Search results for "Tetrodotoxin"

showing 10 items of 78 documents

Postnatal development of the dopaminergic signaling involved in the modulation of intestinal motility in mice

2015

Background:Since antidopaminergic drugs are pharmacological agents employed in the management of gastrointestinal motor disorders at all ages, we investigated whether the enteric dopaminergic system may undergo developmental changes after birth.Methods:Intestinal mechanical activity was examined in vitro as changes in isometric tension.Results:In 2-d-old (P2) mice, dopamine induced a contractile effect, decreasing in intensity with age, replaced, at the weaning (day 20), by a relaxant response. Both responses were tetrodotoxin (TTX)-insensitive. In P2, dopaminergic contraction was inhibited by D1-like receptor antagonist and mimicked by D1-like receptor agonist. In 90-d-old (P90) mice, the …

0301 basic medicineAgonistmedicine.medical_specialtyGastrointestinal Diseasesmedicine.drug_classDopamineTetrodotoxinBiologySettore BIO/09 - FisiologiaEnteric Nervous SystemMice03 medical and health sciences0302 clinical medicineDopamine receptor D3DopamineInternal medicineIntestine SmallCyclic AMPmedicineAnimalsEstrenesReceptorDopaminergicReceptor antagonistPyrrolidinonesMice Inbred C57BL030104 developmental biologyEndocrinologyAnimals NewbornDopamine receptorType C PhospholipasesDideoxyadenosinePediatrics Perinatology and Child Health2345-Tetrahydro-78-dihydroxy-1-phenyl-1H-3-benzazepineSignal transductionGastrointestinal Motility030217 neurology & neurosurgerySignal Transductionmedicine.drugPediatric Research
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TRPM8 Channel Activation Reduces the Spontaneous Contractions in Human Distal Colon

2020

The transient receptor potential-melastatin 8 (TRPM8) is a non-selective Ca2+-permeable channel, activated by cold, membrane depolarization, and different cooling compounds. TRPM8 expression has been found in gut mucosal, submucosal, and muscular nerve endings. Although TRPM8 plays a role in pathological conditions, being involved in visceral pain and inflammation, the physiological functions in the digestive system remain unclear as yet. The aims of the present study were: (i) to verify the TRPM8 expression in human distal colon

0301 basic medicineMaleGene ExpressionPharmacologySettore BIO/09 - Fisiologialcsh:ChemistryTissue Culture Techniqueschemistry.chemical_compound0302 clinical medicineIntestinal MucosaReceptorlcsh:QH301-705.5Spectroscopyhuman colon contractilityAged 80 and overTetraethylammoniumDepolarizationGeneral MedicineIberiotoxinMiddle AgedComputer Science Applications030220 oncology & carcinogenesisTetrodotoxinFemaleMuscle ContractionAgonistSerotoninmedicine.drug_classColonTRPM Cation ChannelsTetrodotoxinApaminCatalysisArticleInorganic Chemistry03 medical and health sciencesIBSmedicineTRPM8HumansPhysical and Theoretical ChemistryMolecular BiologyAgedOrganic ChemistryMuscle SmoothTetraethylammonium chloridePhosphinic Acids1-[Diisopropyl-phosphinoyl]-alkane (DIPA)030104 developmental biologychemistrylcsh:Biology (General)lcsh:QD1-999ApaminTRPM-8PeptidesInternational Journal of Molecular Sciences
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δ 1‐OPIOID receptor‐mediated controlofacetylcholine (ACh) release in human neocortex slices

1998

In slices of human neocortex, prelabelled with [3H]-choline, the release of [3H]-acetylcholine reflects the evoked release of endogenous acetylcholine which was elicited by the same electrical stimulation paradigm. [3H]-Acetylcholine release was depressed by the delta-opioid receptor agonist D-Pen2-D-Pen5-enkephalin. When the nerve endings were depolarized by elevating extracellular potassium the evoked [3H]-acetylcholine release was similarly depressed by D-Pen2-D-Pen5-enkephalin in the absence, but not in the presence, of tetrodotoxin which blocks action potential propagation. Therefore, the delta-opioid receptor inhibiting [3H]-acetylcholine release should not be located to cholinergic n…

AdultAgonistmedicine.medical_specialtymedicine.drug_classNarcotic AntagonistsNeocortexTetrodotoxinIn Vitro TechniquesOctreotideBenzylidene Compoundschemistry.chemical_compoundDevelopmental NeuroscienceInterneuronsOpioid receptorReceptors Opioid deltaInternal medicinemedicineHumansReceptorAgedAged 80 and overNeocortexEnkephalinsMiddle AgedReceptor antagonistAcetylcholineElectric StimulationNaltrexoneEndocrinologymedicine.anatomical_structurenervous systemchemistryTetrodotoxinCholinergicEnkephalin D-Penicillamine (25)-AcetylcholineDevelopmental Biologymedicine.drugInternational Journal of Developmental Neuroscience
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Endothelium-dependent component in the contractile responses of human omental arteries to adrenergic stimulation

1993

Abstract The present study was designed to investigate the influence of endothelium-derived nitric oxide on the contractile responses of isolated human omental arteries to electrical field stimulation and noradrenaline. We measured isometric tension in artery rings obtained from portions of human omentum during the course of abdominal operations (32 patients). Electrical field stimulation induced frequency-dependent contractions which were abolished by tetrodotoxin (10−6 M) and prazosin (10−6 M), thus indicating that this effect was due to noradrenaline released from adrenergic nerves acting on α1-adrenoceptors. The increases in tension induced by electrical field stimulation were of greate…

AdultMalemedicine.medical_specialtyArginineEndotheliumIndomethacinTetrodotoxinIn Vitro TechniquesArginineNitric OxideMuscle Smooth VascularNitric oxideNorepinephrinechemistry.chemical_compoundInternal medicinemedicinePrazosinHumansAgedPharmacologybusiness.industryStereoisomerismArteriesMiddle AgedElectric StimulationNG-Nitroarginine Methyl EsterEndocrinologymedicine.anatomical_structurechemistryVasoconstrictionCirculatory systemTetrodotoxinFemaleEndothelium VascularbusinessOmentumMuscle Contractionmedicine.drugBlood vesselArteryEuropean Journal of Pharmacology
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Exogenous glucagon-like peptide-1 reduces contractions in human colon circular muscle.

2014

Glucagon-like peptide 1 (GLP1) is a naturally occurring peptide secreted by intestinal L-cells. Though its primary function is to serve as an incretin, GLP1 reduces gastrointestinal motility. However, only a handful of animal studies have specifically evaluated the influence of GLP1 on colonic motility. Consequently, the aims of this study were to investigate the effects induced by exogenous GLP1, to analyze the mechanism of action, and to verify the presence of GLP1 receptors (GLP1Rs) in human colon circular muscular strips. Organ bath technique, RT-PCR, western blotting, and immunofluorescence were used. In human colon, exogenous GLP1 reduced, in a concentration-dependent manner, the ampl…

AdultMalemedicine.medical_specialtyColonEndocrinology Diabetes and MetabolismIncretinMotilityDown-RegulationIn Vitro TechniquesSettore BIO/09 - FisiologiaGlucagon-Like Peptide-1 ReceptorNitric oxidechemistry.chemical_compoundEndocrinologyGlucagon-Like Peptide 1Internal medicinemedicineReceptors GlucagonHumansLarge intestineReceptorMyenteric plexusAgedAged 80 and overMusclesGLP-1 human colon contractily intestinal motilityMiddle Agedmedicine.anatomical_structureEndocrinologychemistryMechanism of actionTetrodotoxinFemalemedicine.symptomGastrointestinal MotilityMuscle Contraction
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Involvement of tachykinin NK2 receptors in the modulation of spontaneous motility in rat proximal colon.

2000

The role of endogenous tachykinins and the mechanisms whereby they act on NK2 receptors, modulating spontaneous motility, were investigated in rat isolated proximal colon. The mechanical activity was detected as changes in intraluminal pressure. The NK2 receptor antagonist, MEN 10627, produced a concentration-dependent reduction of the contraction amplitude. [beta-Ala8]-neurokinin A(4-10), an NK2 receptor agonist, and [Sar9, Met(O2)11]-Substance P ([Sar9, Met(O2)11]-SP), an NK1 receptor agonist, induced a concentration-dependent contractile response, characterized by an increase in basal tone with superimposed phasic contractions. MEN 10627 antagonized the response to [beta-Ala8]-neurokinin…

AgonistMalemedicine.medical_specialtyPhysiologymedicine.drug_classColonNeurokinin AInhibitory postsynaptic potentialNitric OxidePeptides CyclicTonic (physiology)chemistry.chemical_compoundPiperidinesInternal medicinemedicineAnimalsEnzyme InhibitorsRats WistarReceptorEndocrine and Autonomic SystemsChemistrymusculoskeletal neural and ocular physiologyGastroenterologyAntagonistReceptors Neurokinin-2RatsEndocrinologyNicotinic agonistNG-Nitroarginine Methyl EsterBenzamidesTetrodotoxinHexamethoniumGastrointestinal MotilityNeurogastroenterology and motility : the official journal of the European Gastrointestinal Motility Society
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Modulation by 5-HT3 and 5-HT4 receptors of the release of 5-hydroxytryptamine from the guinea-pig small intestine.

1993

The effects of agonists and antagonists of 5-hydroxytryptamine (5-HT) receptors on the release of endogenous 5-HT from enterochromaffin cells were studied in the vascularly perfused isolated guinea-pig small intestine. The experiments were done in the presence of tetrodotoxin in order to exclude a neuronally mediated influence on 5-HT release. The 5-HT3 receptor agonist 2-methyl-5-HT increased 5-HT release, and this effect was antagonized by 1 nmol/l tropisetron. Nanomolar concentrations of tropisetron, MDL 72,222 and granisetron decreased 5-HT release. Ondansetron (0.1 and 1 mumol/l) did not modify 5-HT release. 5-Methoxytryptamine, BIMU8 and cisapride concentration-dependently inhibited 5…

AgonistMalemedicine.medical_specialtySerotoninmedicine.drug_classGuinea PigsStimulationTetrodotoxinBiologychemistry.chemical_compoundInternal medicineIntestine SmallmedicineEnterochromaffin CellsAnimalsIntestinal MucosaReceptorPharmacologyGeneral Medicinemusculoskeletal systemSerotonin Receptor AgonistsPerfusionEndocrinologychemistryMetitepineReceptors SerotoninAutoreceptorEnterochromaffin cellTropisetronFemaleSerotoninSerotonin Antagonistsmedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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Evidence for the presence of P2y and P2x receptors with different functions in mouse stomach.

2005

To clarify the function of P2 receptor subtypes in mouse stomach, the motor responses to ATP, alpha,beta-methyleneATP (alpha,beta-MeATP), P2X receptor agonist, 2-methylthioATP (2-MeSATP), P2Y receptor agonist, and the effects of the desensitisation of P2X receptors with alpha,beta-MeATP and of P2Y receptors with ADPbetaS were analysed recording the endoluminal pressure from whole-organ. ATP-induced relaxation was antagonised by suramin, non-selective P2 receptor antagonist, by desensitisation of P2Y receptors with ADPbetaS, and increased by desensitisation of P2X receptors with alpha,beta-MeATP. alpha,beta-MeATP produced biphasic responses: relaxation, reduced by P2X- or P2Y desensitisation…

Agonistmedicine.medical_specialtyP2Y receptorRelaxationContraction (grammar)medicine.drug_classSuraminMuscle RelaxationTetrodotoxinP2 receptorBiologyIn Vitro TechniquesSettore BIO/09 - Fisiologiachemistry.chemical_compoundMiceAdenosine TriphosphateInternal medicinemedicineAnimalsReceptorPharmacologyContractionDose-Response Relationship DrugReceptors Purinergic P2Mouse stomachStomachAntagonistP2Y receptorThionucleotidesATPAdenosine DiphosphateMice Inbred C57BLEndocrinologychemistryP2X receptorReceptors Purinergic P2XTetrodotoxinmedicine.drugMuscle ContractionEuropean journal of pharmacology
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Role for NK1 and NK2 receptors in the motor activity in mouse colon

2007

The present study examined the effects induced by endogenous and exogenous activation of NK(1) and NK(2) receptors on the mechanical activity of mouse proximal colon. Experiments were performed in vitro recording the changes in intraluminal pressure from isolated colonic segments. Electrical field stimulation in the presence of atropine and guanethidine produced a small relaxation, followed by nonadrenergic noncholinergic (NANC) contraction. SR140333, NK(1) receptor antagonist, or SR48968, NK(2) receptor antagonist, significantly reduced the contraction, although SR48968 appeared more efficacious. The co-administration of SR140333 and SR48968 virtually abolished the NANC contraction. [Sar(9…

AtropineAgonistmedicine.medical_specialtyContraction (grammar)Colonmedicine.drug_classNeurokinin AMuscarinic AntagonistsTetrodotoxinSubstance PSettore BIO/09 - FisiologiaNK1 receptorNitric oxideMicechemistry.chemical_compoundNeurokinin-1 Receptor Antagonistsnitric oxideInternal medicinemedicineAnimalsNK2 receptorReceptorGuanethidinePharmacologyAntagonistReceptors Neurokinin-2Receptors Neurokinin-1Electric StimulationPeptide FragmentsMice Inbred C57BLEndocrinologychemistryTetrodotoxinNANC contractionCholinergicTachykininMuscle ContractionSodium Channel Blockersmedicine.drugEuropean Journal of Pharmacology
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Evidence that adenosine is not involved in the non-adrenergic non-cholinergic relaxation in the rat duodenum.

1990

In rat isolated duodenal segments, adenosine induced, in the presence of atropine and guanethidine, a dose-dependent, long-lasting (about 20 s), tetrodotoxin (TTX)-resistant relaxation both in endoluminal pressure and in isometric tension. Electrical field stimulation (EFS) induced, in the presence of atropine and guanethidine, a TTX-sensitive short-lasting (about 6 s) relaxation followed by a sustained rebound contraction. Theophylline, a P1 receptor antagonist, at the concentration of 100 microM caused a marked inhibition of the adenosine-induced relaxation, while the EFS-induced relaxation was not modified. Our results suggest that adenosine induces relaxation of the rat duodenal smooth …

AtropineGuanethidinemedicine.medical_specialtyAdenosinePhysiologyDuodenumMuscle RelaxationTetrodotoxinIn Vitro TechniquesBiochemistrychemistry.chemical_compoundTheophyllineInternal medicineIsometric ContractionmedicinePressureAnimalsTheophyllineNeurotransmitterReceptorGuanethidineReceptors PurinergicMuscle SmoothAdenosineAdenosine receptorElectric StimulationRatsAtropineEndocrinologychemistryTetrodotoxinmedicine.drugArchives internationales de physiologie et de biochimie
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