Search results for "Traumatic"

showing 10 items of 326 documents

Inhibition of myosin light chain kinase reduces brain edema formation after traumatic brain injury.

2010

The role of the endothelial contractile apparatus in the process of brain edema formation after brain trauma is not characterized. Phosphorylation of myosin light chains by myosin light chain kinases (MLCK) activates endothelial contractile elements and results in a rearrangement of the cytoskeleton. This may enhance post-traumatic blood-brain barrier dysfunction. In order to investigate the role of the MLCK on brain edema formation and blood-brain barrier permeability after brain injury, mice were anesthetized and subjected to a controlled cortical impact (CCI). MLCK expression is significantly up-regulated after CCI with a maximum 12 h post-injury. Specific inhibition of MLCK by ML-7 resu…

MaleMyosin light-chain kinaseMyosin Light ChainsTime FactorsEndotheliumIntracranial PressureTraumatic brain injuryCentral nervous systemBrain Edemamacromolecular substancesBrain damageNaphthalenesBlood–brain barrierBiochemistryNeuroprotectionDrug Administration ScheduleFunctional LateralityStatistics NonparametricCerebral edemaCellular and Molecular NeuroscienceMicemedicineAnimalsEnzyme InhibitorsMyosin-Light-Chain KinaseNeurologic Examinationbusiness.industryAzepinesmedicine.diseaseConstrictionCell biologyMice Inbred C57BLDisease Models Animalmedicine.anatomical_structureGene Expression RegulationBlood-Brain BarrierBrain Injuriesmedicine.symptombusinessNeuroscienceEvans BlueJournal of neurochemistry
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Influence of Age on Brain Edema Formation, Secondary Brain Damage and Inflammatory Response after Brain Trauma in Mice

2012

After traumatic brain injury (TBI) elderly patients suffer from higher mortality rate and worse functional outcome compared to young patients. However, experimental TBI research is primarily performed in young animals. Aim of the present study was to clarify whether age affects functional outcome, neuroinflammation and secondary brain damage after brain trauma in mice. Young (2 months) and old (21 months) male C57Bl6N mice were anesthetized and subjected to a controlled cortical impact injury (CCI) on the right parietal cortex. Animals of both ages were randomly assigned to 15 min, 24 h, and 72 h survival. At the end of the observation periods, contusion volume, brain water content, neurolo…

MalePathologyAgingAnatomy and PhysiologyCritical Care and Emergency MedicineMouseT-LymphocytesInterleukin-1beta610 MedizinNitric Oxide Synthase Type IISystemic inflammationMiceAnesthesiologyCell Movement610 Medical sciencesEdemaImmune PhysiologyEdemaLungNeurosurgical CareMultidisciplinaryHematologic TestsQRAging and ImmunityAnimal ModelsOrgan SizeHead Injurymedicine.anatomical_structureNeurologyNeurointensive CareCytokinesMedicinemedicine.symptomResearch Articlemedicine.medical_specialtyTraumatic brain injuryScienceImmunologyInflammationBrain damageAtrophyModel OrganismsNeurorehabilitation and TraumamedicineAnimalsRNA MessengerBiologyCerebrumNeuroinflammationInflammationLungbusiness.industryInterleukin-6Tumor Necrosis Factor-alphaImmunityWatermedicine.diseaseMice Inbred C57BLGene Expression RegulationCyclooxygenase 2Immune SystemBrain InjuriesClinical ImmunologybusinessPhysiological ProcessesPLoS ONE
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The effect of biological sealants and adhesive treatments on matrix metalloproteinase expression during renal injury healing

2017

Background Renal injuries are relatively common in cases of abdominal trauma. Adhesives and sealants can be used to repair and preserve damaged organs. Using a rat model, this study explores the activity of different matrix metalloproteinases (MMP) during the healing of renal injuries treated by two biological adhesives (TachoSil and GelitaSpon) and a new synthetic elastic cyanoacrylate (Adhflex). Methods Renal traumatic injuries were experimentally induced in 90 male Wistar rats by a Stiefel Biopsy Punch in the anterior aspect of the left kidney. Animals were divided into five groups: 1, sham non-injured (n = 3); 2, non-treated standard punch injury (n = 6); 3, punch injury treated with Ta…

MalePathologyCritical Care and Emergency MedicinePhysiology030232 urology & nephrologylcsh:MedicineMatrix metalloproteinasePathology and Laboratory MedicineKidneyMMP8Biochemistrylaw.invention0302 clinical medicinelawMedicine and Health Scienceslcsh:ScienceImmune ResponseTrauma MedicineKidneyMultidisciplinaryProteasesTachoSilEnzymesmedicine.anatomical_structureTraumatic injuryCyanoacrylate030220 oncology & carcinogenesisPhysical SciencesAnatomyTraumatic InjuryResearch Articlemedicine.medical_specialtyHistologyMaterials ScienceImmunologyFibrin Tissue Adhesive03 medical and health sciencesSigns and SymptomsDiagnostic MedicineAdhesivesTissue RepairmedicineAnimalsRats WistarMaterials by AttributeInflammationWound Healingbusiness.industrylcsh:RBiology and Life SciencesProteinsKidneysRenal Systemmedicine.diseaseMatrix MetalloproteinasesAbdominal traumaEnzymologyMetalloproteaseslcsh:QPhysiological ProcessesbusinessWound healingCollagensPLOS ONE
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Proneurotrophin Binding to P75 Neurotrophin Receptor (P75ntr) Is Essential for Brain Lesion Formation and Functional Impairment after Experimental Tr…

2015

Traumatic brain injury (TBI) initiates an excessive mediator release of e.g. neurotrophins, which promote neuronal survival, differentiation, and modulate synaptic plasticity. Paradoxically, mature forms of neurotrophins promote neuronal survival, whereas unprocessed forms of neurotrophins induce cell death through p75 neurotrophin receptor (p75NTR) signaling. p75NTR is widely expressed during synaptogenesis and is subsequently downregulated in adulthood. Repair mechanisms after acute cerebral insults can reactivate its expression. Therefore, the influence of p75NTR on secondary brain damage was addressed. mRNA levels of p75NTR and its ligands were quantified in brain tissue up to 7 days af…

MaleProgrammed cell deathmedicine.medical_specialtyTraumatic brain injurySynaptogenesisReceptors Nerve Growth FactorBrain damageMiceInternal medicineAnimalsMedicineLow-affinity nerve growth factor receptorRNA MessengerMice KnockoutBehavior AnimalCell Deathbiologybusiness.industrymedicine.diseaseMice Inbred C57BLDisease Models AnimalEndocrinologyBrain InjuriesSynaptic plasticitybiology.proteinFemalesense organsNeurology (clinical)medicine.symptomSignal transductionbusinessNeuroscienceProtein BindingSignal TransductionNeurotrophinJournal of Neurotrauma
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Inhibition of Proteasomal Glucocorticoid Receptor Degradation Restores Dexamethasone-Mediated Stabilization of the Blood–Brain Barrier After Traumati…

2013

To establish the molecular background for glucocorticoid insensitivity, that is, failure to reduce edema formation and to protect blood-brain barrier integrity after acute traumatic brain injury.Controlled animal study.University research laboratory.Male C57Bl/6N mice.Mechanical brain lesion by controlled cortical impact.Our study demonstrates that 1) proteasomal glucocorticoid receptor degradation is established in brain endothelial cells after traumatic brain injury as a form of posttranslational glucocorticoid receptor modification; 2) inhibition of the proteasomal degradation pathway with bortezomib (0.2 mg/kg) in combination with the glucocorticoid dexamethasone (10 mg/kg) by subcutane…

MaleProteasome Endopeptidase ComplexTraumatic brain injuryBlotting WesternBrain EdemaPharmacologyReal-Time Polymerase Chain ReactionCritical Care and Intensive Care MedicineBlood–brain barrierSensitivity and SpecificityDexamethasoneStatistics NonparametricBortezomibMiceRandom AllocationReceptors GlucocorticoidGlucocorticoid receptorReference ValuesmedicineAnimalsRNA MessengerReceptorDexamethasonebusiness.industryBortezomibmedicine.diseaseBoronic AcidsImmunohistochemistryMice Inbred C57BLBlotDisease Models Animalmedicine.anatomical_structureBlood-Brain BarrierBrain InjuriesPyrazinesMultivariate AnalysisBlood Gas AnalysisbusinessGlucocorticoidmedicine.drugCritical Care Medicine
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Cognitive estimation: Performance of patients with focal frontal and posterior lesions

2018

The Cognitive Estimation Test (CET) is a widely used test to investigate estimation abilities requiring complex processes such as reasoning, the development and application of appropriate strategies, response plausibility checking as well as general knowledge and numeracy (e.g., Shallice and Evans, 1978; MacPherson et al., 2014). Thus far, it remains unknown whether the CET is both sensitive and specific to frontal lobe dysfunction. Neuroimaging techniques may not represent a useful methodology for answering this question since the complex processes involved are likely to be associated with a large network of brain regions, some of which are not functionally necessary to successfully carry …

MaleRAPM Raven's Advanced Progressive MatricesNo NumberNeuropsychological TestsAudiologyPrefrontal cortexBrain mappingDevelopmental psychologyCVA cerebrovascular accidentExecutive functionsBehavioral NeurosciencePFC prefrontal cortex0302 clinical medicineBrain Injuries TraumaticImage Processing Computer-AssistedPrefrontal cortexprefrontal cortexBrain Mapping05 social sciencesGNT Graded Naming TestNeuropsychologyCognitionMiddle Agedexecutive functionsExecutive functionsMagnetic Resonance ImagingFrontal Lobefluid IntelligenceFrontal lobeFemaleAnalysis of varianceFluid intelligencePsychologyAdultmedicine.medical_specialtyCognitive NeuroscienceExperimental and Cognitive PsychologyCognitive estimation testCognitive Estimation TestArticle050105 experimental psychology03 medical and health sciencesNeuroimagingmedicineHumans0501 psychology and cognitive sciencesAgedAnalysis of VarianceHC healthy comparisonsIQ Intelligence QuotientCognition DisordersNART National Adult Reading Test030217 neurology & neurosurgeryLF left frontalNeuropsychologia
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Low bone mineral density in HIV-positive young Italians and migrants.

2020

Background Human immunodeficiency virus (HIV) infected individuals may have osteoporosis. We aimed to evaluate the bone mineral density (BMD) in naïve antiretroviral (ARV) treated HIV positive patients comparing native Italian group (ItG) to a Migrants group (MiG) upon arrival in Italy. Methods We conducted a cross-sectional study on 83 HIV patients less than 50 years old. We used the dual-energy X-ray absorptiometry (DXA) within six months from the HIV diagnosis. Participants were categorized as having low BMD if the femoral neck or total lumbar spine Z-score was– 2 or less. Results MiG showed low BMD more often than ItG (37.5% vs.13.6%), especially for the female gender (16.7% vs. 0.0%). …

MaleRNA virusesEuropean PeopleCritical Care and Emergency MedicineBone densityEpidemiologyOsteoporosisHIV InfectionsLogistic regressionPathology and Laboratory Medicine0302 clinical medicineAbsorptiometry PhotonImmunodeficiency VirusesBone DensityMedicine and Health SciencesEthnicities030212 general & internal medicineVitamin DConnective Tissue DiseasesMusculoskeletal SystemTrauma MedicineBone mineralTransients and MigrantsMultidisciplinaryQRvirus diseasesHuman immunodeficiency virus (HIV)HIV diagnosis and managementosteoporosis.Middle AgedItalian Peoplemedicine.anatomical_structureAnti-Retroviral AgentsItalyMedical MicrobiologyConnective TissueBone FractureViral PathogensVirusesMedicineInfectious diseasesFemalePathogensAnatomyTraumatic InjuryResearch ArticleAdultMedical conditionsmedicine.medical_specialtyScience030209 endocrinology & metabolismViral diseasesMicrobiology03 medical and health sciencesLumbarSex FactorsRheumatologyInternal medicineRetrovirusesmedicineVitamin D and neurologyHumansBoneMicrobial PathogensSkeletonFemoral neckbusiness.industryLentivirusOrganismsBiology and Life SciencesHIVBone fracturemedicine.diseaseDiagnostic medicineCD4 Lymphocyte CountCross-Sectional StudiesLogistic ModelsBiological TissueMedical Risk FactorsPeople and PlacesOsteoporosisPopulation GroupingsbusinessPloS one
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Neuroprotection by erythropoietin administration after experimental traumatic brain injury.

2007

A large body of evidence indicates that the hormone erythropoietin (EPO) exerts beneficial effects in the central nervous system (CNS). To date, EPO's effect has been assessed in several experimental models of brain and spinal cord injury. This study was conducted to validate whether treatment with recombinant human EPO (rHuEPO) would limit the extent of injury following experimental TBI. Experimental TBI was induced in rats by a cryogenic injury model. rHuEPO or placebo was injected intraperitoneally immediately after the injury and then every 8 h until 2 or 14 days. Forty-eight hours after injury brain water content, an indicator of brain edema, was measured with the wet-dry method and bl…

MaleTime FactorsBrain EdemaFunctional LateralityRats Sprague-Dawleychemistry.chemical_compoundTraumatic brain injuryMedicineAnalysis of Variance Animals Blood-Brain Barrier; drug effects Brain Edema; drug therapy/etiology Brain Infarction; drug therapy/etiology Brain Injuries; complications/drug therapy Disease Models; Animal Erythropoietin; administration /&/ dosage Evans Blue; diagnostic use Functional Laterality Humans Male Neurologic Examination Neuroprotective Agents; administration /&/ dosage Rats Rats; Sprague-Dawley Reaction Time; drug effects Recombinant Proteins Time Factorsadministration /&/ dosageSpinal cord injuryEvans BlueNeurologic ExaminationGeneral Neuroscienceexperimental models of brain and spinal cord injuryExtravasationNeuroprotectionRecombinant Proteinsmedicine.anatomical_structureNeuroprotective AgentsBlood-Brain BarrierAnesthesiadiagnostic usemedicine.drugEvans BlueBrain InfarctionTraumatic brain injuryCentral nervous systemrecombinant human EPO (rHuEPO)PlaceboNeuroprotectionReaction TimeAnimalsHumansMolecular BiologyErythropoietinAnalysis of VarianceNeuroscience (all)business.industryAnimaldrug therapy/etiologymedicine.diseaseRatsDisease Models AnimalchemistryErythropoietindrug effectsBrain InjuriesDisease Modelsrecombinant human EPO (rHuEPO); experimental models of brain and spinal cord injury; NeuroprotectionNeurology (clinical)Sprague-Dawleybusinesscomplications/drug therapyDevelopmental BiologyBrain research
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An early bolus of hypertonic saline hydroxyethyl starch improves long-term outcome after global cerebral ischemia.

2006

Objective: The beneficial effect of hypertonic saline solutions in the emergency treatment of shock and traumatic brain injury is well described. The present study determines effects of a single bolus of hypertonic saline on long-term survival, neurologic function, and neuronal survival 10 days after global cerebral ischemia. In addition, we evaluated the therapeutic window for hypertonic saline treatment (early vs. delayed application). Design: Laboratory experiment. Setting: University laboratory. Subjects: Male Wistar rats weighing 240‐330 g. Interventions: Rats were submitted to temporal global cerebral ischemia using temporary bilateral carotid occlusion combined with hypobaric hypoten…

MaleTime FactorsTraumatic brain injurymedicine.medical_treatmentIschemiaPlasma SubstitutesBlood PressureHydroxyethyl starchCritical Care and Intensive Care MedicineWeight GainBrain IschemiaHydroxyethyl Starch DerivativesBolus (medicine)Intensive caremedicineAnimalsRats WistarSalineNeuronsSaline Solution HypertonicCell Deathbusiness.industrySodiumBrainmedicine.diseaseHypertonic salineRatsCerebral blood flowHematocritAnesthesiaReperfusionPotassiumbusinessmedicine.drugCritical care medicine
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Cranioplasty with autologous bone flaps cryopreserved with Dimethylsulphoxide : does tissue processing matter

2021

Este artículo se encuentra disponible en la siguiente URL: https://www.sciencedirect.com/science/article/abs/pii/S1878875021001625?via%3Dihub En este artículo de investigación también participan: Dolores Ocete, Lucas Aranda, Ana Melero, Antonio J. Guillot, Nuria Yagüe y Carlos Botella. Este es el pre-print del siguiente artículo: Mirabet, V., García, D., Roca, A., Quiroz, A. R., Antón, J., Rodríguez-Cadarso, M., Ocete, D., Aranda, L., Melero, A., Guillot, A. J., Yagüe, N., Guillén, I. & Botella, C. (2021). Cranioplasty with autologous bone flaps cryopreserved with Dimethylsulphoxide: does tissue processing matter. World Neurosurgery, vol. 149 (may.), pp. e582?e591, que se ha publicado de fo…

MaleTime Factorsmedicine.medical_treatmentBrain EdemaSurgical Flaps0302 clinical medicineCryoprotective AgentsPostoperative ComplicationsHuesos - Crioconservación.Brain Injuries TraumaticAutograftsAutologous bone flapMiddle AgedCranioplastyResorptionAnti-Bacterial AgentsStrokeCryopreservacion of organs tissues etc.030220 oncology & carcinogenesisTissue bankVancomycinDecompressive craniectomyFemalemedicine.drugCrioconservación de órganos tejidos etc.Adultmedicine.medical_specialtyDecompressive CraniectomyAdolescentDecompressive craniectomyCráneo - Cirugía.CranioplastySkull - Surgery.03 medical and health sciencesYoung AdultmedicineHumansSurgical Wound InfectionDimethyl SulfoxideBones - Cryopreservacion.Bone ResorptionCryopreservationbusiness.industryBone storageSkullPostoperative complicationBone processingPlastic Surgery Proceduresmedicine.diseaseSurgeryHydrocephalusSurgeryNeurology (clinical)businessComplication030217 neurology & neurosurgery
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