Search results for "Tissue"

showing 10 items of 4413 documents

Changes in the Peripheral Endocannabinoid System as a Risk Factor for the Development of Eating Disorders

2017

BACKGROUND AND OBJECTIVE Eating Disorder (ED) is characterized by persistently and severely disturbed eating behaviours. They arise from a combination of long-standing behavioural, emotional, psychological, interpersonal, and social factors and result in insufficient nutrient ingestion and/or adsorption. The three main EDs are: anorexia nervosa, bulimia nervosa, and binge eating disorder. We review the role of peripheral endocannabinoids in eating behaviour. DISCUSSION The neuronal pathways involved in feeding behaviours are closely related to catecholaminergic, serotoninergic and peptidergic systems. Accordingly, feeding is promoted by serotonin, dopamine, and prostaglandin and inhibited b…

0301 basic medicinemedicine.medical_specialtyCannabinoid receptorEndocrinology Diabetes and Metabolismmedicine.medical_treatmentNutritional StatusFeeding and Eating Disorders03 medical and health sciencesIslets of LangerhansReceptor Cannabinoid CB1Binge-eating disorderInternal medicinemedicineImmunology and AllergyAnimalsHumansOpioid peptideMuscle Skeletal030109 nutrition & dieteticsBulimia nervosabusiness.industrydigestive oral and skin physiologyBody WeightBrainFeeding Behaviormedicine.diseaseEndocannabinoid systemEating disordersEndocrinologyAdipose TissueLiverAnorexia nervosa (differential diagnoses)CannabinoidbusinessEnergy MetabolismEndocannabinoidsSignal Transduction
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Assessing the Contribution of Relative Macrophage Frequencies to Subcutaneous Adipose Tissue

2021

Background: Macrophages play an important role in regulating adipose tissue function, while their frequencies in adipose tissue vary between individuals. Adipose tissue infiltration by high frequencies of macrophages has been linked to changes in adipokine levels and low-grade inflammation, frequently associated with the progression of obesity. The objective of this project was to assess the contribution of relative macrophage frequencies to the overall subcutaneous adipose tissue gene expression using publicly available datasets.Methods: Seven publicly available microarray gene expression datasets from human subcutaneous adipose tissue biopsies (n = 519) were used together with TissueDecod…

0301 basic medicinemedicine.medical_specialtyDOWN-REGULATIONsubcutaneous adipose tissueEndocrinology Diabetes and MetabolismAdipose tissueAdipokine030209 endocrinology & metabolismInflammationBiologycell-type composition03 medical and health scienceschemistry.chemical_compound0302 clinical medicineDownregulation and upregulationINFLAMMATIONInternal medicineGene expressionlipid metabolismmedicinelow-grade inflammationpublicly available dataMacrophagecomputational deconvolutionTX341-641OXIDATIVE STRESSPHOSPHORYLATIONFatty acid synthesisGENE-EXPRESSIONNutritionOriginal ResearchINSULIN-RESISTANCENutrition and DieteticsNutrition. Foods and food supplyWOMENLipid metabolismmacrophages030104 developmental biologyEndocrinologychemistryOBESITYmedicine.symptomSTEM-CELLSFood ScienceACID-METABOLISMFrontiers in Nutrition
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Role of Adipokines and Perivascular Adipose Tissue in Abdominal Aortic Aneurysm: A Systematic Review and Meta-Analysis of Animal and Human Observatio…

2021

Improved understanding of abdominal aortic aneurysms (AAA) pathogenesis is required to identify treatment targets. This systematic review summarized evidence from animal studies and clinical research examining the role of adipokines and perivascular adipose tissue (PVAT) in AAA pathogenesis. Meta-analyses suggested that leptin (Standardized mean difference [SMD]: 0.50 [95% confidence interval (CI): −1.62, 2.61]) and adiponectin (SMD: −3.16 [95% CI: −7.59, 1.28]) upregulation did not significantly affect AAA severity within animal models. There were inconsistent findings and limited studies investigating the effect of resistin-like molecule-beta (RELMβ) and PVAT in animal models of AAA. Clin…

0301 basic medicinemedicine.medical_specialtyEndocrinology Diabetes and Metabolismaortic ruptureAdipose tissueAdipokine030204 cardiovascular system & hematologylcsh:Diseases of the endocrine glands. Clinical endocrinologyGastroenterologyPathogenesis03 medical and health sciencesEndocrinologyabdominal aortic aneurysm0302 clinical medicineAdipokinesInternal medicineAnimalsHumansMedicinelcsh:RC648-665adipokineAdiponectinbusiness.industryLeptinmedicine.diseaseAbdominal aortic aneurysmadipose tissueObservational Studies as Topic030104 developmental biologyResistinSystematic ReviewAnimal studiesperiaortic adipose tissuebusinessAortic Aneurysm AbdominalFrontiers in Endocrinology
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Effects of resveratrol on eNOS in the endothelium and the perivascular adipose tissue

2017

Under physiological conditions, nitric oxide (NO) is produced in the vasculature mainly by the endothelial NO synthase (eNOS). Experiments using gene-disrupted mice have demonstrated that eNOS has antihypertensive, antithrombotic, and antiatherosclerotic effects. Recent studies show that eNOS is expressed not only in the endothelium but also in the perivascular adipose tissue (PVAT). Resveratrol prevents eNOS uncoupling and upregulates eNOS expression and activity. These effects of resveratrol are well established for the eNOS enzyme in the endothelium. Interestingly, resveratrol also improves PVAT function. However, a causal role for eNOS in the effects of resveratrol on PVAT function has …

0301 basic medicinemedicine.medical_specialtyEndotheliumAdipose tissue030204 cardiovascular system & hematologyResveratrolGeneral Biochemistry Genetics and Molecular BiologyNitric oxide03 medical and health scienceschemistry.chemical_compound0302 clinical medicineHistory and Philosophy of ScienceEnosInternal medicineAntithromboticmedicinebiologyChemistryGeneral NeuroscienceNitric Oxide Synthase Type IIIbiology.organism_classificationNitric oxide synthase030104 developmental biologyEndocrinologymedicine.anatomical_structurebiology.proteinAnnals of the New York Academy of Sciences
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Mechanisms underlying the influence of oestrogen on cardiovascular physiology in women.

2019

Women show a lower incidence of cardiovascular diseases than age-matched men, but this benefit disappears after menopause. Oestrogen-mediated vascular actions are mainly attributed to oestradiol and exerted by oestrogen receptors (ERα, ERβ and G protein-coupled oestrogen receptor), through rapid and/or genomic mechanisms, but these effects depend on ageing and inflammation. A cardiovascular approach in women's health has arisen due to controversy regarding oestrogen's beneficial impact as reported in experimental and observational studies and large randomized trials. These can be explained, in part, by two mutually non-exclusive hypotheses. On the one hand, the timing hypothesis, which stat…

0301 basic medicinemedicine.medical_specialtyEndotheliumPhysiologyMedicinaEstrogen receptorInflammationProstacyclin03 medical and health sciences0302 clinical medicineInternal medicinemedicineHumansEndothelial dysfunctionReceptorskin and connective tissue diseasesMolecular Structurebusiness.industryEstrogen Receptor alphaEstrogensmedicine.diseaseCardiovascular physiologyPostmenopause030104 developmental biologymedicine.anatomical_structureEndocrinologyAgeingFemaleEndothelium Vascularmedicine.symptombusiness030217 neurology & neurosurgerymedicine.drugThe Journal of physiologyReferences
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Decreases in Circulating Concentrations of Long-Chain Acylcarnitines and Free Fatty Acids During the Glucose Tolerance Test Represent Tissue-Specific…

2019

Background: Insulin plays a pivotal role in the regulation of both carbohydrate and lipid intermediate turnover and metabolism. In the transition from a fasted to fed state, insulin action inhibits lipolysis in adipocytes, and acylcarnitine synthesis in the muscles and heart. The aim of this study was to measure free fatty acid (FFA) and acylcarnitine levels during the glucose tolerance test as indicators of tissue-specific insulin resistance. Results: Insulin release in response to glucose administration decreased both FFA and long-chain acylcarnitine levels in plasma in healthy control animals by 30% (120 min). The glucose tolerance test and [3H]-deoxy-D-glucose uptake in tissues revealed…

0301 basic medicinemedicine.medical_specialtyGlucose uptakemedicine.medical_treatmentEndocrinology Diabetes and MetabolismAdipose tissuelong-chain acylcarnitines030209 endocrinology & metabolismType 2 diabetesglucose tolerance testlcsh:Diseases of the endocrine glands. Clinical endocrinology03 medical and health sciences0302 clinical medicineInsulin resistanceEndocrinologyInternal medicineinsulin resistancemedicineLipolysisOriginal ResearchGlucose tolerance testlcsh:RC648-665medicine.diagnostic_testChemistryInsulinfree fatty acidsmedicine.disease030104 developmental biologyEndocrinologyPostprandialtype 2 diabetesFrontiers in Endocrinology
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Altered synaptic phospholipid signaling in PRG-1 deficient mice induces exploratory behavior and motor hyperactivity resembling psychiatric disorders.

2017

Abstract Plasticity related gene 1 (PRG-1) is a neuron specific membrane protein located at the postsynaptic density of glutamatergic synapses. PRG-1 modulates signaling pathways of phosphorylated lipid substrates such as lysophosphatidic acid (LPA). Deletion of PRG-1 increases presynaptic glutamate release probability leading to neuronal over-excitation. However, due to its cortical expression, PRG-1 deficiency leading to increased glutamatergic transmission is supposed to also affect motor pathways. We therefore analyzed the effects of PRG-1 function on exploratory and motor behavior using homozygous PRG-1 knockout (PRG-1−/−) mice and PRG-1/LPA2–receptor double knockout (PRG-1−/−/LPA2−/−)…

0301 basic medicinemedicine.medical_specialtyGlutamic AcidNerve Tissue ProteinsBiologyHyperkinesisHippocampusOpen field03 medical and health sciencesBehavioral NeuroscienceGlutamatergicchemistry.chemical_compoundMice0302 clinical medicineLysophosphatidic acidmedicineAnimalsReceptors Lysophosphatidic AcidPsychiatryMice KnockoutNeuronsMental DisordersGlutamate receptorSomatosensory CortexMice Inbred C57BL030104 developmental biologymedicine.anatomical_structurechemistrySynapsesExploratory BehaviorGABAergicCalmodulin-Binding ProteinsFemaleNeuronSignal transductionLysophospholipidsPostsynaptic density030217 neurology & neurosurgerySignal TransductionBehavioural brain research
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Obesity as a Risk Factor for Alzheimer’s Disease: Implication of Leptin and Glutamate

2019

Obesity is known to induce leptin and insulin resistance. Leptin is a peptide hormone synthesized in adipose tissue that mainly regulates food intake. It has been shown that insulin stimulates the production of leptin when adipocytes are exposed to glucose to encourage satiety; while leptin, via a negative feedback, decreases the insulin release and enhances tissue sensitivity to it, leading to glucose uptake for energy utilization or storage. Therefore, resistance to insulin is closely related to leptin resistance. Obesity in middle age has also been related to Alzheimer’s disease (AD). In recent years, the relation between impaired leptin signaling pathway and the onset of AD has been stu…

0301 basic medicinemedicine.medical_specialtyMini Reviewmedicine.medical_treatmentGlucose uptakeExcitotoxicityAdipose tissuemedicine.disease_causelcsh:RC321-57103 medical and health sciences0302 clinical medicineInsulin resistanceInternal medicinemedicineoverweightleptin-resistanceReceptorlcsh:Neurosciences. Biological psychiatry. Neuropsychiatrybusiness.industryGeneral NeuroscienceInsulinLeptindigestive oral and skin physiologyGlutamate receptormedicine.disease030104 developmental biologyEndocrinologyLTPbusinessexcitotoxicity030217 neurology & neurosurgeryhormones hormone substitutes and hormone antagonistsNeurosciencedementiaFrontiers in Neuroscience
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Coronally advanced flap and connective tissue graft with or without plasma rich in growth factors (PRGF) in treatment of gingival recession

2017

Background Several researchers have tried to improve the results of gingival recession treatment techniques. One of the methods is to use growth factors The present study was undertaken to evaluate the effect of CAF (coronally advanced flap) + CTG (connective tissue graft) + PRGF (plasma rich in growth factors) in the treatment of Miller Class I buccal gingival recession. Material and methods Twenty-two teeth with Miller Class I gingival recession in 6 patients 26 ‒ 47 years of age were included in a split-mouth designed randomized controlled trial (RCT). In each patient, one side was treated with CAF + CTG + PRGF (test) and the other side was treated with CAF + CTG (control). The following…

0301 basic medicinemedicine.medical_specialtyMucogingival junctionbusiness.industryResearchConnective tissueAttachment levelRepeated measures designDentistryMucogingival surgery030206 dentistry:CIENCIAS MÉDICAS [UNESCO]Root coverage03 medical and health sciencesPlastic surgery030104 developmental biology0302 clinical medicinemedicine.anatomical_structureUNESCO::CIENCIAS MÉDICASmedicinePeriodontologymedicine.symptombusinessGeneral DentistryGingival recession
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Supporting recovery from brain injury

2018

The beauty and intricacy of the human brain is unfortunately also mirrored by its vulnerability. Damage to the brain is typically permanent. Because cells of the adult brain, apart from rare exceptions, no longer divide, there is essentially no regrowth of damaged brain tissue. Acquired brain injury in the majority of cases occurs directly through traumatic events such as an accident involving a blow to the head or indirectly through interruption of the blood supply, namely a stroke. Brain injury is a major burden, with an estimated 1.7 million people in the United States suffering a traumatic brain injury and nearly 800,000 Americans suffering a stroke each year ( 1 , 2 ). However, current…

0301 basic medicinemedicine.medical_specialtyMultidisciplinarybusiness.industryTraumatic brain injuryTreatment optionsRecovery of FunctionBrain tissueHuman brainmedicine.disease03 medical and health sciences030104 developmental biology0302 clinical medicinePhysical medicine and rehabilitationmedicine.anatomical_structureBrain InjuriesmedicineHumansBlood supplybusinessAcquired brain injury030217 neurology & neurosurgeryScience
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