Search results for "Thyronine"

showing 10 items of 61 documents

Influence of vitamin A status on the regulation of uridine (5′-)diphosphate-glucuronosyltransferase (UGT) 1A1 and UGT1A6 expression by L-triiodothyro…

2001

The uridine (5′-)diphosphate-glucuronosyltransferases (UGT) are involved in the phase II of various xenobiotics and endogenous compounds. They are responsible for glucuronidation of many substrates, especially including bilirubin (UGT1A1) and phenolic compounds (UGT1A6). We previously showed that the expression of both isoforms is regulated at the transcriptional level by thyroid hormone in rat liver. In this present study, effects of vitamin A dietary intake (0, 1.72, 69 ug retinol acetate/g food) on the regulation of UGT1A1 and UGT1A6 activity and expression by 3,5,3′ triiodo-L-THYRONINE (l-T3) were examined in the same organ. Activities were determined toward bilirubin and 4-nitrophenol.…

MaleUGT1A6Vitaminmedicine.medical_specialtyGlucuronosyltransferaseTriiodothyronine ReverseCellular detoxificationGlucuronidationMedicine (miscellaneous)digestive systemchemistry.chemical_compoundInternal medicinemedicineAnimalsRNA MessengerGlucuronosyltransferaseRats WistarVitamin ANutrition and DieteticsTriiodothyroninebiologyReverse Transcriptase Polymerase Chain ReactionRetinolRatsEndocrinologyGene Expression RegulationLiverchemistryMicrosomes Liverbiology.proteinHormoneBritish Journal of Nutrition
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Abnormal thyroid hormones and non-thyroidal illness syndrome in obstructive sleep apnea, and effects of CPAP treatment

2016

Objective In obstructive sleep apnea (OSA), while both hypothyroidism and hyperthyroidism have been studied, the occurrence of non-thyroidal illness syndrome (NTIS) (normal thyroid stimulating hormone [TSH] with low triiodotironine) has not been investigated. We explored the occurrence of NTIS in patients with moderate to severe OSA and its relationship to the severity of nocturnal respiratory disorders. We also studied the occurrence of subclinical hypothyroidism (SH, ie, high TSH with normal thyroxine) in OSA and changes in circulating TSH, free triiodotironine (fT3) and free thyroxine (fT4) after CPAP treatment. Methods After a nocturnal respiratory polysomnography, 125 consecutive patie…

Maleendocrine system diseasesSettore MED/10 - MALATTIE DELL'APPARATO RESPIRATORIOPolysomnographyGastroenterologyHypoxemiaHypoxemia0302 clinical medicineSubclinical hypothyroidismThyroid stimulating hormoneobstructive sleep apneaOxygen saturation (medicine)Subclinical infectionSleep Apnea ObstructiveContinuous Positive Airway Pressuremedicine.diagnostic_testMedicine (all)apnee ostruttive nel sonnoGeneral MedicineMiddle Agedfunzione tiroideaTriiodothyronineFemalemedicine.symptomhormones hormone substitutes and hormone antagonistsThyroid Hormonesendocrine systemmedicine.medical_specialtyPolysomnography03 medical and health sciencesThyroid-stimulating hormoneInternal medicinemedicineHumansthyroid functionbusiness.industrymedicine.diseaseEuthyroid Sick Syndromesrespiratory tract diseasesThyroid hormoneObstructive sleep apneaThyroxineEndocrinology030228 respiratory systemCase-Control Studiesbusiness030217 neurology & neurosurgeryEuthyroid sick syndromeHormoneEuthyroid sick syndromeSleep Medicine
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Plasma levels of lipoproteins and apolipoproteins in congenital hypothyroidism: Effects of l-thyroxine substitution therapy

1995

Thyroid status in humans is an important factor in the regulation of lipoprotein metabolism. There are several data on hypothyroidism in the adult population, but less information is available about congenital hypothyroidism. Since lipid metabolism at birth is substantially different from that of adults, it is not likely that the same abnormalities that occur in adult hypothyroidism are also present when this is diagnosed at early life. We studied 16 subjects with congenital hypothyroidism, seven at the time of diagnosis and also after normalization of thyroid hormone levels over a period of 2.0 +/- 1.0 months of substitution therapy with L-thyroxine (5.9 +/- 1.2 micrograms/kg/d) and nine a…

Maleendocrine systemmedicine.medical_specialtyApolipoprotein BLipoproteinsEndocrinology Diabetes and MetabolismThyrotropinTriglyceridechemistry.chemical_compoundEndocrinologyHypothyroidismInternal medicineBlood plasmaCongenital HypothyroidismHumansMedicineLipoproteinTriglyceridesTriiodothyronineApolipoprotein A-Ibiologybusiness.industryCholesterolThyroidInfant NewbornInfantmedicine.diseaseApolipoproteinApolipoproteins; Humans; Infant Newborn; Lipoproteins HDL; Apolipoprotein A-I; Cholesterol; Thyrotropin; Infant; Congenital Hypothyroidism; Triglycerides; Lipoproteins; Thyroxine; Hypothyroidism; Lipoproteins LDL; Female; MaleCongenital hypothyroidismLipoproteins LDLThyroxineApolipoproteinsCholesterolEndocrinologymedicine.anatomical_structurechemistrybiology.proteinFemalelipids (amino acids peptides and proteins)Lipoproteins HDLbusinessHumanLipoproteinHormoneMetabolism
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Relapses of hyperthyroidism in patients treated with radioiodine for nodular toxic goiter: relation to thyroid autoimmunity.

2003

Relapses of hyperthyroidism after treatment with radioiodine for uni- or multi-nodular goiter may be accompanied by the appearance of TSAb. However, this phenomenon has only emerged from one retrospective study on Northern European patients, in which it was not possible to determine whether TSAb also appeared in treated patients who did not relapse. The present study aimed to assess the appearance, immunogenic nature and clinical characteristics of hyperthyroidism relapse after treatment with 131I for nodular toxic goiter in patients from the Mediterranean area. A retrospective study was performed on 76 consecutive patients, born and resident in Sicily and aged 56-80 yr at diagnosis, who we…

Maleendocrine systemmedicine.medical_specialtyGoiterendocrine system diseasesEndocrinology Diabetes and MetabolismGraves' diseaseThyrotropinDiseaseGastroenterologyHyperthyroidismIodine RadioisotopesEndocrinologyRecurrenceInternal medicineImmunopathologymedicineToxic goiterHumansAgedAutoantibodiesRetrospective StudiesAutoimmune diseaseAged 80 and overbusiness.industryIncidence (epidemiology)Retrospective cohort studyMiddle Agedmedicine.diseaseeye diseasesSurgeryThyroxineTriiodothyronineFemalebusinessGoiter NodularJournal of endocrinological investigation
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Fos-related antigen 2 (Fra-2) memorizes photoperiod in the rat pineal gland

2004

As the physiological role of fos-related antigen-2 (Fra-2) is largely unknown and since the pineal plays an important role in the photoperiodic control of the body, we have tested the hypothesis that Fra-2 expression is photoperiod-dependent and may be involved in imprinting photoperiod on the pineal gland and the body as a whole. To this end, we have investigated Fra-2 mRNA expression and Fra-2 protein expression under various light/dark (LD) cycles. A clear nocturnal increase occurs for both monitored parameters under all photoperiodic conditions studied. The level of Fra-2 protein expression clearly depends on photoperiod, because the amount of protein at dark onset and during the night …

Maleendocrine systemmedicine.medical_specialtyTime FactorsPhotoperiodBlotting WesternDeiodinaseFos-Related Antigen-2CREBPineal GlandRats Sprague-DawleyPhenylephrinePineal glandOrgan Culture TechniquesAcetyltransferasesInternal medicineCyclic AMPmedicineAnimalsDrug InteractionsProtein phosphorylationRNA MessengerCircadian rhythmCyclic GMPHeat-Shock ProteinsbiologyReverse Transcriptase Polymerase Chain ReactionGeneral NeuroscienceIsoproterenolAdrenergic beta-AgonistsAdaptation PhysiologicalPeptide FragmentsRatsDNA-Binding ProteinsEndocrinologymedicine.anatomical_structureGene Expression RegulationIodothyronine deiodinasebiology.proteinArylalkylamineFemaleAdrenergic alpha-Agonistshormones hormone substitutes and hormone antagonistsTranscription FactorsEndocrine glandNeuroscience
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Congenital hypothyroidism in a child with unsuspected familial dysalbuminemic hyperthyroxinemia caused by a mutation (R218H) in the human albumin gene

2001

We found familial dysalbuminemic hyperthyroxinemia (FDH) in a 5-month-old boy with congenital hypothyroidism (CH) who had a blood thyrotropin (TSH) level of 479 mU/L but normal total serum thyroxine (T4) and higher than normal total triiodothyronine (T3) levels. Thyroid hormone substitution began at 5 weeks of age when T4 and T3 concentrations were below normal. Until the age of 5 months, treatment with levothyroxine was suboptimal on the basis of high serum TSH levels despite above-normal T4 levels. FDH was confirmed by isoelectric focusing and testing of other family members. DNA analysis of the patient revealed R218H, a mutation in the serum albumin gene associated with FDH, which was al…

Maleendocrine systemmedicine.medical_specialtyendocrine system diseasesSerum albuminLevothyroxineThyrotropinHypothyroidismAlbuminsInternal medicineCongenital HypothyroidismAlbuminuriaHumansMedicineEuthyroidTriiodothyroninebiologybusiness.industryThyroidAlbuminInfantmedicine.diseaseCongenital hypothyroidismHyperthyroxinemiaThyroxinemedicine.anatomical_structureEndocrinologyFamilial dysalbuminemic hyperthyroxinemiaMutationPediatrics Perinatology and Child Healthbiology.proteinbusinessmedicine.drugThe Journal of Pediatrics
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Correlation between severity of growth hormone deficiency and thyroid metabolism and effects of long-term growth hormone treatment on thyroid functio…

2013

<b><i>Background/Aim:</i></b> The significance of changes in thyroid function in children during growth hormone (GH) treatment remains uncertain. We aimed to evaluate the impact of GH replacement on thyroid status in children with idiopathic GH deficiency (GHD). <b><i>Methods:</i></b> Data of 105 GHD children (82 M, 23 F; aged 11.13 years) during a 36-month follow-up were analyzed. At diagnosis the areas under the curve of GH (AUC<sub>GH</sub>) were calculated during a GH-releasing hormone + arginine (GHRH-Arg) and insulin tolerance test. <b><i>Results:</i></b> A significant ΔfT<sub>3</su…

Malemedicine.medical_specialtyAdolescentEndocrinology Diabetes and MetabolismThyroid GlandArginineGrowth Hormone-Releasing HormoneGrowth hormone deficiencythyroidSettore MED/13 - Endocrinologiagrowth hormone; thyroidEndocrinologyInternal medicineMedicineHumansskin and connective tissue diseasesChildRetrospective StudiesTriiodothyroninebusiness.industryHuman Growth HormoneThyroidMetabolismGrowth hormone–releasing hormonemedicine.diseaseGrowth hormone treatmentEndocrinologymedicine.anatomical_structurePediatrics Perinatology and Child Healthgrowth hormoneTriiodothyronineFemalesense organsThyroid functionbusinessHormoneHormone research in paediatrics
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A new heterozygous mutation (L338N) in the human Gsalpha (GNAS1) gene as a cause for congenital hypothyroidism in Albright's hereditary osteodystroph…

2003

OBJECTIVE: To identify the molecular defect by which psychomotor retardation is caused in two brothers with congenital hypothyroidism who received adequate treatment with l-thyroxine. CASE REPORT: A six-year-old boy presented with psychomotor retardation and congenital primary hypothyroidism (CH). The patient had a normal blood thyrotrophin (TSH) level on neonatal screening, but low total serum thyroxine and triiodothyronine concentrations prompting thyroid hormone substitution shortly after birth. Nevertheless, psychomotor development was retarded and the patient underwent further investigation. Typical features of Albright's hereditary osteodystrophy (AHO) such as round face, obesity, and…

Malemedicine.medical_specialtyHeterozygoteGenotypeEndocrinology Diabetes and MetabolismThyrotropinFibrous Dysplasia PolyostoticEndocrinologyHypothyroidismInternal medicinemedicineGTP-Binding Protein alpha Subunits GsHumansOsteodystrophyChildAlbright's hereditary osteodystrophyPseudohypoparathyroidismPsychomotor retardationbusiness.industryThyroidErythrocyte MembranePrimary hypothyroidismGeneral MedicineSequence Analysis DNAmedicine.diseaseCongenital hypothyroidismPedigreeThyroxineEndocrinologymedicine.anatomical_structureMutationTriiodothyroninePseudopseudohypoparathyroidismCalciummedicine.symptomMetacarpusPsychomotor DisordersbusinessEuropean journal of endocrinology
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Role of thyroid state on induction by ciprofibrate of laurate hydroxylase and peroxisomal enzymes in rat liver microsomes.

1993

The effects of hypothyroidism and hyperthyroidism upon liver microsomal omega-laurate hydroxylase activity (cytochrome P450 IV A1-dependent), peroxisome proliferation marker enzyme activities and acyl CoA oxidase (AOX) expression induced by ciprofibrate (2 mg/kg/day during 8 days) were studied in the male Wistar rat so as to clarify firstly the possible involvement of thyroid hormones in the modification of peroxisomal ciprofibrate-induced enzyme activities in relation to hepatic microsomal cytochrome P450 IV A1 induction, and secondly the possible direct effect of thyroid hormones on the gene expression of specific peroxisomal enzymes. No significant change was found in the ciprofibrate-in…

Malemedicine.medical_specialtyThyroid HormonesPeroxisome ProliferationGlucuronatesGlycerolphosphate DehydrogenaseBiochemistryMicrobodiesMixed Function OxygenasesClofibric AcidCytochrome P-450 Enzyme SystemInternal medicinemedicineAnimalsEnzyme inducerRats WistarPharmacologyTriiodothyroninebiologyBody WeightFibric AcidsCytochrome P450Organ SizePeroxisomeEnoyl-CoA hydrataseRatsEndocrinologyGene Expression RegulationEnzyme InductionProtein Biosynthesisbiology.proteinMicrosomes LiverCiprofibrateCytochrome P-450 CYP4Amedicine.drugHormoneBiochemical pharmacology
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The effects of insulin on transport and metabolism of glucose in skeletal muscle from hyperthyroid and hypothyroid rats.

1997

The effects of insulin on the rates of glucose disposal were studied in soleus muscles isolated from hyper- or hypothyroid rats. Treatment with triiodothyronine for 5 or 10 days decreased the sensitivity of glycogen synthesis but increased the sensitivity of lactate formation to insulin. The sensitivity of 3-O methylglucose to insulin was increased only after 10 days of treatment and was accompanied by an increase in the sensitivity of 2-deoxyglucose phosphorylation; however, 2-deoxyglucose and glucose 6-phosphate in response to insulin remained unaltered. In hypothyroidism, insulin-stimulated rates of 3-O-methylglucose transport and 2-deoxyglucose phosphorylation were decreased; however, a…

Malemedicine.medical_specialtymedicine.medical_treatmentClinical BiochemistryBiological Transport ActiveBiologyCarbohydrate metabolismIn Vitro TechniquesBiochemistryHyperthyroidismDinoprostonechemistry.chemical_compoundHypothyroidismInternal medicinemedicineAnimalsInsulinGlycolysisDrug InteractionsLactic AcidPhosphorylationRats WistarGlycogen synthaseMuscle SkeletalHexokinaseInsulinGlucose transporterGeneral MedicineMetabolismRatsCortisoneEndocrinologyGlucosechemistryBasal (medicine)Growth Hormonebiology.proteinTriiodothyronineGlycolysisGlycogenEuropean journal of clinical investigation
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