Search results for "Transduction"

showing 10 items of 2149 documents

Resveratrol modulates the levels of microRNAs targeting genes encoding tumor-suppressors and effectors of TGFbeta signaling pathway in SW480 cells.

2010

International audience; Resveratrol (trans-3,4',5-trihydroxystilbene) is a natural antioxidant with cardiovascular and cancer preventive properties that is currently at the stage of pre-clinical studies for human cancer prevention. Beside its known effects on protein coding genes, one possible mechanism for resveratrol protective activities is by modulating the levels of non-coding RNAs. Here, we analyzed the effects of resveratrol on microRNA populations in human SW480 colon cancer cells. We establish that resveratrol treatment decreases the levels of several oncogenic microRNAs targeting genes encoding Dicer1, a cytoplasmic RNase III producing mature microRNAs from their immediate precurs…

Antineoplastic AgentsSmad ProteinsResveratrolBiochemistryAntioxidantsArticleTransforming Growth Factor beta1chemistry.chemical_compoundTGFβTransforming Growth Factor betaCell Line TumormicroRNAStilbenesPTENHumansRibonuclease III[ SDV.BBM ] Life Sciences [q-bio]/Biochemistry Molecular BiologyPharmacologyOncogene ProteinsbiologyEffectorTumor Suppressor ProteinsTransforming growth factor betaMolecular biologyColon cancer; microRNAs; miR-663; Resveratrol; SW480 cells; TGFβmiR-663Cell biologyColon cancerMicroRNAsSW480 cellschemistryResveratrolbiology.proteinSignal transductionTransforming growth factorSignal Transduction
researchProduct

PTEN Mediates the Antioxidant Effect of Resveratrol at Nutritionally Relevant Concentrations

2014

Introduction.Antioxidant properties of resveratrol have been intensively studied for the last years, bothin vivoandin vitro. Its bioavailability after an oral dose is very low and therefore it is very important to make sure that plasma concentrations of free resveratrol are sufficient enough to be active as antioxidant.Aims.In the present study, using nutritionally relevant concentrations of resveratrol, we aim to confirm its antioxidant capacity on reducing peroxide levels and look for the molecular pathway involved in this antioxidant effect.Methods.We used mammary gland tumor cells (MCF-7), which were pretreated with different concentrations of resveratrol for 48 h, and/or a PTEN inhibit…

Antioxidantendocrine system diseasesArticle Subjectmedicine.medical_treatmentlcsh:MedicineResveratrolGeneral Biochemistry Genetics and Molecular BiologyAntioxidantschemistry.chemical_compoundDownregulation and upregulationStilbenesmedicinePTENHumansPhosphorylationskin and connective tissue diseasesHydrogen peroxidePI3K/AKT/mTOR pathwayGeneral Immunology and MicrobiologybiologyAkt/PKB signaling pathwaySuperoxide Dismutaseorganic chemicalslcsh:RPTEN Phosphohydrolasefood and beveragesGeneral MedicineHydrogen PeroxideCatalaseUp-RegulationEnzyme ActivationBiochemistrychemistryCatalaseResveratrolbiology.proteinMCF-7 CellsProto-Oncogene Proteins c-akthormones hormone substitutes and hormone antagonistsSignal TransductionResearch Article
researchProduct

Ursolic acid ameliorates stress and reactive oxygen species in C. elegans knockout mutants by the dopamine Dop1 and Dop3 receptors.

2020

Abstract Background Depression and stress-related disorders are leading causes of death worldwide. Standard treatments elevating serotonin or noradrenaline levels are not sufficiently effective and cause adverse side effects. A connection between dopamine pathways and stress-related disorders has been suggested. Compounds derived from herbal medicine could be a promising alternative. We examined the neuroprotective effects of ursolic acid (UA) by focusing on dopamine signalling. Methods Trolox equivalent capacity assay was used to determine the antioxidant activities of UA in vitro. C. elegans N2 wildtype and dopamine receptor-knockout mutants (dop-1-deficient RB665 and dop-3-deficient LX70…

Antioxidantmedicine.medical_treatmentDopamineLongevityPharmaceutical SciencePharmacologyNeuroprotectionAntioxidants03 medical and health scienceschemistry.chemical_compoundGene Knockout Techniques0302 clinical medicineDopamineStress PhysiologicalDrug DiscoverymedicineAnimalsHumansReceptorCaenorhabditis elegansCaenorhabditis elegans Proteins030304 developmental biologyPharmacologychemistry.chemical_classification0303 health sciencesReactive oxygen speciesChemistryReceptors Dopamine D2Receptors Dopamine D1Receptors Dopamine D3TriterpenesMolecular Docking SimulationComplementary and alternative medicineDopamine receptor030220 oncology & carcinogenesisMutationMolecular MedicineSerotoninTroloxReactive Oxygen Speciesmedicine.drugSignal TransductionPhytomedicine : international journal of phytotherapy and phytopharmacology
researchProduct

Apolipoprotein E Regulates Amyloid Formation within Endosomes of Pigment Cells.

2015

International audience; Accumulation of toxic amyloid oligomers is a key feature in the pathogenesis of amyloid-related diseases. Formation of mature amyloid fibrils is one defense mechanism to neutralize toxic prefibrillar oligomers. This mechanism is notably influenced by apolipoprotein E variants. Cells that produce mature amyloid fibrils to serve physiological functions must exploit specific mechanisms to avoid potential accumulation of toxic species. Pigment cells have tuned their endosomes to maximize the formation of functional amyloid from the protein PMEL. Here, we show that ApoE is associated with intraluminal vesicles (ILV) within endosomes and remain associated with ILVs when th…

Apolipoprotein EAmyloidAmyloidEndosome[SDV.BC]Life Sciences [q-bio]/Cellular BiologyEndosomesBiologyExosomesGeneral Biochemistry Genetics and Molecular BiologyMiceApolipoproteins Emental disordersAnimalsHumansamyloid-related diseaseslcsh:QH301-705.5[SDV.BC] Life Sciences [q-bio]/Cellular BiologyMelanosomeMice KnockoutMelanosomesEndosomal Sorting Complexes Required for TransportVesicleMicrovesiclesPMELCell biologyMice Inbred C57BLlcsh:Biology (General)BiochemistryGene Expression RegulationMelanocytesSignal transductionHeLa CellsSignal TransductionCell reports
researchProduct

Superoxide Flux in Endothelial Cells via the Chloride Channel-3 Mediates Intracellular Signaling

2007

Reactive oxygen species (ROS) have been implicated in both cell signaling and pathology. A major source of ROS in endothelial cells is NADPH oxidase, which generates superoxide (O2.−) on the extracellular side of the plasma membrane but can result in intracellular signaling. To study possible transmembrane flux of O2.−, pulmonary microvascular endothelial cells were preloaded with the O2.−-sensitive fluorophore hydroethidine (HE). Application of an extracellular bolus of O2.−resulted in rapid and concentration-dependent transient HE oxidation that was followed by a progressive and nonreversible increase in nuclear HE fluorescence. These fluorescence changes were inhibited by superoxide dism…

ApoptosisMembrane PotentialsSuperoxide dismutasechemistry.chemical_compoundChloride ChannelsSuperoxidesExtracellularAnimalsHumansEnzyme InhibitorsRNA Small InterferingMolecular BiologyLungCells CulturedFluorescent Dyeschemistry.chemical_classificationReactive oxygen speciesNADPH oxidasebiologySuperoxideAngiotensin IIThrombinAcetophenonesEndothelial CellsNADPH OxidasesCell BiologyArticlesCell biologyMitochondriaPhenanthridinesOxygenchemistryDIDSbiology.proteinCalciumSignal transductionOxidation-ReductionIntracellularSignal Transduction
researchProduct

Activation of nitric oxide signaling by the rheumatoid arthritis shared epitope

2006

Objective. Susceptibility to rheumatoid arthritis (RA) is closely associated with HLA–DRB1 alleles encoding a shared epitope (SE) in positions 70–74 of the HLA–DR chain. The mechanistic basis for this association is unknown. Given the proposed pathogenic role of nitric oxide (NO) in RA, this study was undertaken to examine whether the SE can trigger NO signaling events. Methods. The intracellular levels of NO were measured with the fluorescent NO probe 4,5diaminofluorescein diacetate and by the 2,3diaminonaphthalene method. NO synthase activity was determined by measuring the rate of conversion of radioactive arginine to citrulline. Levels of cGMP were measured with a commercial enzyme-link…

ArginineT-LymphocytesMolecular Sequence DataImmunologyCellBiologyNitric OxideEpitopeCell LineNitric oxideArthritis Rheumatoidchemistry.chemical_compoundRheumatologymedicineCitrullineHumansImmunology and AllergyPharmacology (medical)Amino Acid SequenceB cellB-LymphocytesLymphoblastHLA-DR1 AntigenFibroblastsMolecular biologyChromium Radioisotopesmedicine.anatomical_structurechemistryImmunologyEpitopes B-LymphocyteFluoresceinIndicators and ReagentsSignal transductionSignal TransductionArthritis & Rheumatism
researchProduct

The rheumatoid arthritis shared epitope increases cellular susceptibility to oxidative stress by antagonizing an adenosine-mediated anti-oxidative pa…

2006

We have recently demonstrated that the rheumatoid arthritis (RA) shared epitope (SE) acts as a ligand that triggers nitric oxide (NO) signaling in opposite cells. Given the known pro-oxidative effect of NO and the proposed role of oxidative stress in the pathogenesis of RA, this study explores whether SE-triggered signaling can increase cellular oxidative stress. cAMP levels, adenylyl cyclase activity, and protein kinase A activity were measured using commercial kits. Generation of reactive oxygen species (ROS) was quantified using the fluorochrome dichlorofluorescein diacetate. Oxidative DNA damage was quantified using the single-cell electrophoresis technique. Here, we report that cells e…

Arthritis RheumatoidB-LymphocytesOxidative StressAdenosineEpitopes B-LymphocyteHumansDisease SusceptibilityReactive Oxygen SpeciesAntioxidantsSignal TransductionResearch ArticleArthritis researchtherapy
researchProduct

JAK3/STAT5/6 Pathway Alterations Are Associated with Immune Deviation in CD8+ T Cells in Renal Cell Carcinoma Patients

2010

To investigate the molecular mechanisms underlying altered T cell response in renal cell carcinoma (RCC) patients, we compared autologous and allogeneic CD8(+) T cell responses against RCC line from RCC patients and their HLA-matched donors, using mixed lymphocyte/tumor cell cultures (MLTCs). In addition, we analyzed the expression of molecules associated with cell cycle regulation. Autologous MLTC responder CD8(+) T cells showed cytotoxic activity against RCC cell lines; however the analysis of the distribution of CD8(+) T-cell subsets revealed that allogenic counterparts mediate superior antitumor efficacy. In RCC patients, a decreased proliferative response to tumor, associated with defe…

Article SubjectCell Survivallcsh:Biotechnologylcsh:MedicineEnzyme-Linked Immunosorbent AssayE2F4 Transcription FactorCD8-Positive T-Lymphocytesurologic and male genital diseaseslcsh:TP248.13-248.65Chromium IsotopesSTAT5 Transcription FactorTumor Cells CulturedHumansCarcinoma Renal CellInhibitor of Differentiation Protein 2Microscopy Confocallcsh:RCell CycleIntracellular Signaling Peptides and ProteinsJanus Kinase 3Flow Cytometryfemale genital diseases and pregnancy complicationsKidney NeoplasmsGene Expression Regulation NeoplasticCase-Control StudiesLymphocyte Culture Test MixedSTAT6 Transcription FactorCyclin-Dependent Kinase Inhibitor p27Research ArticleSignal TransductionJournal of Biomedicine and Biotechnology
researchProduct

Thermal sensation and cell adaptability

2013

Whole person adaptive comfort is discussed with reference to recent findings in molecular scale systems biology. The observations are upscaled to hypotheses relating to less traditional interpretations of thermal processes, which have new implications for indoor climate management and design. Arguments are presented for a revision of current focus, model and paradigm. The issue is seen as a problem of integrating theoretical development, conceptual modeling and as an investigation of the extent to which environments and acclimatization can be used to achieve individual fitness and health, not only at the subjective comfort level, as hitherto promoted. It is argued that there are many questi…

Atmospheric ScienceShock proteinsAcclimatizationmedia_common.quotation_subjectHealth Toxicology and MutagenesisScale integrationTransduction (psychology)Thermal sensationBiologyMechanotransduction CellularModels BiologicalAdaptabilityCell Physiological PhenomenaTransductionAdaptive comfortAnimalsHumansThermosensingEcosystemmedia_commonReview PaperCommunicationIndoor designEcologybusiness.industryScale (chemistry)TemperatureMolecular scale adaptationThermoreceptorsAdaptation PhysiologicalASHRAE standardsAdaptability modelCell energyGene expressionSystems biologybusinessCognitive psychologyInternational Journal of Biometeorology
researchProduct

PACAP induces bradycardia in guinea-pig heart by stimulation of atrial cholinergic neurones

1996

Based on previous studies which indicated that pituitary adenylate cyclase activating peptide (PACAP) acts as a positive inotropic and chronotropic substance in different species via the cAMP signal transduction pathway, the objective of the present work was to investigate cAMP-regulated myocardial key proteins in response to PACAP in isolated ventricular cells of the guinea pig. Surprisingly, the two molecular forms of PACAP, PACAP(1-27) and PACAP(1-38), showed no effect on intracellular cAMP-levels, L-type Ca2+ channel current or phosphorylation of troponin inhibitor (TnI) and phospholamban (PLB). Additionally, inotropy of isolated guinea-pig ventricular strips was not affected by the neu…

AtropineMaleChronotropicendocrine systemmedicine.medical_specialtyGuinea PigsReceptors Pituitary Adenylate Cyclase-Activating PolypeptideStimulationIn Vitro TechniquesBiologyInternal medicineBradycardiaCyclic AMPmedicineAnimalsHeart AtriaReceptors Pituitary HormonePatch clampNeuronsPharmacologyNeurotransmitter AgentsMyocardiumCalcium-Binding ProteinsNeuropeptidesGeneral MedicineAcetylcholineRatsPhospholambanElectrophysiologyAtropinePituitary adenylate cyclase-activating peptideEndocrinologycardiovascular systemPituitary Adenylate Cyclase-Activating PolypeptideCholinergicFemalehormones hormone substitutes and hormone antagonistsAcetylcholineReceptors Pituitary Adenylate Cyclase-Activating Polypeptide Type ISignal Transductionmedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
researchProduct