Search results for "Techniques"

showing 10 items of 4426 documents

In-vitro and Ex-vivo Inhibition of Blood Platelet Aggregation by Naftazone

1996

Abstract Because of the considerable interest in the role of platelets and antiplatelet therapy in cardiovascular disease, including the aggregation of platelets to each other during arterial thrombosis and atherogenesis, we have studied the effect of naftazone (Etioven), an original vasculotropic drug on platelet aggregation. Rat and human platelets were prepared and incubated in-vitro with different concentrations of naftazone. We found that naftazone inhibited both platelet secretion and aggregation in platelet-rich plasma (PRP) and washed platelets after stimulation with thrombin or ADP. Rats were also treated intraperitoneally for five days with various naftazone doses (0.125-10 mg kg−…

Blood PlateletsMaleSerotoninPlatelet AggregationPharmaceutical ScienceStimulationIn Vitro TechniquesPharmacologyRats Sprague-DawleyThrombinmedicineAnimalsHumansPlateletTiclopidinePharmacologyAspirinChemistryThrombinBiological activityRatsAdenosine DiphosphateDipyridamoleBiochemistryPlatelet Aggregation InhibitorsEx vivoNaphthoquinonesmedicine.drugJournal of Pharmacy and Pharmacology
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Clonidine increases membrane-associated phospholipase A2

2005

Background and objective: An anti-inflammatory effect of α 2 -adrenoreceptor agonists has been suggested. Phospholipase A 2 is a key enzyme in the production of precursors of inflammatory lipid mediators. The aim of the present study was to investigate the effect of clonidine on phospholipase A 2 activity in an established in vitro model. Methods: Human being platelet membranes containing active phospholipase A 2 were exposed to buffer control or to three increasing concentrations of clonidine. Phospholipase A 2 was measured by a radioisotope technique. Results: A massive increase in phospholipase A 2 activity was measured after clonidine exposure leading to final values of 92.5 ′ 3.1 pmol …

Blood PlateletsMalemedicine.medical_specialtyAnti-Inflammatory AgentsInflammationIn Vitro TechniquesClonidinePhospholipases AInternal medicinemedicineHumansPlateletchemistry.chemical_classificationPhospholipase Abusiness.industryGroup IV Phospholipases A2Cell MembraneSubstrate (chemistry)Lipid signalingClonidinePhospholipases A2Anesthesiology and Pain MedicineEndocrinologyEnzymeMembranechemistryFemalemedicine.symptombusinessAdrenergic alpha-AgonistsAdjuvants Anesthesiamedicine.drugEuropean Journal of Anaesthesiology
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Non-steroidal Anti-inflammatory Agents, Part 19:E-2-Pyrrolizin-5-yl Acrylic Acids as Potent Dual or Selective Inhibitors of Bovine Cyclooxygenase and…

1995

The pyrrolizinyl substituted acrylic acid derivatives represent another class of dual and selective inhibitors of cyclooxygenase and 5-lipoxygenase. By modifying their substitution pattern at the phenyl moiety of C-6 the balance between the activity against cyclooxygenase and against 5-lipoxygenase can be shifted. Structure-activity relationships are discussed. Compound 6k is the most potent and well-balanced dual inhibitor of both enzymes, while the highest selectivity of lipoxygenase inhibition was found for 6j. The activity and selectivity of compounds with an additional sulfur moiety depend on the oxidation status of this atom, giving an indication of the discussed coupling between pero…

Blood PlateletsNeutrophilsStereochemistryCarboxylic acidPharmaceutical ScienceIn Vitro TechniquesSulfoneStructure-Activity Relationshipchemistry.chemical_compoundLipoxygenaseDrug DiscoveryAnimalsStructure–activity relationshipMoietyCyclooxygenase InhibitorsPyrrolesLipoxygenase Inhibitorschemistry.chemical_classificationbiologyAnti-Inflammatory Agents Non-SteroidalAcrylateschemistryEnzyme inhibitorArachidonate 5-lipoxygenasebiology.proteinCattleCyclooxygenaseArchiv der Pharmazie
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Synergistic interaction of adenylate cyclase activators and nitric oxide donor SIN-1 on platelet cyclic AMP

1995

Abstract The molecular mechanism of the synergistic platelet inhibition by activators of adenylate cyclase and guanylate cyclase in human platelets was investigated. The adenylate cyclase activators iloprost and prostaglandin E 1 and the guanylate cyclase activator 3-morpholino-synonimine (SIN-1) dose-dependently inhibited thrombin-induced aggregation of washed human platelets. Furthermore, SIN-1 at a concentration inhibiting platelet aggregation by only 10% shifted the IC 50 values of iloprost and prostaglandin E 1 by one order of magnitude to the left, indicating a synergistic action of adenylate cyclase and guanylate cyclase activators. Iloprost and prostaglandin E 1 dose-dependently ele…

Blood Plateletsmedicine.medical_specialtyGUCY1B3Platelet Aggregationmedicine.medical_treatmentAdenylate kinaseIn Vitro TechniquesNitric OxideCyclasechemistry.chemical_compoundInternal medicineCyclic AMPmedicineHumansPlateletIloprostAlprostadilCyclic GMPPharmacologyForskolinGUCY1A3PhosphodiesteraseDrug SynergismEnzyme ActivationEndocrinologychemistryGuanylate CyclaseMolsidominelipids (amino acids peptides and proteins)Platelet Aggregation InhibitorsAdenylyl CyclasesProstaglandin EEuropean Journal of Pharmacology: Molecular Pharmacology
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Amelioration of spinal cord compressive injury by pharmacological preconditioning with erythropoietin and a nonerythropoietic erythropoietin derivati…

2006

Object Spinal cord injury (SCI) is a devastating clinical syndrome for which no truly efficacious therapy has yet been identified. In preclinical studies, erythropoietin (EPO) and its nonerythropoietic derivatives asialoEPO and carbamylated EPO have markedly improved functional outcome when administered after compressive SCI. However, an optimum treatment paradigm is currently unknown. Because the uninjured spinal cord expresses a high density of EPO receptor (EPOR) in the basal state, signaling through these existing receptors in advance of injury (pharmacological preconditioning) might confer neuroprotection and therefore be potentially useful in situations of anticipated damage. Methods…

Blotting WesternAsialoglycoproteinsPharmacologyNeuroprotectionCentral nervous system diseaseImmunoenzyme TechniquesRats Sprague-DawleySpinal cord compressionReceptors ErythropoietinMedicineAnimalsReceptorSpinal cord injuryErythropoietinSpinal Cord InjuriesAnalysis of Variancebusiness.industryGeneral MedicineSpinal cordmedicine.diseaseErythropoietin receptorRatsDisease Models Animalmedicine.anatomical_structureNeuroprotective AgentsErythropoietinImmunologybusinessmedicine.drugJournal of neurosurgery. Spine
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DNA methylation and histone acetylation of rat methionine adenosyltransferase 1A and 2A genes is tissue-specific.

2000

Methionine adenosyltransferase (MAT) catalyzes the biosynthesis of S-adenosylmethionine (AdoMet). In mammals MAT activity derives from two separate genes which display a tissue-specific pattern of expression. While MAT1A is expressed only in the adult liver, MAT2A is expressed in non-hepatic tissues. The mechanisms behind the selective expression of these two genes are not fully understood. In the present report we have evaluated MAT1A and MAT2A methylation in liver and in other tissues, such as kidney, by methylation-sensitive restriction enzyme digestion of genomic DNA. Our data indicate that MAT1A is hypomethylated in liver and hypermethylated in non-expressing tissues. The opposite situ…

Blotting WesternBiologyIn Vitro TechniquesKidneyBiochemistryHistonesHistone methylationAnimalsRats WistarEpigenomicsDNA methylationMyocardiumAnti-acetylated H4Kidney metabolismAcetylationCell BiologyMethylationMethionine AdenosyltransferaseDNA MethylationMolecular biologyRatsBlotting SouthernHistoneHistone acetylationLiverOrgan SpecificityMethionine AdenosyltransferaseHistone methyltransferaseDNA methylationbiology.proteinMethionine adenosyltransferaseGene expressionSpleenThe international journal of biochemistrycell biology
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In-vitro test system for the evaluation of cyclooxygenase-1 (COX-1) and cyclooxygenase-2 (COX-2) inhibitors based on a single HPLC run with UV detect…

2001

Objective and Design: The aim of this study was to develop a new, whole-cell test system which is easy to handle and requires a standard equipment for the parallel screening of COX-1 and COX-2 inhibitors.¶Materials: Bovine aortic endothelial cells (BAECs).¶Treatment and methods: Unstimulated bovine aortic coronary endothelial cells (BAECs) were used as a source of COX-1 and BAECs pretreated with ASA (100 μM) and activated with phorbol myristate acetate (PMA) were used as a source of COX-2. The time- and concentration-dependent induction of COX-2 expression in the BAECs was evaluated by a kinetic profile (HPLC analysis) and detected by Western-Blot analysis using polyclonal antibodies agains…

Blotting WesternImmunologyDrug Evaluation PreclinicalAorta ThoracicIn Vitro TechniquesHigh-performance liquid chromatographyLipoxygenaseDiclofenacmedicineAnimalsCyclooxygenase InhibitorsLipoxygenase InhibitorsIC50Chromatography High Pressure LiquidPharmacologyCyclooxygenase 2 InhibitorsbiologyChemistryMolecular biologyIsoenzymesKineticsMeloxicamBiochemistryCyclooxygenase 2Prostaglandin-Endoperoxide SynthasesPolyclonal antibodiesCyclooxygenase 1biology.proteinTetradecanoylphorbol AcetateAceclofenacCattleSpectrophotometry UltravioletEndothelium VascularCyclooxygenasemedicine.drugInflammation Research
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Macrophage-mediated angiogenic activation of outgrowth endothelial cells in co-culture with primary osteoblasts.

2014

The successful vascularisation of complex tissue engineered constructs for bone regeneration is still a major challenge in the field of tissue engineering. In this context, co-culture systems of endothelial cells and osteoblasts represent a promising approach to advance the formation of a stable vasculature as well as an excellent in vitro model to identify factors that positively influence bone healing processes, including angiogenesis. Under physiological conditions, the activation phase of angiogenesis is mainly induced by hypoxia or inflammation. Inflammatory cells such as macrophages secrete proinflammatory cytokines and proangiogenic growth factors, finally leading to the formation of…

Bone Regenerationlcsh:Diseases of the musculoskeletal systemAngiogenesislcsh:SurgeryNeovascularization PhysiologicInflammationBone healingBone and BonesProinflammatory cytokineTissue engineeringCell Line TumormedicineHumansBone regenerationOsteoblastsTissue EngineeringCell adhesion moleculeChemistryMacrophagesEndothelial CellsCell Differentiationlcsh:RD1-811Coculture TechniquesCell biologyCulture Media ConditionedMicrovesselsImmunologyCytokinesTumor necrosis factor alphalcsh:RC925-935medicine.symptom
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Inhibition of miR-21 restores RANKL/OPG ratio in multiple myeloma-derived bone marrow stromal cells and impairs the resorbing activity of mature oste…

2015

// Maria Rita Pitari 1 , Marco Rossi 1 , Nicola Amodio 1 , Cirino Botta 1 , Eugenio Morelli 1 , Cinzia Federico 1 , Annamaria Gulla 1 , Daniele Caracciolo 1 , Maria Teresa Di Martino 1 , Mariamena Arbitrio 2 , Antonio Giordano 3, 4 , Pierosandro Tagliaferri 1 , Pierfrancesco Tassone 1, 4 1 Department of Experimental and Clinical Medicine and T. Campanella Cancer Center, Magna Graecia University, S. Venuta University Campus, Catanzaro, Italy 2 ISN-CNR, Roccelletta di Borgia, Catanzaro, Italy 3 Department of Human Pathology and Oncology, University of Siena, Siena, Italy 4 Sbarro Institute for Cancer Research and Molecular Medicine, Center for Biotechnology, College of Science and Technology,…

Bone diseaseMessengerOsteoclastsTumor Microenvironment3' Untranslated RegionsMultiple myelomaTumorbiologyMesenchymal Stromal CellsRANKLProtein Inhibitors of Activated STATUp-Regulationmedicine.anatomical_structureOncologyRANKLmiRNAsmiR-21MiRNAMultiple MyelomaMiR-21; MiRNAs; Multiple myeloma bone disease; OPG; RANKL; 3' Untranslated Regions; Bone Marrow Cells; Bone Resorption; Cell Adhesion; Cell Line Tumor; Coculture Techniques; HEK293 Cells; Humans; Interleukin-6; Lentivirus; Mesenchymal Stromal Cells; MicroRNAs; Molecular Chaperones; Multiple Myeloma; Osteoclasts; Osteoprotegerin; Protein Inhibitors of Activated STAT; RANK Ligand; RNA Messenger; STAT3 Transcription Factor; Stromal Cells; Tumor Microenvironment; Up-Regulation; OncologyResearch Papermusculoskeletal diseasesSTAT3 Transcription FactorStromal cellBone Marrow CellsBone resorptionCell LineOsteoprotegerinCell Line TumormedicineCell AdhesionHumansRNA MessengerBone Resorptionbusiness.industryInterleukin-6LentivirusRANK LigandOsteoprotegerinMesenchymal Stem Cellsmedicine.diseaseMolecular medicineCoculture TechniquesMicroRNAsmultiple myeloma bone diseaseHEK293 CellsImmunologyCancer researchbiology.proteinRNAOPGBone marrowStromal CellsbusinessMolecular ChaperonesOncotarget
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Experimental evidence of pharmacological management of anchorage in Orthodontics: A systematic review

2015

Introduction: Orthodontic anchorage is one of the most challenging aspects of Orthodontics. Preventing undesired movement of teeth could result in safer and less complicated orthodontic treatment. Recently, several reviews have been published about the effects of different molecules on bone physiology and the clinical side effects in Orthodontics. However, the effects of local application of these substances on the rate of orthodontic tooth movement have not been assessed.Objectives: The aim of this research was to analyze the scientific evidence published in the literature about the effects of different molecules on orthodontic anchorage.Methods: The literature was systematically reviewed …

Bone remodeling periodBisfosfonatosTooth Movement TechniquesMovimentação dentáriaPharmacological managementAnti-Inflammatory AgentsOsteoclastsPamidronateDentistryZoledronic AcidAntioxidantsLactonesMiceStilbenesOrthodontic Anchorage ProceduresMedicineAgentes anti-inflamatóriosSulfonesAnti-inflammatory agentsOrthodonticsDiphosphonatesOsteoprotegerinaImidazolesOrthodontic anchorage proceduresArticlesBisphosphonatesProcedimentos de ancoragem ortodônticaTooth movementInclusion and exclusion criteriaBone RemodelingTooth MobilityOral SurgeryDiclofenacMEDLINEOrthodonticsKappa indexInterferon-gammaAnimalsHumansbusiness.industryOsteoprotegerinBone physiologyIsoxazolesOrthodontic Anchorage ProceduresAcetylcysteineRatslcsh:RK1-715CelecoxibResveratrolTooth movementlcsh:DentistryClodronic Acidbusiness
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