Search results for "Tumor necrosis factor-alpha"

showing 10 items of 504 documents

TNFalpha, IFNgamma and IL-10 gene polymorphisms in a sample of Sicilian patients with coeliac disease.

2005

Coeliac disease is associated with DQ2 and DQ8 alleles, but other genes also confer an additional genetic risk.Defining whether the genetic profiles of interleukin-10, tumour necrosis factor alpha and interferon gamma are associated with an increased coeliac disease risk.The functionally gene polymorphisms of tumour necrosis factor alpha (-308G/A), interferon gamma (+874T/A) and interleukin-10 (-1082G/A) were typed using sequence specific primer-polymerase chain reaction in 110 Sicilian coeliac disease patients and in 220 Sicilian healthy controls.No differences in genotype frequencies of interleukin-10 polymorphisms were found between coeliac disease patients and healthy controls. A signif…

AdultMaleNecrosisAdolescentGenotypeCoeliac diseaseInterferon-gammaGene FrequencymedicineHumansGenetic Predisposition to DiseaseAlleleChildGeneSicilyPolymorphism GeneticHepatologybusiness.industryTumor Necrosis Factor-alphaGastroenterologynutritional and metabolic diseasesInfantGluten intoleranceMiddle Agedmedicine.diseaseGenotype frequencyInterleukin-10Interleukin 10Celiac DiseaseCase-Control StudiesChild PreschoolImmunologyTumor necrosis factor alphaFemalemedicine.symptombusinessDigestive and liver disease : official journal of the Italian Society of Gastroenterology and the Italian Association for the Study of the Liver
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Viral and host factors in the prediction of response to interferon-alpha therapy in chronic hepatitis C after long-term follow-up.

1998

Acute infection with hepatitis C virus (HCV) develops into a chronic hepatitis in about 50-70% of patients. Treatment of these patients with interferon-alpha (IFN-alpha) results in a sustained long-term response in only 15-20% but causes numerous unwanted side-effects in a higher percentage of patients. The aim of our study was to define host or viral parameters that would allow identification of responders and non-responders to IFN-alpha prior to the onset of treatment. We studied a group of 87 patients suffering from chronic hepatitis C who were treated with IFN-alpha. After long-term follow-up, 18 patients (21%) showed a sustained response to IFN-alpha therapy (normalization of serum tra…

AdultMaleNecrosisGenotypeHepatitis C virusAlpha interferonInflammationHepacivirusmedicine.disease_causeAntiviral AgentsFibrosisVirologyGenotypemedicineHumansViremiaHepatologymedicine.diagnostic_testbusiness.industryTumor Necrosis Factor-alphaInterferon-alphaHLA-DR AntigensHepatitis C ChronicMiddle AgedViral Loadmedicine.diseaseInfectious DiseasesTreatment OutcomeLiver biopsyImmunologyRNA ViralFemalemedicine.symptombusinessViral hepatitisFollow-Up StudiesJournal of viral hepatitis
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Oxidative stress and inflammation in long-term renal transplanted hypertensives.

2006

INTRODUCTION Several studies have shown that chronic renal failure (CRF) is characterized by "accelerated atherosclerosis". More recent studies emphasize that inflammation and oxidative stress play a central role in atherosclerosis, and it is well-established that C-reactive protein (CRP) is a cardiovascular risk marker in the general population, in end-stage renal disease (ESRD) patients and in allograft recipients. METHODS We measured the serum concentration of high sensitivity CRP, TNFalpha, 8-iso-prostaglandin F2alpha (8-iso-PGF2alpha, an in vivo oxidative stress marker) in 15 CRF patients and in 15 transplant recipients. Exclusion criteria were age 65 years, smoking, diabetes mellitus …

AdultMaleNephrologymedicine.medical_specialtyTime FactorsPopulationRenal functionDinoprostchemistry.chemical_compoundInternal medicineDiabetes mellitusmedicineHumanseducationeducation.field_of_studyCreatinineTumor Necrosis Factor-alphabusiness.industryGeneral MedicineMiddle Agedmedicine.diseaseKidney Transplantationoxidative stress inflammation hypertension renal transplantationTransplantationOxidative StressC-Reactive ProteinEndocrinologyBlood pressurechemistryNephrologyCase-Control StudiesHypertensionKidney Failure ChronicInflammation MediatorsbusinessKidney disease
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Identification of epithelial gaps in human small and large intestine by confocal endomicroscopy.

2007

Background & Aims: Confocal endomicroscopy is an emerging technology that poses the endoscopist with challenges for identifying epithelial structures in the human intestine. We have shown previously that the murine intestinal epithelium is punctuated by gaps caused by cell shedding. The goals of this study were to determine if confocal endomicroscopy could resolve the presence of human epithelial gaps and whether a proinflammatory cytokine could increase cell shedding. Methods: Intestinal mucosa was imaged after staining with acriflavine. Confocal endomicroscopy of 17 patients yielded 6277 images from the human terminal ileum and rectum. Results were validated by parallel studies of anesthe…

AdultMalePathologymedicine.medical_specialtyAdolescentConfocalBiologylaw.inventionMiceIntestinal mucosaConfocal microscopylawMicroscopyIntestine SmallEndomicroscopymedicineAnimalsHumansIntestine LargeBarrier functionAgedMicroscopy ConfocalHepatologyTumor Necrosis Factor-alphaGastroenterologyEpithelial CellsColonoscopyMiddle AgedIntestinal epitheliumEpitheliummedicine.anatomical_structureFemaleGastroenterology
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Increased expression of interleukin-32 in the inflamed ileum of ankylosing spondylitis patients

2012

Objective. To study the mRNA expression and protein tissue distribution of IL-32 in ileal biopsy specimens from patients with AS. Methods. Quantitative gene expression analysis, by real-time PCR, of IL-32, IL-1b, IL-10, TNF-a and IFN-g was performed on ileal biopsies of 15 AS and 15 Crohn’s disease (CD) patients and 10 healthy subjects (HSs). IL-32 tissue distribution was evaluated by immunohistochemistry. The effect of IL-32 on the production of IL-10 by intestinal epithelial cell lines was also evaluated. Results. In the ileal specimens of patients with AS and intestinal chronic inflammation, significant up-regulation of IL-32 at both the mRNA and protein levels was found as compared with…

AdultMalePathologymedicine.medical_specialtyAdolescentmedicine.medical_treatmentInterleukin-1betaInflammationInterferon-gammaYoung AdultCrohn DiseaseRheumatologyIleumBiopsyintestinal inflammationmedicineHumansSpondylitis AnkylosingPharmacology (medical)IleitisRNA MessengerCrohn's diseasemedicine.diagnostic_testTumor Necrosis Factor-alphabusiness.industryInterleukinsMacrophagesIL-32 ankylosing spondylitis IL-10 intestinal inflammationInterleukinEpithelial CellsIleitisMiddle AgedHCT116 Cellsmedicine.diseaseImmunity InnateInterleukin-10Settore MED/16 - ReumatologiaInterleukin 10Interleukin 32ankylosing spondylitiCytokineCase-Control StudiesImmunologyIL-32IL-10Femalemedicine.symptombusiness
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IL-34 is overexpressed in the inflamed salivary glands of patients with Sjogren's syndrome and is associated with the local expansion of pro-inflamma…

2013

Objectives To investigate the expression of IL-34 in labial salivary glands (LSGs) of patients with primary SS (p-SS) and its role in inducing a pro-inflammatory monocyte phenotype. Methods LSG biopsies were obtained from 20 patients with p-SS and 10 patients with non-Sjogren's sicca syndrome (n-SS). The expression of IL-34, IL-1β, TNF-α, IL-17 and IL-23 was assessed by real-time PCR. IL-34 expression was also investigated in LSGs by immunohistochemistry. The frequencies of subpopulations of CD14(+) monocytes were evaluated by flow cytometry among isolated mononuclear cells from peripheral blood and salivary glands from both patients and controls. The role of recombinant IL-34 on isolated p…

AdultMalePathologymedicine.medical_specialtyCD14Interleukin-1betaLipopolysaccharide ReceptorsInflammationCD16Interleukin-23Peripheral blood mononuclear cellMonocytesSalivary GlandsRheumatologySicca syndromemedicineHumansPharmacology (medical)interleukin-34 Sjögren’s syndrome monocytesAgedInflammationSalivary glandTumor Necrosis Factor-alphabusiness.industryInterleukinsMonocyteInterleukin-17Receptors IgGMiddle AgedFlow CytometrySettore MED/16 - ReumatologiaSjogren's Syndromemedicine.anatomical_structureImmunologyInterleukin 34Femalemedicine.symptombusinessRheumatology
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Immunohistological and immunoelectron microscopic identification of TNF alpha in normal human and murine epidermis.

1992

The presence, distribution and cellular localization of tumour necrosis factor-alpha (TNF alpha) were investigated in normal human and murine epidermis using immunohistological and immunoelectron microscopic methods with monoclonal and polyclonal antibodies. The immunostaining revealed an intercellular plasma membrane and cytoplasmic labelling of the epidermal keratinocytes, but no labelling of Langerhans cells, melanocytes and Merkel cells. Large amounts of TNF alpha were regularly found in the sebaceous glands. These findings demonstrate that epidermal keratinocytes and especially sebocytes produce and release TNF alpha and that this keratinocyte-derived cytokine may be important for the …

AdultMalePathologymedicine.medical_specialtyImmunoelectron microscopymedicine.medical_treatmentDermatologyMicemedicineAnimalsHumansMicroscopy ImmunoelectronCellular localizationMice Inbred BALB Cintegumentary systemEpidermis (botany)biologyTumor Necrosis Factor-alphaGeneral MedicineMiddle AgedMolecular biologyImmunohistochemistryCytokinemedicine.anatomical_structurePolyclonal antibodiesbiology.proteinImmunohistochemistryFemaleEpidermisMerkel cellImmunostainingArchives of dermatological research
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Functional profile of cytomegalovirus (CMV)-specific CD8+ T cells and kinetics of NKG2C+ NK Cells associated with the resolution of CMV DNAemia in al…

2011

Immune mechanisms involved in control of cytomegalovirus (CMV) infection in the allogeneic stem cell transplantation setting have not been fully disclosed. CMV pp65 and IE-1-specific CD8(+) T cells expressing IFN-γ, TNF-α, and CD107a, alone or in combination, and NKG2C(+) NK cells were prospectively enumerated during 13 episodes of CMV DNAemia. The expansion of monofunctional and polyfunctional CD8(+) T cells was associated with CMV DNAemia clearance. The size and functional diversity of the expanding CD8(+) T-cell population was greater in self-resolved episodes than in episodes treated with antivirals. These differences were related to the magnitude of expansion of cognate antigen IFN-γ C…

AdultMalePopulationCongenital cytomegalovirus infectionCytomegalovirusCD8-Positive T-LymphocytesBiologyViral Matrix ProteinsInterferon-gammaYoung AdultInterleukin 21AntigenLysosomal-Associated Membrane Protein 1VirologymedicineHumansTransplantation HomologousCytotoxic T celleducationeducation.field_of_studyTumor Necrosis Factor-alphaReceptors IgGvirus diseasesMiddle AgedPhosphoproteinsmedicine.diseaseVirologyCD56 AntigenKiller Cells NaturalTransplantationInfectious DiseasesCytomegalovirus InfectionsDNA ViralImmunologyFemaleStem cellNK Cell Lectin-Like Receptor Subfamily CCD8Stem Cell TransplantationJournal of Medical Virology
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Accumulation of dysfunctional effector CD8+T cells in the liver of patients with chronic HCV infection

2005

Background/Aims Hepatitis C virus (HCV) causes a chronic infection that can lead to fibrosis and carcinoma. Immune responses mediated by cytotoxic T lymphocytes (CTLs) could be involved in viral clearance or persistence, and therefore in determining the course of the disease. Methods Intrahepatic and peripheral blood CD8+T cells were obtained from 32 HCV-chronically infected patients and analysed by flow-cytometry for surface markers of differentiation, IFNγ and TNFα production, degranulation capacity and perforin content, after CD3 triggering. Results were compared with those obtained from 13 patients with a non-viral liver disease. Results Intrahepatic CD8+T cells of HCV-infected patients…

AdultMalePore Forming Cytotoxic ProteinsCD3ApoptosisCD8-Positive T-LymphocytesInterferon-gammaLiver diseaseImmune systemHumansMedicineCytotoxic T cellAgedMembrane GlycoproteinsHepatologybiologyPerforinTumor Necrosis Factor-alphabusiness.industryDegranulationHepatitis C ChronicMiddle Agedmedicine.diseaseAcquired immune systemPhenotypeLiverPerforinImmunologybiology.proteinFemalebusinessCD8Follow-Up StudiesT-Lymphocytes CytotoxicJournal of Hepatology
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Myocardial infarction marker levels are influenced by prothrombin and tumor necrosis factor-α gene polymorphisms in young patients.

2012

Polymorphisms of genes encoding key factors for the control and activation of inflammatory response and coagulation cascade regulation may play a role in genetic susceptibility to acute myocardial infarction (AMI). This study sought to analyze the effect of TNF - 308G/A and pro-thrombin (FII) 20210G/A polymorphisms on the laboratory parameters of young patients affected by AMI. Results indicated that TNF - 308A positive genotype frequencies were increased in these patients and that a genetically determined higher production of TNF-alpha is associated in young subjects to a more severe cardiac damage as depicted by higher levels of troponin, Creatine kinase-MB Isoenzyme (mCK-MB) and a signif…

AdultMalePro-thrombinGenotypeImmunologyMyocardial InfarctionSNPSingle-nucleotide polymorphismAcute myocardial infarctionPolymorphism Single NucleotideBiochemistryYoung AdultGene FrequencyGenotypeTroponin IGenetic predispositionCreatine Kinase MB FormHumansImmunology and AllergyMedicineGenetic Predisposition to DiseaseMolecular BiologyAllele frequencyInflammationbiologyTumor Necrosis Factor-alphabusiness.industryAge FactorsFibrinogenHematologyMiddle AgedTroponinTumor necrosis factor-a.TroponinGenotype frequencyImmunologyHaematochemical parameterbiology.proteinProthrombinCreatine kinasebusinessBiomarkers
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