Search results for "Transmitter"

showing 10 items of 348 documents

Synthesis and characterization of biotinylated and photoactivatable neuroleptics. Novel bifunctional probes for dopamine receptors

1992

Abstract We have synthesized and characterized a series of novel derivatives of established antagonists of the neurotransmitter dopamine, i.e. butyrophenones, hexahydrocarbolines and phenothiazenes. All derivatives were biotinylated, some of them carried an additional (photoactivatable) azido group. In the case of butyrophenones, the structural modifications were introduced at the aliphatic keto group and/or the heterocyclic ring system, both modifications resulting in significant decreases in binding affinity to dopamine D 2 and dopamine D 1 receptor subtypes. Biotinylation of hexahydrocarbolines significantly increased their binding affinity to D 1 receptors, with the affinity for D 2 rec…

Magnetic Resonance SpectroscopySpectrophotometry InfraredPhotochemistryButyrophenoneStereochemistryBiotinIn Vitro TechniquesLigandsBinding CompetitiveReceptors DopamineStructure-Activity Relationship03 medical and health scienceschemistry.chemical_compound0302 clinical medicineDopaminemedicineAnimalsNeurotransmitterReceptor030304 developmental biologyPharmacology0303 health sciencesDopamine antagonistAffinity LabelsBenzazepineschemistryBiochemistrySpiperoneDopamine receptorBiotinylationCattleButyrophenones030217 neurology & neurosurgeryAntipsychotic Agentsmedicine.drugEuropean Journal of Pharmacology: Molecular Pharmacology
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Early-infantile onset epilepsy and developmental delay caused by bi-allelic GAD1 variants

2020

Mice lacking GAD1 show neonatal mortality, but the human phenotype associated with GAD1 disruption is poorly characterized. Neuray et al. describe six patients with biallelic GAD1 mutations, presenting with early-infantile onset epilepsy, neurodevelopmental delay, muscle weakness and non-CNS manifestations.

Male0301 basic medicineGlutamate decarboxylaseMalalties cerebralsNeurotransmissorsNeurodevelopmental delayEpilepsy0302 clinical medicineMESH: ChildAge of OnsetChildcleft palateGAD1AcademicSubjects/SCI01870Glutamate DecarboxylaseGlutamate receptorMuscle weakness//purl.org/becyt/ford/3.1 [https]NeurotransmittersMESH: InfantHypotoniamuscle weakneCleft palateMESH: EpilepsyChild PreschoolMuscle Hypotonia[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]//purl.org/becyt/ford/3 [https]FemaleBrain diseasesAbnormalitiesmedicine.symptomMultiplemedicine.drugcleft palate; epilepsy; GAD1; muscle weakness; neurodevelopmental delayMESH: Glutamate Decarboxylasemedicine.medical_specialtyMESH: Abnormalities MultipleMESH: MutationMESH: Age of OnsetBiologyInhibitory postsynaptic potentialGAD1 cleft palate epilepsy muscle weakness neurodevelopmental delay.gamma-Aminobutyric acidGAD1neurodevelopmental delay03 medical and health sciencesExcitatory synapseInternal medicinemedicineHumansAbnormalities MultiplePreschoolAllelesMESH: Neurodevelopmental Disordersmuscle weaknessMESH: HumansEpilepsyMESH: Muscle HypotoniaMESH: AllelesMESH: Child PreschoolInfantmedicine.diseaseMESH: MaleEpilèpsiaEditor's Choice030104 developmental biologyEndocrinologyNeurodevelopmental DisordersMutationepilepsyAcademicSubjects/MED00310Neurology (clinical)Cleft palate; Epilepsy; GAD1; Muscle weakness; Neurodevelopmental delay; Abnormalities Multiple; Age of Onset; Alleles; Child; Child Preschool; Epilepsy; Female; Glutamate Decarboxylase; Humans; Infant; Male; Muscle Hypotonia; Mutation; Neurodevelopmental DisordersMESH: Female[SDV.MHEP]Life Sciences [q-bio]/Human health and pathology030217 neurology & neurosurgeryReports
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Characterization of the muscarinic receptor subtype(s) mediating contraction of the guinea-pig lung strip and inhibition of acetylcholine release in …

1997

1 The muscarinic receptor subtypes mediating contraction of the guinea-pig lung strip and inhibition of the release of acetylcholine from cholinergic vagus nerve endings in the guinea-pig trachea in vitro have previously been characterized as M-2-like, i.e. having antagonist affinity profiles that are qualitatively similar but quantitatively dissimilar compared to cardiac M-2 receptors. The present study sought to establish definitely the identity of these receptor subtypes by using the selective muscarinic receptor antagonist, tripitramine. Guinea-pig atria and guinea-pig trachea (postjunctional contractile response) were included for reference.2 It was found that tripitramine antagonized …

MaleAUTORECEPTORSlung strip guinea-pigsubtypes ofatria guinea-pigBenzodiazepinesFUNCTIONAL-CHARACTERIZATIONMuscarinic acetylcholine receptorReceptorLungAIRWAYSeducation.field_of_studyguinea-pigSMOOTH-MUSCLEMuscarinic acetylcholine receptor M3Muscarinic acetylcholine receptor M2METHOCTRAMINE-RELATED TETRAAMINESAtrial FunctionReceptors MuscarinicSchild regressionTracheaDepression ChemicalPapersHEARTFemaleAcetylcholineBINDING-PROPERTIESmedicine.drugMuscle Contractionmedicine.medical_specialtyCardiotonic Agentstrachea guinea-piglung stripPopulationGuinea PigsMuscarinic AntagonistsBiologyTritiummuscarinic receptorRABBITInternal medicinemedicineAnimalsNEUROTRANSMITTER RELEASEHeart AtriaeducationAcetylcholine receptorPharmacologyprejunctional muscarinic autoreceptorMuscle SmoothMyocardial ContractionAcetylcholineElectric StimulationEndocrinologyatriaCELLSBritish journal of pharmacology
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Behavioral Effects of GABAA Receptor Stimulation and GABA-Transporter Inhibition

2000

Abstract The present analysis addressed behavioral changes after treatment with 4.5 mg/kg or 18.5 mg/kg of the GABA-uptake inhibitor tiagabine combined with either the benzodiazepine diazepam (1.5 mg/kg) or the imidazopyridine zolpidem (0.05 mg/kg), the latter two acting differentially on GABA A receptor subtypes. The study included 97 male PVG/OIaHsd rats. A standard open field, an enriched open field, and an elevated plus-maze was used to study rat behavior. Treatment with the low dose of tiagabine alone induced no specific behavioral effects, whereas the high dose had an anxiolytic-like potential. Furthermore, diazepam but not zolpidem displayed anxiolytic-like effects. Combination of ea…

MaleAgonistGABA Plasma Membrane Transport Proteinsmedicine.medical_specialtyZolpidemTiagabinePyridinesmedicine.drug_classmedicine.medical_treatmentClinical BiochemistryNipecotic AcidsOrganic Anion TransportersMotor ActivityPharmacologyToxicologyBiochemistryOpen fieldBehavioral NeuroscienceInternal medicinemedicineAnimalsHypnotics and SedativesDrug InteractionsNeurotransmitter Uptake InhibitorsTiagabineBiological PsychiatryPharmacologyBenzodiazepineBehavior AnimalChemistryGABAA receptorMembrane ProteinsMembrane Transport ProteinsReceptors GABA-ARatsZolpidemEndocrinologyAnticonvulsantDrug Therapy CombinationCarrier ProteinsDiazepammedicine.drugPharmacology Biochemistry and Behavior
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Selective activation of 5-HT(2C) receptors stimulates GABA-ergic function in the rat substantia nigra pars reticulata: a combined in vivo electrophys…

2007

In vivo electrophysiology and microdialysis were used to investigate the physiological role of 5-HT(2C) receptors in the control of substantia nigra pars reticulata (SNr) function. Extracellular single-unit recordings were performed from putative GABA-containing neurons in the SNr of anesthetized rats, and local GABA release was studied by in vivo microdialysis in the SNr of awake freely-moving rats. Systemic administration of the selective 5-HT(2C) receptor agonist (S)-2-(chloro-5-fluoro-indol-1-yl)-1-methylethylamine 1:1 C(4)H(4)O(4) (RO 60-0175) caused a dose-dependent excitation of about 30% of the SNr neurons recorded. However, the remaining neurons were either inhibited or unaffected …

MaleAgonistSerotoninMicrodialysismedicine.drug_classMicrodialysisAction PotentialsBiologyPharmacologyInhibitory postsynaptic potentialSynaptic TransmissionRats Sprague-Dawleychemistry.chemical_compoundReceptor Serotonin 5-HT2CmedicineAnimalsDrug InteractionsNeurotransmittergamma-Aminobutyric Acid5-HT receptorNeuronsDose-Response Relationship DrugGeneral NeuroscienceExcitatory Postsynaptic PotentialsExtracellular FluidNeural InhibitionReceptor antagonistRatsSerotonin Receptor AgonistsUp-RegulationElectrophysiologySubstantia Nigranervous systemchemistrySB-243213Serotonin 5-HT2 Receptor AntagonistsSystemic administrationSerotonin AntagonistsNeuroscienceSerotonin 5-HT2 Receptor Agonists
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Increase by 5-hydroxykynuramine of spontaneous acetylcholine release from myenteric neurons: mediated by serotonin M receptors

1987

The effects of 5-hydroxykynuramine (5-OH-K) and of 3-(2-amino-5-hydroxyphenyl)-propaneamine (AHPP) on spontaneous and electrically evoked release of [3H]acetylcholine were studied in the guinea-pig myenteric plexus longitudinal muscle preparation preincubated with [3H]choline. 5-OH-K caused a concentration-dependent increase in spontaneous [3H]acetylcholine release (EC50 5.3 microM). This effect was diminished in the presence of a desensitizing concentration of 5-hydroxytryptamine (5-HT). AHPP (1-100 microM) did not affect the spontaneous outflow of [3H]acetylcholine. The electrically evoked release of [3H]acetylcholine was significantly reduced in the presence of 100 microM of either 5-OH-…

MaleAgonistSerotoninmedicine.medical_specialtymedicine.drug_classKynuramineMethiothepinGuinea PigsMyenteric PlexusIn Vitro TechniquesBiologychemistry.chemical_compoundInternal medicinemedicineAnimalsCholineReceptorNeurotransmitterMyenteric plexusPharmacologyPropiophenonesPropylaminesAcetylcholineElectric StimulationEndocrinologychemistryReceptors SerotoninMetitepineFemaleSerotoninAcetylcholinemedicine.drugEuropean Journal of Pharmacology
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Neurotransmitters involved in the fast inhibitory junction potentials in mouse distal colon

2003

We investigated, in murine colon circular muscle, the role of adenosine 5′-triphosphate (ATP) and pituitary adenylate cyclase activating peptide (PACAP) as inhibitory neurotransmitters of the fast component of nerve-evoked inhibitory junction potential (fast IJP). Fast IJP was antagonised by apamin or suramin, abolished by desensitisation with the P2Y receptor agonist, adenosine 5′-O-2-thiodiphosphate (ADPβS), unaffected by desensitisation with P2X receptor agonist, α,β-methylene ATP (α,β-meATP), and reduced by PACAP-(6-38), a PACAP receptor antagonist. ATP induced membrane hyperpolarization resistant to tetrodotoxin, Nω-nitro-L-arginine methyl ester (L-NAME) or PACAP-(6-38), but antagonise…

MaleAgonistendocrine systemmedicine.medical_specialtyP2Y receptorColonmedicine.drug_classPurinoceptorNeuromuscular JunctionSuraminTetrodotoxinBiologyApaminSettore BIO/09 - FisiologiaMembrane PotentialsCellular and Molecular NeuroscienceMicechemistry.chemical_compoundAdenosine TriphosphateInternal medicinemedicineAnimalsMurinePharmacologyNeurotransmitter AgentsDose-Response Relationship Drugmusculoskeletal neural and ocular physiologyNeuropeptidesMembrane hyperpolarizationThionucleotidesHyperpolarization (biology)Receptor antagonistAdenosinePeptide FragmentsATPAdenosine DiphosphatePituitary adenylate cyclase-activating peptideNG-Nitroarginine Methyl EsterEndocrinologyApaminchemistryPituitary Adenylate Cyclase-Activating PolypeptideFast inhibitory junction potentialPACAP (pituitary adenylate cyclase activating peptide)hormones hormone substitutes and hormone antagonistsmedicine.drugEuropean Journal of Pharmacology
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Long-term effects on cortical glutamate release induced by prenatal exposure to the cannabinoid receptor agonist (r)-(+)-[2,3-dihydro-5-methyl-3-(4-m…

2003

The aim of the present in vivo microdialysis study was to investigate whether prenatal exposure to the CB1 receptor agonist WIN55,212-2 mesylate (WIN; (R)-()-(2,3- dihydro-5-methyl-3-(4-morpholinyl-methyl)pyrrolo(1,2,3-de)- 1,4-benzoxazin-6-yl)-1-naphthalenylmethanone), at a dose of 0.5 mg/kg (s.c. from the fifth to the 20th day of gestation), that causes neither malformations nor overt signs of toxicity, influences cortical glutamate extracellular levels in adult (90- day old) rats. Dam weight gain, pregnancy length and litter size at birth were not significantly affected by prenatal treatment with WIN. Basal and K-evoked dialysate glutamate levels were lower in the cerebral cortex of adul…

MaleAgonistmedicine.medical_specialtyMicrodialysisTime FactorsCannabinoid receptormedicine.drug_classMicrodialysisMorpholinesGlutamic Acidmaternal marijuana consumptionNaphthalenesBiologyTimechemistry.chemical_compoundGlutamatergicPiperidinesPregnancyInternal medicinebasal and K -evoked glutamate levelsmedicineAnimalsDrug InteractionsWakefulnessNeurotransmitterReceptorSR141716A; basal and K+-evoked glutamate levels; maternal marijuana consumptionCerebral CortexAnalysis of VarianceDose-Response Relationship DrugCannabinoidsGeneral NeuroscienceGlutamate receptorBenzoxazinesRatsEndocrinologyAnimals NewbornchemistryPrenatal Exposure Delayed EffectsSR141716AToxicityPotassiumPyrazolesSR141716A; basal and K -evoked glutamate levels; maternal marijuana consumption.CalciumFemaleRimonabantExtracellular Space
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The effects of several muscarinic antagonists on pre- and postsynaptic receptors in the isolated rabbit heart

1981

In order to reveal possible differences between pre- and postsynaptic muscarine receptors, seven antagonists were tested for their affinities on these receptor sites in the rabbit isolated perfused heart. Methacholine was used as an agonist to inhibit the noradrenaline overflow evoked by electrical stimulation (3 Hz, 3 min) of the sympathetic nerves (presynaptic parameter) and to decrease the systolic tension development of the right atrium (postsynaptic parameter). The affinity of an antagonist was expressed as pA2. A decreasing order of potency was obtained with ipratropium, scopolamine, atropine, trihexyphenidyl, amitriptyline, and gallamine, both for pre- and postsynaptic responses. The…

MaleAgonistmedicine.medical_specialtymedicine.drug_classPopulationStimulationIn Vitro TechniquesNorepinephrinechemistry.chemical_compoundPostsynaptic potentialInternal medicineMuscarinic acetylcholine receptormedicineAnimalsMethacholine CompoundsReceptors CholinergicReceptoreducationPharmacologyeducation.field_of_studyBinding SitesMuscarineDose-Response Relationship DrugGallamine TriethiodideMyocardiumParasympatholyticsGeneral MedicineReceptors MuscarinicReceptors NeurotransmitterEndocrinologychemistryFemaleMethacholineRabbitsmedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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Rewarding effects and reinstatement of MDMA-induced CPP in adolescent mice.

2007

Although the rewarding effects of 3,4-methylenedioxy-metamphetamine (MDMA) have been demonstrated in self-administration and conditioned place preference (CPP) procedures, its addictive potential (ie, the vulnerability to relapse, measured by its ability to induce reinstatement of an extinguished response), remains poorly understood. In this study, the effects of MDMA (5, 10, and 20 mg/kg) on the acquisition, extinction and reinstatement of CPP were evaluated in mice, using two different protocols during acquisition of CPP. In the first experiment, animals were trained using a two-session/day schedule (MDMA and saline for 4 consecutive days), whereas in the second experiment, they were trai…

MaleBiogenic AminesReinforcement ScheduleN-Methyl-34-methylenedioxyamphetamineStriatumPharmacologyExtinction Psychologicalchemistry.chemical_compoundMiceRewardDopaminemental disordersmedicineAnimalsNeurotransmitterPharmacologyBrain ChemistryAnalysis of VarianceBehavior AnimalDose-Response Relationship DrugMDMAExtinction (psychology)Conditioned place preferenceRatsPsychiatry and Mental healthchemistryAnimals NewbornAnesthesiaHallucinogensConditioning OperantSerotoninAnalysis of variancePsychologypsychological phenomena and processesmedicine.drugNeuropsychopharmacology : official publication of the American College of Neuropsychopharmacology
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