Search results for "Type I"

showing 10 items of 966 documents

Painful scar neuropathy: principles of diagnosis and treatment

2015

Nerve-tissue interactions are critical. Peripheral nerve injuries may involve intraneural and extraneural scar formation and affect nerve gliding planes, sometimes leading to complex clinical presentations. All of these pathological entities involve pain as the main clinical symptom and can be subsumed under the term "painful scar neuropathy". The authors review the literature on treatment approaches to peripheral nerve scar neuropathy and the outcomes of neurolysis-associated procedures and propose a simple classification and a therapeutic approach to scar neuropathy. The search retrieved twenty-one papers, twenty of which reported pain reduction or resolution with various techniques. Ther…

Painful scarLesion typemedicine.medical_specialtybusiness.industrySettore MED/19 - Chirurgia PlasticaLogical approachExtraneuralComplex regional pain syndrome type II painful neuropathy painful scar neuropathy scar neuritis traction neuropathySurgeryTherapeutic approachPeripheral nerveNeuropathic painMedicinebusinessPathological
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Prediction of bone mass gain by bone turnover parameters after parathyroidectomy for primary hyperparathyroidism: neural network software statistical…

2006

Background Primary hyperparathyroidism (pHPT) is the most frequent endocrine hypersecretion disease, and parathyroidectomy is the only curative option, since pharmacologic therapy reduces hypercalcemia but does not impede parathyroid hormone hypersecretion. According to guidelines from the National Institutes of Health, parathyroidectomy is associated with bone mass increase in some asymptomatic patients, while in others bone mass is not changed after surgery. Therefore, we performed the present study in an attempt to elucidate whether a preoperative biochemical bone parameter can be predictive of a significant vertebral bone mass increase in patients with pHPT. Methods For each patient we …

Parathyroidectomymedicine.medical_specialtymedicine.medical_treatmentUrinary systemUrologyParathyroid hormoneCollagen Type Ibone mass bone mineralization bone turnoverBone remodelingBone DensityHumansMedicineAgedAged 80 and overParathyroidectomyHyperparathyroidismbiologybusiness.industryMiddle AgedHyperparathyroidism Primarymedicine.diseaseUrinary calciumSurgeryOsteocalcinbiology.proteinSurgeryBone RemodelingNeural Networks ComputerPeptidesbusinessPrimary hyperparathyroidismSurgery
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Familial hypercholesterolaemia: A global call to arms

2015

Familial Hypercholesterolaemia (FH) is the commonest autosomal co-dominantly inherited condition affecting man. It is caused by mutation in one of three genes, encoding the low-density lipoprotein (LDL) receptor, or the gene for apolipoprotein B (which is the major protein component of the LDL particle), or in the gene coding for PCSK9 (which is involved in the degradation of the LDL-receptor during its cellular recycling). These mutations result in impaired LDL metabolism, leading to life-long elevations in LDL-cholesterol (LDL-C) and development of premature atherosclerotic cardiovascular disease (ASCVD) [1], [2] and [3]. If left untreated, the relative risk of premature coronary artery d…

PathologyApolipoprotein BDisease030204 cardiovascular system & hematologymedicine.disease_causeGlobal HealthDISEASEDoenças Cardio e Cérebro-vasculares0302 clinical medicineHyperlipoproteinemia Type IISocieties MedicalRISK0303 health sciencesMutationbiology3. Good healthPREVALENCEEuropelipids (amino acids peptides and proteins)Cardiology and Cardiovascular MedicineFamilial hypercholesterolaemiaLife Sciences & Biomedicinemedicine.medical_specialtyHeterozygote1102 Cardiovascular Medicine And HaematologyHyperlipoproteinemia Type II03 medical and health sciencesInternal medicinemedicineHumans030304 developmental biologyScience & Technologybusiness.industryGUIDANCEPCSK9Heterozygote advantage1103 Clinical SciencesEndocrinologyPeripheral Vascular DiseaseCardiovascular System & HematologyReceptors LDLRECEPTORES DE LIPOPROTEÍNASRelative riskMutationbiology.proteinCardiovascular System & CardiologyFamilial HypercholesterolaemiabusinessCLINICIANLipoprotein
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Shell Extracts from the Marine Bivalve Pecten maximus Regulate the Synthesis of Extracellular Matrix in Primary Cultured Human Skin Fibroblasts

2014

International audience; Mollusc shells are composed of more than 95% calcium carbonate and less than 5% of an organic matrix consisting mostly of proteins, glycoproteins and polysaccharides. Previous studies have elucidated the biological activities of the shell matrices from bivalve molluscs on skin, especially on the expression of the extracellular matrix components of fibroblasts. In this work, we have investigated the potential biological activities of shell matrix components extracted from the shell of the scallop Pecten maximus on human fibroblasts in primary culture. Firstly, we demonstrated that shell matrix components had different effects on general cellular activities. Secondly, …

Pathologylcsh:Medicine[SDV.BC.BC]Life Sciences [q-bio]/Cellular Biology/Subcellular Processes [q-bio.SC][CHIM.THER]Chemical Sciences/Medicinal ChemistryBiochemistryExtracellular matrixCell SignalingMolecular Cell Biologylcsh:ScienceSkinConnective Tissue Cellschemistry.chemical_classificationPectenMultidisciplinary[ CHIM.THER ] Chemical Sciences/Medicinal ChemistryExtracellular MatrixCell biologymedicine.anatomical_structureConnective TissueScallopCytochemistryAnatomyCellular Structures and OrganellesType I collagenResearch ArticleBiotechnologySignal Transductionmedicine.medical_specialtyPrimary Cell CultureExtracellular Matrix SignalingBiologyBiomaterialsDermisAnimal ShellsmedicineAnimalsHumansPecten maximus14. Life underwaterTissue Extractslcsh:R[ SDV.BC.BC ] Life Sciences [q-bio]/Cellular Biology/Subcellular Processes [q-bio.SC]Biology and Life SciencesCell BiologyFibroblastsbiology.organism_classificationIn vitroBiological TissuechemistryCell culturelcsh:QGlycoprotein
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Collagenase-3 (MMP-13) Enhances Remodeling of Three-Dimensional Collagen and Promotes Survival of Human Skin Fibroblasts

2006

Collagenase-3 (MMP-13) is a matrix metalloproteinase capable of cleaving a multitude of extracellular matrix proteins in addition to fibrillar collagens. Human MMP-13 is expressed by fibroblasts in chronic cutaneous ulcers, but not in normally healing adult skin wounds. However, MMP-13 is produced by fibroblasts in adult gingival and in fetal skin wounds characterized by rapid collagen remodeling and scarless healing. Here, we have examined the role of human MMP-13 in remodeling of three-dimensional (3D) collagenous matrix by primary adult human skin fibroblasts. The high level of human MMP-13 expression by fibroblasts achieved by adenoviral gene delivery resulted in potent enhancement of r…

Pathologymedicine.medical_specialtyCell SurvivalHuman skinDermatologyMatrix Metalloproteinase InhibitorsMatrix metalloproteinaseBiologyBiochemistryFilamentous actinAdenoviridaeDermal fibroblastExtracellular matrixMatrix Metalloproteinase 13medicineHumansFibroblastMolecular BiologyCells CulturedCell ProliferationWound HealingTissue Inhibitor of Metalloproteinase-1integumentary systemCell BiologyFibroblastsActinsCell biologyEnzyme ActivationCollagen type I alpha 1medicine.anatomical_structureCollagenaseCollagenmedicine.drugJournal of Investigative Dermatology
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Endothelial nitric oxide synthase upregulation in the guinea pig organ of Corti after acute noise trauma.

2004

Endothelial nitric oxide synthase (eNOS) upregulation was identified 60 h after acute noise trauma in morphologically intact cells of the reticular lamina in the organ of Corti of the guinea pig in the second turn of the cochlea. Using gold-coupled anti-eNOS antibodies and electron microscopy, it was shown that eNOS expression was upregulated in all cell areas and cell types except inner hair cells. Furthermore, eNOS was found in the organelle-free cytoplasm and in mitochondria of various cell types. The density of eNOS in mitochondria was considerably higher compared with the surrounding cytoplasm. Since eNOS activity is regulated by calcium, the eNOS detection was combined with calcium pr…

Pathologymedicine.medical_specialtyCytoplasmNitric Oxide Synthase Type IIIGuinea Pigschemistry.chemical_elementCalciumMicrotubulesDownregulation and upregulationMicroscopy Electron TransmissionEnosStress PhysiologicalHair Cells AuditorymedicineAnimalsCalcium SignalingMolecular BiologyOrgan of CortiCytoskeletonbiologyGeneral NeuroscienceNitric Oxide Synthase Type IIIbiology.organism_classificationImmunohistochemistryCell biologyMitochondriaUp-RegulationNitric oxide synthaseActin CytoskeletonDisease Models Animalmedicine.anatomical_structureDrosophila melanogasterchemistryAcoustic StimulationHearing Loss Noise-InducedCytoplasmOrgan of Cortibiology.proteinCalciumNeurology (clinical)Nitric Oxide SynthaseNoiseIntracellularDevelopmental BiologyBrain research
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"Bull’s eye” appearance of hepatocellular adenomas in patients with glycogen storage disease type I — atypical magnetic resonance imaging findings: T…

2021

BACKGROUND Hepatocellular adenomas are rare tumors that can occur in patients with glycogen storage disease type I. CASE SUMMARY We herein report two cases of histologically proven hepatocellular adenomas in patients with glycogen storage disease type I. Magnetic resonance imaging (MRI) was performed after bolus injection of gadoxetate disodium, a liver-specific gadolinium-based MRI contrast agent. In the present cases, some of the hepatocellular adenomas showed unexpectedly a “bull’s eye” appearance on T2-weighted and post-contrast images, which was not previously described as imaging findings of hepatocellular adenomas in glycogen storage disease. A bull’s eye appearance on T2-weighted im…

Pathologymedicine.medical_specialtyGadoxetate Disodium03 medical and health sciencesMagnetic resonance imaging0302 clinical medicineCase reportmedicineGlycogen storage diseaseIn patientGlycogen storage diseaseGlycogen storage disease type Imedicine.diagnostic_testbusiness.industryMagnetic resonance imagingGeneral MedicineHepatocellular adenomaequipment and suppliesmedicine.diseaseHepatocellular adenomadigestive system diseasesBull’s eye030220 oncology & carcinogenesis030211 gastroenterology & hepatologyGadoxetate disodiumBull's Eyebusinesshuman activitiesWorld Journal of Clinical Cases
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Structure and Function of Matrix Components in the Cruciate Ligaments

1992

In the present study, the matrix components of 100 cruciate ligaments were analyzed by conventional electron microscopy, immunohistology, morphometry, and immunoelectron microscopy. The anterior (ACL) and the posterior (PCL) cruciate ligaments contained collagen types III, IV, and VI. Several structural glycoproteins, like fibronectin, laminin, entactin, tenascin, and undulin were detected using monoclonal antibodies. Whereas laminin and entactin were higher concentrated in the PCL, type VI collagen was more frequently found in the ACL. The ACL had a critical nourishment in its distal and middle thirds. In all ligament parts the PCL revealed a better vascular supply with strong correlation …

Pathologymedicine.medical_specialtyHistologybiologyChemistryImmunoelectron microscopyTenascinmusculoskeletal systemExtracellular matrixFibronectinCruciate ligamentType IV collagenmedicine.anatomical_structureLamininbiology.proteinmedicineLigamentAnatomyhuman activitiesCells Tissues Organs
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Hypothetical molecular mechanisms by which local iron overload facilitates the development of venous leg ulcers and multiple sclerosis lesions.

2008

Summary This paper presents a hypothetical model of role for iron in the development of venous leg ulcers and multiple sclerosis. Elevated concentrations of iron were found in the skin affected by venous hypertension and also in the areas of brain with multiple sclerosis lesions. Individuals with hemochromatosis gene (HFE) mutations: C282Y and H63D, which result in a less efficient transport of iron by macrophages, are characterized by an increased risk for venous leg ulcer and multiple sclerosis. Multiple sclerosis is a T cell-mediated disease, and T cells probably participate in the development of venous ulcers. This deleterious role of ferric ions could be related to the regulation of T …

Pathologymedicine.medical_specialtyIron OverloadMultiple SclerosisT cellT-LymphocytesDown-RegulationNitric Oxide Synthase Type IIApoptosisVenous leg ulcerModels BiologicalNitric oxideVaricose Ulcerchemistry.chemical_compoundDownregulation and upregulationMedicineAnimalsHumansReceptorReceptors Interferonbiologybusiness.industryMultiple sclerosisMacrophagesLeg UlcerGeneral MedicineModels Theoreticalmedicine.diseaseNitric oxide synthasemedicine.anatomical_structurechemistryApoptosisImmunologybiology.proteinNitric Oxide SynthasebusinessMedical hypotheses
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Post‐pneumonectomy Lung Deformation is Associated with Alveolar Type II Cell Apoptosis and Altered Parenchymal Mechanics

2015

Pathologymedicine.medical_specialtyLungbusiness.industrymedicine.medical_treatmentAnatomyDeformation (meteorology)BiochemistryPneumonectomymedicine.anatomical_structureApoptosisParenchymaGeneticsMedicineAlveolar type II cellbusinessMolecular BiologyBiotechnologyThe FASEB Journal
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