Search results for "VTRNA2-1"

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Variable maternal methylation overlapping the nc886/vtRNA2-1 locus is locked between hypermethylated repeats and is frequently altered in cancer.

2014

Cancer is as much an epigenetic disease as a genetic one; however, the interplay between these two processes is unclear. Recently, it has been shown that a large proportion of DNA methylation variability can be explained by allele-specific methylation (ASM), either at classical imprinted loci or those regulated by underlying genetic variants. During a recent screen for imprinted differentially methylated regions, we identified the genomic interval overlapping the non-coding nc886 RNA (previously known as vtRNA2-1) as an atypical ASM that shows variable levels of methylation, predominantly on the maternal allele in many tissues. Here we show that the nc886 interval is the first example of a …

AdultCancer ResearchLung NeoplasmsRNA UntranslatedLoss of HeterozygosityLocus (genetics)Breast NeoplasmsBiologyLoss of heterozygosityGenomic ImprintingYoung Adultnc886NeoplasmsHumansEpigeneticsAllelePromoter Regions GeneticMolecular BiologyvtRNA2-1GeneticsDNA methylationMethylationMiddle Agedvault RNAsMolecular biologyDifferentially methylated regionsUrinary Bladder NeoplasmsGenetic LociTandem Repeat SequencesDNA methylationColonic NeoplasmsmiRNAsFemaleimprintingGenomic imprintingResearch PaperEpigenetics
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Methylation status of VTRNA2-1/nc886 is stable across populations, monozygotic twin pairs and in majority of tissues.

2022

Aims & methods: The aim of this study was to characterize the methylation level of a polymorphically imprinted gene, VTRNA2-1/nc886, in human populations and somatic tissues.48 datasets, consisting of more than 30 tissues and >30,000 individuals, were used. Results: nc886 methylation status is associated with twin status and ethnic background, but the variation between populations is limited. Monozygotic twin pairs present concordant methylation, whereas similar to 30% of dizygotic twin pairs present discordant methylation in the nc886 locus. The methylation levels of nc886 are uniform across somatic tissues, except in cerebellum and skeletal muscle. Conclusion: The nc886 imprint may be est…

VTRNA2-1EXPRESSIONCancer Researchpolymorphic imprintingväestötutkimusDISEASEnc886Geneticsnoncoding 886COHORTPLACENTAEXPOSUREgeeniekspressioBRAINEPIGENOME-WIDE ASSOCIATIONRISKDNA methylationgeenit1184 Genetics developmental biology physiologyDna Methylation ; Vtrna2-1 ; Developmental Origins Of Health And Disease Hypothesis ; Imprinting ; Metastable Epiallele ; Nc886 ; Noncoding 886 ; Polymorphic Imprinting ; Population Studiespopulation studies217 Medical engineeringmetastable epialleleDNA-metylaatiodevelopmental origins of health and disease hypothesisHEALTH3111 Biomedicineimprinting
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