Search results for "Tumor necrosis factor."

showing 10 items of 848 documents

IL-34 is overexpressed in the inflamed salivary glands of patients with Sjogren's syndrome and is associated with the local expansion of pro-inflamma…

2013

Objectives To investigate the expression of IL-34 in labial salivary glands (LSGs) of patients with primary SS (p-SS) and its role in inducing a pro-inflammatory monocyte phenotype. Methods LSG biopsies were obtained from 20 patients with p-SS and 10 patients with non-Sjogren's sicca syndrome (n-SS). The expression of IL-34, IL-1β, TNF-α, IL-17 and IL-23 was assessed by real-time PCR. IL-34 expression was also investigated in LSGs by immunohistochemistry. The frequencies of subpopulations of CD14(+) monocytes were evaluated by flow cytometry among isolated mononuclear cells from peripheral blood and salivary glands from both patients and controls. The role of recombinant IL-34 on isolated p…

AdultMalePathologymedicine.medical_specialtyCD14Interleukin-1betaLipopolysaccharide ReceptorsInflammationCD16Interleukin-23Peripheral blood mononuclear cellMonocytesSalivary GlandsRheumatologySicca syndromemedicineHumansPharmacology (medical)interleukin-34 Sjögren’s syndrome monocytesAgedInflammationSalivary glandTumor Necrosis Factor-alphabusiness.industryInterleukinsMonocyteInterleukin-17Receptors IgGMiddle AgedFlow CytometrySettore MED/16 - ReumatologiaSjogren's Syndromemedicine.anatomical_structureImmunologyInterleukin 34Femalemedicine.symptombusinessRheumatology
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Molecular interactions between human cartilaginous endplates and nucleus pulposus cells: a preliminary investigation.

2014

Study Design. Conditioned media (CM) of cartilaginous endplates (CEPs) of intervertebral discs were analyzed in a bioassay with regard to their influence on matrix turnover and inflammatory factors on nucleus pulposus (NP) cells of the same patient. CEP tissue underwent further histological and ultrastructural analysis. Objective. To identify possible interactions between the CEP and the disc via molecular factors that may influence disc matrix degradation and to determine degenerative changes of CEP tissue. Summary of Background Data. Impaired endplate perme-ability due to degeneration and calcification is considered to be a key contributor to disc degeneration. An upregulation of metallop…

AdultMalePathologymedicine.medical_specialtyCellIntervertebral Disc DegenerationMatrix metalloproteinaseMatrix (biology)Proinflammatory cytokineDownregulation and upregulationMatrix Metalloproteinase 13MedicineHumansOrthopedics and Sports MedicineAggrecansIntervertebral DiscAggrecanCells CulturedAgedbusiness.industryInterleukin-6Interleukin-8Middle AgedCell biologyTissue Degenerationmedicine.anatomical_structureCartilageTumor necrosis factor alphaFemaleMatrix Metalloproteinase 3Neurology (clinical)businessSpine
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CD27 distinguishes two phases in bone marrow infiltration of splenic marginal zone lymphoma.

2004

Aims:  To investigate CD27 expression in splenic marginal zone lymphoma (SMZL), an indolent low-grade B-cell lymphoma with constant involvement of the bone marrow, especially with an intrasinusoidal pattern. It is not clear if the neoplastic clone is composed of virgin or somatically mutated B cells. CD27 is reported to be a hallmark of memory B cells. Methods and results:  We evaluated 64 bone marrow biopsy specimens (BMBs) from 36 patients with SMZL for the expression of CD27. For comparison, splenectomy specimens of patients with traumatic splenic rupture or with SMZL were used. All BMBs showed lymphomatous infiltration. When located in the marrow sinusoids, neoplastic cells were CD27– i…

AdultMalePathologymedicine.medical_specialtyHistologybone marrowLymphomaBiopsyNaive B cellsplenic marginal zone lymphomaSplenic NeoplasmSpleenPathology and Forensic MedicineBiopsyintrasinusoidal infiltrationmedicineHumansSplenic marginal zone lymphomaCD27naive and memory B cellsAgedAged 80 and overmedicine.diagnostic_testbusiness.industrySplenic NeoplasmsGeneral MedicineMiddle Agedmedicine.diseaseBone Marrow NeoplasmImmunohistochemistryTumor Necrosis Factor Receptor Superfamily Member 7bone marrow; CD27; intrasinusoidal infiltration; naive and memory B cells; splenic marginal zone lymphomamedicine.anatomical_structureBone marrow neoplasmFemaleBone marrowbusinessBone Marrow NeoplasmsInfiltration (medical)
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Immunohistological and immunoelectron microscopic identification of TNF alpha in normal human and murine epidermis.

1992

The presence, distribution and cellular localization of tumour necrosis factor-alpha (TNF alpha) were investigated in normal human and murine epidermis using immunohistological and immunoelectron microscopic methods with monoclonal and polyclonal antibodies. The immunostaining revealed an intercellular plasma membrane and cytoplasmic labelling of the epidermal keratinocytes, but no labelling of Langerhans cells, melanocytes and Merkel cells. Large amounts of TNF alpha were regularly found in the sebaceous glands. These findings demonstrate that epidermal keratinocytes and especially sebocytes produce and release TNF alpha and that this keratinocyte-derived cytokine may be important for the …

AdultMalePathologymedicine.medical_specialtyImmunoelectron microscopymedicine.medical_treatmentDermatologyMicemedicineAnimalsHumansMicroscopy ImmunoelectronCellular localizationMice Inbred BALB Cintegumentary systemEpidermis (botany)biologyTumor Necrosis Factor-alphaGeneral MedicineMiddle AgedMolecular biologyImmunohistochemistryCytokinemedicine.anatomical_structurePolyclonal antibodiesbiology.proteinImmunohistochemistryFemaleEpidermisMerkel cellImmunostainingArchives of dermatological research
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Immune-mediated necrotizing myopathy is characterized by a specific Th1-M1 polarized immune profile.

2012

Immune-mediated necrotizing myopathy (IMNM) is considered one of the idiopathic inflammatory myopathies, comprising dermatomyositis, polymyositis, and inclusion body myositis. The heterogeneous group of necrotizing myopathies shows a varying amount of necrotic muscle fibers, myophagocytosis, and a sparse inflammatory infiltrate. The underlying immune response in necrotizing myopathy has not yet been addressed in detail. Affected muscle tissue, obtained from 16 patients with IMNM, was analyzed compared with eight non-IMNM (nIMNM) tissues. Inflammatory cells were characterized by IHC, and immune mediators were assessed by quantitative real-time PCR. We demonstrate that immune- and non–immune-…

AdultMalePathologymedicine.medical_specialtyT cellBiopsyCell CountBiologyCD8-Positive T-LymphocytesMajor histocompatibility complexReal-Time Polymerase Chain ReactionPolymyositisPathology and Forensic MedicineYoung AdultImmune systemSarcolemmamedicineHumansAgedAged 80 and overB-LymphocytesMyositisMacrophagesMusclesHistocompatibility Antigens Class IAutoantibodyImmunityComplement System ProteinsDermatomyositisMiddle AgedTh1 Cellsmedicine.diseaseCapillariesmedicine.anatomical_structureChild PreschoolImmunologybiology.proteinTumor necrosis factor alphaFemaleInclusion body myositisThe American journal of pathology
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Functional profile of cytomegalovirus (CMV)-specific CD8+ T cells and kinetics of NKG2C+ NK Cells associated with the resolution of CMV DNAemia in al…

2011

Immune mechanisms involved in control of cytomegalovirus (CMV) infection in the allogeneic stem cell transplantation setting have not been fully disclosed. CMV pp65 and IE-1-specific CD8(+) T cells expressing IFN-γ, TNF-α, and CD107a, alone or in combination, and NKG2C(+) NK cells were prospectively enumerated during 13 episodes of CMV DNAemia. The expansion of monofunctional and polyfunctional CD8(+) T cells was associated with CMV DNAemia clearance. The size and functional diversity of the expanding CD8(+) T-cell population was greater in self-resolved episodes than in episodes treated with antivirals. These differences were related to the magnitude of expansion of cognate antigen IFN-γ C…

AdultMalePopulationCongenital cytomegalovirus infectionCytomegalovirusCD8-Positive T-LymphocytesBiologyViral Matrix ProteinsInterferon-gammaYoung AdultInterleukin 21AntigenLysosomal-Associated Membrane Protein 1VirologymedicineHumansTransplantation HomologousCytotoxic T celleducationeducation.field_of_studyTumor Necrosis Factor-alphaReceptors IgGvirus diseasesMiddle AgedPhosphoproteinsmedicine.diseaseVirologyCD56 AntigenKiller Cells NaturalTransplantationInfectious DiseasesCytomegalovirus InfectionsDNA ViralImmunologyFemaleStem cellNK Cell Lectin-Like Receptor Subfamily CCD8Stem Cell TransplantationJournal of Medical Virology
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Accumulation of dysfunctional effector CD8+T cells in the liver of patients with chronic HCV infection

2005

Background/Aims Hepatitis C virus (HCV) causes a chronic infection that can lead to fibrosis and carcinoma. Immune responses mediated by cytotoxic T lymphocytes (CTLs) could be involved in viral clearance or persistence, and therefore in determining the course of the disease. Methods Intrahepatic and peripheral blood CD8+T cells were obtained from 32 HCV-chronically infected patients and analysed by flow-cytometry for surface markers of differentiation, IFNγ and TNFα production, degranulation capacity and perforin content, after CD3 triggering. Results were compared with those obtained from 13 patients with a non-viral liver disease. Results Intrahepatic CD8+T cells of HCV-infected patients…

AdultMalePore Forming Cytotoxic ProteinsCD3ApoptosisCD8-Positive T-LymphocytesInterferon-gammaLiver diseaseImmune systemHumansMedicineCytotoxic T cellAgedMembrane GlycoproteinsHepatologybiologyPerforinTumor Necrosis Factor-alphabusiness.industryDegranulationHepatitis C ChronicMiddle Agedmedicine.diseaseAcquired immune systemPhenotypeLiverPerforinImmunologybiology.proteinFemalebusinessCD8Follow-Up StudiesT-Lymphocytes CytotoxicJournal of Hepatology
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Myocardial infarction marker levels are influenced by prothrombin and tumor necrosis factor-α gene polymorphisms in young patients.

2012

Polymorphisms of genes encoding key factors for the control and activation of inflammatory response and coagulation cascade regulation may play a role in genetic susceptibility to acute myocardial infarction (AMI). This study sought to analyze the effect of TNF - 308G/A and pro-thrombin (FII) 20210G/A polymorphisms on the laboratory parameters of young patients affected by AMI. Results indicated that TNF - 308A positive genotype frequencies were increased in these patients and that a genetically determined higher production of TNF-alpha is associated in young subjects to a more severe cardiac damage as depicted by higher levels of troponin, Creatine kinase-MB Isoenzyme (mCK-MB) and a signif…

AdultMalePro-thrombinGenotypeImmunologyMyocardial InfarctionSNPSingle-nucleotide polymorphismAcute myocardial infarctionPolymorphism Single NucleotideBiochemistryYoung AdultGene FrequencyGenotypeTroponin IGenetic predispositionCreatine Kinase MB FormHumansImmunology and AllergyMedicineGenetic Predisposition to DiseaseMolecular BiologyAllele frequencyInflammationbiologyTumor Necrosis Factor-alphabusiness.industryAge FactorsFibrinogenHematologyMiddle AgedTroponinTumor necrosis factor-a.TroponinGenotype frequencyImmunologyHaematochemical parameterbiology.proteinProthrombinCreatine kinasebusinessBiomarkers
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Inflammatory capacity of exosomes released in the early stages of acute pancreatitis predicts the severity of the disease

2021

As acute pancreatitis progresses to the severe form, a life-threatening systemic inflammation is triggered. Although the mechanisms involved in this process are not yet well understood, it has been proposed that circulating exosomes may be involved in the progression of inflammation from the pancreas to distant organs. Here, the inflammatory capacity and protein profile of plasma exosomes obtained during the first 24 h of hospitalization of patients diagnosed with acute pancreatitis were characterized and compared with the final severity of the disease. We found that the final severity of the disease strongly correlates with the inflammatory capacity of exosomes in the early stages of acute…

AdultMaleProteomicsInflammationSystemic inflammationExosomesS100A9Pathology and Forensic MedicineS100A8medicineHumansS100A8PancreasS100A9AgedAged 80 and overInflammationbusiness.industryMiddle Agedmedicine.diseaseMicrovesiclesAcute pancreatitisPancreatitisAcute DiseaseImmunologyDisease ProgressionPancreatitisAcute pancreatitisFemaleTumor necrosis factor alphamedicine.symptombusinessSignal TransductionJournal of Pathology
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Asthmatic Bronchial Epithelium Is More Susceptible to Oxidant-Induced Apoptosis

2002

Abnormal apoptotic mechanisms are associated with disease pathogenesis. Because the asthmatic bronchial epithelium is characteristically damaged with loss of columnar epithelial cells, we postulated that this is due to unscheduled apoptosis. Using an antibody directed toward the caspase cleavage product of poly(ADP-ribose) polymerase, immunohistochemistry applied to endobronchial biopsies showed higher levels of staining in the bronchial epithelium of subjects with asthma as compared with normal control subjects (% epithelial staining [median (range) = 10.5 (1.4-24.5) versus 0.4 (0.0-9.7)]; P < 0.001). Because we were unable to determine whether this difference was due to ongoing inflammati…

AdultMalePulmonary and Respiratory MedicinePathologymedicine.medical_specialtyCell Biology; Molecular Biology; Pulmonary and Respiratory MedicineBiopsyClinical BiochemistryCellApoptosisBronchiInflammationRespiratory MucosaBiologyIn vivomedicineHumansMolecular BiologyCells CulturedAgedTumor Necrosis Factor-alphaEpithelial CellsHydrogen PeroxideCell BiologyMiddle AgedFlow CytometryOxidantsAsthmaIn vitroStainingmedicine.anatomical_structureApoptosisbiology.proteinImmunohistochemistryFemalePoly(ADP-ribose) Polymerasesmedicine.symptomAntibodyAmerican Journal of Respiratory Cell and Molecular Biology
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