Search results for "VITRO"

showing 10 items of 2786 documents

Toxicity of a dental adhesive compared with ionizing radiation and zoledronic acid

2015

Background: To determine the toxicity of aqueous dilutions of a universal self-priming dental adhesive (DA) and comparing these with those elicited by exposure to ionizing radiation (IR), Zoledronic acid (Z) treatment and the synergic effects of the combined treatment with IR+Z. Material and Methods: The genotoxic effect of DA was determined by the increase in the frequency of micronuclei in cytokinesis-blocked in cultured human lymphocytes before and after exposure to 2Gy of X-rays. The cytotoxic effect was studied by using the MTT cell viability test in normal prostate cell lines (PNT2) after exposure to different X-ray doses (0Gy-20Gy). The cell lines divided into different groups and tr…

Programmed cell deathDental CementsOdontologíaPharmacologymedicine.disease_causeZoledronic AcidIonizing radiationToxicologyPolymethacrylic AcidsRadiation IonizingToxicity TestsmedicineCytotoxic T cellHumansViability assayLymphocytesGeneral DentistryCells CulturedOral Medicine and PathologyDiphosphonatesChemistryResearchImidazoles:CIENCIAS MÉDICAS [UNESCO]Ciencias de la saludIn vitroOtorhinolaryngologyToxicityMicronucleus testUNESCO::CIENCIAS MÉDICASSurgeryGenotoxicity
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Involvement of Microglia in Neurodegenerative Diseases: Beneficial Effects of Docosahexahenoic Acid (DHA) Supplied by Food or Combined with Nanoparti…

2021

Neurodegenerative diseases represent a major public health issue and require better therapeutic management. The treatments developed mainly target neuronal activity. However, an inflammatory component must be considered, and microglia may constitute an important therapeutic target. Given the difficulty in developing molecules that can cross the blood–brain barrier, the use of food-derived molecules may be an interesting therapeutic avenue. Docosahexaenoic acid (DHA), an omega-3 polyunsaturated fatty acid (22:6 omega-3), has an inhibitory action on cell death and oxidative stress induced in the microglia. It also acts on the inflammatory activity of microglia. These data obtained in vitro or…

Programmed cell deathDocosahexaenoic AcidsQH301-705.5microgliaApoptosisInflammationReviewPharmacologyProtective AgentsInhibitory postsynaptic potentialmedicine.disease_causeCatalysisInorganic ChemistryDrug Delivery Systemsneurodegenerative diseasemedicineAnimalsHumansBiology (General)Physical and Theoretical ChemistryQD1-999Molecular BiologySpectroscopychemistry.chemical_classificationMicrogliabusiness.industryOrganic ChemistryNeurodegenerative DiseasesGeneral Medicinedocosahexaenoic acidnanomedicineIn vitroComputer Science ApplicationsDisease Models AnimalOxidative StressChemistryTreatment Outcomemedicine.anatomical_structurechemistryBlood-Brain BarrierinflammationDocosahexaenoic acidDietary SupplementsNanoparticlesmedicine.symptombusinessOxidative stressPolyunsaturated fatty acidInternational Journal of Molecular Sciences
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P7142-AG impacts on endothelial cell activation and endothelial cell viability in vitro and impairs endothelial repair in vivo

2019

Abstract Background The endocannabinoid (eCB) 2-arachidonoylglycerol (2-AG) is a known modulator of inflammation and few studies have addressed its influence on myeloid cells in the context of atherogenesis. However, the impact of 2-AG on endothelial cell function has not been studied before. Methods Endothelial repair was studied in two treatment groups of wildtype mice following electrical denudation of the common carotid artery at a length of 3000 μm. One group received the monoacylglycerol lipase (MAGL)-inhibitor JZL184 [5 mg/kg i.p.], which impairs 2-AG degradation and thus causes elevated 2-AG levels, the other group received vehicle. The residual endothelial gap at five days in eithe…

Programmed cell deathEndotheliumbusiness.industrymedicine.medical_treatmentInflammationIn vitroCell biologyEndothelial stem cellmedicine.anatomical_structureCytokineIn vivomedicineTHP1 cell linemedicine.symptomCardiology and Cardiovascular MedicinebusinessEuropean Heart Journal
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Cell cooperation in coelomocyte cytotoxic activity of Paracentrotus lividus coelomocytes

2007

The coelomic fluid from the sea urchin Paracentrotus lividus contains several coelomocyte types including amoebocytes and uncoloured spherulocytes involved in immune defences. In the present paper, we show a Ca(2+)-dependent cytotoxic activity for the unfractionated coelomocytes assayed in vitro, with rabbit erythrocytes and the K562 tumour cell line. In a plaque-forming assay, whole coelomocyte preparations as well as density gradient separated coelomocyte populations revealed that cell populations enriched in uncoloured spherulocytes, exerted high cytotoxic activity by releasing lysins in the presence of amoebocytes. This cooperative effect could be dependent on soluble factors released b…

Programmed cell deathErythrocytesPhysiologyCytotoxicityCell CommunicationCell SeparationBiochemistryParacentrotus lividusbiology.animalCentrifugation Density GradientAnimalsHumansCytotoxic T cellCytotoxicityMolecular BiologySea urchinCoelomocyteCoelomocyte cooperationInnate immunityCell DeathEchinodermbiologyAnatomybiology.organism_classificationIn vitroCell biologyParacentrotus lividusCell cultureParacentrotusRabbitsCoelomocyteK562 CellsComparative Biochemistry and Physiology Part A: Molecular & Integrative Physiology
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Oxygen Consumption and Oxygen Diffusion Properties of Multicellular Spheroids from two Different Cell Lines

1984

Multicellular spheroids are an in vitro tissue model in which the cells are supplied by diffusion of oxygen and substrates from the environmental growth medium (Sutherland et al., 1971). Since these substances are consumed when diffusing to the spheroid center, their concentration should decrease continuously towards the inner parts of the spheroids. Therefore, the location of the cells within the spheroid is an important determinant of the efficiency of the O2 and nutrient supply. The restriction of the O2 availability in the inner part of the spheroids may influence the metabolic and cell cycle state, and may even cause cell death, indicated by central necrosis in larger spheroids. Also, …

Programmed cell deathMulticellular organismCell cultureChemistryembryonic structuresSpheroidMetabolismCell cycleIn vitroCell biologyOxygen tension
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Excitotoxin-induced changes in transglutaminase during differentiation of cerebellar granule cells

2002

Excitotoxicity induced by NMDA receptor stimulation is able to increase the activity of many enzymes involved in neuronal cell death. Primary cultures of rat cerebellar granule cells were used to elucidate the role of transglutaminase reaction in the excitotoxic cell response, and to evaluate the role of glutamate receptors in cell survival and degeneration. Granule neurons, maintained in vitro for two weeks, were exposed to NMDA at different stages of differentiation. Following NMDA receptor activation, increases in transglutaminase activity were observed in cell cultures. The levels of enzyme activity were higher in cells at 5 days in vitro than in those at 8-9 or 13-14 days in vitro. Mor…

Programmed cell deathN-MethylaspartateTime FactorsCell SurvivalTissue transglutaminaseNeurotoxinsClinical BiochemistryExcitotoxicityStimulationmedicine.disease_causeReceptors N-Methyl-D-AspartateBiochemistryCerebellummedicineAnimalsRats WistarNeuronsTransglutaminasesbiologyOrganic ChemistryGlutamate receptorCell DifferentiationIn vitroRatsCell biologyAnimals Newbornnervous systemApoptosisNerve Degenerationbiology.proteinNMDA receptorTransglutaminase – Excitotoxicity – Neurodegenerative diseases – Apoptosis – Glutamate – Cerebellar granule neuronsAmino Acids
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Studies on the apoptotic activity of natural and synthetic retinoids: discovery of a new class of synthetic terphenyls that potently support cell gro…

2005

New terphenyl derivatives have been synthesized and tested for their effect on cell survival in serum-free cultures. These compounds protected HL60 cells from death and supported their growth with an activity higher than that of the natural 14-hydroxy-retro-retinol. Terphenyls 26 and 28 also possess antiapoptotic activity on neuronal cells, proving them as possible candidates for the treatment of neurodegenerative and ischemic diseases.

Programmed cell deathNecrosisreceptor-alphamedicine.drug_classmechanismApoptosisHL-60 Cellsnecrosischemistry.chemical_compoundRetinoidsdeathTerphenylDrug DiscoverymedicineHumansRetinoidNeuronsCell growthbiphenyl-4-carboxylic acidarotinoidIn vitroCell biologyCultured cortical-neuronchemistryBiochemistryCell cultureApoptosisretinobenzoic acidMolecular MedicineIndicators and Reagentsmultidrugmedicine.symptomCell Division(14R)-14-hydroxy-414-retro-retinolJournal of medicinal chemistry
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Caspase-8 regulates TNF-alpha induced epithelial necroptosis and terminal ileitis

2011

Two groups identify the regulation of death-receptor-induced necroptosis as an epithelial intrinsic mechanism that is important for the maintenance of immune homeostasis and the prevention of intestinal inflammation in mice. Welz et al. describe an unexpected physiological function for FADD (Fas-associated protein with death domain), an adaptor protein required for death-receptor-induced apoptosis. Mice with intestinal epithelial specific knockout of FADD develop severe colon inflammation due to increased death of FADD-deficient colonic epithelial cells. Gunther et al. report a novel and unexpected function of caspase-8 in maintaining immune homeostasis in the gut. Caspase-8 expression by g…

Programmed cell deathPaneth CellsNecroptosisInflammationApoptosisBiologyIn Vitro Techniquesdigestive systemArticle03 medical and health sciencesMiceNecrosis0302 clinical medicineCrohn DiseasemedicineAnimalsHumansFADD030304 developmental biology0303 health sciencesCaspase 8MultidisciplinaryInnate immune systemTumor Necrosis Factor-alphaColitisIntestinal epithelium3. Good healthmedicine.anatomical_structure030220 oncology & carcinogenesisReceptor-Interacting Protein Serine-Threonine KinasesPaneth cellImmunologybiology.proteinCancer researchTumor necrosis factor alphaGoblet Cellsmedicine.symptomGene DeletionNature
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In Vitro and in Vivo Evaluation of Water-Soluble Iminophosphorane Ruthenium(II) Compounds. A Potential Chemotherapeutic Agent for Triple Negative Bre…

2014

A series of organometallic ruthenium(II) complexes containing iminophosphorane ligands have been synthesized and characterized. Cationic compounds with chloride as counterion are soluble in water (70–100 mg/mL). Most compounds (especially highly water-soluble 2) are more cytotoxic to a number of human cancer cell lines than cisplatin. Initial mechanistic studies indicate that the cell death type for these compounds is mainly through canonical or caspase-dependent apoptosis, nondependent on p53, and that the compounds do not interact with DNA or inhibit protease cathepsin B. In vivo experiments of 2 on MDA-MB-231 xenografts in NOD.CB17-Prkdc SCID/J mice showed an impressive tumor reduction (…

Programmed cell deathStereochemistryPhosphoranesAntineoplastic AgentsTriple Negative Breast NeoplasmsMice SCIDPharmacologyIn Vitro TechniquesArticleRutheniumIn vivoCoordination ComplexesMice Inbred NODDrug DiscoverymedicineOrganometallic CompoundsCytotoxic T cellAnimalsHumansCathepsinCisplatinChemistryWaterIn vitro3. Good healthHEK293 CellsSolubilityCell cultureApoptosisMolecular MedicineFemalemedicine.drugJournal of Medicinal Chemistry
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Apoptosis causes lumen formation during angiogenesis in vitro.

2002

Programmed cell deathUmbilical VeinsAngiogenesisLumen (anatomy)ApoptosisBiologyBiochemistryUmbilical veinmedicineIn Situ Nick-End LabelingHumansCells CulturedNeovascularization PathologicCell BiologyIntercellular Adhesion Molecule-1ImmunohistochemistryIn vitroCell biologyEndothelial stem cellPlatelet Endothelial Cell Adhesion Molecule-1Microscopy Electronmedicine.anatomical_structureApoptosisImmunologyCollagenEndothelium VascularCardiology and Cardiovascular MedicineBlood vesselMicrovascular research
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