonine KinasesVascular injurySmooth muscle cell proliferation ; Restenosis ; Vascular injury ; Vascular development ; Basement membraneCatheterizationProto-Oncogene ProteinsAnimalsPTENProtein kinase BRestenosisCell growthVascular developmentOligonucleotides AntisenseFibronectinsRatsFibronectinFocal Adhesion Kinase 1Focal Adhesion Protein-Tyrosine Kinasesbiology.proteinCancer researchHeparitin SulfateCarotid Artery InjuriesProtein Processing Post-TranslationalProto-Oncogene Proteins c-aktHeparan Sulfate ProteoglycansCirculation Research
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The endocannabinoid system controls key epileptogenic circuits in the hippocampus.

2006

SummaryBalanced control of neuronal activity is central in maintaining function and viability of neuronal circuits. The endocannabinoid system tightly controls neuronal excitability. Here, we show that endocannabinoids directly target hippocampal glutamatergic neurons to provide protection against acute epileptiform seizures in mice. Functional CB1 cannabinoid receptors are present on glutamatergic terminals of the hippocampal formation, colocalizing with vesicular glutamate transporter 1 (VGluT1). Conditional deletion of the CB1 gene either in cortical glutamatergic neurons or in forebrain GABAergic neurons, as well as virally induced deletion of the CB1 gene in the hippocampus, demonstrat…

MaleVesicular glutamate transporter 1HUMDISEASEHippocampusGene ExpressionHippocampal formationHippocampusMembrane Potentialschemistry.chemical_compoundMice0302 clinical medicineReceptor Cannabinoid CB1Premovement neuronal activitygamma-Aminobutyric Acid0303 health sciencesKainic AcidbiologyBehavior AnimalReverse Transcriptase Polymerase Chain Reactionmusculoskeletal neural and ocular physiologyGeneral NeurosciencePyramidal CellsCalcium Channel BlockersEndocannabinoid systemlipids (amino acids peptides and proteins)psychological phenomena and processesmedicine.drugKainic acidNeuroscience(all)MorpholinesGlutamic AcidMice TransgenicNaphthalenesMOLNEUROgamma-Aminobutyric acid03 medical and health sciencesGlutamatergicCannabinoid Receptor ModulatorsmedicineAnimals030304 developmental biologyAnalysis of VarianceEpilepsyBenzoxazinesMice Inbred C57BLnervous systemchemistryCalcium-Calmodulin-Dependent Protein KinasesVesicular Glutamate Transport Protein 1biology.proteinNerve NetSYSNEUROCalcium-Calmodulin-Dependent Protein Kinase Type 2Neuroscience030217 neurology & neurosurgeryEndocannabinoidsNeuron
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