Search results for "Tritium"

showing 10 items of 165 documents

Synthesis of tritium labeled (�)-1-[2(triphenylmethoxy)ethyl]-3-piperidinecarboxylic acid: a possible compound to determine the efficacy of potential…

2000

(±)-1-[2-(Triphenyl[ 3 H]methoxy)ethyl]-3-piperidinecarboxylic acid ([ 3 H]SNAP-5114) with a specific activity of 40 Ci/mmol was prepared in a two step synthesis starting from ethyl (2-(4-hydroxyphenyl)bis(4-methoxyphenyl)methoxy)-ethyl)piperidine-3-carboxylate and [ 3 H]methyliodide with subsequent hydrolysis of the resulting ester with lithium hydroxide to yield the desired [ 3 H]SNAP-5114.

LigandStereochemistryOrganic ChemistryEtherBiochemistryChemical synthesisMedicinal chemistryLithium hydroxideAnalytical Chemistrychemistry.chemical_compoundHydrolysischemistryYield (chemistry)Drug DiscoveryRadiology Nuclear Medicine and imagingTritiumSpecific activitySpectroscopyJournal of Labelled Compounds and Radiopharmaceuticals
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COMPLEMENT-DEPENDENT B-CELL ACTIVATION BY COBRA VENOM FACTOR AND OTHER MITOGENS?

1974

It has been proposed that two distinct signals are required for the triggering of the precursors of antibody-forming bone marrow-derived cells (B cells): (a) the binding of antigen or of a mitogen to the corresponding receptor sites on B-cell membranes and (b) the interaction of activated C3 with the C3 receptor of B lymphocytes. There is growing evidence that B-cell mitogens and T (thymus-derived cell)-independent antigens are capable of activating the alternate pathway of the complement system (bypass). Therefore, the effect of another potent bypass inducer was investigated with regard to B-cell activation and the role of C3. Purified, pyrogen-free cobra venom factor was mitogenic for bot…

LipopolysaccharidesErythrocytesT-LymphocytesImmunologyHemolytic Plaque TechniqueMice Inbred StrainsLymphocyte ActivationTritiumArticleMiceAntigenPolysaccharidesLectinsConcanavalin AEscherichia coliAnimalsImmunology and AllergyCells CulturedImmune adherence reactionAntigens BacterialB-LymphocytesSheepbiologyVenomsPokeweed mitogenSnakesComplement System ProteinsMolecular biologyImmune Adherence ReactionComplement systemKineticsCell cultureConcanavalin AAntibody Formationbiology.proteinMitogensAntibodyFetal bovine serumThymidineJournal of Experimental Medicine
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Characterization of the muscarinic receptor subtype(s) mediating contraction of the guinea-pig lung strip and inhibition of acetylcholine release in …

1997

1 The muscarinic receptor subtypes mediating contraction of the guinea-pig lung strip and inhibition of the release of acetylcholine from cholinergic vagus nerve endings in the guinea-pig trachea in vitro have previously been characterized as M-2-like, i.e. having antagonist affinity profiles that are qualitatively similar but quantitatively dissimilar compared to cardiac M-2 receptors. The present study sought to establish definitely the identity of these receptor subtypes by using the selective muscarinic receptor antagonist, tripitramine. Guinea-pig atria and guinea-pig trachea (postjunctional contractile response) were included for reference.2 It was found that tripitramine antagonized …

MaleAUTORECEPTORSlung strip guinea-pigsubtypes ofatria guinea-pigBenzodiazepinesFUNCTIONAL-CHARACTERIZATIONMuscarinic acetylcholine receptorReceptorLungAIRWAYSeducation.field_of_studyguinea-pigSMOOTH-MUSCLEMuscarinic acetylcholine receptor M3Muscarinic acetylcholine receptor M2METHOCTRAMINE-RELATED TETRAAMINESAtrial FunctionReceptors MuscarinicSchild regressionTracheaDepression ChemicalPapersHEARTFemaleAcetylcholineBINDING-PROPERTIESmedicine.drugMuscle Contractionmedicine.medical_specialtyCardiotonic Agentstrachea guinea-piglung stripPopulationGuinea PigsMuscarinic AntagonistsBiologyTritiummuscarinic receptorRABBITInternal medicinemedicineAnimalsNEUROTRANSMITTER RELEASEHeart AtriaeducationAcetylcholine receptorPharmacologyprejunctional muscarinic autoreceptorMuscle SmoothMyocardial ContractionAcetylcholineElectric StimulationEndocrinologyatriaCELLSBritish journal of pharmacology
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Characterization of γ-aminobutyrate type A receptors with atypical coupling between agonist and convulsant binding sites in discrete brain regions

2001

Abstract γ-Aminobutyric acid type A (GABA A ) receptor ionophore ligand t -[ 35 S]butylbicyclophosphorothionate ([ 35 S]TBPS) was used in an autoradiographic assay on brain cryostat sections to visualize and characterize atypical GABA-insensitive [ 35 S]TBPS binding previously described in certain recombinant GABA A receptors and the cerebellar granule cell layer. Picrotoxinin-sensitive but 1-mM GABA-insensitive [ 35 S]TBPS binding was present in the rat cerebellar granule cell layer, many thalamic nuclei, subiculum and the internal rim of the cerebral cortex, amounting in these regions up to 6% of the basal binding determined in the absence of exogenous GABA. Similar binding properties wer…

MaleAgonistAzidesmedicine.medical_specialtyCerebellumSesterterpenesmedicine.drug_classLoreclezoleConvulsantsBiologySulfur RadioisotopesTritiumBinding CompetitiveBenzodiazepinesRadioligand AssayCellular and Molecular Neurosciencechemistry.chemical_compoundThalamusCerebellumInternal medicinemedicineAnimalsHumansPicrotoxinRats WistarBinding siteReceptorGABA AgonistsMolecular Biologygamma-Aminobutyric AcidMuscimolGABAA receptorAffinity LabelsBridged Bicyclo Compounds HeterocyclicReceptors GABA-AGranule cellRatsEndocrinologymedicine.anatomical_structurenervous systemMuscimolchemistryBiophysicsChickensmedicine.drugMolecular Brain Research
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STIMULATION OF ?1-ADRENOCEPTORS ENHANCES ELECTRICALLY EVOKED [3H]-ACETYLCHOLINE RELEASE FROM RAT PHRENIC NERVE

1990

1. The effects of isoprenaline, noradrenaline and fenoterol on the electrically evoked release of [3H]-acetylcholine from the rat phrenic nerve were investigated. 2. Isoprenaline (0.1 mumol/L) and noradrenaline (1 mumol/L) enhanced evoked [3H]-acetylcholine release by about 90%, an effect which was abolished by CGP 20712A (0.1 mumol/L), a specific antagonist at beta 1-adrenoceptors. Noradrenaline still enhanced [3H]-acetylcholine release in the presence of phentolamine (1 mumol/L). 3. The enhancing effect of both isoprenaline and noradrenaline decreased at prolonged exposure times (24-32 min). A pre-exposure of the tissue to a low concentration (0.01 mumol/L) of isoprenaline prevented the e…

MaleAgonistmedicine.medical_specialtyPhysiologymedicine.drug_classAdrenergic beta-AntagonistsNeuromuscular transmissionStimulationPropranololIn Vitro TechniquesTritiumNorepinephrinePhentolaminePhysiology (medical)IsoprenalineInternal medicineReceptors Adrenergic betamedicineAnimalsPhentolamineFenoterolFenoterolPharmacologyChemistryImidazolesIsoproterenolRats Inbred StrainsPropranololAcetylcholineElectric StimulationRatsPhrenic NerveEndocrinologyFemaleAcetylcholinemedicine.drugClinical and Experimental Pharmacology and Physiology
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Cyclosporine A Impairs the Macrophage Reverse Cholesterol Transport in Mice by Reducing Sterol Fecal Excretion

2012

Despite the efficacy in reducing acute rejection events in organ transplanted subjects, long term therapy with cyclosporine A is associated with increased atherosclerotic cardiovascular morbidity. We studied whether this drug affects the antiatherogenic process of the reverse cholesterol transport from macrophages in vivo. Cyclosporine A 50 mg/kg/d was administered to C57BL/6 mice by subcutaneous injection for 14 days. Macrophage reverse cholesterol transport was assessed by following [(3)H]-cholesterol mobilization from pre-labeled intraperitoneally injected macrophages, expressing or not apolipoprotein E, to plasma, liver and feces. The pharmacological treatment significantly reduced the …

MaleApolipoprotein EMouselcsh:MedicineCardiovascularBiochemistryFecesMiceSubcutaneous injectionchemistry.chemical_compoundIntestinal Mucosalcsh:ScienceCholesterol 7-alpha-HydroxylaseMultidisciplinaryReverse cholesterol transportAnimal ModelsLipidsIntestinesCholesterolLiverCyclosporineMedicinelipids (amino acids peptides and proteins)Research Articlemedicine.medical_specialtyLipoproteinsTritiumCholesterol 7 alpha-hydroxylaseCardiovascular PharmacologyExcretionApolipoproteins EModel OrganismsIn vivoInternal medicinemedicineAnimalsBiologyCholesterollcsh:RProteinsBiological TransportLipid MetabolismAtherosclerosisSitosterolsSterolMice Inbred C57BLKineticsEndocrinologyGene Expression RegulationchemistryMacrophages Peritoneallcsh:QATP-Binding Cassette TransportersPLoS ONE
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Activation of L-arginine transport by protein kinase C in rabbit, rat and mouse alveolar macrophages

1998

1 The role of protein kinase C in controlling L-arginine transport in alveolar macrophages was investigated. 2 L-[3H]Arginine uptake in rabbit alveolar macrophages declined by 80 % after 20 h in culture. 4β-Phorbol 12-myristate 13-acetate (PMA), but not 4α-phorbol 12-myristate 13-acetate (α-PMA), present during 20 h culture, enhanced L-[3H]arginine uptake more than 10-fold. Staurosporine and chelerythrine opposed this effect. 3 L-[3H]Arginine uptake was saturable and blockable by L-lysine. After PMA treatment Vmax was increased more than 5-fold and Km was reduced from 0.65 to 0.32 mM. 4 Time course experiments showed that PMA increased L-[3H]arginine uptake almost maximally within 2 h. This…

MaleArgininePhysiologyMice Inbred StrainsStimulationCycloheximideArginineTritiumL-arginine transportRats Sprague-DawleyMicechemistry.chemical_compoundSpecies SpecificityLeucineMacrophages AlveolarmedicineAnimalsStaurosporineRNA MessengerEnzyme InhibitorsProtein Kinase CProtein kinase CbiologySodiumMembrane ProteinsBiological TransportRabbit ratOriginal Articlesbiology.organism_classificationMolecular biologyRatsKineticsChelerythrinechemistryEthylmaleimideCarcinogensAmino Acid Transport Systems BasicTetradecanoylphorbol AcetateFemaleRabbitsCarrier Proteinsmedicine.drugThe Journal of Physiology
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Altered receptor subtypes in the forebrain of GABAA receptor δ subunit-deficient mice: recruitment of γ2 subunits

2002

A GABA(A) receptor delta subunit-deficient mouse line was created by homologous recombination in embryonic stem cells to investigate the role of the subunit in the brain GABA(A) receptors. High-affinity [(3)H]muscimol binding to GABA sites as studied by ligand autoradiography was reduced in various brain regions of delta(-/-) animals. [(3)H]Ro 15-4513 binding to benzodiazepine sites was increased in delta(-/-) animals, partly due to an increment of diazepam-insensitive receptors, indicating an augmented forebrain assembly of gamma 2 subunits with alpha 4 subunits. In the western blots of forebrain membranes of delta(-/-) animals, the level of gamma 2 subunit was increased and that of alpha …

MaleAzidesProtein subunitBiologyTritiumSynaptic TransmissionIon ChannelsGABAA-rho receptorInterleukin 10 receptor alpha subunitBenzodiazepinesMiceRadioligand Assaychemistry.chemical_compoundAnimalsReceptorGABA Agonistsgamma-Aminobutyric AcidMice KnockoutNeuronsBinding SitesMuscimolGABAA receptorGeneral NeuroscienceBrainAffinity LabelsNeural InhibitionReceptors GABA-AMolecular biologynervous systemMuscimolchemistryMutationForebrainFemaleCys-loop receptorsNeuroscience
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Beta-adrenoceptor density and subtype distribution in cerebellum and hippocampus from patients with Alzheimer's disease

1993

beta-Adrenoceptor density and beta 1- and beta 2-subtype distribution were examined in hippocampi and cerebella from patients with Alzheimer's disease (AD/SDAT). Tissues from age-, sex and post-mortem delay matched non-demented patients served as controls. The total beta-adrenoceptor density as evaluated in saturation experiments with the hydrophilic radioligand [3H]CGP 12177 was higher in hippocampal (36-39 fmol/mg protein) than cerebellar tissues (20-21 fmol/mg), however, no differences were found in either brain region between AD/SDAT patients and controls. Subtype distribution using the highly selective beta 1-adrenoceptor antagonist CGP 20712A revealed a slightly higher proportion of b…

MaleCerebellummedicine.medical_specialtyAdrenergic beta-AntagonistsHippocampal formationTritiumBinding CompetitiveHippocampusPropanolaminesAdenylyl cyclaseBasal (phylogenetics)chemistry.chemical_compoundDegenerative diseaseAlzheimer DiseaseCerebellumInternal medicineReceptors Adrenergic betamedicineHumansHippocampus (mythology)AgedAged 80 and overPharmacologybusiness.industryImidazolesAntagonistGeneral MedicineMiddle Agedmedicine.diseaseKineticsmedicine.anatomical_structureEndocrinologychemistryDementiaFemaleAlzheimer's diseasebusinessNaunyn-Schmiedeberg's Archives of Pharmacology
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alpha-Bungarotoxin, kappa-bungarotoxin, alpha-cobratoxin and erabutoxin-b do not affect [3H]acetylcholine release from the rat isolated left hemidiap…

1995

Endplate preparations of the rat left hemidiaphragm were incubated with [3H]choline to label neuronal transmitter stores. Nerve evoked release of newly-synthesized [3H]acetylcholine was measured in the absence of cholinesterase inhibitors to investigate whether snake venom neurotoxins by blocking presynaptic nicotinic autoreceptors affect evoked transmitter release. Contractions of the indirectly stimulated hemidiaphragm were recorded to characterize the blocking effect of alpha-neurotoxins at the post-synaptic nicotinic receptors. Neither the long chain neurotoxins alpha-cobratoxin (1 microgram ml-1) and alpha-bungarotoxin (5 microgram ml-1) nor the short chain neurotoxin erabutoxin-b (0.1…

MaleDiaphragmNeurotoxinsPharmacologyReceptors NicotinicTritiumSynaptic TransmissionPostsynaptic potentialmedicineAnimalsCobra Neurotoxin ProteinsChromatography High Pressure LiquidCholinesterasePharmacologyErabutoxinsbiologyChemistryMuscle SmoothGeneral MedicineBungarotoxinmusculoskeletal systemBungarotoxinsAcetylcholineRatsPhrenic NerveNicotinic agonistSnake venomIsotope Labelingbiology.proteinAutoreceptorFemaleCobratoxinNeuroscienceAcetylcholinemedicine.drugMuscle ContractionSnake VenomsNaunyn-Schmiedeberg's archives of pharmacology
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