Search results for "Toxicity"

showing 10 items of 2261 documents

T cell-mediated cytotoxicity: discrimination between antigen recognition, lethal hit and cytolysis phase.

1974

Using a 51Cr release cytotoxicity assay, the cytotoxic effector phase of in vitro activated mouse T lymphocytes (killer cells) against 51Cr-labeled target cells has been investigated. It is shown that within 5–10 minutes of contact between killer cells and target cells, the target cells are already committed to lysis, therefore, antigen recognition and “lethal hit” must have taken place within this period of time. In contrast, target cell lysis (cytolysis phase) requires up to 3–4 h in order to be completed; it occurs independently of killer cells and it is highly temperature dependent. The killer cell-dependent phase (antigen-recognition and “lethal hit”) is dissociated into two consecutiv…

C57BL/6MaleLysisTime FactorsCell SurvivalT-LymphocytesImmunologyAntigen-Antibody ReactionsMiceAntibody SpecificityImmunology and AllergyCytotoxic T cellAnimalsCytotoxicitybiologyEffectorTemperatureNeoplasms Experimentalbiology.organism_classificationCytotoxicity Tests ImmunologicVirologyIn vitroChromium RadioisotopesCell biologyMice Inbred C57BLCytolysisKineticsMice Inbred DBAMice Inbred CBAFemaleT cell mediated cytotoxicityLymphocyte Culture Test MixedEuropean journal of immunology
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Delivering all in one: Antigen-nanocapsule loaded with dual adjuvant yields superadditive effects by DC-directed T cell stimulation

2018

Therapeutic vaccination is and remains a major challenge, particularly in cancer treatment. In this process, the effective activation of dendritic cells by a combination of distinctly acting adjuvants and an antigen is crucial for success. While most common vaccine formulations lack the efficiency to trigger sufficient T cell responses in a therapeutic tumor treatment, nanovaccines offer unique properties to tackle that challenge. Here, we report the stepwise development of a nanocapsule for vaccination approaches, comprising a shell consisting of antigen and loaded with a superadditive adjuvant combination. In a first initial step, we identified the combination of resiquimod (R848) and mur…

CD4-Positive T-Lymphocytes0301 basic medicineCell SurvivalOvalbuminT-Lymphocytesmedicine.medical_treatmentT cellPharmaceutical ScienceMice Transgenic02 engineering and technologyCD8-Positive T-LymphocytesCancer VaccinesCell Line03 medical and health scienceschemistry.chemical_compoundNanocapsulesAntigenmedicineAnimalsHumansAntigensCytotoxicityAdjuvants PharmaceuticCell ProliferationChemistryImidazolesDextransDendritic CellsDendritic cell021001 nanoscience & nanotechnologyCell biologyMice Inbred C57BL030104 developmental biologymedicine.anatomical_structureCytokinesSpermineResiquimod0210 nano-technologyAcetylmuramyl-Alanyl-IsoglutamineAdjuvantMuramyl dipeptideCD8Journal of Controlled Release
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CD8+ cytotoxic T lymphocytes isolated from allogeneic healthy donors recognize HLA class Ia/Ib–associated renal carcinoma antigens with ubiquitous or…

2004

AbstractAllogeneic hematopoietic stem cell transplantation can induce considerable tumor remissions in metastatic renal-cell carcinoma (RCC) patients. The precise effector mechanisms mediating these graft-versus-tumor reactions are unknown. We studied RCC-directed CD8+ T-cell responses in blood lymphocytes of healthy individuals matched with established RCC cell lines for HLA-class I. In 21 of 22 allogeneic mixed lymphocyte/tumor-cell cultures (MLTCs), RCC-reactive cytotoxic T-lymphocytes (CTLs) were readily obtained. From MLTCs, 121 CD8+ CTL clones with memory phenotype were isolated. Their anti–RCC reactivity was restricted by multiple classical HLA-Ia molecules, in particular by HLA-A2, …

CD4-Positive T-LymphocytesCytotoxicity ImmunologicGenotypemedicine.medical_treatmentMolecular Sequence DataImmunologyCell SeparationHuman leukocyte antigenHematopoietic stem cell transplantationCross ReactionsBiologyurologic and male genital diseasesBiochemistryEpitheliumCell therapyEpitopesAntigenAntigens NeoplasmmedicineHumansTransplantation HomologousCytotoxic T cellAmino Acid SequenceCarcinoma Renal CellHistocompatibility Antigens Class ICell BiologyHematologyImmunotherapyFlow CytometryHematopoietic Stem CellsTissue DonorsCTL*HealthSpectrometry Mass Matrix-Assisted Laser Desorption-IonizationColonic NeoplasmsImmunologyMitogen-Activated Protein KinasesPeptidesCD8T-Lymphocytes CytotoxicBlood
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Uptake and presentation of exogenous antigen and presentation of endogenously produced antigen by skin dendritic cells represent equivalent pathways …

2008

Gene gun-mediated biolistic DNA vaccination with beta-galactosidase (betaGal)-encoding plasmid vectors efficiently modulated antigen-induced immune responses in an animal model of type I allergy, including the inhibition of immunoglobulin E (IgE) production. Here we show that CD4(+) as well as CD8(+) T cells from mice biolistically transfected with a plasmid encoding betaGal under the control of the fascin promoter (pFascin-betaGal) are capable of inhibiting betaGal-specific IgE production after adoptive transfer into naïve recipients. Moreover, suppression of IgE production was dependent on interferon (IFN)-gamma. To analyse the modalities of activation of CD4(+) and CD8(+) T cells regardi…

CD4-Positive T-LymphocytesCytotoxicity ImmunologicKeratinocytesAdoptive cell transferGenetic VectorsImmunologyAntigen presentationPriming (immunology)CD8-Positive T-LymphocytesBiologyImmunoglobulin GDNA vaccinationInterferon-gammaMiceCross-PrimingImmune systemAntigenHypersensitivityVaccines DNAAnimalsImmunology and AllergyCytotoxic T cellPromoter Regions GeneticMice KnockoutAntigen PresentationInterleukin-12 Subunit p40Keratin-15VaccinationT-Lymphocytes Helper-InducerOriginal ArticlesBiolisticsImmunoglobulin Ebeta-GalactosidaseAdoptive TransferMolecular biologyImmunoglobulin GLangerhans CellsImmunologybiology.proteinKeratin-5FemaleImmunology
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Clonal analysis of human T lymphocytes infiltrating the liver in chronic active hepatitis B and primary biliary cirrhosis

1988

Human T lymphocytes infiltrating the liver in chronic active hepatitis B (CAH-B) and primary biliary cirrhosis were isolated from liver biopsy cores, cloned by limiting dilution technique and expanded in vitro. Phenotypic and functional analysis demonstrates that this tissue infiltrate represents a heterogeneous cell population. However, when compared to peripheral blood lymphocytes of the same patients, a marked enrichment for T8+ cytotoxic T cells was found to exist at a local site in both types of chronic liver disease. These data provide support for the notion that liver cell injury in CAH-B and PBC may be mediated by a common immunologic mechanism likely executed by cells of the T line…

CD4-Positive T-LymphocytesCytotoxicity ImmunologicPathologymedicine.medical_specialtyT-LymphocytesBiliary cirrhosisImmunologyChronic liver diseasePrimary biliary cirrhosismedicineHumansImmunology and AllergyCytotoxic T cellHepatitis Chronicmedicine.diagnostic_testLiver Cirrhosis Biliarybusiness.industryLiver cellHepatobiliary diseaseAntibodies MonoclonalT lymphocyteHepatitis Bmedicine.diseaseClone CellsKiller Cells NaturalLiver biopsyImmunologybusinessT-Lymphocytes CytotoxicEuropean Journal of Immunology
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Perforin deficiency attenuates inflammation and tumor growth in colitis-associated cancer

2010

Background: Patients with inflammatory bowel disease (IBD) have a markedly increased risk to develop colon cancer, but there are only limited data about the host antitumor response in such colitis-associated cancer. In the present study we aimed at assessing the role of perforin-dependent effector mechanisms in the immune response in a murine model of colitis-associated colon cancer. Methods: Wildtype and perforin-deficient mice were analyzed in a mouse model of colitis-associated colon cancer using azoxymethane (AOM) and dextran sodium sulfate (DSS). Results: Tumors of wildtype mice showed infiltration of CD4+, CD8+ T cells, natural killer (NK) cells, high numbers of apoptotic cells, and e…

CD4-Positive T-LymphocytesCytotoxicity ImmunologicPore Forming Cytotoxic ProteinsT-LymphocytesMedizinInflammationCD8-Positive T-LymphocytesBiologymedicine.disease_causeInflammatory bowel diseaseMiceImmune systemmedicineAnimalsImmunology and AllergyCytotoxic T cellIntestinal MucosaColitisReverse Transcriptase Polymerase Chain ReactionPerforin DeficiencyDextran SulfateGastroenterologyColitismedicine.diseaseSpecific Pathogen-Free OrganismsKiller Cells NaturalMice Inbred C57BLDisease Models AnimalPerforinChronic DiseaseColonic NeoplasmsImmunologybiology.proteinmedicine.symptomCarcinogenesisInflammatory Bowel Diseases
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Characterization of lung γδ T cells following intranasal infection with Mycobacterium bovis bacillus Calmette-Guérin

2002

The lungs are considered to have an impaired capacity to contain infection by pathogenic mycobacteria, even in the presence of effective systemic immunity. In an attempt to understand the underlying cellular mechanisms, we characterized the gammadelta T cell population following intranasal infection with Mycobacterium bovis bacillus Calmette-Guerin (BCG). The peak of gammadelta T cell expansion at 7 days postinfection preceded the 30 day peak of alphabeta T cell expansion and bacterial count. The expanded population of gammadelta T cells in the lungs of BCG-infected mice represents an expansion of the resident Vgamma2 T cell subset as well as an influx of Vgamma1 and of four different Vdelt…

CD4-Positive T-LymphocytesCytotoxicity ImmunologicT cellImmunologyGene Rearrangement delta-Chain T-Cell Antigen ReceptorEpitopes T-Lymphocytechemical and pharmacologic phenomenaBiologyCD8-Positive T-LymphocytesLymphocyte ActivationLymphocyte DepletionInterleukin 21MiceAntigenT-Lymphocyte SubsetsmedicineImmunology and AllergyCytotoxic T cellAnimalsTuberculosisIL-2 receptorAntigen-presenting cellLungAdministration IntranasalCells CulturedGene Rearrangement gamma-Chain T-Cell Antigen ReceptorT-cell receptorhemic and immune systemsReceptors Antigen T-Cell gamma-deltaAcquired immune systemFlow CytometryMycobacterium bovisMice Inbred C57BLstomatognathic diseasesmedicine.anatomical_structureImmunologyCytokinesCell DivisionT-Lymphocytes Cytotoxic
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Mage-3 and influenza-matrix peptide-specific cytotoxic T cells are inducible in terminal stage HLA-A2.1+ melanoma patients by mature monocyte-derived…

2000

Abstract Dendritic cell (DC) vaccination, albeit still in an early stage, is a promising strategy to induce immunity to cancer. We explored whether DC can expand Ag-specific CD8+ T cells even in far-advanced stage IV melanoma patients. We found that three to five biweekly vaccinations of mature, monocyte-derived DC (three vaccinations of 6 × 106 s.c. followed by two i.v. ones of 6 and 12 × 106, respectively) pulsed with Mage-3A2.1 tumor and influenza matrix A2.1-positive control peptides as well as the recall Ag tetanus toxoid (in three of eight patients) generated in all eight patients Ag-specific effector CD8+ T cells that were detectable in blood directly ex vivo. This is the first time …

CD4-Positive T-LymphocytesCytotoxicity Immunologicmedicine.medical_treatmentInjections SubcutaneousImmunologyImmunization SecondaryEpitopes T-LymphocyteCD8-Positive T-LymphocytesLymphocyte ActivationCancer VaccinesMonocytesViral Matrix ProteinsAntigens NeoplasmTetanus ToxoidImmunology and AllergyMedicineCytotoxic T cellHumansMelanomaCells Culturedbusiness.industryMelanomaToxoidCell DifferentiationDendritic cellDendritic Cellsmedicine.diseaseNeoplasm ProteinsImmunizationImmunologyInjections IntravenousIntercellular Signaling Peptides and ProteinsbusinessPeptidesAdjuvantCD8Ex vivoT-Lymphocytes Cytotoxic
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Targeting the activation-induced antigen CD137 can selectively deplete alloreactive T cells from antileukemic and antitumor donor T-cell lines.

2006

AbstractIn HLA-incompatible hematopoietic stem cell transplantation, alloreactive donor T cells recognizing recipient mismatch HLA cause severe graft-versus-host disease (GVHD). Strategies allowing the selective depletion of alloreactive T cells as well as the enhancement of graft-versus-malignancy immunity would be beneficial. We generated donor CD8 T-cell lines in vitro using allogeneic recipient cells mismatched at a single HLA class I allele or haplotype as stimulators. Recipient cells were obtained from acute myeloid leukemias, renal-cell carcinomas, and CD40L-induced B lymphoblasts. Resulting alloreactive T cells were activated by incubating day 21 T-cell cultures with HLA-mismatch tr…

CD4-Positive T-LymphocytesHerpesvirus 4 HumanIsoantigensT cellImmunologyCD40 LigandCytomegalovirusGraft vs Host DiseaseHuman leukocyte antigenBiologyCD8-Positive T-LymphocytesIn Vitro TechniquesLymphocyte ActivationTransfectionBiochemistryImmunotherapy AdoptiveLymphocyte DepletionTumor Necrosis Factor Receptor Superfamily Member 9AntigenHLA AntigensT-Lymphocyte SubsetsmedicineCytotoxic T cellHumansCarcinoma Renal CellCells CulturedSkinB-LymphocytesImmunomagnetic SeparationLymphoblastCD137Cell BiologyHematologyT lymphocyteFibroblastsCytotoxicity Tests ImmunologicKidney Neoplasmsmedicine.anatomical_structureLeukemia MyeloidHistocompatibilityImmunologyK562 CellsCD8Blood
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Higher Incidence of Cancer Therapy-Related Cardiac Dysfunction in the COVID-19 Era: A Single Cardio-Oncology Center Experience

2023

Aim: COVID-19 pandemic had a big impact on our life, it has revolutionized the practice of cardiology and the organization of hospital and outpatient activities. Thus the aim of our study was to assess the impact of the COVID-19 pandemic on the development of cancer therapy-related cardiac dysfunction (CTRCD). Methods and results: A single center retrospective study was carried out evaluating 96 cancer patients treated with anthracyclines and admitted to our Cardio-Oncology unit from June to August 2019 and 60 patients from June to August 2021. The incidence of CTRCD was assessed performing an echocardiogram at the time of the enrollment. We found a significantly higher incidence of CTRCD i…

COVID-19 CTRCD cardio-oncology cardiotoxicity pandemiccardio-oncology; cardiotoxicity; COVID-19; pandemic; CTRCDPharmacology (medical)General Pharmacology Toxicology and PharmaceuticsJournal of Cardiovascular Development and Disease; Volume 10; Issue 1; Pages: 23
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