Search results for "Transduction"

showing 10 items of 2149 documents

Development of a microwave transduction gas sensor

2013

In this work, we have developed a new gas-sensing transduction based on the propagation of microwave inside a sensitive material to detect atmospheric pollutants at room temperature. First, we present a short overview of the main gas-sensing transduction in the litterature. These techniques operate in frequency ranges different from microwaves. The dielectric permittivity is the main physical parameter in the microwave transduction. The principle is based on the excitation of a sensitive material by a microwave electromagnetic field in a propagative structure. The reflected wave is specific to the excitation's frequency. It depends on the interaction between the sensitive material and the p…

[SPI.OTHER]Engineering Sciences [physics]/OtherToluèneAmmoniacOxyde inorganique[ SPI.OTHER ] Engineering Sciences [physics]/Other[SPI.OTHER] Engineering Sciences [physics]/OtherCapteur de gazNo english keywordMatériau moléculaire[ PHYS.COND.CM-GEN ] Physics [physics]/Condensed Matter [cond-mat]/Other [cond-mat.other]Transduction microondePhtalocyanine[PHYS.COND.CM-GEN] Physics [physics]/Condensed Matter [cond-mat]/Other [cond-mat.other][PHYS.COND.CM-GEN]Physics [physics]/Condensed Matter [cond-mat]/Other [cond-mat.other]
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Pharmacogenomic identification of small molecules for lineage specific manipulation of subventricular zone germinal activity

2017

Strategies for promoting neural regeneration are hindered by the difficulty of manipulating desired neural fates in the brain without complex genetic methods. The subventricular zone (SVZ) is the largest germinal zone of the forebrain and is responsible for the lifelong generation of interneuron subtypes and oligodendrocytes. Here, we have performed a bioinformatics analysis of the transcriptome of dorsal and lateral SVZ in early postnatal mice, including neural stem cells (NSCs) and their immediate progenies, which generate distinct neural lineages. We identified multiple signaling pathways that trigger distinct downstream transcriptional networks to regulate the diversity of neural cells …

animal diseasesGene Identification and AnalysisGenetic NetworksAPC-PAIDMiceNeural Stem CellsCell SignalingLateral VentriclesDatabases GeneticGene Regulatory NetworksBiology (General)WNT Signaling CascadeNotch SignalingOrganic CompoundsBB/M029379/1GenomicsSignaling CascadesOligodendrogliaChemistryBBSRCPhysical Sciences[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Network AnalysisNeurovetenskaperSignal TransductionResearch ArticleBiotechnologyComputer and Information SciencesSignal InhibitionQH301-705.5NeurogenesisResearch and Analysis MethodsSmall Molecule LibrariesGenetics/dk/atira/pure/core/subjects/biomedicalsciencesAnimalsAdultsCell LineageComputer Simulation[SDV.NEU] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Molecular Biology TechniquesMolecular BiologyOrganic ChemistryGene MappingChemical CompoundsNeurosciencesBiology and Life SciencesRCUKBiomedical SciencesCell BiologyNerve RegenerationSignaling NetworksGene Expression Regulationnervous systemSmall MoleculesAge GroupsPeople and PlacesPopulation GroupingsTranscriptome
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Intraflagellar transport protein 172 is essential for primary cilia formation and plays a vital role in patterning the mammalian brain

2008

AbstractIFT172, also known as Selective Lim-domain Binding protein (SLB), is a component of the intraflagellar transport (IFT) complex. In order to evaluate the biological role of the Ift172 gene, we generated a loss-of-function mutation in the mouse. The resulting Slb mutant embryos die between E12.5 and 13.0, and exhibit severe cranio-facial malformations, failure to close the cranial neural tube, holoprosencephaly, heart edema and extensive hemorrhages. Cilia outgrowth in cells of the neuroepithelium is initiated but the axonemes are severely truncated and do not contain visible microtubules. Morphological and molecular analyses revealed a global brain-patterning defect along the dorsal–…

animal structuresBody PatterningNodal ProteinSlbNodalBiologyArticleMiceFGF8Intraflagellar transportHoloprosencephalymedicineMHB boundaryAnimalsHedgehog ProteinsRNA MessengerCiliaNodeMolecular BiologyAdaptor Proteins Signal TransducingBody PatterningGeneticsMammalsCell DeathCiliumEndodermNeural tubeIntracellular Signaling Peptides and ProteinsBrainGene Expression Regulation DevelopmentalCell BiologyEmbryo MammalianCell biologyNeuroepithelial cellGastrulationCytoskeletal Proteinsmedicine.anatomical_structurePhenotypeIFT172Gene Targetingembryonic structuresNODALBiomarkersGene DeletionDevelopmental BiologySignal TransductionDevelopmental Biology
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Extremely low frequency electromagnetic fields (ELF-EMFs) induce in vitro angiogenesis process in human endothelial cells.

2008

Effects of extremely low frequency (ELF) electromagnetic fields (EMFs) on activation of angiogenesis were analysed using cultured umbilical human vein endothelial cells (HUVECs). The cultures were exposed to a sinusoidal EMF to intensity of 1 mT, 50 Hz for up to 12 h. EMFs increased the degree of endothelial cell proliferation and tubule formation, coupled by an acceleration in the process of wound healing. Since this process is physiologically accompanied by a large modification in the structural organization of actin and focal adhesions, we analyzed the rearrangement of some cytoskeleton elements demonstrating a major reorganization of the fibres and of the focal adhesion complexes after …

animal structuresCytoskeleton organizationPhysiologyAngiogenesisBiophysicsNeovascularization PhysiologicBiologyRadiation DosageFocal adhesionElectromagnetic FieldsEndothelial cellElectricityHumansRadiology Nuclear Medicine and imagingTherapeutic angiogenesisCytoskeletonCells CulturedEndothelial CellsDose-Response Relationship RadiationGeneral MedicineCell biologyEndothelial stem cellAngiogenesiSignal transductionWound healingExtremely low frequency electromagnetic fieldsBioelectromagnetics
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Sea urchin deciliation induces thermoresistance and activates the p38 mitogen-activated protein kinase pathway.

2003

In this study, we demonstrate by a variety of approaches (ie, morphological analysis, Western blots, immunolocalization, and the use of specific antibodies) that hyperosmotic deciliation stress of sea urchin embryos induces a thermotolerant response. Deciliation is also able to activate a phosphorylation signaling cascade the effector of which might be the p38 stress-activated protein kinase because we found that the administration of the p38 inhibitor SB203580 to sea urchin deciliated gastrula embryos makes the hyperosmotic deciliation stress lethal.

animal structuresHot TemperaturePyridinesp38 mitogen-activated protein kinasesSEA URCHIN DECILIATION p38MAP KINASEBiochemistryp38 Mitogen-Activated Protein KinasesEnzyme activatorStress Physiologicalbiology.animalAnimalsCiliaSettore BIO/06 - Anatomia Comparata E CitologiaPhosphorylationProtein kinase ASea urchinbiologyEffectorImidazolesAntibodies MonoclonalCell BiologyGastrulaOriginal ArticlesMolecular biologyBlotEnzyme ActivationSea Urchinsembryonic structuresPhosphorylationElectrophoresis Polyacrylamide GelSignal transductionMitogen-Activated Protein KinasesSignal TransductionCell stresschaperones
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Toll signal transduction pathway in bivalves: Complete cds of intermediate elements and related gene transcription levels in hemocytes of immune stim…

2014

Based on protein domain structure and organization deduced from mRNA contigs, 15 transcripts of the Toll signaling pathway have been identified in the bivalve, Mytilus galloprovincialis. Identical searches performed on publicly available Mytilus edulis ESTs revealed 11 transcripts, whereas searches performed in genomic and new transcriptome sequences of the Pacific oyster, Crassostrea gigas, identified 21 Toll-related transcripts. The remarkable molecular diversity of TRAF and IKK coding sequences of C. gigas, suggests that the sequence data inferred from Mytilus cDNAs may not be exhaustive. Most of the Toll pathway genes were constitutively and ubiquitously expressed in M. galloprovinciali…

animal structuresMolluskToll signaling pathwayInnate immunity; Mollusks; Mytilus; Signal transduction; Toll pathway; NF-κBImmunologyProtein domainSettore BIO/05 - ZoologiaMytiluSignal transductionNF-κBTranscriptomeTranscription (biology)Animals[SDU.STU.HY]Sciences of the Universe [physics]/Earth Sciences/HydrologyGenePhylogenyComputingMilieux_MISCELLANEOUSMytilusInnate immunityMessenger RNAInnate immune systemMollusksToll-like receptors; signal transduction; Mytilus-galloprovincialis Lmk (bivalvia)biologyEcologyfungiMytilus-galloprovincialis Lmk (bivalvia)biology.organism_classificationMytilusToll-like receptorsCell biologyInnate immunity; Mollusks; Mytilus; NF-κB; Signal transduction; Toll pathwayToll pathwayNF-jBDevelopmental Biology
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Cabut, a C2H2 zinc finger transcription factor, is required during Drosophila dorsal closure downstream of JNK signaling.

2005

AbstractDuring dorsal closure, the lateral epithelia on each side of the embryo migrate dorsally over the amnioserosa and fuse at the dorsal midline. Detailed genetic studies have revealed that many molecules are involved in this epithelial sheet movement, either with a signaling function or as structural or motor components of the process. Here, we report the characterization of cabut (cbt), a new Drosophila gene involved in dorsal closure. cbt is expressed in the yolk sac nuclei and in the lateral epidermis. The Cbt protein contains three C2H2-type zinc fingers and a serine-rich domain, suggesting that it functions as a transcription factor. cbt mutants die as embryos with dorsal closure …

animal structuresMorphogenesisBiologyCabutZinc fingerMorphogenesismedicineAnimalsDrosophila ProteinsDorsal closureYolk sacMolecular BiologyTranscription factorYolk nucleiCytoskeletonGeneticsZinc fingerEpidermis (botany)C2H2 Zinc FingerJNK Mitogen-Activated Protein KinasesZinc FingersCell BiologyDorsal closureCell biologymedicine.anatomical_structureDrosophila melanogasterEpidermal Cellsembryonic structuresMutationJNK cascadeDrosophilaJNKDevelopmental BiologySignal TransductionTranscription FactorsDevelopmental biology
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Notch and Numb are required for normal migration of peripheral glia in Drosophila

2006

Abstract A prominent feature of glial cells is their ability to migrate along axons to finally wrap and insulate them. In the embryonic Drosophila PNS, most glial cells are born in the CNS and have to migrate to reach their final destinations. To understand how migration of the peripheral glia is regulated, we have conducted a genetic screen looking for mutants that disrupt the normal glial pattern. Here we present an analysis of two of these mutants: Notch and numb. Complete loss of Notch function leads to an increase in the number of glial cells. Embryos hemizygous for the weak NotchB-8X allele display an irregular migration phenotype and mutant glial cells show an increased formation of …

animal structuresMutantNotch signaling pathwayBiologyCell MovementGlial cell migrationAnimalsDrosophila ProteinsMolecular BiologyGeneticsReceptors NotchCell migrationCell BiologyImmunohistochemistryEmbryonic stem cellPhenotypeCell biologyJuvenile Hormonesnervous systemMutagenesisNUMBDrosophilaNeurogliaSignal TransductionGenetic screenDevelopmental BiologyDevelopmental Biology
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Sonic hedgehog promotes the migration and proliferation of optic nerve oligodendrocyte precursors.

2007

Optic nerve (ON) oligodendrocyte precursors (OPCs) are generated under the influence of the Sonic hedgehog (Shh) in the preoptic area from where they migrate to colonise the entire nerve. The molecular events that control this migration are still poorly understood. Recent studies suggested that Shh is often used by the same cell population to control different processes, including cell proliferation and migration, raising the possibility that Shh could contribute to these aspects of OPC development. In support of this idea, we show here that Shh induces the proliferation of OPCs derived from embryonic mouse ON explants and acts as a chemoattractant for their migration. In ovo injections of …

animal structuresPopulationCellChick EmbryoAntibodiesCellular and Molecular NeuroscienceMyelinMiceCell MovementCell Line TumormedicineAnimalsHedgehog ProteinsSonic hedgehogeducationMolecular BiologyCells CulturedMyelin SheathCell Proliferationeducation.field_of_studybiologyCell growthChemotaxisStem CellsCell DifferentiationOptic NerveCell BiologyEmbryonic stem cellOligodendrocyteCell biologystomatognathic diseasesOligodendrogliamedicine.anatomical_structurenervous systemembryonic structuresOptic nervebiology.proteinNeuroscienceSignal TransductionMolecular and cellular neurosciences
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Plexin-B1 activates NF-κB and IL-8 to promote a pro-angiogenic response in endothelial cells.

2011

Background The semaphorins and their receptors, the plexins, are proteins related to c-Met and the scatter factors that have been implicated in an expanding signal transduction network involving co-receptors, RhoA and Ras activation and deactivation, and phosphorylation events. Our previous work has demonstrated that Semaphorin 4D (Sema4D) acts through its receptor, Plexin-B1, on endothelial cells to promote angiogenesis in a RhoA and Akt-dependent manner. Since NF-κB has been linked to promotion of angiogenesis and can be activated by Akt in some contexts, we wanted to examine NF-κB in Sema4D treated cells to determine if there was biological significance for the pro-angiogenic phenotype o…

animal structuresRHOAProto-Oncogene Proteins c-aktAngiogenesisSignaling in cellular processesG-protein signalingCancer TreatmentSEMA4Dlcsh:MedicineSignal transductionBiology03 medical and health sciencesMolecular cell biology0302 clinical medicineSemaphorinSettore BIO/10 - BiochimicaAkt Signaling CascadeMembrane Receptor SignalingInterleukin 8lcsh:ScienceBiologyProtein kinase BGTPase signalingRas signaling030304 developmental biology0303 health sciencesMultidisciplinaryMechanisms of Signal Transductionlcsh:RSignaling Cascades3. Good healthCell biologyPlexin B1RNA interferencepro-angiogenicendothelial cellsOncology030220 oncology & carcinogenesisembryonic structuresCancer researchbiology.proteinMedicinelcsh:QAntiangiogenesis TherapyAntiapoptotic signalingSignal transductionResearch ArticlePLoS ONE
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