Search results for "Transferases"

showing 10 items of 426 documents

Evolution of arginine deiminase (ADI) pathway genes

2002

We have analyzed the evolution of the three genes encoding structural enzymes of the arginine deiminase (ADI) pathway, arginine deiminase (ADI), ornithine transcarbamoylase (OTC), and carbamate kinase (CK) in a wide range of organisms, including Archaea, Bacteria, and Eukarya. This catabolic route was probably present in the last common ancestor to all the domains of life. The results obtained indicate that these genes have undergone a complex evolutionary history, including horizontal transfer events, duplications, and losses. Therefore, these genes are not adequate to infer organismal relationships at deep branching levels, but they provide an insight into how catabolic genes evolved and …

Most recent common ancestorHydrolasesMolecular Sequence DataBiologyModels BiologicalEvolution MolecularGeneticsAmino Acid SequenceMolecular BiologyArginine deiminase pathwayGeneArginine deiminaseOrnithine CarbamoyltransferasePhylogenyEcology Evolution Behavior and SystematicsGeneticsLikelihood FunctionsBacteriaSequence Homology Amino AcidPhylogenetic treeCarbamate kinaseFungiSequence Analysis DNAPhosphotransferases (Carboxyl Group Acceptor)ArchaeaMetabolic pathwayHorizontal gene transferMolecular Phylogenetics and Evolution
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Exploring new roles for the rpoS gene in the survival and virulence of the fire blight pathogen Erwinia amylovora

2014

Erwinia amylovora causes fire blight in economically important plants of the family Rosaceae. This bacterial pathogen spends part of its life cycle coping with starvation and other fluctuating environmental conditions. In many Gram-negative bacteria, starvation and other stress responses are regulated by the sigma factor RpoS. We obtained an E. amylovora rpoS mutant to explore the role of this gene in starvation responses and its potential implication in other processes not yet studied in this pathogen. Results showed that E. amylovora needs rpoS to develop normal starvation survival and viable but nonculturable (VBNC) responses. Furthermore, this gene contributed to stationary phase cross-…

MutantVirulenceSigma FactorErwiniaApplied Microbiology and BiotechnologyMicrobiologyViable but nonculturableMicrobiologyPyrusBacterial ProteinsOsmotic PressureSigma factorErwinia amylovoraRosaceaePathogenPlant Diseases2. Zero hungerVirulenceEcologybiologyAgriculturaPolysaccharides Bacterialbiology.organism_classificationOxidative StressEriobotryaHexosyltransferasesGenes BacterialMutationFire blightbacteriarpoSHeat-Shock Response
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MOZ/TIF2-induced acute myeloid leukaemia in transgenic fish.

2008

The inv(8)(p11q13) chromosomal abnormality, described in acute myeloid leukaemias (AML), fuses the histone acetyl-transferase (HAT) MYST3 (MOZ) gene with another HAT gene, NCOA2 (TIF2). We generated a transgenic zebrafish in which the MYST3/NCOA2 fusion gene was expressed under control of the spi1 promoter. An AML developed in 2 of 180 MYST3/NCOA2-EGFP-expressing embryos, 14 and 26 months after injection of the fusion gene in a one-cell embryo, respectively. This leukaemia was characterised by an extensive invasion of kidneys by myeloid blast cells. This model, which is the first zebrafish model of AML, demonstrates the oncogenic potency of MYST3/NCOA2 fusion gene.

MyeloidMicroinjectionsOncogene Proteins FusionTransgeneBiologyKidneyMYST3Fusion geneAnimals Genetically ModifiedNuclear Receptor Coactivator 2hemic and lymphatic diseasesmedicineAnimalsZebrafishGeneZebrafishHistone AcetyltransferasesSPI1Reverse Transcriptase Polymerase Chain ReactionHematologymedicine.diseasebiology.organism_classificationMolecular biologyLeukemiaDisease Models AnimalLeukemia Myeloid Acutemedicine.anatomical_structureembryonic structuresCancer researchGene FusionBritish journal of haematology
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Redox signaling in acute pancreatitis

2015

Acute pancreatitis is an inflammatory process of the pancreatic gland that eventually may lead to a severe systemic inflammatory response. A key event in pancreatic damage is the intracellular activation of NF-κB and zymogens, involving also calcium, cathepsins, pH disorders, autophagy, and cell death, particularly necrosis. This review focuses on the new role of redox signaling in acute pancreatitis. Oxidative stress and redox status are involved in the onset of acute pancreatitis and also in the development of the systemic inflammatory response, being glutathione depletion, xanthine oxidase activation, and thiol oxidation in proteins critical features of the disease in the pancreas. On th…

NecrosisGSH reduced glutathioneSTAT3 signal transducer and activator of transcription 3ERK extracellular signal-regulated kinasesClinical BiochemistryCCK cholecystokininTRAFs TNF receptor associated factorsReview ArticleIκB kinasePharmacologymedicine.disease_causeBiochemistrySHP small heterodimer partnerSTIM1 stromal interaction molecule 1chemistry.chemical_compoundHATs histone acetyltransferasesMedicineASK1GCL glutamate cysteine ligaseTNF-α tumor necrosis factor alphaIKK IκB kinaseNOS nitric oxide synthaseAcute inflammationHIF hypoxia inducible factorlcsh:QH301-705.5NF-κB nuclear factor kappa BDAMPs damage-associated molecular pattern moleculeslcsh:R5-920biologyGSSG oxidized glutathioneNF-kappa BNLRs nucleotide-binding oligomerization domain (NOD) like receptorsTRADD tumor necrosis factor receptor type 1-associated DEATH domain proteinTRPC3 transient receptor potential channel 3VEGF vascular endothelial growth factorGlutathioneTNFR tumor necrosis factor receptorHMGB1 high-mobility group Box 1 proteinIP3R inositol 145-trisphosphate receptor type 3VCAM-1 Vascular Cell adhesion protein 1Acute DiseaseJNK c-Jun N-terminal kinaseAcute pancreatitisTLRs toll-like receptorsmedicine.symptomlcsh:Medicine (General)Oxidation-ReductionAP-1 activator protein-1Signal TransductionmRNA messenger ribonucleic acidHMGB1ASC apoptosis-associated speck-like protein containing a carboxy-terminal CARDRNS reactive nitrogen speciesPTPs protein tyrosine phosphatasesROS reactive oxygen speciesNADH nicotinamide adenine dinucleotidepHe extracellular pHFAEE fatty acid ethyl estersAP acute pancreatitisHumansXanthine oxidaseCBP CREB-binding proteinRyR endoplasmic reticulum membrane ryanodine receptorsMDA malondialdehydeNO nitric oxideXO xanthine oxidaseASK1 apoptosis signal-regulating kinase-1business.industryOrganic ChemistryAutophagyNADPH nicotinamide adenine dinucleotide phosphateHDACs histone deacetylasesmedicine.diseaseCARS compensatory anti-inflammatory response syndromeXDH xanthine dehydrogenaseIL interleukinIκB inhibitor of kappa BAcute pancreatitisETC Electron transport chainPancreatitisMKPs MAPK phosphatasesSAP severe acute pancreatitischemistrylcsh:Biology (General)DTT dithiothreitolOxidative stressNAC N-acetyl cysteineImmunologybiology.proteinCalciumLysosomesReactive Oxygen SpeciesbusinessMAPK mitogen-activated protein kinaseOxidative stressERCP endoscopic retrograde cholangiopancreatographyRedox Biology
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Metabolic profiling reveals distinct variations linked to nicotine consumption in humans--first results from the KORA study.

2008

Exposure to nicotine during smoking causes a multitude of metabolic changes that are poorly understood. We quantified and analyzed 198 metabolites in 283 serum samples from the human cohort KORA (Cooperative Health Research in the Region of Augsburg). Multivariate analysis of metabolic profiles revealed that the group of smokers could be clearly differentiated from the groups of former smokers and non-smokers. Moreover, 23 lipid metabolites were identified as nicotine-dependent biomarkers. The levels of these biomarkers are all up-regulated in smokers compared to those in former and non-smokers, except for three acyl-alkyl-phosphatidylcholines (e.g. plasmalogens). Consistently significant r…

Nicotinemedicine.medical_specialtyPublic Health and Epidemiology/Environmental HealthMetabolitelcsh:MedicineBiologyPharmacologyCohort StudiesNicotinechemistry.chemical_compoundInternal medicineDiabetes and Endocrinology/EndocrinologyGene expressionmedicineMetabolomeCluster AnalysisHumansAlkylglycerone-phosphate synthaselcsh:Sciencechemistry.chemical_classificationAlkyl and Aryl TransferasesMultidisciplinarySmokinglcsh:RLipid metabolismPublic Health and Epidemiology/Global HealthChemical Biology/Small Molecule ChemistryEnzymeEndocrinologychemistryBiochemistry/Small Molecule ChemistryGlycerophospholipidMetabolomePhosphatidylcholineslcsh:Qbiology.geneMental Health/Personality DisordersBiomarkersResearch Articlemedicine.drugPLoS ONE
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Modulation of biotransformation and elimination systems by BM-21, an aqueous ethanolic extract from Thalassia testudinum, and thalassiolin B on human…

2012

Abstract BM-21 is an extract obtained from Thalassia testudinum marine plant with pharmacological properties. The effects of BM-21 and thalassiolin B (TB), its main component, on enzyme and transport proteins involved in drug metabolism and excretion in human cultured hepatocytes were evaluated. Cells were exposed for 48 h to sub-cytotoxic concentrations of BM-21 or TB. Effects on P450 isoforms revealed significant reductions of CYP1A2, 3A4 and 2D6 activities (up to 56%, 66% and 44% inhibition, respectively) after exposition to BM-21, no changes on CYP2A6 and 2C9 activities. TB produced a concentration-dependent reduction of all P450 activities. In addition, a decrease in total UGT and UGT2…

Nutrition and DieteticsbiologyCYP3A4Nutrition. Foods and food supplyThalassiolin BCYP1A2PolyphenolsMedicine (miscellaneous)Cytochrome P450Cytochrome P450P-glycoproteinPharmacologyExcretionBiotransformationIn vivobiology.proteinThalassia testudinumTX341-641UDP-glucuronosyltransferasesCYP2A6Drug metabolismFood ScienceJournal of Functional Foods
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Nature of sterols affects plasma membrane behavior and yeast survival during dehydration.

2011

International audience; The plasma membrane (PM) is a main site of injury during osmotic perturbation. Sterols, major lipids of the PM structure in eukaryotes, are thought to play a role in ensuring the stability of the lipid bilayer during physicochemical perturbations. Here, we investigated the relationship between the nature of PM sterols and resistance of the yeast Saccharomyces cerevisiae to hyperosmotic treatment. We compared the responses to osmotic dehydration (viability, sterol quantification, ultrastructure, cell volume, and membrane permeability) in the wild-type (WT) strain and the ergosterol mutant erg6Δ strain. Our main results suggest that the nature of membrane sterols gover…

Osmotic stressCell Membrane PermeabilityChromatography GasSaccharomyces cerevisiae ProteinsOsmotic shockMembrane permeabilitySaccharomyces cerevisiaeBiophysicsSterol evolutionSaccharomyces cerevisiaeBiologyBiochemistryCell survival03 medical and health scienceschemistry.chemical_compoundOsmotic Pressure[ SDV.MP ] Life Sciences [q-bio]/Microbiology and ParasitologyErgosterolpolycyclic compoundsLipid bilayer030304 developmental biology0303 health sciencesErgosterolOsmotic concentrationDehydration030306 microbiologyCell MembraneMethyltransferasesCell Biologybiology.organism_classificationSterolMicroscopy ElectronSterols[SDV.MP]Life Sciences [q-bio]/Microbiology and ParasitologyBiochemistrychemistryMutationlipids (amino acids peptides and proteins)Osmotic dehydrationPlasma membrane
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Microbial community structure in soils with decomposing residues from plants with genetic modifications to lignin biosynthesis

2006

Lignin is a major determinant of the decomposition of plant materials in soils. Advances in transgenic technology have led to the possibility of modifying lignin to improve the pulping properties of plant materials for papermaking. Previous studies have shown that lignin modifications also affect the rate of plant material decay in soil. The aim of this work was to investigate short-term changes in soil microbial community structures when tobacco residues with reduced activity of enzymes in the monolignol pathway decompose. The residues from lignin-modified plants all decomposed faster than unmodified plant materials. The relative proportions of some of the structural groups of microbial ph…

PaperNitrogenAlcohol oxidoreductaseLignincomplex mixturesMicrobiologysoilchemistry.chemical_compoundTobaccoBotanyGeneticsgenetic modificationLigninMolecular BiologySoil MicrobiologydecompositionBacteriaPlant StemsbiologyfungiFungifood and beveragesMethyltransferasesPlants Genetically Modifiedbiology.organism_classificationDecompositionCarbonAlcohol Oxidoreductases[SDV.MP]Life Sciences [q-bio]/Microbiology and ParasitologyMicrobial population biologychemistrymicrobial community structureSoil waterMonolignolSoil microbiologyBacteriaFEMS Microbiology Letters
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Effect of pulp and paper mill effluent (BKME) on physiological parameters of roach (Rutilus rutilus) infected by the digenean Rhipidocotyle fennica.

2002

Physiological parameters were measured after experimental infection of roach (Rutilus rutilus L.) with Rhipidocotyle fennica Gibson, Valtonen et Taskinen, 1992 (Digenea) cercariae. The fish were caught from two lakes: a eutrophic bleached kraft mill effluent (BKME)-contaminated lake and an oligotrophic unpolluted lake. The intensity of infection was followed up to 10 days post infection (p.i.) and physiological parameters indicating non-specific stress responses and the condition of fish were examined simultaneously. The mean abundance, the number of parasites per fish, of R. fennica was significantly higher in the fish from the contaminated water during the first two days p.i., probably re…

PaperVeterinary medicineCyprinidaeIndustrial WasteTransferases (Other Substituted Phosphate Groups)Trematode InfectionsBiologyengineering.materialDigeneaHost-Parasite InteractionsFish DiseasesFish physiologyChloridesparasitic diseasesAnimalsEffluentAlkyl and Aryl Transferasesbusiness.industryEcologyPulp (paper)Paper millbiology.organism_classificationAlkaline PhosphataseRhipidocotyle fennicaHematocritengineeringParasitologyCalciumTrematodaRutilusEutrophicationbusinessWater Pollutants ChemicalFolia parasitologica
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CD15 – A new marker of pathological villous immaturity of the term placenta

2014

Abstract Introduction Idiopathic immaturity is one of the main reasons for latent placental insufficiency and antenatal hypoxia. Postnatal identification of the immature placental phenotype may help early stratification of a heterogeneous population of newborns and individually identify risk of disease in the immediate postnatal life. The aim of the study was to determine the relevant diagnostic markers associated with pathological placental immaturity. Methods 111 tissue samples from normal and pathological term placentas with persisting villous immaturity comprised the comparative immunohistochemical study (CD15, CD34). Positive immunohistochemical reactions were quantitatively assessed i…

Pathologymedicine.medical_specialtyEndotheliumLewis X AntigenAntigens CD34Placental insufficiencyBiologyPregnancyChronic VillitisFetal macrosomiamedicineHumansPathologicalPlacental villous immaturityAsphyxiaObstetrics and GynecologyHypoxia (medical)FucosyltransferasesPlacental Insufficiencymedicine.diseasemedicine.anatomical_structureReproductive MedicineCase-Control Studiesembryonic structuresImmunologyFemalemedicine.symptomBiomarkersDevelopmental BiologyPlacenta
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