Search results for "Vessel"

showing 10 items of 751 documents

Short‐term hypoxia promotes vascularization in co‐culture system consisting of primary human osteoblasts and outgrowth endothelial cells

2019

Prevascularization of tissue constructs before implantation has been developed as a novel and promising concept for successful implantation. Since hypoxia might induce angiogenesis, we have investigated the effects of hypoxic treatment on vascularization by using co-cultures of primary human osteoblasts (POBs) and outgrowth endothelial cells. Our results show that: (a) repeated short-term hypoxia (2% O2 for 8 hr), not long-term hypoxia (2% O2 for 24 hr), over 1 or 2 weeks, significantly enhances microvessel formation in co-cultures; (b) sustained hypoxia, not short-term or long-term hypoxia, causes cytotoxicity in mono- and co-cultures; (c) the expression of some angiogenic and inflammatory…

Time FactorsMaterials scienceCell SurvivalAngiogenesisProtein subunitmedicine.medical_treatment0206 medical engineeringBiomedical EngineeringNeovascularization Physiologic02 engineering and technologyBone tissueBiomaterialschemistry.chemical_compoundmedicineHumansRNA MessengerCytotoxicityMicrovesselCells CulturedOsteoblastsCell DeathGrowth factorMetals and AlloysEndothelial CellsHypoxia (medical)021001 nanoscience & nanotechnology020601 biomedical engineeringCell HypoxiaCoculture TechniquesUp-RegulationVascular endothelial growth factormedicine.anatomical_structurechemistryCeramics and CompositesCancer researchInflammation Mediatorsmedicine.symptom0210 nano-technologyJournal of Biomedical Materials Research Part A
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Risk Burden of Coronary Perforation in Chronic Total Occlusion Recanalization: Latin American CTO Registry Analysis

2022

Background Coronary perforation is a life‐threatening complication of acute percutaneous coronary intervention (PCI) for chronic total occlusions (CTO), but data on midterm outcomes are limited. Methods and Results Data from LATAM (Latin American)‐CTO Registry (57 centers; 9 countries) were analyzed. We assessed the risk of 30‐day, 1‐year major adverse cardiac events of coronary perforation using time‐to‐event and weighted composite end point analysis having CTO PCI without perforation as comparators. Additionally, we studied the independent predictors of perforation in these patients. Of 2054 patients who underwent CTO PCI between 2015 and 2018, the median Multicenter CTO Registry in Japa…

Time Factorspercutaneous coronary interventionacute myocardial infarctionCoronary Angiographytarget vessel revascularization.Latin AmericaTreatment OutcomeHeart InjuriesRisk FactorsChronic DiseaseHumansProspective StudiesRegistriescoronary perforationCardiology and Cardiovascular Medicinechronic total occlusionJournal of the American Heart Association
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What do we know about growth of vessel elements of secondary xylem in woody plants?

2021

Despite extensive knowledge about vessel element growth and the determination of the axial course of vessels, these processes are still not fully understood. They are usually explained as resulting primarily from hormonal regulation in stems. This review focuses on an increasingly discussed aspect - mechanical conditions in the vascular cambium. Mechanical conditions in cambial tissue are important for the growth of vessel elements, as well as other cambial derivatives. In relation to the type of stress acting on cambial cells (compressive versus tensile stress) we: (i) discuss the shape of the enlarging vessel elements observed in anatomical sections; (ii) present hypotheses regarding the …

Transport watervascular cambiumdiffuse-porous woodEnvironmentGeneral Biochemistry Genetics and Molecular BiologyTreesAuxinXylemdiurnal strainVascular cambiumchemistry.chemical_classificationCambiumbiologyring-porous woodintrusive growthmechanical stressXylembiology.organism_classificationWoodvessel elementchemistrycardiovascular systemPlant speciesBiophysicsPlant hormoneGeneral Agricultural and Biological SciencesVessel elementWoody plantBiological Reviews
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Inflammation Determines the Pro-Adhesive Properties of High Extracellular D-Glucose in Human Endothelial Cells In Vitro and Rat Microvessels In Vivo

2010

BACKGROUND: Hyperglycemia is acknowledged as an independent risk factor for developing diabetes-associated atherosclerosis. At present, most therapeutic approaches are targeted at a tight glycemic control in diabetic patients, although this fails to prevent macrovascular complications of the disease. Indeed, it remains highly controversial whether or not the mere elevation of extracellular D-glucose can directly promote vascular inflammation, which favors early pro-atherosclerotic events. METHODS AND FINDINGS: In the present work, increasing extracellular D-glucose from 5.5 to 22 mmol/L was neither sufficient to induce intercellular adhesion molecule-1 (ICAM-1) and vascular cell adhesion mo…

Umbilical VeinsEndotheliumCardiovascular Disorders/Coronary Artery Diseaselcsh:MedicineInflammationIn vivoDiabetes mellitusCell AdhesionmedicineExtracellularAnimalsHumansLeukocyte RollingCardiovascular Disorders/Vascular BiologyUNESCO::CIENCIAS DE LA VIDA::Microbiología ::Otraslcsh:ScienceCell adhesionInflammationMultidisciplinaryInflammation; Pro-adhesive properties; High extracellular D-glucose; Human endothelial cells In Vitro; Rat microvessels In VivoDose-Response Relationship DrugChemistrylcsh:RCardiovascular Disorders/Peripheral Vascular DiseaseAdhesivenessEndothelial CellsChemotaxismedicine.diseaseIn vitroRatsChemotaxis LeukocyteDiabetes and EndocrinologyCell Biology/Cell AdhesionGlucosemedicine.anatomical_structureHyperglycemiaMicrovesselsImmunologyCancer researchlcsh:QEndothelium Vascularmedicine.symptomResearch Article
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Micromechanisms of load transfer in a unidirectional carbon fibre-reinforced epoxy composite due to fibre failures: Part 3. Multiscale reconstruction…

2008

International audience; This third article describes a multiscale process which takes into account the most important microscopic phenomena associated with composite degradation, including fibre fractures and interfacial debonding, overloading of fibres neighbouring a fibre break as well as viscoelastic behaviour of the matrix. The results have been used to accurately predict the macroscopic failure of unidirectional carbon fibre-reinforced epoxy and quantify damage accumulation in pressure vessels made of the same material. The approach described has allowed the acoustic emission activity resulting from fibres breaks to be evaluated and shown how the residual lifetimes of such vessels, whe…

Unidirectional compositeMaterials scienceComposite number[ PHYS.COND.CM-MS ] Physics [physics]/Condensed Matter [cond-mat]/Materials Science [cond-mat.mtrl-sci]02 engineering and technologyViscoelasticity0203 mechanical engineeringComposite materialCivil and Structural EngineeringFibre failuresDelaminationPressure vesselsMicromechanicsEpoxy021001 nanoscience & nanotechnologyDurabilityPressure vessel020303 mechanical engineering & transportsAcoustic emissionFailure predictionvisual_artCeramics and Compositesvisual_art.visual_art_medium[PHYS.COND.CM-MS]Physics [physics]/Condensed Matter [cond-mat]/Materials Science [cond-mat.mtrl-sci]Multiscale processMicromechanics0210 nano-technology
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Human Fetal Aorta Contains Vascular Progenitor Cells Capable of Inducing Vasculogenesis, Angiogenesis, and Myogenesis in Vitro and in a Murine Model …

2007

Vasculogenesis, the formation of blood vessels in embryonic or fetal tissue mediated by immature vascular cells (ie, angioblasts), is poorly understood. We report the identification of a population of vascular progenitor cells (hVPCs) in the human fetal aorta composed of undifferentiated mesenchymal cells that coexpress endothelial and myogenic markers. Under culture conditions that promoted cell differentiation, hVPCs gave rise to a mixed population of mature endothelial and mural cells when progenitor cells were stimulated with vascular endothelial growth factor-A or platelet-derived growth factor-betabeta. hVPCs grew as nonadherent cells and, when embedded in a three-dimensional collagen…

Vascular Endothelial Growth Factor AAngiogenesisBecaplerminNeovascularization PhysiologicAntigens CD34BiologyMuscle DevelopmentMural cellPathology and Forensic MedicineAngiopoietin-2MiceFetusVasculogenesisAntigens CDIschemiaAnimalsHumansCell LineageAC133 AntigenProgenitor cellAortaCells CulturedGlycoproteinsPlatelet-Derived Growth FactorStem CellsProto-Oncogene Proteins c-sisVascular Endothelial Growth Factor Receptor-2Cell biologyEndothelial stem cellVascular endothelial growth factor BVascular endothelial growth factor AVascular endothelial growth factor CImmunologyBlood VesselsPeptidesBiomarkersRegular ArticlesThe American Journal of Pathology
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Integrin-dependent and -independent functions of astrocytic fibronectin in retinal angiogenesis

2011

Fibronectin (FN) is a major component of the extracellular matrix and functions in cell adhesion, cell spreading and cell migration. In the retina, FN is transiently expressed and assembled on astrocytes (ACs), which guide sprouting tip cells and deposit a provisional matrix for sprouting angiogenesis. The precise function of FN in retinal angiogenesis is largely unknown. Using genetic tools, we show that astrocytes are the major source of cellular FN during angiogenesis in the mouse retina. Deletion of astrocytic FN reduces radial endothelial migration during vascular plexus formation in a gene dose-dependent manner. This effect correlates with reduced VEGF receptor 2 and PI3K/AKT signalli…

Vascular Endothelial Growth Factor AIntegrinsAngiogenesisIntegrinNeovascularization PhysiologicMice TransgenicExtracellular matrixMicePhosphatidylinositol 3-KinasesCell MovementAnimalsProtein Interaction Domains and MotifsMolecular BiologyResearch ArticlesMice KnockoutSprouting angiogenesisbiologyRetinal VesselsCell migrationKinase insert domain receptorVascular Endothelial Growth Factor Receptor-2Extracellular MatrixFibronectinsCell biologyMice Inbred C57BLFibronectinVascular endothelial growth factor AAstrocytesbiology.proteinHeparitin SulfateOligopeptidesProto-Oncogene Proteins c-aktIntegrin alpha5beta1Signal TransductionDevelopmental BiologyDevelopment
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Scaffold vascularization in vivo driven by primary human osteoblasts in concert with host inflammatory cells.

2011

Successful cell-based tissue engineering requires a rapid and thorough vascularization in order to ensure long-term implant survival and tissue integration. The vascularization of a scaffold is a complex process, and is modulated by the presence of transplanted cells, exogenous and endogenous signaling proteins, and the host tissue reaction, among other influencing factors. This paper presents evidence for the significance of pre-seeded osteoblasts for the in vivo vascularization of a biodegradable scaffold. Human osteoblasts, cultured on silk fibroin micronets in vitro, migrated throughout the interconnected pores of the scaffold and produced extensive bone matrix. When these constructs we…

Vascular Endothelial Growth Factor AScaffoldMaterials scienceTime FactorsAngiogenesisCellBiophysicsFibroinNeovascularization PhysiologicBioengineeringMice SCIDBiomaterialsProsthesis ImplantationMiceTissue engineeringIn vivomedicineAnimalsHumansCells CulturedInflammationOsteoblastsTissue ScaffoldsIn vitroCell biologymedicine.anatomical_structureMechanics of MaterialsGiant cellCeramics and CompositesBlood VesselsFibroinsBiomedical engineeringBiomaterials
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Tbx1 regulates Vegfr3 and is required for lymphatic vessel development

2010

Defects in lymphangiogenesis are added to the broad clinical manifestations of DiGeorge syndrome, caused by deletion of the T box transcription factor Tbx1.

Vascular Endothelial Growth Factor ATBX1Cellular differentiationBiologyMice03 medical and health sciences0302 clinical medicinestomatognathic systemReportLymphatic vesselmedicineAnimalsHumansLymphangiogenesisEnhancerCells CulturedResearch ArticlesLymphatic Vessels030304 developmental biology0303 health sciencesABNORMAL CAROTID ARTERIES; TRANSGENIC MICE; VELOCARDIOFACIAL SYNDROME; CARDIOVASCULAR DEFECTS; LYMPHANGIOGENESIS; LYMPHEDEMA; MOUSE; RECEPTOR-3; MUTATION; SYSTEMEndothelial CellsGene Expression Regulation DevelopmentalCell DifferentiationCell BiologyEmbryo Mammalian3. Good healthLymphangiogenesisCell biologyVascular endothelial growth factor ALymphatic systemmedicine.anatomical_structureVascular endothelial growth factor Cembryonic structuresImmunologyT-Box Domain Proteins030217 neurology & neurosurgeryJournal of Cell Biology
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Microphthalmia, persistent hyperplastic hyaloid vasculature and lens anomalies following overexpression of VEGF-A188 from the αA-crystallin promoter

2007

Purpose During growth of the embryonic eye, dose- and site-specific expression of heparin-binding growth factors is critical for the formation of an appropriate vascular supply. Overexpression of vascular endothelial growth factor-A188 (VEGF-A188), a strongly heparin-binding, endothelial-specific mitogen, leads to severe disturbance of vascular and overall ocular morphology. This study aimed to evaluate the effects of VEGF-A188 overexpression on growth of ocular tissue components. Methods Stereological and immunohistochemical methods were employed to identify the vascular profiles, ocular tissue proportions, and cell types in VEGF-A188 transgenic mice and compare them with wild-type mice. R…

Vascular Endothelial Growth Factor Agenetic structuresMyocytes Smooth MuscleCell CountMice TransgenicEyealpha-Crystallin A ChainCongenital AbnormalitiesCorneaMiceLens CrystallineAnimalsMicrophthalmosVascular DiseasesPromoter Regions GeneticHyperplasiaEndothelial CellsHypertrophyEmbryo MammalianAntigens DifferentiationImmunohistochemistryeye diseasesActinsDisease Models AnimalAnimals NewbornBlood Vesselssense organsPericytesHeparan Sulfate ProteoglycansResearch ArticleMolecular Vision
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